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tnf production
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  tnf产生
     RESULTS: TGP(0 5 ̄10 mg·L -1 ) markedly increased both 45 Ca influx and TNF production in MΦ induced by lipopolysaccharide(LPS,5 mg·L -1 ).
     结果:TGP(0.5~10mg·L-1)明显促进LPS诱导MΦ的45Ca内流和TNF产生
短句来源
     The consensus oligonucleotide competitive binding with NF-κB inhibits TNF production
     共有序列寡核苷酸竞争结合NF-κB以减少TNF产生的实验研究
短句来源
     AIM: To investigate whether a consensus oligonucleotide, which contains nuclear factor-kappa B(NF-κB) binding site, decreases TNF production stimulated by lipopolysaccharide(LPS).
     目的 :了解使用含核因子 -κB(NF -κB)结合位点序列的共有序列寡核苷酸在胞浆内与NF -κB结合 ,以减少LPS刺激TNF产生的效果。
短句来源
     Linear regression analysis demonstrated a positive correlation between 45 Ca influx and TNF production.
     线性回归分析表明,45Ca内流和TNF产生呈明显正相关。
短句来源
     CONCLUSIONS: Up regulation of lower concentrations of TGP on TNF production may be associated with increasing 45 Ca influx and activating the calmodulin activity and then enhancing PGE 2 production, and down regulation of higher concentrations may be mediated through the excess PGE 2 production by MΦ itself.
     结论:低浓度TGP对TNF产生上调作用可能与促进45Ca内流,提高钙调蛋白活性从而促进PGE2分泌等有关,而高浓度下调作用是可能与MΦ自身产生大量PGE2介导有关。
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  产生tnf
     Trifluoperazine(40 μmol·L -1 ) could block the enhancing TNF production in TGP LPS activated MΦ cultures.
     三氟拉嗪(40μmol·L-1)可阻断TGP促进LPS诱导MΦ产生TNF
短句来源
     Results: Decrease of TNF production by human monocytes induced by LPS was striking at the concentration of EsA arranged from 1 to 10 μmol/L.
     结果:当EsA浓度在1和10μmol/L时,受LPS刺激的人单核细胞产生TNF显著减少。
短句来源
     The in vitro refolded prokaryotic HLA-G5 promotes the TNF production in human peripheral blood mononuclear cells
     原核表达、体外折叠的HLA-G5促进人外周血单核细胞产生TNF
短句来源
     However, GL 0.01~10 μg/ml promoted TNF production by macrophages in vitro .
     但灵芝0.01~10μg/ml在试管内促进小鼠腹腔巨噬细胞产生TNF
短句来源
     Influences of Types of HLA Class Ⅱ Molecules on TNF Production from Human T-cells Stimulated with Superantigens
     HLAⅡ类抗原对细菌超抗原诱导人T细胞产生TNF的影响
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  分泌tnf
     Results JDP-N stimulated TNF production that reached the maximum at around 6 h, demonstrating similar effect to that of lipopolysaccharide on TNF production.
     结果 JDP-N能诱导Mφ分泌TNF,诱生TNF达峰时间约为6 h,与脂多糖相比,时效关系相仿;
短句来源
     Protein kinase C played a key role in mediating TNF production of Mf induced by JDP-N, in the process of which enhancement of TNF-a mRNA expression in Mφ was also observed.
     蛋白激酶C激活在JDP-N诱导Mφ分泌TNF过程中具有关键性作用; JDP-N能促进MφTNF-α mRNA的表达。
短句来源
     Conclusion Promotion of TNF production might be one of the mechanisms by which JDP-N enhances the immune function.
     结论 JDP-N增强小鼠免疫功能的机制之一是促进Mφ分泌TNF
短句来源
     The results showed the prokaryotic HLA-G5-antigenic peptide complex was successfully produced,sharing the epitope of HLA class I,and the complex was able to promote the TNF production of human PBMC in vitro.
     TNF分泌量采用TNF敏感的L929细胞进行生物法测定,结果显示原核表达、体外折叠的HLA-G5促进人PBMC分泌TNF-α。
短句来源
     The conformation of the refolded complex was detected by HLA class I specific mAb W6/32 with native-PAGE/Western blot and ELISA,the effect of the refolded HLA-G5 on the TNF production of human(PBMC) was observed with bioassay by using TNF-sensitive L929 cell line.
     利用PBMC受LPS刺激后产生TNF的特点,观察原核表达、体外折叠的HLA-G5对人PBMC分泌TNF的影响。
短句来源
  “tnf production”译为未确定词的双语例句
     However, the level of TNF production of LCs from recurrent CA patients was significantly less than that from non-recurrent CA patients (24.75% ± 7.51 vs 36.62% ± 10.96,P = 0.00016).
     而尖锐湿疣复发组的TNF分泌能力明显低于尖锐湿疣未复发组(24.75%±7.51%与36.62%±10.96%,P=0.00016);
短句来源
     TNF production in ET + GLY group was definitely inhibited.
     说明ET+Gly组TNF的产生被明显抑制,而单纯 Gly不能诱导 TNF的产生。
短句来源
     (2)BSP distinctly enhanced the activity of IL 1 and TNF production by mouse peritoneal macrophages in vitro.
     ( 2 ) BSP在体外还能促进正常小鼠腹腔巨噬细胞产生 IL - 1和 TNF。
短句来源
     Results Serum IL 6、IL 2、TNF production in HFRS patients were significantly higher than the control( P <0 01),IL 6、IL 2 increased to peak at oliguria statge,and TNF to peak at hypotension stage.
     结果 老年 HFRS患者血清 IL - 6、IL - 2和 TNF含量较对照组明显增高(P<0 .0 1 ) ,IL- 6、IL- 2的高峰值在少尿期 ,TNF的高峰值在低血压期。 PHA刺激 HFRS患者 PBMC产生 IL- 6、IL- 2和 TNF在发热期已达高水平 ,低血压期、少尿期持续在高水平 ,多尿期开始下降。
短句来源
     The LCL were stimulated with LPS to produce TNF. Results The average level of TNF production of LCL from all CA patients (including recurrent and non-recurrent CA patients) was similar to that of normal controls (30.14% ± 12.27 vs 34.06% ± 12.06,P = 0.1136).
     结果实验结果显示尖锐湿疣患者组(包括复发患者和未复发患者)LCL的TNF分泌能力与正常人对照组差异无显著性(30.14%±12.27%与34.06%±12.06%,P=0.1136);
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  tnf production
Linoleic acid (LA) diminished TNF production by 16%.
      
Human α-1-acid Glycoprotein Inhibits TNF Production in the CNS of Rabbits with Meningitis: A Report of Preliminary Observations
      
There was a significant effect of thermal injury on cytokine production by LPS-stimulated Kupffer cells at post burn day 8 and on TNF production by stimulated splenic macrophages also at post burn day eight.
      
TNF production was generally higher for one-day compared to four-day cultures (i.e., TNF was disappearing in the cultures), but IL-6 and PGE2 production was greater in four-day cultures.
      
In vitro experiments showed an augmentation of TNF production by bronchoalveolar cells in the presence of silica.
      
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We have carried out to investigate whether 30 kinds of Chinese medicines could effect on TNF production in mice. The results showed that oral administration of RADIX GINSENG, RHIZOMA ZEDOARIAE, HERBA ARTEMISIAE SCOPARIAE or RADIX PUERARIAE as priming agents could induce TNF production in serum of mice triggered with OK432 In other hand, Pretreated with CORTEX MAGNOLIAE OFFICINALIS, FOLIUM PERILLAE, RHIZOMA ZINGIBERIS RECENS, RADIX SCUTELLARIAE, FRUCTUS GARDENIAE, RAMULUS CINNAMOMf or CHAIPOTO, the...

We have carried out to investigate whether 30 kinds of Chinese medicines could effect on TNF production in mice. The results showed that oral administration of RADIX GINSENG, RHIZOMA ZEDOARIAE, HERBA ARTEMISIAE SCOPARIAE or RADIX PUERARIAE as priming agents could induce TNF production in serum of mice triggered with OK432 In other hand, Pretreated with CORTEX MAGNOLIAE OFFICINALIS, FOLIUM PERILLAE, RHIZOMA ZINGIBERIS RECENS, RADIX SCUTELLARIAE, FRUCTUS GARDENIAE, RAMULUS CINNAMOMf or CHAIPOTO, the high TNF pr-oducibility of C3H/He mice stimulated by MDP and OK432 could be depressed. The res- . ults above suggest that Chinese medicines and prescriptions might play a pharmacological role through influence on TNF level in the body

用C_3H/He小鼠对30种中医方药的TNF诱生作用进行了研究,发现灌服人参、莪术、茵陈蒿、葛根等可诱导OK_(432)激发小鼠血清产生TNF活性;灌服厚朴、苏叶、生姜、黄芩、山栀子、桂皮及柴朴汤等却能使MDP-OK_(432)诱生的血清高水平TNP降低。

It has been confirmed that macrophages(M)play an impor- tant role in the pathogenesis of proliferative glomerulonephritis.Interleu- kin 1(IL-1)and tumor necrosis factor(TNF)are the important mediat- ors.In this paper,we investigated the effects of Rheum Officinal(RO)on the production of IL-1 and TNF by Mφ in vitro.The sera from rats fed with RO(RO sera),Emodin and Polysaccharide(ROP)were used.RO sera (10%)and Emodin(12.5 and 25μg/ml)significantly inhibited the TNF secretion by M.IL-1...

It has been confirmed that macrophages(M)play an impor- tant role in the pathogenesis of proliferative glomerulonephritis.Interleu- kin 1(IL-1)and tumor necrosis factor(TNF)are the important mediat- ors.In this paper,we investigated the effects of Rheum Officinal(RO)on the production of IL-1 and TNF by Mφ in vitro.The sera from rats fed with RO(RO sera),Emodin and Polysaccharide(ROP)were used.RO sera (10%)and Emodin(12.5 and 25μg/ml)significantly inhibited the TNF secretion by M.IL-1 was inhibited by Emodin as well,while ROP has no effects on IL-1 or TNF production.The inhibitory effects of RO on the cy- tokines production,together with its effect on inhibiting glomerular mesangi- al cell proliferation,may be the important mechanisms of RO in the treat- ment of glomerulonephritis and chronic renal failure.

本文观察了灌服大黄醇提膏后的大鼠血清以及大黄素、大黄多糖对巨噬细胞(M)白细胞介素Ⅰ(IL—1)及肿瘤坏死因子(TNF)产生的影响.发现含大黄血清以及大黄素可抑制 MIL—1及 TNF 的产生,其抑制作用与剂量有关。大黄能抑制炎症因子的产生,可能是其治疗慢性肾炎及延缓慢性肾衰进展的又一作用机理。

In this report we employed L 929 cell lytic assay for tumor necrosis factor (TNF) to observe the effects of three trace elements(Zinc, Copper and Selenium) on TNF production in normal human peripheral blood mononuclear cells (PBMC). The results showed that zinc and copper were able to augment TNF production in PBMO in vitro. The most optimal concentration of zinc and copper was 0.25 namol/L. They were both able to maintain TNF secretion for as long as 96 hours. The effect of copper was...

In this report we employed L 929 cell lytic assay for tumor necrosis factor (TNF) to observe the effects of three trace elements(Zinc, Copper and Selenium) on TNF production in normal human peripheral blood mononuclear cells (PBMC). The results showed that zinc and copper were able to augment TNF production in PBMO in vitro. The most optimal concentration of zinc and copper was 0.25 namol/L. They were both able to maintain TNF secretion for as long as 96 hours. The effect of copper was stronger than that of zinc. The results also showed that selenium alone had no effect on TNF production. However, when lipopolysacharide (LPS) was added, they were able to enhance TNF secretion. The present study strongly suggested that zinc, copper and selenium might play an important role in regulating TNF production and their immune regulation function may be mediated by TNF releases.

本文采用L929细胞毒试验,在体外观察了三种微量元素锌、铜、硒对正常人外周血单个核细胞产生肿瘤坏死因子(TNF)的影响。结果发现锌、铜在体外能明显促进TNF产生,以0.25mmol/L为最适浓度,并能维持96小时以上。铜的作用较锌为强。硒虽然单独不能诱导TNF分泌,但与LPS协同能促使单个核细胞释放TNF。本文结果对研究上述三种元素参与免疫反应调节机理及TNF分泌调控均有裨益。

 
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