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阿尔茨海默病ad
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  “阿尔茨海默病(ad)”译为未确定词的双语例句
     Objective To detect the relation between 5-HT2A receptor gene polymorphism and Alzheimerdisease(AD) in Chinese.
     目的探讨中国汉族人群中5-羟色胺(5-HT)2A(5-HT2A)受体基因多态性与阿尔茨海默病(AD)的相互关系。
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     To study the feature of rCMRglc in patients with mild cognitive impairment (MCI) and in patients with Alzheimer's disease (AD) by positron emission tomography (PET) and its relationship with neuropsychological test;
     (2) 探讨轻度认知功能障碍(MCI)与阿尔茨海默病(AD)的脑葡萄糖代谢、神经心理学特点及其之间的关系,以及MCI与AD的正电子发射计算机断层扫描(PET)诊断,从神经心理及脑葡萄糖代谢的角度探讨MCI与AD之间的关系。
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     Objective To study the association of Alzheimer′s disease(AD) with the apolipoprotein E (ApoE) ε4 allele in Chinese of Beijing.
     目的研究北京地区阿尔茨海默病(AD)与载脂蛋白E(ApoE)基因的ε4等位基因的相互关系。
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     Objective To study glucose metabolism of the brain in Azheimer's disease by positron emission tomography (PET) , and to investigate the imaging characteristics and diagnosis criteria of 18F-FDG PET in Chinese Alzheimer's disease(AD).
     目的 探讨阿尔茨海默病(AD)脑葡萄糖代谢及其18F-脱氧葡萄糖正电子发射计算机断层扫描(18F-FDG PET)显像的影像学特征和PET诊断标准。
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     Methods: Odd ball auditory evoked model was used to test the P300 of 24 patients with vascular dementia(VD), 25 patients with Alzheimer's dementia (AD) and 22 normal controls (NC).
     方法:选用听觉。 odd ball诱发范式对24例血管性痴呆(VD)、25例阿尔茨海默病(AD)和22例正常老年人(NC)进行P300测试。
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     An investigation of the rehabilitation efficacy in Alzheimer's disease
     阿尔茨海默的康复治疗
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     Amyloid-β immunotherapy for Alzheimer's disease
     阿尔茨海默的免疫治疗
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     Aim:To study the nature of the olfactory dysfunction associated with Alzheimer's disease(AD).
     目的 :研究阿尔茨海默 (AD)患者嗅觉障碍。
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     Objective To investigate the etiology and pathogenesis of Alzheimer's disease(AD).
     目的 探讨阿尔茨海默 (AD)的因及发机制。
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     Objective It is known that dementia is a multi-factorial disorder, but the etiological factors other than aging remain to be explored, hence we sought to investigate the risk factors of dementia and Alzheimer' s disease (AD).
     目的探讨老年痴呆和阿尔茨海默(AD)的危险因素。
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  alzheimer ' s disease (ad)
β-Amyloid peptide (Aβ), a normal constituent of neuronal and non-neuronal cells, has been proved to be the major component of extracellular plaque of Alzheimer's disease (AD).
      
A significant proportion of early onset Alzheimer's disease (AD) is caused by mutations in human genes for amyloid precursor protein (APP), presenilins 1 and 2 (PSEN1,2).
      
Missense mutations in human presenilin 1 gene (hPS1) cause an autosomal dominant, early onset form of Alzheimer's disease (AD).
      
Among three isoforms, the apoE4 isoform is associated with increased frequency of atherosclerosis and Alzheimer's disease (AD).
      
Studies of the molecular mechanisms of Alzheimer's disease (AD) have led to two major achievements.
      
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Objective The purpose of this study was to explore the relation between Ca 2+ in brain cells and Alzheimer′s disease(AD). Methods The experimental rats were injected an excitotoxin, kanic acid (KA) to destroy the nucleus basal of Meynert (NBM) to made a brain pathological model of Alzheimer ,s disease. The effect of KA on brain cells Ca 2+ in sucking rats were tested with Fura 2/AM. Nikadipine, KCl, as the voltage dependent calcium channel ,s activator and blockade were used to determine that...

Objective The purpose of this study was to explore the relation between Ca 2+ in brain cells and Alzheimer′s disease(AD). Methods The experimental rats were injected an excitotoxin, kanic acid (KA) to destroy the nucleus basal of Meynert (NBM) to made a brain pathological model of Alzheimer ,s disease. The effect of KA on brain cells Ca 2+ in sucking rats were tested with Fura 2/AM. Nikadipine, KCl, as the voltage dependent calcium channel ,s activator and blockade were used to determine that KA through what channel made Ca 2+ to flow into brain cells. Results KA with different concentration could increase the [Ca 2+ ]i in brain cells of sucking rats ( P <0.05). KA caused Ca 2+ to flow into brain cells through the voltage dependent calcium channel. In addition, glutamate, as an excitotoxin, though receptor dependent calcium channel increased [Ca 2+ ]i in brain cells ( P <0.05). Conclusion It is suggested that KA could cause the overload of Ca 2+ in brain cells, which may be linked to the development of AD.

目的探讨脑内细胞钙离子与发生阿尔茨海默病(AD)之间的关系。方法用兴奋性神经毒海人藻酸(KA)破坏Meynert基底核(NBM),复制出与人类AD相似的大鼠脑神经病理改变模型。采用Fura-2/AM测定乳鼠脑细胞游离钙的方法观察KA的影响。同时应用尼卡地平、氯化钾作为电压依赖性钙通道阻滞剂与激动剂,确定KA是通过何种钙通道促使钙内流的。结果用不同浓度KA均可以使乳鼠脑细胞胞浆游离钙浓度明显升高(P<0.05),并证实KA是通过电压依赖性钙通道使钙离子流入脑细胞的。用同属兴奋性神经毒的谷氨酸可通过受体依赖性钙通道使脑细胞胞浆游离钙增多(P<0.05)。结论KA可使鼠脑细胞内钙超载,后者与AD的发生有关。

Objective The study was to investigate the characteristics of brainstem auditory evoked potentials (BAEP) in patients with Alzheimer′s disease (AD) and senile schizophrenics. Methods The BAEP was elicited from 32 AD patients, 34 senile schizophrenics and 40 normally agematched controls by presenting them clicks using a Dantec Concerto Instrument. Results The variability of BAEP waveform of AD patients became increased. The AD group presented prolonged III, IV and VI latencies, decreased II, IV, VI and VII...

Objective The study was to investigate the characteristics of brainstem auditory evoked potentials (BAEP) in patients with Alzheimer′s disease (AD) and senile schizophrenics. Methods The BAEP was elicited from 32 AD patients, 34 senile schizophrenics and 40 normally agematched controls by presenting them clicks using a Dantec Concerto Instrument. Results The variability of BAEP waveform of AD patients became increased. The AD group presented prolonged III, IV and VI latencies, decreased II, IV, VI and VII amplitudes. The V amplitude was 18% lower than that of schizophrenics and 34% lower than that of normal controls. Compared with normal controls, schizophrenics showed prolonged III and VI latencies, and their VII amplitude was decreased by 20%.Conclusion The abnormal BAEP of these aged patients with AD and schizophrenia is suggested to be related to the brain area between the auditory nerves and the pons.

目的探讨阿尔茨海默病(AD)和老年精神分裂症(SS)脑干听觉诱发电位(BAEP)的特点。方法应用丹麦电生理仪及Click短声刺激,测查32例AD、34例SS和40名正常老年人(NC)的BAEP。结果AD组患者波形变异大,绝对潜伏期波Ⅲ、波Ⅳ和波Ⅵ明显延迟,主波绝对波幅波Ⅴ平均波幅分别低于SS组和NC组18%和34%,波Ⅱ、波Ⅳ、波Ⅴ、波Ⅵ和Ⅶ绝对波幅也显著低于SS组和NC组,与NC组间差异有显著性。SS组波Ⅲ、波Ⅵ绝对潜伏期较NC组延迟,绝对波幅波Ⅶ平均波幅低于NC组20%。结论老年精神疾病BAEP异常表现在听神经至桥脑下段之间

Objective To explore the incidence of dementia and Alzheimer disease (AD) and the impact of gender, age and education. Methods Study subjects were stratified clustering sampled from community and assessed by a two stage procedure twicely during a period of 5 years. The screening instrument was Mini Mental Status Examination and the clinical diagnosis of dementia and AD were base upon The 3rd Edition Diagnostic and Statistical of Manual of Mental Disorder, Revised version and NINCDS ADRDA criteria. Results...

Objective To explore the incidence of dementia and Alzheimer disease (AD) and the impact of gender, age and education. Methods Study subjects were stratified clustering sampled from community and assessed by a two stage procedure twicely during a period of 5 years. The screening instrument was Mini Mental Status Examination and the clinical diagnosis of dementia and AD were base upon The 3rd Edition Diagnostic and Statistical of Manual of Mental Disorder, Revised version and NINCDS ADRDA criteria. Results 3 024 subjects of non dementia in 1987 were re surveyed in 1992, and of them 141 new cases of dementia including 92 cases of AD were identified. The annual incidence of dementia and AD are as follows: 0.71% and 0.42% in those of 55 years and over, 0.91% and 0.56% in 60 years and over, 1.31% and 0.89% in 65 and over. Analysis from single factor, two factors and logistic regression showed that age and education but not gender were closely related to occurring of dementia and AD. Conclusion The incidence of dementia and AD in Shanghai cohort is similar to that from other countries. Both of them are age related diseases. Education shows a great protective effect and lack of formal education is one of the risk factors.

目的调查痴呆和阿尔茨海默病(AD)的发病率及其与性别、年龄、教育程度的关系。方法采用二阶段法,在1987年调查的基础上,于1992年对上海市静安区的居民进行调查。按美国精神障碍诊断和统计手册第3版修订本、美国神经病和言语障碍和中风研究所/阿尔茨海默病和相关疾病协会的标准诊断痴呆和AD。结果1992年在实访的3024人中,痴呆和AD的年发病率:55岁以上为0.71%和0.42%,60岁以上为0.91%和0.56%,65岁以上为1.31%和0.89%。经单因素、双因素和Logistic回归分析示,发病率与性别无关,与年龄及教育程度密切相关。结论痴呆和AD的发病率与世界各国报道相似。教育对痴呆的发生具保护作用,文盲为危险因素

 
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