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toxin of
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  毒素
     Mechanisms of NO and H_2O_2 in the Resistance Responses of Cotton Suspension Cells to Toxin of Verticilium Dahliae
     棉花悬浮培养细胞对黄萎病菌毒素响应中NO和H_2O_2的作用机制
短句来源
     Mixtures of the final whole cultures of recombinant strains Bt B601, Bt B611, Bt B640, Bt U 30(expressing Cry4Aa, Cry4Ba, Cry11Aa and Cyt1Aa protein of Bacillus thurigiensis subsp. israelensis respectively) and Bt CW 3(expressing the Binary toxin of Bacillus sphaericus ) were used in bioassays against resistant Cpq larvae to value the synergism among the mosquito larvicidal toxins.
     本研究测定了分别表达苏云金芽孢杆菌Cry4Aa、Cry4Ba、Cry11Aa、Cyt1Aa和球形芽孢杆菌二元毒素Bin的转化菌株Bt B60 1、Bt B611、Bt B640、Bt U 30和Bt CW 3全发酵培养物两两或两两以上不同组合对抗性库蚊的毒力 ,分析了杀蚊毒素间的协同作用。
短句来源
     Influence of Anti-ulcer Toxin of East Asia Scorpion on NO and PGE_2 of Different Modelled Rats with Ulcer
     东亚钳蝎抗溃疡毒素对不同模型胃溃疡大鼠NO、PGE_2的影响
短句来源
     Toxin of Microcystis aeruginosa Küitz in dianchi and its change in water body
     滇池铜绿微囊藻(Microcystis aeruginosa Küitz)毒素及其在水体中的变化
短句来源
     RELATIONSHIP OF SA,NO AND H_2O_2 SIGNALS IN THE REPONSES OF ARABIDOPSIS TO TOXIN OF VERTICILLIUM DAHLIAE.
     大丽轮枝菌毒素与拟南芥互作反应中SA、NO 与H_2O_2信号的相关性
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  “toxin of”译为未确定词的双语例句
     Study on the Determination of Antibody against Type B Toxin of Clostridium Botulinum by ABC-ELISA
     ABC-ELISA检测B型肉毒毒素抗体的研究
短句来源
     The LD_ 50 of crude toxin of Ricinus was 11.494 mg/kg for 48 h in the laboratory.
     蓖麻粗毒对小鼠灌胃实验测得LD50为11.494mg/kg。
短句来源
     Results GM1 ELISA assay of CT toxin of Vibrio cholerae showed that the level of CT production of the toxR gene deficient strain, 569B 43 was much lower than that of donor strain 569B, the P/N value of toxR gene deficient strain 569B 43 was 1.82, the P/N value of 569B was 4.52. No difference was found between IEM101 and the toxR gene deficient strains IEM101 4 in CT production.
     结果 采用GM1 ELISA检测受测菌CT基因表达 ,toxR基因缺失株 5 6 9B 43的P/N值为 1 82 ,而其原出发菌株 5 6 9B的P/N为 4 5 2 ,而IEM10 1和其toxR基因缺失株的P/N值均低于 2。
短句来源
     ① Record of normal persistent sodium current of neuron in cerebral hippocampal CA1 area 0.5 mmol/L CdCl2 calcium channel blocking agent and 20 mmol/L TEA kalium channel blocking agent were used to perform 400 ms square-wave stimulation under -105 mV claw voltage and -30 mV stimulated voltage. Introversion current slight late activation and lasting for a long time was recorded and determined as persistent sodium currents by blocking toxin of puffer fish.
     ①脑海马CA1区神经元正常持续钠电流的记录:使用钙通道阻滞剂CdCl20.5mmol/L及钾通道阻滞剂TEA20mmol/L,在钳制电压-105mV、刺激电压-30mV下给予长为400ms的方波刺激,可记录到一个较小的、激活较晚且持续时间较长的内向电流,经河豚毒素阻断证实为持续性钠电流。
短句来源
     APPLICATION OF MONOCLONAL ANTIBODY TO ELISA FOR DETECTING TYPE B TOXIN OF CLOSTRIDIUM BOTULINUM
     单克隆抗体在ELISA法检测B型肉毒毒素中的应用
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  相似匹配句对
     Phytopathogenic Toxin
     植物病原毒素
短句来源
     2.toxin.
     2疫毒学说;
短句来源
     Analysis of Biological Toxin
     生物毒素的分析
短句来源
     Advances in the Study of Microcystion Toxin
     微囊藻毒素研究进展
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  toxin of
The inhibitory effect induced by CTC toxin of the phagocytic function not only was abolished completely, but phagocytosis was enhanced in the presence of interferon-gamma (IFN-γ).
      
Changes in serum acid phosphatase activity in guinea pigs poisoned with toxin of Clostridium perfringens type and with a mixture
      
Effect of toxin of burned skin on the state of the hemodynamics in rats
      
Effects of pathogenic action of botulinus toxin of spinal motoneurons of various types
      
Effect of the toxin of Clostridium perfringens type A and its lethal factor (lecithinase) on the microcirculation
      
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Thirteen adult guinea-pigs were divided into 3 groups:2 as normal controls;6 and 5 being treated with streptomycin sulphate 200 and 400mg per kilo body weight res- pectively for 21—60 days.The vestibular and auditory functions were tested.Their brains were prepared for Bielschowsky and Nissl sections.Microscopic examination re- vealed the following facts. Vascular disturbances prevail in the nervous system.The blood vessels are crumpled with concommittent perivascular edema.The adjacent nervous tissue shows...

Thirteen adult guinea-pigs were divided into 3 groups:2 as normal controls;6 and 5 being treated with streptomycin sulphate 200 and 400mg per kilo body weight res- pectively for 21—60 days.The vestibular and auditory functions were tested.Their brains were prepared for Bielschowsky and Nissl sections.Microscopic examination re- vealed the following facts. Vascular disturbances prevail in the nervous system.The blood vessels are crumpled with concommittent perivascular edema.The adjacent nervous tissue shows ischemic softening and the nerve fibres become demyelinated. The intoxicated nerve cells are generally dehydrated and shrunken often with eccen- tricity of nuclei and chromophilic lumps.Gliosis and neurophagosis are common. The vestibular and cochlear systems of the eighth or stato-acoustic nerve exhibit intoxicative changes in the 2 groups of animals.On the peripheral as well as the central side,the degeneration of the vestibular structures supercedes that of the cochlear system. The site and nature of the intoxication are much alike in the light and heavy-dose groups,yet the latter shows a higher severity. The degenerative alterations appear simultaneously in the perpheral and central struc- tures.A question of primacy in intoxication—peripheral or central—is not existent. There is a chain of structures on the peripheral and central sides of the stato-acoustic system;their degeneration does not proceed along the conduction path,nor the degree of intoxication varies with their relative positions on the conduction scale.The peri- pheral and central units get intoxicated independently. Besides the stato-acoustic structures streptomycin afflicts other sensory(e.g.trige- minal) and motor systems,the viscero-motor in particular.Streptomycin intoxication is general in nature,yet some structures are especially sensitive to it. The relative vulnerability to streptomycin of the nervous structures is apparently conditioned by their chemical constitution. Streptomycin injuries higher brain parts which are mainly motor,especially viscero- motor.The diencephalic viscero-motor nuclei,the striate complex and the visceral cor- tex are excessively disintegrated.The extensive intoxicative manifestations have a neurological basis. The toxin of streptomycin affects the entire nervous system and hence the whole bodily mechanisms.Its medical application calls for a meticulous consideration with respect to the patient's sensitivity to the antibiotic,the size of the dose and the length of the therapeutic course.

十三个成长的豚鼠分为三粗,2个作正常对照,此外6个和5个每日分别注射链霉秦硫酸盐,一公斤体重200和400毫克,共经21至60天。未了,豚鼠都经过平衡和听觉的测验。各脑分别染制Nissl和Bielschowsky切片。显微镜的检查得到以下的结果。神经系中普见血管的病变。血管塌陷,管外间质水肿。邻近的神经组织失血软化。神经纤维表现髓鞘溶解。中毒的神经细胞脱水萎缩,常见胞核偏位,染色质偏聚、结块。也时有神经胶质增生和神经吞噬现象。二组豚鼠在位听神经的前庭系和耳蜗系都有中毒的征象。在周缘部和中枢部,前庭系各极结构的病变,与耳蜗系相较,更为严重。在注射轻剂和重剂的二组豚鼠,神经系中毒的部位和性质是相似的,但重剂组的病变程度较高。在位听系,中毒的病变同时见于周缘部和中枢部。中毒的第一性的问题——在周缘, 抑在中枢——是不存在的。周缘和中枢各有一系结构,病变的表现不沿神经传导的路线;病变的轻重也不随路线上地位的高低。周缘和中枢的各项结构是各自中毒的。链霉素不仅毒害位听神经一系,而更损伤其它感觉系(如三叉神经系)和运动系——特是内脏运动系——的结构。链霉素中毒是一般性的,但有些结构特易感受它的毒害。在各级结构中,神经细胞的...

十三个成长的豚鼠分为三粗,2个作正常对照,此外6个和5个每日分别注射链霉秦硫酸盐,一公斤体重200和400毫克,共经21至60天。未了,豚鼠都经过平衡和听觉的测验。各脑分别染制Nissl和Bielschowsky切片。显微镜的检查得到以下的结果。神经系中普见血管的病变。血管塌陷,管外间质水肿。邻近的神经组织失血软化。神经纤维表现髓鞘溶解。中毒的神经细胞脱水萎缩,常见胞核偏位,染色质偏聚、结块。也时有神经胶质增生和神经吞噬现象。二组豚鼠在位听神经的前庭系和耳蜗系都有中毒的征象。在周缘部和中枢部,前庭系各极结构的病变,与耳蜗系相较,更为严重。在注射轻剂和重剂的二组豚鼠,神经系中毒的部位和性质是相似的,但重剂组的病变程度较高。在位听系,中毒的病变同时见于周缘部和中枢部。中毒的第一性的问题——在周缘, 抑在中枢——是不存在的。周缘和中枢各有一系结构,病变的表现不沿神经传导的路线;病变的轻重也不随路线上地位的高低。周缘和中枢的各项结构是各自中毒的。链霉素不仅毒害位听神经一系,而更损伤其它感觉系(如三叉神经系)和运动系——特是内脏运动系——的结构。链霉素中毒是一般性的,但有些结构特易感受它的毒害。在各级结构中,神经细胞的化学合成决定它们反应链霉素的灵钝和轻重。链霉素伤害高级的脑部,这多属运动性,特是内脏运动性。间脑的内脏性核团、纹状体簇和内脏性皮质中毒特别剧烈。链霉素中毒症状广泛,这是有神经基础的。链霉素的毒质伤害全神经系,也影响全身;为医疗使用这个抗菌素,关于患者对此药的敏感,剂量和疗程是要慎重考虑的。

ETEG, EIEC and EPEC are im-portant pathogens responsible for humanbacterial diarrhea. In recent years,the investigation of ETEC pathogenesishas advanced to a level of molecularbiology, and the study of EIEC hasalso made significant development.However, little is known about thepathogenesis of EPEC. Therefore, thereis some doubt about the value of clinicalapplication of classical EPEC diagno-stic sera to identify the cause ofdiarrhea in infants. The authors made a preliminarystudy of the pathogenesis of EPECdiarrhea....

ETEG, EIEC and EPEC are im-portant pathogens responsible for humanbacterial diarrhea. In recent years,the investigation of ETEC pathogenesishas advanced to a level of molecularbiology, and the study of EIEC hasalso made significant development.However, little is known about thepathogenesis of EPEC. Therefore, thereis some doubt about the value of clinicalapplication of classical EPEC diagno-stic sera to identify the cause ofdiarrhea in infants. The authors made a preliminarystudy of the pathogenesis of EPECdiarrhea. Enterotoxin and enteroinva-siveness tests were carried out in 5strains of EPEC. Only one of the fivestrains produced LT enterotoxin whilethe other four showed neither entero-toxin nor enteroinvasiveness. Then,these four strains were fed to domesticinfant rabbits in order to find outwhether they can induce diarrhea. Twostrains caused diarrhea in infant rabbitsand bacteria were found in the fecesof these rabbits much longer than thosethat were free from diarrhea. Further-more, they were the dominant strainsin the excrement. Histological sectionof rabbits intestine reyealed somepathological changes. These findingsindicate that the toxin of EPEC differsfrom LT and ST enterotoxin becauseit damages the structure of intestinaltissue. At the same time, the chiefcondition for EPEC to cause diarrheais that EPEC can colonize in the wallof small intestine and propagate on alarge scale. Possibly, these have some-thing to do with a colonization factor.A determination of the hemagglutina-tion patterns of seven strains of EPECrevealed that the four strains causedmannose-resistant hemagglutination ofguineapig, human and cow erythrocytes,which is different from the hemagglu-tination patterns produced by type Ⅰpili and CFA/Ⅰ, CFA/Ⅱ. Only one ofthirteen strains of common Escherichiacoli caused this hemagglutination pat-tern. Through statistic treatment, P<0.05, it shows clearly that this kindof hemagglutination is chiefly producedby strains of EPEC rather than bycommon Escherichia coli. Owing to the difference of pathoge-nesis between EPEC and ETEC and EIEC,it is still necessary to classify theserotype of classical EPEC in searchingfor the cause of diarrhea in infants.

作者使用传统的肠道致病性大肠杆菌株进行了肠毒素、肠侵袭性及幼兔实验性腹泻等试验。实验结果表明,肠道致病性大肠杆菌是大肠杆菌性腹泻的重要病原,其致病机理既不同于产肠毒素性大肠杆菌,也不同于肠道侵袭性大肠杆菌。因此,临床实验室在检查婴幼儿腹泻的病原时,常规作传统的肠道致病性大肠杆菌血清分型仍属必要。

The tested materials included: 35 inbred strains of known male-sterile cytoplasm belonging to a certain group,17 inbred strains of unknown male-sterile cytoplasm,and 200 hybrids crossed with different inbred lines.The tested results indicated that their reaction to the toxin of the southern leaf blight race T,the phenotype and genotype of male-fertility,and the reactions to environmental conditions are different.A simple convenient and accurate way of synthetic identification and classification of the...

The tested materials included: 35 inbred strains of known male-sterile cytoplasm belonging to a certain group,17 inbred strains of unknown male-sterile cytoplasm,and 200 hybrids crossed with different inbred lines.The tested results indicated that their reaction to the toxin of the southern leaf blight race T,the phenotype and genotype of male-fertility,and the reactions to environmental conditions are different.A simple convenient and accurate way of synthetic identification and classification of the cytoplasmic male-sterility type in maize is presented on the basis of the test and the study of the data.

供试材料包括35个已知其不育胞质所属类群的不育系、17个待鉴定不育胞质类型的品系、200个用这些不育系和不同自交系测交的杂交种。研究结果表明,这些不育系对小斑病T小种毒素的反应、育性的表现和遗传及对环境条件的反应是不同的。在试验研究资料的基础上,提出了一种简便准确的玉米不育细胞质综合鉴定分类方法。

 
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