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   急性肝功能衰竭的 在 基础医学 分类中 的翻译结果: 查询用时:0.882秒
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急性肝功能衰竭的
相关语句
  acute liver failure
    Result: All 8 porcines died in 16h after the diffluence. High serum ammonia,increases of hepatic enzymes, metabolic disorder of blood suger,and the blood coagulation fit the manifestation of acute liver failure. Some porcinesmanifested with lethargy, dysphoria, vomiting and like hepatic coma.
    结果:8只实验猪均在门体分流后16小时内死亡,出现血氨升价J、月一「酶谱升高、纤维蛋白原减低、血糖代谢紊乱和血流动力学紊乱等情况符合急性肝功能衰竭的表现。部分猪出现嗜睡、烦躁、呕叫_等川二性脑病表现。病理显示肝脏大片坏死,脑组织亦有水肿和坏少巨。
短句来源
  acute hepatic failure
    [Objective] To establish an animal model with acute hepatic failure by feeding carbon tetrachloride.
    目的不同剂量的四氯化碳可以引起不同的肝脏损害,本试验用四氯化碳灌服制造急性肝功能衰竭的动物模型。
短句来源
    The group fed 4 mL carbon tetrachloride per kilogram body weight comes forth the character of acute hepatic failure and pathologic alteration.
    每公斤体重4mL四氯化碳组出现急性肝功能衰竭的临床表现以及肝脏病理改变。
短句来源
  “急性肝功能衰竭的”译为未确定词的双语例句
    A pig animal model of fulminant hepatic failure
    急性肝功能衰竭的猪动物模型
短句来源
    ConclusionThe porcine model of AHF induced by a combination of D-gal ( 0.5 g/kg) and LPS (1 μg/kg) will be of much use in the development of APOLT for AHF.
    结论通过门静脉注射0.5g/kgDGal和1μg/kg脂多糖的方法可建立一种理想的猪急性肝功能衰竭模型。 该模型可用于辅助性部分原位肝移植治疗急性肝功能衰竭的研究。
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  acute liver failure
Acetaminophen overdose and acute liver failure: Modern management
      
Renal extracorporeal support should be initiated using a continuous method when recovery of liver function is expected (acute liver failure, waiting for liver transplantation).
      
Acute liver failure represents a serious life-threatening event comparable to acute heart failure with cardiogenic shock or acute renal failure.
      
He developed fulminant hepatitis with acute liver failure and an acute respiratory distress syndrom (ARDS).
      
Three months later the infant had acute liver failure.
      
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  acute hepatic failure
A molecular adsorbent recycling system in treating posthepatectomy acute hepatic failure patients with hepatocellular carcinoma:
      
Differential gene expression profiles in acute hepatic failure model in mice infected with MHV-3 virus intervened by anti-hepati
      
Differential gene expression profiles in Balb/cJ mouse model of acute hepatic failure infected with MHV-3 virus intervened by anti-hepatic failure compound (AHFC) and the changes of cytokines regulated by genes were investigated.
      
Acute hepatic failure model of Balb/cJ mice infected with MHV-3 virus was established.
      
Changes of high mobility group box 1 in serum of pig acute hepatic failure model and significance
      
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Human hepatic stimulator substance (hHSS) was extracted from fetal liver of 4-6 month gastation according to the method of LaBrecque. The stimulatory activity of hHSS was assayed by specific incorporation rate of 3H-thymidine into the hepatic DNA. hHSS ( 75mg / kg, ip) was given six hours before D-galactosamine (2.5g/kg , ip.) injection. The experimental results were as follows; (1) The mortality induced by D-galactosamine could be reduced by hHSS. (2) The high level of sGPT and sGOT in D-galactosamine-intoxicated...

Human hepatic stimulator substance (hHSS) was extracted from fetal liver of 4-6 month gastation according to the method of LaBrecque. The stimulatory activity of hHSS was assayed by specific incorporation rate of 3H-thymidine into the hepatic DNA. hHSS ( 75mg / kg, ip) was given six hours before D-galactosamine (2.5g/kg , ip.) injection. The experimental results were as follows; (1) The mortality induced by D-galactosamine could be reduced by hHSS. (2) The high level of sGPT and sGOT in D-galactosamine-intoxicated mice could be lowered by hHSS. (3) hHSS could decrease the liver malonaldehyde content of D-galactosamine-intoxicated mice. (4) Hepatic histological findings on light and electron microscopic examination indicated that the D-galactosamine-induced hepatic lesions could be improved by hHSS. The results suggest that hHSS has obvious protective effect on experimental acute liver failure and its mechanisms might be related to inhibition of lipid peroxidation and maintenance of normal metabolism of liver cells.

取健康孕妇水囊引产的4~6个月龄的胎儿肝脏,按LaBreque法提取肝刺激因子(hHSS)。将hHSS注入肝部分切除的大鼠体内,用~3H-TdR掺入肝DNA测定其生物学活性。给昆明种小鼠腹腔注射D-半乳糖胺(D-Gal)以造成急性肝功能衰竭,观察hHSS对这种急性肝衰的保护效应,结果如下:①hHSS明显降低D-Gal所致的小鼠死亡率(P<0.01);②hHSS显著降低D-Gal所致sGPT和sGOT水平增高(sGPT 208.2±26.1比103.6±21.2 U/100ml;sGOT 502.6±65.4比287.8±39.4 U/100m1);③hHSS明显降低D-Gal所致的小鼠肝组织丙二醛水平增高(330.4±38.9比115.5±43.8nmol/100mg蛋白质);④光镜和电镜的组织学观察表明hHSS明显减轻D-Gal对肝组织的损害。上述实验结果表明,hHSS对D-Gal所致的急性肝衰具有明显的保护作用,其机制可能是保护肝细胞正常代谢和防止膜脂质过氧化。

epatitis virus infection is a special problem

肝炎病毒感染是亚洲人包括中国的特殊感染问题,肝炎病毒感染与骨髓移植之间的关系尚不很明确。笔者通过对106例骨髓移植患者乙型、丙型肝炎病毒感染及其与临床结果的综合分析,发现骨髓移植后患者乙型、丙型肝炎病毒感染率分别为7/106(6.6%)、65/106(61.3%),乙型、丙型肝炎病毒活动均可致轻到中度肝功能上升,1例HBV、HCV混合感染患者因急性肝功能衰竭死亡,但无论乙型或丙型肝炎病毒感染均不影响骨髓的植活,亦不导致急、慢性移植物抗宿主病及肝静脉阻塞综合征发病率上升,因而肝炎病毒感染并非骨髓移植禁忌症,但肝炎病毒感染的防治仍是我们面临的一重大课题。人类重组干扰素可防治丙型肝炎病毒活动,笔者对此进行了初步探讨。

he renal function was observed in rats of

本实验对半乳糖胺造成急性肝功能衰竭动物的肾功能进行观察,结果显示,在肝脏功能发生严重障碍的同时,肾功能也出现明显异常,尤以肾小球滤过功能障碍为甚(Pcr、BUN升高,Ccr降低);组织学改变符合临床急性肝衰时的肾脏变化,是较为理想的肝肾综合征动物模型,可用于急性肝衰时肾功能障碍发病机理及防治的研究。

 
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