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急性肝衰竭的
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  acute liver failure
     EXPERIMENTAL STUDY OF SV_(40) Tag HEPATOCYTE TRANSPLANTATION IN THE TREATMENT OF SURGICAL ACUTE LIVER FAILURE
     猿病毒40大T抗原基因(SV_(40)Tag)肝细胞移植治疗手术介导的急性肝衰竭的实验研究
短句来源
     The protective effect of Gabexate mesilate on D-galactosamine-lipopolysaccharide-induced acute liver failure in rats
     加贝酯对D-氨基半乳糖加脂多糖引起的大鼠急性肝衰竭的保护作用
短句来源
     Objective:To investigate the therapeutic effect of microencapsulated pig hepatocyte transplantation on acute liver failure rats.
     目的:探讨微囊化猪肝细胞移植治疗大鼠急性肝衰竭的效果。
短句来源
     Therapeutic effect of transplantation of microencapsulated human immortalized hepatocytes on acute liver failure in mice
     微囊化人肝细胞移植对小鼠急性肝衰竭的治疗作用
短句来源
     Treatment of acute liver failure with bone marrow stem cells transplantation in rats
     骨髓干细胞移植治疗急性肝衰竭的实验研究
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  acute hepatic failure
     Objective:To research the relation of Th1/Th2 imbalance and acute hepatic failure induced by D-glactosamine/endotoxin.
     目的:研究Th1/Th2失衡与D-氨基半乳糖/内毒素所致大鼠急性肝衰竭的关系。
短句来源
     Objective To evaluate the protective effect of recombinant human granulocyte colony stimulating factor (rhG-CSF) on acute hepatic failure induced by galactosamine (D-GalN) and lipopolysaccharide (LPS) in mice, and to explore its mechanism.
     目的:观察重组人粒细胞集落刺激因子(recombinant human granulocyte colony stimulating factor;rhG-CSF)对D-氨基半乳糖(D-galactosamine,D-GalN)加脂多糖(lipopolysaccharide,LPS)联合诱导的小鼠急性肝衰竭的保护作用,并探讨其机制。
短句来源
     With verified rather good effect of Qingkai Granule on acute hepatic failure in rats in previous study, acute hepatic failure model in mice is established to investigate roles of hepatic cells apoptosis and caspase-3in fulminant hepatitis and action mechanism of Qingkai Granule against acute hepatic failure.
     然而对重型肝炎肝细胞凋亡的药物阻断方面却研究较少,以往的研究表明清开颗粒对实验性急性肝衰竭大鼠有一定的治疗和预防作用,为了探讨肝细胞凋亡、Caspase-3在重型肝炎中的作用及清开颗粒对肝细胞凋亡的保护作用,我们建立小鼠急性肝衰竭模型来从分子水平进一步研究清开颗粒抗急性肝衰竭的作用机制。
     Results(1)Different extent liver injury can be induced by different dosages of D-gal,and the pathophysiology of acute hepatic failure can be copied very well at the dose of 1.4g/kg,and the model is very steady.
     结果(1)不同剂量的D-gal可导致不同程度的肝损伤,而1.4 g/kg剂量较好地模拟了急性肝衰竭的病理生理改变且稳定性较好。
短句来源
     Study on Effect of Hepatocytes Transplantation in Rats with Acute Hepatic Failure
     肝细胞移植治疗大鼠急性肝衰竭的研究
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  “急性肝衰竭的”译为未确定词的双语例句
     Results The expression of toll like receptor 4 mRNA was significantly increased at 12 hour and significantly different from control group(P<0.05). The expression of CD14 mRNA was significantly different from control group at 6 hour(P<0.05). They both reached the peak at 48 hour,gradually deseased at 72,120,148 hour point.
     结果Toll样受体4mRNA在急性肝衰竭的12 h有显著升高,与正常组差异有显著性(P<0.05),CD14 mRNA在6 h与正常组差异有非常显著性(P<0.01),两者均在48 h达到高峰,72、120和148 h逐渐下降。
短句来源
     Toll like receptor 4 and CD14 are intimately associated with actue hepatic failure by D-galactosamine. They may become a new treat target in clinical.
     Toll样受体4,CD14与D-氨基半乳糖介导的急性肝衰竭有着密切的联系,可能成为临床上治疗急性肝衰竭的新靶点。
短句来源
     Methods 16 patients with MODS including ALF were treated with PE and CBP except general management, various biochemical parameters and serum TNF-α, IL-6 were determined before and after treatment.
     方法  16例伴有急性肝衰竭的MODS患者除经内科常规治疗外 ,行PE联合连续性静脉 -静脉血液滤过 (CVVH)治疗 ,在治疗前、PE后、CVVH结束时 ,分别抽血测肝肾功能、血氨、TNF -α及IL- 6的水平 ,并进行ICU监护。
短句来源
     ALF carries a very high mortality of 50-80% because of the lack of effective medical therapeutic measures.
     由于缺乏有效的内科治疗手段,目前病毒性肝炎所致急性肝衰竭的病死率仍高达50-80%。
短句来源
     Effect of fetal hepatocyte culture supernatant on acute hepatic failure in mice
     胎肝细胞培养上清液对小鼠急性肝衰竭的保护作用
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  acute liver failure
Acetaminophen overdose and acute liver failure: Modern management
      
Renal extracorporeal support should be initiated using a continuous method when recovery of liver function is expected (acute liver failure, waiting for liver transplantation).
      
Acute liver failure represents a serious life-threatening event comparable to acute heart failure with cardiogenic shock or acute renal failure.
      
He developed fulminant hepatitis with acute liver failure and an acute respiratory distress syndrom (ARDS).
      
Three months later the infant had acute liver failure.
      
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  acute hepatic failure
A molecular adsorbent recycling system in treating posthepatectomy acute hepatic failure patients with hepatocellular carcinoma:
      
Differential gene expression profiles in acute hepatic failure model in mice infected with MHV-3 virus intervened by anti-hepati
      
Differential gene expression profiles in Balb/cJ mouse model of acute hepatic failure infected with MHV-3 virus intervened by anti-hepatic failure compound (AHFC) and the changes of cytokines regulated by genes were investigated.
      
Acute hepatic failure model of Balb/cJ mice infected with MHV-3 virus was established.
      
Changes of high mobility group box 1 in serum of pig acute hepatic failure model and significance
      
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It has been accepted that the Dgalactosamine (GaIN) induced fulminant hepatic failure (FHF) in the rat is a good model for assessment of artificial liver support system. However, in such a study extrahepatic factors other than the extensive and profound liver damage during hemoperfusion can also be of utmost importance in the correct evaluation of the results of hemoperfusion in FHF rats. It is important to select an optimum dosage of GalN in inducing FHF animal models for experimentation. The liver damage induced...

It has been accepted that the Dgalactosamine (GaIN) induced fulminant hepatic failure (FHF) in the rat is a good model for assessment of artificial liver support system. However, in such a study extrahepatic factors other than the extensive and profound liver damage during hemoperfusion can also be of utmost importance in the correct evaluation of the results of hemoperfusion in FHF rats. It is important to select an optimum dosage of GalN in inducing FHF animal models for experimentation. The liver damage induced should be neither too severe nor too mild, and the level of SGPT is a useful guide for choosing suitable FHF rats for hemoperfusion.The ambient temperature has been shown to be an interferring factor. The inhibiting effect of trauma or operation given before the injection of GaIN for induction of hepatic lesion is another important factor to be considered in establishing a chronic model for hemoperfusion.Extra-hepatic factors with the possibility of influencing the effects of hemoperfusion must also be taken into consideration: hypovolemia and hemoconcentration should be corrected as soon as possible, metabolic acidosis avoided and blood pressure kept within normal range.Lowering of blood cortisol level in FHF rat remains a problem to be studied. However, our investigations revealed no cortisol adsorption by the adsorbent ACAC.There may be still other factors deserving of investigation besides those observed in the present study. Since hemoperfusion of FHF rat model is a method now widely used in experiments, further studies of the factors influencing the effect of hemoperfusoin are urgently needed for a better understanding of the pathophysiologic status of FHF rat models during hemoperfusion.

将D-氨基半乳糖形成的大鼠急性肝衰竭模型用于考核人工肝辅助装置的疗效,这一方法已经证明是令人满意的,但必须掌握实验条件,即一些影响因素,才能正确判断其疗效。例如D-氨基半乳糖的用量必须恰如其分,才能使肝损害不致偏轻或偏重,而且能及时测定SGPT值,可供选择时的有益指标。对室温的影响必须重视,高温及低温有使肝损害加重或减轻的不良后果。在建立慢性血液灌流模型时,不同性质的损伤,可能会形成一种干扰因素而影响结果。其他一些肝外因素也须加以考虑:低血容量及血浓缩现象宜尽快纠正;代谢性酸中毒须加以避免;血压须始终保持在接近正常水平。模型大鼠的低皮质醇血浓度值得进一步研究,但炭吸附剂ACAC血液灌流对它并无影响。 我们的研究仅限于问题的一个方面。鉴于大鼠模型的血液灌流,目前为一实用的研究方法,进一步探讨这些病理生理性的改变也很有必要。

In this study, acute liver failure was induced in rats by d-galactosamine (D-GalN) injection. Intraperitoneal injection,24 h later, of viable syngeneic hepatocytes apparently increased the survival rate of these animals (48%), while transplantation of non-viable hepatocytes was ineffective and even showed a certain degree of clotting disorders.A microscopical observation of the liver specimens and estimation of DNA synthesis cy 3H-TdR suggest that viable hepatocellular transplantation may minimize the hepatic...

In this study, acute liver failure was induced in rats by d-galactosamine (D-GalN) injection. Intraperitoneal injection,24 h later, of viable syngeneic hepatocytes apparently increased the survival rate of these animals (48%), while transplantation of non-viable hepatocytes was ineffective and even showed a certain degree of clotting disorders.A microscopical observation of the liver specimens and estimation of DNA synthesis cy 3H-TdR suggest that viable hepatocellular transplantation may minimize the hepatic injury and promote hepatic cell regeneration.

本文报道,用D-氨基半乳糖(D-GAIN)诱发大鼠急性肝衰竭。于大鼠D-GaIN中毒后24h,腹腔移植同种完整肝细胞,提高活存率为48%。移植破碎肝细胞无效,而且出现凝血系统紊乱现象。本实验通过肝脏病理现察及~3H-胸隙嘧啶核苷(~3H-TdR)肝内掺入量测定结果表明,早期移植完整肝细胞,能阻止肝损害发展,并能刺激和促进病肝细胞再生、复原等作用。

著者等已报告812树脂活性炭膜的研制,系活性炭粉与二甲基亚砜、812树脂混合构成,具有吸附和超滤两种功能,及较好血液相容性。本文报告该膜用于人工肝肾方面的体外与体内实验结果。用急性肝衰竭和黄疸家犬作直接血液灌流,证明在2小时能清除血浆氨54.36%、胆红素33.82%,血浆支链氨基酸与芳香氨基酸比值(BCAA:AAA)由1.7升至3.0。在超滤液中未检出蛋白,其中钠、钾、氯等电解质与血浆水平相同。本文实验表明812树脂活性炭膜可以用于人工肝、肾辅助装置。

 
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