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abnormal release
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  异常释放
    Findings of this serial studies strongly suggested that the uncontrollable "mediator disease" resuIted from ischemic-reperfusion damage, swelling and edema of visceraI tissues, the endothelial cell damage and abnormal release mediators might be of importance in the pathogenesis of early PB organ damage。
    缺血和再灌流损伤、脏器水肿、内皮细胞损伤及多种炎症介质的异常释放,导致难以控制的介质病,是脏器损害的重要因素;
短句来源
    Objective To study the effect of extracorporeal circulation on the abnormal release of proinflammatory and anti inflammatory cytokines, and explore their effect on systemic inflammatory response after operation.
    目的 研究体外循环术对致炎性细胞因子和抗炎性细胞因子异常释放的影响 ,并探讨其在术后炎症反应中的作用。
短句来源
    BACKGROUND: As unspecific antagonist of opiate receptor, naloxone is widely used for multiple diseases which are related with abnormal release of endogenous opium.
    背景:纳洛酮作为阿片受体的非特异性拮抗剂,已被用于治疗内源性阿片类物质异常释放有关的多种疾病。
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  “abnormal release”译为未确定词的双语例句
    CONCLUSION The abnormal release of the cellular factors is an important cause of MOF after openheartsurgery.
    结论 细胞因子过多释放是心脏直视手术后多器官衰竭的重要因素 .
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  abnormal release
We hypothesized that some motor changes of the gut might be secondary to impaired neural input to smooth muscle or abnormal release of gut endocrine peptides.
      
Abnormal release of glutamate, which may trigger auras, and abnormal platelet behaviour, which constitutes a possible predisposing factor to MwA, are possible targets for MwA-specific prophylactic therapy.
      
Abnormal release of glutamate, that may trigger auras, and abnormal platelet behaviour, that constitute a possible predisposing factor to MwA, may be possible targets for MwA specific prophylactic therapy.
      
There is thus no reason to suspect any abnormal release of BB to the blood.
      
In addition, in CF patients, impaired neutrophil functions may contribute to an abnormal release of inflammatory mediators.
      


The pathogenesis of postburn organ damage as well as the effective measures for its prevention and treatment were studied. The present clinical prospective studies and animal experiments revealed or further confirmed that all the main organs could be damaged in the early stage following severe burns. Findings of this serial studies strongly suggested that the uncontrollable "mediator disease" resuIted from ischemic-reperfusion damage, swelling and edema of visceraI tissues, the endothelial cell damage and...

The pathogenesis of postburn organ damage as well as the effective measures for its prevention and treatment were studied. The present clinical prospective studies and animal experiments revealed or further confirmed that all the main organs could be damaged in the early stage following severe burns. Findings of this serial studies strongly suggested that the uncontrollable "mediator disease" resuIted from ischemic-reperfusion damage, swelling and edema of visceraI tissues, the endothelial cell damage and abnormal release mediators might be of importance in the pathogenesis of early PB organ damage。 Fast fluid infusion for delayed resuscitation, the use of the mediator antagonixtxanisodamine and gensinosides or escharectomy by one operation are effective in the prevention and treatment of organ damage in the early stage of severe burns。

通过系列的临床前瞻性研究与动物实验,发现并从多方面进一步证实了严重烧伤早期各主要脏器均可发生损害;缺血和再灌流损伤、脏器水肿、内皮细胞损伤及多种炎症介质的异常释放,导致难以控制的介质病,是脏器损害的重要因素;延迟复苏快速补液和使用介质拮抗剂山莨菪碱及人参皂甙或一次性切痂是防治严重烧伤早期脏器损害的有效方法。

AIM To evaluate the clinical significance of the changes in serum tumor necrosis factor-α (TNF-α), interleukin-β (IL-1β), interleukin-6 (IL-6), interleukin-8 (IL-8). METHODS Thirteen patients with multiple organ failure following openheartsurgery were selected. Serum TNFα, IL1β, IL6, IL8 levels were measured by enzymelinked immunosorbent assay (ELISA). RESULTS The levels of plasma TNFα (0.61±0.08), (0.81±0.09) vs (0.20±0.05) μg·L -1 (p<0.05, 0.01); IL1β (0.52±0.09), (0.69±0.04) vs (0.16±0.03) μg·L -1...

AIM To evaluate the clinical significance of the changes in serum tumor necrosis factor-α (TNF-α), interleukin-β (IL-1β), interleukin-6 (IL-6), interleukin-8 (IL-8). METHODS Thirteen patients with multiple organ failure following openheartsurgery were selected. Serum TNFα, IL1β, IL6, IL8 levels were measured by enzymelinked immunosorbent assay (ELISA). RESULTS The levels of plasma TNFα (0.61±0.08), (0.81±0.09) vs (0.20±0.05) μg·L -1 (p<0.05, 0.01); IL1β (0.52±0.09), (0.69±0.04) vs (0.16±0.03) μg·L -1 (P<0.01, 0.01); IL6 (0.41± 0.08 ), (0.56±0.07) vs (0.17±0.03) μg·L -1 (P<0.05,0.05); IL8 (3.70±0.80), (4.92±0.78) vs (1.6±0.40) μg·L -1 (P<0.05, 0.05) rose markedly after MOF. The time of reaching the peak value in IL6, IL8 was slower than that in TNFα, IL1β. CONCLUSION The abnormal release of the cellular factors is an important cause of MOF after openheartsurgery. TNFα,IL1β are triggling factors.

目的 检测心脏直视手术后多器官功能衰竭 ( MOF)患者血清肿瘤坏死因子 ( TNF- α)、白细胞介素 1β( IL- 1β)、白细胞介素 6 ( IL- 6 )、白细胞介素 8( IL- 8)的含量变化并探讨其意义 .方法 选择心脏直视手术后发生多器官衰竭患者 13例 ,采用放射免疫方法检测患者血清 TNF- α,IL- 1β,IL- 6 ,IL- 8的含量 .结果 多器官衰竭发生存活组和死亡组 3 d后 TNF-α( 0 .6 1± 0 .0 8) ,( 0 .81± 0 .0 9) vs ( 0 .2 0± 0 .0 5 )μg.L- 1 ( P<0 .0 5 ,0 .0 1) ;IL- 1β( 0 .5 2± 0 .0 9) ,( 0 .6 9± 0 .0 4) vs( 0 .16±0 .0 3) μg.L- 1 ( P<0 .0 1,0 .0 1) ;IL- 6 ( 0 .41± 0 .0 8) ,( 0 .5 6± 0 .0 7) vs( 0 .17± 0 .0 3) μg.L- 1 ( P<0 .0 5 ,0 .0 5 ) ;IL- 8( 3.70± 0 .80 ) ,( 4.92± 0 .7...

目的 检测心脏直视手术后多器官功能衰竭 ( MOF)患者血清肿瘤坏死因子 ( TNF- α)、白细胞介素 1β( IL- 1β)、白细胞介素 6 ( IL- 6 )、白细胞介素 8( IL- 8)的含量变化并探讨其意义 .方法 选择心脏直视手术后发生多器官衰竭患者 13例 ,采用放射免疫方法检测患者血清 TNF- α,IL- 1β,IL- 6 ,IL- 8的含量 .结果 多器官衰竭发生存活组和死亡组 3 d后 TNF-α( 0 .6 1± 0 .0 8) ,( 0 .81± 0 .0 9) vs ( 0 .2 0± 0 .0 5 )μg.L- 1 ( P<0 .0 5 ,0 .0 1) ;IL- 1β( 0 .5 2± 0 .0 9) ,( 0 .6 9± 0 .0 4) vs( 0 .16±0 .0 3) μg.L- 1 ( P<0 .0 1,0 .0 1) ;IL- 6 ( 0 .41± 0 .0 8) ,( 0 .5 6± 0 .0 7) vs( 0 .17± 0 .0 3) μg.L- 1 ( P<0 .0 5 ,0 .0 5 ) ;IL- 8( 3.70± 0 .80 ) ,( 4.92± 0 .78) vs( 1.6± 0 .40 )μg.L- 1 ( P<0 .0 5 ,0 .0 5 )的含量比对照组明显升高 ,且 IL - 6 ,IL - 8升高时限滞后于 TNF-α,IL - 1β.结论 细胞因子过多释放是心脏直视手术后多器官衰竭的重要因素 . TNF-α,IL - 1β是引发多器官衰竭的始动因子

Objective To study the effect of extracorporeal circulation on the abnormal release of proinflammatory and anti inflammatory cytokines, and explore their effect on systemic inflammatory response after operation. Methods Seven patients with rheumatic heart diseases underwent extracorporeal circulation were investigated. The concentrations of TNFα, IL 1β, IL 6 and IL 10 in plasma were measured by ELISA at 5 different time points, such as before operation (T1), 15th minute (T2) and 60th minute (T3)...

Objective To study the effect of extracorporeal circulation on the abnormal release of proinflammatory and anti inflammatory cytokines, and explore their effect on systemic inflammatory response after operation. Methods Seven patients with rheumatic heart diseases underwent extracorporeal circulation were investigated. The concentrations of TNFα, IL 1β, IL 6 and IL 10 in plasma were measured by ELISA at 5 different time points, such as before operation (T1), 15th minute (T2) and 60th minute (T3) during operation and 4th hour(T4) and 24th hour after operation (T5), at the same time, the expression of intracellular cytokines (TNFα, IL 6 and IL 10 ) in lymphocytes were measured by flow cytometry. Results The concentrations of TNFα, IL 1β, IL 6 and IL 10 in plasma increased significantly ( P <0.05) after the beginning of extracorporeal circulation, and had been increasing with the extracorporeal circulation longthening. The peak concentration of TNFα was (24.5±4.9)pg/ml at T3, IL 1β(18.4±2.8)pg/ml at T4, IL 6(361.8±75.1)pg/ml at T4, IL 10(957.4±206.6)pg/ml at T5. The dominating cytokines during operation were proinflammatory cytokines (TNFα, IL 1β and IL 6), and after operation, anti inflammatory cytokine (IL 10). The changes of expression in intracellular cytokines such as TNFα,IL 6 and IL 10 was no significance before, during and after operation. Conclusion The unbalance of proinflammatory and anti inflammatory cytokines during and after extracorporeal circulation triggerd the systemic inflammatory response, and aggravated it with suppressing cellular and humoral immunity.

目的 研究体外循环术对致炎性细胞因子和抗炎性细胞因子异常释放的影响 ,并探讨其在术后炎症反应中的作用。方法 ELISA测定 7例风湿性心脏病瓣膜置换术不同时点血浆肿瘤坏死因子α(TNFα)、白细胞介素 (IL) 6、IL 10和IL 1β浓度 ,同时用流式细胞术检测淋巴细胞内相应细胞因子的表达。结果  4种细胞因子在体外循环术中和 (或 )术后均明显增高 (P <0 .0 5 ) ,它们之间存在明显相关。手术开始后以TNFα、IL 6和IL 1β为主 [峰值时间和浓度分别为 :体外循环术开始后30minTNFα(2 4.5± 4.9)pg/ml,体外循环术停止后 4hIL 6 (36 1.8± 75 .1)pg/ml和IL 1β (18.4± 2 .8)pg/ml],而术后 2 4h以IL 10为主 [(95 7.4± 2 0 6 .6 )pg/ml]。淋巴细胞内 3种细胞因子表达无明显变化 (P >0 .0 5 )。结论 体外循环术中致炎性细胞因子与抗炎性细胞因子平衡失调启动炎症反应并通过抑制细胞和体液免疫反应促进炎症反应。

 
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