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溃疡
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  ulcer
    Study of Psychosomatic Neuroendocrine-immunoregulatory Network Basis of Stress Ulcer and Effects of Xin Shen1hao
    应激性溃疡心身相关的神经内分泌免疫网络基础及心身1号干预的研究
短句来源
    EFFECT OF HIGH PLASMA HCO_3~- ON THE HEALING PROCESS OF ACETIC ULCER IN RAT
    血浆高HCO_3~-对大鼠乙酸溃疡愈合过程影响的研究
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    THE RELATIONSHIP AMONG TISSUE CALCIUM, SERUM CALCIUM AND DUODENAL ULCER
    大鼠组织及血清钙含量与十二指肠溃疡关系
短句来源
    THE EXPERIMENTAL RESEARCH ON THE MODEL OF ACUTE STRESS ULCER
    急性应激性溃疡模型的实验研究
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    Study on Therapeutic Mechanism of Xilei Powder for Peptic Ulcer
    锡类散治疗消化性溃疡作用机制的研究
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  ulceration
    Inhibitory effects of Ginkgolide A on stress ulceration in rats
    银杏内酯A对应激性溃疡发生的抑制作用
短句来源
    With the application of H2 receptor antagonist (H2-RA) and proton pumpinhibitor (PPI) in recent years, the ulceration got faster short-term healing.
    近年来随着 H2受体拮抗剂(H2-RA)及质子泵抑制剂(PPI)的应用,虽然能较快地促进溃疡近期愈合,但停药后,1 年内 65%~80%的病人复发,2 年内几乎 100%复发;
短句来源
    Therefore, according to the primary efficacy and attending symptom of HWJND, referring to the research of literature, we mostly study the affection of HWJND to gastric juice of SU's model in rat、 the index of gastric ulceration> the content of GMBF、 PGE_2、 NO and iNOSmRNA in gastric mucosal etc.
    通过和胃降逆方对SU模型大鼠的实验研究,发现和胃降逆方按15、20、25g生药/kg体重给予SU模型大鼠,能减少SU模型大鼠的胃液的分泌量和胃粘膜溃疡指数,增加胃粘膜血流量、前列腺素含量,降低胃粘膜NO含量及其iNOSmRNA表达水平等,结果具有统计学意义(p<0.05-0.01)。
短句来源
    The rate of gastric ulceration was 41.0 per cent when injected different dose of red pepper alkaloid extract to submucosa of the antrum pyloricum of rats.
    将不同剂量的辣椒生物硷提取液注入大鼠胃窦前壁浆膜下,6周后观察表明,在39只大鼠中有16只诱发胃窦前壁粘膜溃疡,溃疡发生率为41.0%。
短句来源
    Results:Pretreatment with EGb (1040 mg/kg) not only significantly inhibited the myoelectric activity response,but significantly reduced MDA levels and gastric mucosal ulceration index induced by stress in a dosedependent manner ( P <0 05 and P <0 01 vs.control group).
    结果:EGb10~40 m g/kg预处理可显著压抑应激所致的胃肌电活动紊乱及血浆和胃粘膜组织MDA 水平的异常升高(P< 0.05 或P< 0.01),使胃粘膜溃疡指数明显降低(P均< 0.05),并具有剂量依从性。
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  gastric ulcer
    Antiulcer mechanism of Kuiyang powder (溃疡散) on experimental gastric ulcer in rats
    溃疡散对大鼠实验性胃溃疡的保护机制
短句来源
    Kuiyangsan's Protection on Experimental Gastric Ulcer and Its Mechanism
    溃疡散对实验性胃溃疡的保护作用及机制
短句来源
    Experimental Study of Pingwei Capsule's Inhibition of Gastric Ulcer and Its Anti-inflammatory and Analgesic Action
    平胃胶囊抑制溃疡及消炎、止痛作用的实验研究
短句来源
    1. JQNT Capsule could obviously diminish ulcer area ofreserpine gastric ulcer on rats. It could increase the content ofPOE2 and regulate the counterbalance of TXB2/6-K-PGF1 a mgastric mucosa that improved microcirculation.
    1、九气拈痛胶囊能明显缩小利血平胃溃疡大鼠的溃疡面积,增加胃粘膜前列腺素(PGE_2)的含量,调节TXB_2/6-K-PGF_(1α)的平衡,改善微循环。
短句来源
    The volume (4.75±0.88mm~3) and area (4.05±0.93mm~2) of gastric ulcer decreased in the rats with slight hyperthyroidism, and the healing rate of gastric ulcer accelerated (37±3.8% for volume healing rate, 49±3.2% for area healing rate).
    甲轻亢组溃疡容积(4.75±0.88mm~3)和面积(4.05±0.93mm~2)都减小,但无统计学意义,溃疡容积愈合率(37±3.8%)和面积愈合率(49±3.2%)显著升高;
短句来源
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  “溃疡”译为未确定词的双语例句
    ANTIULCER EFFECT OF THE POLYSACCHARIDES FROM TREMELLA FUCIFORMIS SPORES OF TREMELLA FUCIFORMIS AND AURICULARIA AURICULA
    银耳多糖、银耳孢子多糖及黑木耳多糖的抗溃疡作用
短句来源
    [Conclusion]Cuyu granule can improve the level of EGF and EGFR and prevent GU from recurring by improving maturation of regenerative mucosa in GU rats.
    [结论]促愈颗粒可能通过增加胃黏膜EGF和EGFR的水平,进而提高GU再生黏膜结构和功能成熟度,从而促进溃疡愈合,提高溃疡愈合质量,并防止溃疡复发。
短句来源
    Methods The change of the collagen type Ⅰ、Ⅲ mRNA expressions were measured by semi-quantitative RT-PCR. the collagen type Ⅰ、Ⅲ content was used to detect by radioimmunity.
    方法用含/未含黄芪的培养液培养糖尿病足溃疡处成纤维细胞,RT-PCR检测Ⅰ、Ⅲ型胶原mRNA表达,放免法检测培养上清液中Ⅰ、Ⅲ型胶原含量。
短句来源
    Results It showed that the collagen type Ⅰ、Ⅲ mRNA expressions and collagen type Ⅰ、Ⅲ content were significantly higher compared with those cultured without Astraglus (P
    结果用含黄芪的培养液培养的糖尿病足溃疡处成纤维细胞Ⅰ、Ⅲ型胶原mRNA表达及培养上清液中Ⅰ、Ⅲ型胶原含量高于未含黄芪的培养液培养的细胞,且差异有统计学意义(P<0.05)。
短句来源
    Anti-ulcer Action of Extracts from Opuntia Dillenii on Rats
    仙人掌提取物抗溃疡作用
短句来源
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  ulcer
Diclofenac (25 mg kg-1) and meloxicam (7.5 mg kg-1) produced 9.3 and 19.6 mm2 ulcer areas in stomachs of rats, respectively, when used alone, but when combined with nimesulide diclofenac and meloxicam did not cause any injury in rat stomachs.
      
While ranitidine at 100 mg kg-1 dose prevented diclofenac-induced ulcer formation, it reduced meloxicam-induced ulcer formation significantly.
      
Antiulcer Effects of the Tripeptide PGP and Its Possible Metabolites (PG, GP, Glycine, and Proline) in Different Models of Ulcer
      
PG reduced the ethanol- and indomethacin-induced lesions approximately twofold but was considerably less effective in stress-induced ulcer.
      
GP decreased the stress- and indometacin-induced ulcer but tended to stimulate the ethanol-induced lesions.
      
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  ulceration
We propose to treat deep corneal burns by grafting a living stromal equivalent, since keratocyte death and stromal destruction are the main factors of ulceration and destruction of the eye.
      
The Pro-Gly-Pro glyproline increases a resistance of the gastric mucosa (GM) to ulcerogenic factors in various models of ulceration and exhibits therapeutic properties in acceleration of the healing of an acetate ulcer.
      
Statistically reliable antiulcer effects in all the ulceration models were found for only two peptides: PGP and GPGPGP.
      
The PGP tripeptide displays a wide spectrum of physiological activities and affects a number of mechanisms that cause the development of GM ulceration.
      
The damage of barrier in mucosa with erosion or ulceration, and the thickness of mucin were examined by scanned electron microscope (SEM).
      
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  gastric ulcer
About 30% of patients with gastritis, active duodenal or gastric ulcer had antibody levels as low as the seroconverted dyspeptic patients but with normal gastroduodenal mucosa.C.
      
Study on clinical effect and mechanism of Jianpi Qingre Huayu Recipe (健脾清热化瘀方) in treating patients with gastric ulcer
      
Effect of mexican tea herb and pilular adina herb on concrescence of gastric mucosa in experimental gastric ulcer rats
      
pylori-infected child, and current guidelines recommend eradication therapy for infected children with duodenal and gastric ulcer, gastric lymphoma, and atrophic gastritis with intestinal metaplasia.
      
Other "less traditional" therapies, including probiotics, heparins, and anti-gastric ulcer remedies, challenge our understanding of the pathogenesis of ulcerative colitis and may provide further insights into future therapies.
      
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Experimental intestinal amoebiasis was pro- duced by inoculating directly into the caecum of guinea pigs faeces containing 100,000 trophozoites from a patient with acute intestinal amoebiasis. The results obtained from 18 guinea pigs showed that the rate of infection was 83%, while the rate of morbidity was 44%. The pathological changes of the intestinal tract were observed as follows:gross examination showed that the lesions occurred mainly in the caecum,most of them were localized in distribu- tion,varied...

Experimental intestinal amoebiasis was pro- duced by inoculating directly into the caecum of guinea pigs faeces containing 100,000 trophozoites from a patient with acute intestinal amoebiasis. The results obtained from 18 guinea pigs showed that the rate of infection was 83%, while the rate of morbidity was 44%. The pathological changes of the intestinal tract were observed as follows:gross examination showed that the lesions occurred mainly in the caecum,most of them were localized in distribu- tion,varied in size,and numbered from one to two in each guinea pig. The lesions consisted of greyish-white fibrin- ous pseudomembrane,some were ulcerated. Microscopically,two types of pathological changes were encountered.Type one showed loca- lized necrosis with disseminated amoebic tropho- zoites in the lymph follicle but without any inf- lammatory reaction.In type two there was a considerable amount of fibrinous exudation and necrosis on the surface of the mucous membrane. This necrosis often extended far into the submu- cous layer to form a typical flask-shaped ulcer, where a large number of amoebic trophozoites were found lining its wall.Marked inflammatory reaction in the surrounding tissue was seen. It was demonstrated that the pathological lesions found in the guinea pigs were those of typical intestinal amoebiasis.The problems of site of infection,the pathway of amoebic invasion and the pathogenesis of amoebiasis were discussed.

我们从一例急性阿米巴痢疾患者的粪中所得阿米巴虫栋(Strain C_2),直接以盲肠内接种方法感染豚鼠,接种量为100,000个滋养体。对感染后的观察结果,作本文报告。通过18只豚鼠的实验感染表明,所致感染率为83%;致病率为44%。肠道的病变表现为:纤维素假膜炎和假膜脱落后形成的溃疡。对豚鼠的病变是由阿米巴感染所致提出论据,并对病变部位与侵袭途径,阿米巴的感染与致病问题作了探讨。

The Chian-Chung-Tang has been used clinically for treatment of gastric ulceration. It is composed of Hedysarum polybotrys,Cinnamom carsia,Paeonia lactiflora Gly- cyrrhiza uralensis, Angelica sinenss, Rhizoma Zingiberis, Fructus Zizyphi jujubae, and Saccharum Grannorum. 1.The induction rate of Shay rat ulceration together with the quantity of gastric secretion, free acidity, and total acidity was markedly dereased by Chian-Chung-Tang given subcutaneously, but the same effect was not found, when the drug was given...

The Chian-Chung-Tang has been used clinically for treatment of gastric ulceration. It is composed of Hedysarum polybotrys,Cinnamom carsia,Paeonia lactiflora Gly- cyrrhiza uralensis, Angelica sinenss, Rhizoma Zingiberis, Fructus Zizyphi jujubae, and Saccharum Grannorum. 1.The induction rate of Shay rat ulceration together with the quantity of gastric secretion, free acidity, and total acidity was markedly dereased by Chian-Chung-Tang given subcutaneously, but the same effect was not found, when the drug was given orally. 2.The movement of the stomach of the pigeon as well as the movement of the intestine of the rabbit in situ and that of rabbit and rat in vitro was depressed by the drug. 3.The drug was also found to antagonize the effect of pilocarpine and prostigmine upon the movement of intestine of rabbit in situ, and that by acetylcholine and pilocar- pine upon the movement of intestine of rabbit and rat in vitro. 4.The LD_(50) of Chian-Chung-Tang in mice was found to be 48g/kg given sub- cutaneously.

本文对加味黄芪建中湯的抗大白鼠胃溃疡作用及毒性作了初步探讨.建中湯煎剂给大白鼠皮下注射10克/公斤,可防止幽门结扎所致胃溃疡的发生;还能抑制胃液分泌、减少游离酸和总酸度;并使胃液pH值上升.但灌胃给药25克/公斤则不能防止溃疡的发生.建中湯煎剂对鸽胃正常运动及家兔腸运动都有抑制作用,并在一定程度上能对抗乙酰胆碱和毛果云香碱所致的腸痉挛.建中湯煎剂给小白鼠皮下注射的LD_(50)为48±7.2克/公斤.

Antioxidant D,with N-phenyl-2-naphthylamine as its main ingre-dient,was suspected to be carcinogenic.Opinions differed as to whether the carcinogenicity was due to N-phenyl-2-naphthylamine itself or its probable contaminant,2-naphthylamine.In the present experiment 2-naphthylamine-free antiexidant D,containing more than 85% N-phenyl-2-naphthylamine and a small amount of N-phenyl-1-naphthylamine,was dissolved in sesame oil and given to male albino rats 5 times weekly at individual dosages of 16 mg,10 mg and 4...

Antioxidant D,with N-phenyl-2-naphthylamine as its main ingre-dient,was suspected to be carcinogenic.Opinions differed as to whether the carcinogenicity was due to N-phenyl-2-naphthylamine itself or its probable contaminant,2-naphthylamine.In the present experiment 2-naphthylamine-free antiexidant D,containing more than 85% N-phenyl-2-naphthylamine and a small amount of N-phenyl-1-naphthylamine,was dissolved in sesame oil and given to male albino rats 5 times weekly at individual dosages of 16 mg,10 mg and 4 mg/100 mg b.w.for 11/2 Yrs.All surviving exprimental andcontrol rats were sacrificed at the end of 2nd exprimental year and subjected to detailed anatomical and histologi-cal examination.57 Rats given Antioxid-ant D developed 47% various malig-nant tumors with predominance of car-cinomas of the lungs,prostate,pan-creas and thyroid glands,43 Rats given sesame oil only as well as 62 untreated rats had 11.6% and 9.7% malignant tumors respectively,without above-mentioned carcinomas Results of the experiment demonstrated the car-cinogenecity of Antioxidant D and suggested its possible selective affinity for the lungs.

长期灌喂大白鼠防老剂D的麻油溶液,在总剂量0.5至10克、实验135—735天时,引起显著高于麻油及自然对照组动物的癌瘤,分散于多个器官系统,以肺癌、前列腺癌及胰腺癌为多见。实验组动物的胃和阑肠溃疡高发。所用防老剂D不含2-萘胺,其所诱发肿瘤和2-萘胺无关。

 
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