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急性肝功能衰竭的
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  acute liver failure
    The protective effect of Gan Kang on carbon tetrachloride induced liver injuries had been investigated. Gan Kang significantly decreased the necroses and degenerations of Liver,the SGPT level,the BSP retentive amount,and the mortality of animals with acute liver failure.
    肝康对四氯化碳引起的急性肝损害有显著的保护作用,能减轻肝细胞的变性坏死、降低SGPT、BSP 滞留量和急性肝功能衰竭的死亡率.
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  “急性肝功能衰竭的”译为未确定词的双语例句
    Effects of Human Fetal Liver Cytosol on Experimental Acute Hepatic Failure in Rats
    人胎肝细胞液对大鼠实验性急性肝功能衰竭的治疗作用
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  acute liver failure
Acetaminophen overdose and acute liver failure: Modern management
      
Renal extracorporeal support should be initiated using a continuous method when recovery of liver function is expected (acute liver failure, waiting for liver transplantation).
      
Acute liver failure represents a serious life-threatening event comparable to acute heart failure with cardiogenic shock or acute renal failure.
      
He developed fulminant hepatitis with acute liver failure and an acute respiratory distress syndrom (ARDS).
      
Three months later the infant had acute liver failure.
      
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Using the radioactive nuclide 113m In as the tracer, the change of blood flow in the liver was studied on rats contracting experimental acute hepatic failure. The results indicated that during acute hepatic failure, the K-value decreased from 0.447 as in the normal control to 0.327. Following the treatment with 654-2, the blood flow of liver increased markedly and the K-value increased as well to 0.413, nearing the normal value. It shews that 654-2 possesses the effect of improving the blood flow in the liver...

Using the radioactive nuclide 113m In as the tracer, the change of blood flow in the liver was studied on rats contracting experimental acute hepatic failure. The results indicated that during acute hepatic failure, the K-value decreased from 0.447 as in the normal control to 0.327. Following the treatment with 654-2, the blood flow of liver increased markedly and the K-value increased as well to 0.413, nearing the normal value. It shews that 654-2 possesses the effect of improving the blood flow in the liver of rat contracting experimental acute hepatic failure.

应用放射性核素~(113m)铟为示踪剂,对实验性大鼠急性肝功能衰竭时肝血流的变化进行研究。结果表明急性肝衰时K值由正常对照组0.447降至0.327。经用654—2治疗后,肝血流量明显增加,K值增至0.413,接近正常。说明654—2有改善实验性大鼠急性肝功能衰竭时肝血流量的作用。

The protective effect of Gan Kang on carbon tetrachloride induced liver injuries had been investigated.Gan Kang significantly decreased the necroses and degenerations of Liver,the SGPT level,the BSP retentive amount,and the mortality of animals with acute liver failure.In rats with chronic liver failure,Gan Kang markedly inhibited the liver fibrosis,increased the serum aiumin level,and decreased the globulin contents.In addition,the mortality of rats with Liver cirrhosis could also be decreased by Gan Kang.

肝康对四氯化碳引起的急性肝损害有显著的保护作用,能减轻肝细胞的变性坏死、降低SGPT、BSP 滞留量和急性肝功能衰竭的死亡率.此外,肝康还能减少慢性肝损害大鼠的肝纤维组织形成和脂肪交性,提高血浆白蛋白和降低球蛋白的含量,增强肝脏 BSP 排泄功能及降低肝硬化大鼠的死亡率.

Calf Hepatic Stimulatory Substance (cHSS) was extracted from new-born calf, and its preventing lethal effect was observed on the hepatic injury in rats treated with D-galactosamine. After injected with D-galactosamine for 24 hours, the percentage of rats surviving in each grup was determined daily for 2 weeks. The results are as follows: (1)55.5% in physical saline group; (2)50% in piglet hepatic growth factor group; (3)88. 5% in cHSS group. Only the third group attained statistical significance with respect...

Calf Hepatic Stimulatory Substance (cHSS) was extracted from new-born calf, and its preventing lethal effect was observed on the hepatic injury in rats treated with D-galactosamine. After injected with D-galactosamine for 24 hours, the percentage of rats surviving in each grup was determined daily for 2 weeks. The results are as follows: (1)55.5% in physical saline group; (2)50% in piglet hepatic growth factor group; (3)88. 5% in cHSS group. Only the third group attained statistical significance with respect to other two groups. These results in dicated that the calf liver contains cHSS, which successfully reversed the lethality of D-galactosamine-induced hepatic necrosis in rats. By luther studies, the cHSS might become the effective drug for Acute Hepatic Failure and Fatal Hepatic Failure.

从新生牛肝提取再生刺激因子(cHSS),并观察其对D—Galn所致急性肝功能衰竭大鼠的救治效果。Wistar大鼠注射D—Galn(2.2g/kgBW)24h后分组进行试验,观察2周存活率分别为:(1)生理盐水组55.5%;(2)乳猪肝细胞生长素组50%;(3)cHSS组88.5%。第(3)组与第①、②组比较,P<0.01,有显著差异。表明新生牛肝中存在肝再生刺激物质,可显著提高药物性肝衰竭大鼠的存活率。

 
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