Objective: To study the effects and clinical significance of plasmal thromboxane B_2 (TXB_2), platelet alpha granule membrance protein-140 (GMP-140), 6-Keto-PGF1a (6-K- PGF1α), endothelins(ET), tissue type plasminogen activator(tPA)and plasmingen activator inhibitor-1 PAI-1) in patients with cerebral arteriosclerosis and cerebral thrombosis.
Employing Platelet Alpha Granule Membrance Protein 140 (GMP 140) as indicator for platelet activation,determination has been made for the plasma's GMP 140 concentration for 74 cases of diabetes,36 cases of cardiovascular disease and 53 cases of rend disease and comparison has been made for the GMP 140 values determined respectively among the above mentioned three groups of diseases and between the group of bolld stasis syndrome and the group of non blood stasis syndrome.
Objective:To study the effects of tissue-type plasminogen activator(t-PA),plasminogen activator inhibitor-1 (PAI-1),thrombomodulin(TM)and platelet alpha granule membrance protein-140(GMP-140) in patients with acute cerebral infarction(CI).
Objective To study the changes and the role of platelet alpha granule membrane protein (GMP 140) and thrombomodulin (TM) in children with Henoch Schenlein purpura (HSP).
Objective:To investigate the changes of lipid peroxide (LPO) ,superoxide dismutase (SOD) and platelet alpha- granule membrane protein1 40 (GMP- 1 40 ) in patients of acute myocardial infarction (AMI) and unstable angina pectoris (UA) and the significance.
The levels of platelet alpha-granule membrane protein (GMP-140),tissue-type plasminogen activator (t-PA) and its inhibitor (PAI-1) were measured before and after treatment.
Methods:Enzyme-linked immunosorbent assay(ELISA) was used to measure plasma plasminogen activator inhibitor-1(PAI-1), platelet alpha-granule membrane protein-140(GMP-140) and von Willebrand factor(vWF) in 60 patients with CHF and in 20 normal controls.
Objective: To study the effect of Maitork (a sodium chloride injection containing Ginkgo bioba extract and ligustrazine phosphate) on plasma levels of fibrinogen (Fg), platelet granule membrane protein 140 (GMP-140), tissue plasminogen activator (tPA) after focal cerebral ischemia-reperfusion in rats.
Four thrombogenic and thrombolytic markers including endothelin 1(ET 1), platelet granule membrane protein 140 (GMP 140), thrombin active fragment (FⅡa) and D Dimer were determined before and after ILLLI or aspirin treatment.
With monoclonal antibody SZ 51, the activated levels of the platelet granule membrane protein(GMP 140) of 94 cases with coronary heart disease (CHD) are assayed by RIA.
Objective To study the changes of plasma platelet granule membrane protein-140 (GMP-140) in patients with cerebral thrombosis in acute and recovery phases.
Animals of pseudooperated and model groups were fed with water (20 ml/kg),PGHZS experiment group with PGHZS (25.40 g/kg) for 2 weeks,the activity of tissue plasminogen activator(tPA),plasminogen activator inhibitor(PAI) and concentration of platelet αgranule membrane protein140(GMP140) in blood samples from model abdominal aorta were analyzed.
Objective:To observe the dynamic changes in oxygen free radical (OFR),endothelin (ET),platelet αgranule membrane protein (GMP140) before and after intravenous thrombolytic therapy with urokinase and their relationship with early reperfusion in patients with acute myocardial infarction (AMI). Methods:38 patients were included in the study.
5-HT can also antagonize the apoptotic effect induced by thrombospondin-1 (TSP-1) which is a platelet α granule protein and has synergic effect with platelet-derived growth factor (PDGF) to enhance megakaryocytes proliferation. Therefore, 5-HT is likely to be an important substance in the feedback regulation of thrombopoiesis.
Our findings support the conclusion that exercise-induced peripheral ischemia with severe symptoms of claudication does not produce platelet alpha-granule release.
The results showed that the platelet granule membrane protein 140 (GMP-140) level increased obviously in CHD groups compared with normal control, suggesting that platelet activation existed in CHD.
These data suggest that IgG-Agg releases platelet granule ADP by a cyclooxygenase-dependent mechanism and the released ADP, in combination with AGEPC, activates platelets synergistically.
Platelet function was examined by a cytofluorimetric method, using an anti-GPM-140 antibody which is directed against a platelet α granule membrane protein.
血小板α颗粒
platelet alpha granule
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