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肾间质病变
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  interstitial pathological changes
     The expression of HBAg of renal tubule was related to renal tubule and interstitial pathological changes.
     肾小管HBAg表达与肾小管和肾间质病变有关。
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     Conclusion Adrenomedullin play an important role in the process of cell proliferation in renal interstitial pathological changes by UUO.
     结论肾上腺髓质素在UUO介导的肾间质病变的细胞增殖过程中有着重要的作用。
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  “肾间质病变”译为未确定词的双语例句
     Renal CTGF expression in LN was positively related with tubulointerstitial fibrosis(r=0.863,P<0.01).
     LN患者肾组织CTGF表达量与肾间质病变程度呈正相关(r=0.863,P<0.01);
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     Objective The role of bFGF in renal tubular injury, regeneration and interstitial lesion of rats was studied.
     目的 研究碱性成纤维细胞生长因子 (bFGF)在大鼠肾小管损伤、再生以及肾间质病变过程中的作用。
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     Prostaglandin and interstitial nephropathy
     前列腺素与肾间质病变
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     Action of NF-κB p65 in renal interstitium in rats with active Heymann nephritis
     主动性Heymann肾炎中NF-κBp65在肾间质病变中的作用
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     A positive correlation was found between the positive rate of NF-κB p65 in renal tubule and the lesion score of renal interstitium, the levels of urinary protein (r=(0.7138) and (0.6376), P<0.05).
     肾小管NF -κBp6 5阳性数与尿蛋白水平和肾间质病变评分呈明显正相关 (r分别为 0 7138和 0 6 376 ,P <0 0 5 )。
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     Prostaglandin and interstitial nephropathy
     前列腺素与间质病变
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     The pathological changes were examined with Masson staining.
     Masson染色观察间质病变程度。
短句来源
     Renal Tubulointerstitial Lesions in 820 Cases of Glomerular Diseases
     820例小球疾病的小管间质病变
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     Tubulointerstitial lesion was classified by using Katafuchi scale.
     小管间质病变分级采用Katafuchi积分。
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     There is no significant difference in the pathological scores of tubule between the two groups.
     两组间间质病变病理评分无差异。
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o evaluate the pathological change of remissiveand

本文动态对比分析了20例长期缓解与9例有反复的小儿肾病综合征病理学改变,结果表明缓解组lgG,C3沉积随病情好转而明显减少(P<0.05)。反复组IgG沉积下减少,C3,Fib因病情反复而明显增多(P<0.05)。缓解组IgG,C3,Fib沉积减少程度明显优于反复组(P<0.05~0.001)。虽然在首次活检时反复组病理改变比缓解组严重,但是缓解组前后的病理形态改善程度明显优于反复组(P<0.001),肾小球病变的消长与肾间质病变具有同步性,小儿肾病综合征病理类型之间具有可变性。因此作者认为,两组病人肾脏对免疫复合物清除能力不同决定了病理学的改变及临床转归,肾间质病变与肾小球病变在发病机理上可能有一定的联系,另外,小儿肾病综合征病理改变是一动态变化的过程,病理学的变化决定了临床表现。

in order to clarify the role of adhesion molecules in unilateral ureteral obstruction(UUO)model,distribution of ICAM-1 and macropbages in the renal tubulointerstitium were studied and the therapeutic effects ofanti-mouse ICAM-1 mAb on this model were also investigated in the present study.90 NIH mice were divided intothree groups.After UUO operation,group A(n=42)were treated with anti-mouse ICAM-1 mAb(9mg·kg-1/d)for 5 days;group B(n=42)were given the same volume of buffered saline and used as controls.Sham opera-tion...

in order to clarify the role of adhesion molecules in unilateral ureteral obstruction(UUO)model,distribution of ICAM-1 and macropbages in the renal tubulointerstitium were studied and the therapeutic effects ofanti-mouse ICAM-1 mAb on this model were also investigated in the present study.90 NIH mice were divided intothree groups.After UUO operation,group A(n=42)were treated with anti-mouse ICAM-1 mAb(9mg·kg-1/d)for 5 days;group B(n=42)were given the same volume of buffered saline and used as controls.Sham opera-tion was performed in group C(n=6)which served as normal controls. All the mice were sacrificed on the day of1,3,5,7,9.12 and 15 after UUO respectively. Renal frozen sections were stained with anti-mouse ICAM-1 mAband anti-mouse Mac-1 mAb separately. The expression of ICAM-1 mRNA in the frozen sections were also detectedwith in situ hybridization technique.The relative areas of renal interstitium were measured with Masson stainingsections using a computer image analysis system. The results showed that there are high expression of ICAM-1and ICAM-1 mRNA in the renal interstitium at the first day after UUO and it could be lasted for 3~5days.Theexpression of ICAM-1 decreased from the 7th day after UUO,but the numbers of macrophages increased at thesame time,The relative areas of renal interstitium increased as time past after UUO,After the treatment of anti-mouse ICAM-1 mAb,the ICAM-1 expression delayed and decreased.The infiltration of inflammatory cells de-creased in the renal interstitium of the mice. The results indicate that there is close relationship between the ex-pression of ICAM-1 and the infiltration of macrophages in kidney of UUO mice.

利用单侧输尿管梗阻制备小鼠肾间质病变模型,观察了肾间质中细胞间粘附分子-1(ICAM-1)及Mac-1阳性细胞的变化,并利用抗小鼠ICAM-1单克隆抗体对此模型进行了治疗。结果发现在单侧输尿管梗阻后第一天,梗阻侧肾间质小血管附近及近端肾小管上即有ICAM-1及ICAM-1mRNA的表达,第3~5天达高峰,第7天时开始下降。与此同时,肾间质内Mac-1阳性的巨噬细胞也于梗阻后第3天开始出现,第7天达高峰。经抗ICAM-1单克隆抗体治疗后,ICAM-1及Mac-1的表达均延迟出现并明显减弱,肾间质病理改变亦有所减轻。

We determined the factors for postoperative persistent hypertension in the patients with aldosterone producing adrenal adenoma (APA) in 53 patients with APA who were followed up for average 3 1 years. All had normal serum potassium concentration postoperatively.Blood pressure was normal in 37 patients (69 8%) but 18 7/12 7kPa or more in 16 patients (30 2%) with persistent hypertension.Also compared were sex,age, history of hypertension, effect of reducing blood pressure to antisterone, preoperative blood...

We determined the factors for postoperative persistent hypertension in the patients with aldosterone producing adrenal adenoma (APA) in 53 patients with APA who were followed up for average 3 1 years. All had normal serum potassium concentration postoperatively.Blood pressure was normal in 37 patients (69 8%) but 18 7/12 7kPa or more in 16 patients (30 2%) with persistent hypertension.Also compared were sex,age, history of hypertension, effect of reducing blood pressure to antisterone, preoperative blood pressure, time of persistent hypertension, serum potassium concentration, aldosterone concentration in 24 hour urine,amount of PRA, and the type of operation.The results showed that an APA patient aged 50 years or more appears to have a great chance of persistent hypertension than an APA patient under age of 40 years, and the odds ratio is 3∶ 1.There was a significant difference between the mean age for persistent hypertension and for normal blood pressure, and varioas response of reducing blood pressure to antisterone( P <0 05). It is suggested that for an older APA patient and the patient without of reducing blood pressure to antisterone, there are other factors for hypertension such as renal veinlet change or renal interstitial lesions except for hyperaldosteronism. We recommend renal biopsy (using kidney puncture) at the operating table for those patients in order to understand pathological change and guide treatment after operation.

为了解产生醛固酮的肾上腺腺瘤(APA)术后部分患者持续高血压的病因,作者对53例产生醛固酮的肾上腺腺瘤术后患者进行了平均3.1年的随访。全部患者术后血钾正常,血压正常者37例,血压≥18.7/12.7kPa16例。将正常血压与持续高血压2组患者从性别、年龄、有无高血压史,对安体舒通的降压反应、术前高血压值、高血压时间、血清钾值、24小时尿醛固酮值、PRA值以及手术方式进行比较。结果显示:≥50岁APA患者发生持续高血压机会与<40岁APA患者比值比3∶1;在持续高血压的平均年龄与正常血压平均年龄以及对安体舒通的降压反应之间存在显著差异(P<0.05)。提示年龄愈大的APA患者以及对安体舒通降压反应不明显的患者除了醛固酮增高引起高血压的原因之外,尚有肾内小血管病变或肾间质病变等原因。作者建议对该类患者手术时行肾穿刺活检,以明确病因,有利术后进一步治疗。

 
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