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   肝纤维化形成 的翻译结果: 查询用时:0.032秒
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肝纤维化形成
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  hepatic fibrogenesis
     Conclusion Rho/ROCK signaling pathways are changed in the process hepatic fibrogenesis.
     结论Rho/Rho激酶信号转导通路在肝纤维化形成过程中发生变化。
短句来源
     Objective To investigate the effect of prostaglandin E 1 (PGE 1) on hepatic fibrogenesis and its mechanism in rats with schistosomiasis japonica.
     目的 探讨前列腺素 E1 (PGE1 )对日本血吸虫病肝纤维化形成的影响及其机制。
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     Differentiation of bone marrow stem cells in rat hepatic fibrogenesis environment
     骨髓干细胞在大鼠肝纤维化形成环境中的分化
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     The levels of 6 K PGF 1α and GG presented the trend of up going with the development of hepatic fibrogenesis,which was identified by pathology.
     在肝纤维化形成过程中 ,大鼠血浆 6 K PGF1α及GG水平呈逐渐上升趋势。
短句来源
     The changes of serum aminotransferase (ALT),albumin (ALB),Laminin (LN),hyaluronic acid (HA) and Ⅳ collagen (CⅣ) as well as the content of hepatic hydroxyproline (HYP) and the formation of hepatic fibrosis were comparatively observed to evaluate the effects of SEY on hepatic fibrogenesis in rats.
     检测大鼠血清丙氨酸氨基转换酶(ALT)、白蛋白(ALB)、层粘蛋白(LN)、透明质酸(HA)和Ⅳ型胶原的(CⅣ)水平以及肝脏纤维化程度和羟脯氨酸(HYP)的含量,以此评价所用药物对大鼠肝纤维化形成的影响。
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  liver fibrogenesis
     Effect of cyclooxygenase-2 on liver fibrogenesis induced by CCl_4 in rats
     环氧合酶-2在CCl_4诱导大鼠肝纤维化形成中的作用
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     Possibility of rat liver fibrogenesis inhibited by bone marrow-derived mesenchymal stem cells:a 7-week observation on survival time of stem cells and activation of hepatic stellate cells in liver of recipients and a survival analysis
     骨髓间质干细胞抑制大鼠肝纤维化形成的可能性:受体肝内干细胞存活时间、肝星状细胞活化程度及生存分析的7周观察
短句来源
     Purpose To investigate the effect of cyclooxygenase-2(COX-2) on liver fibrogenesis induced by CCl_4. Methods Male S-D rats were used.
     目的探讨环氧合酶-2(cyclooxygenase-2,COX-2)在四氯化碳(CCl4)诱导大鼠肝纤维化形成过程中的作用。
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     Role of lipid peroxidation in liver fibrogenesis induced by dimethylnitrosamine in rats
     脂质过氧化在二甲基亚硝胺大鼠肝纤维化形成过程中的作用
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     THE ROLE OF TRANSFORMING GROWTH FACTOR β 1 AND ITS RECEPTOR IN IMMUNOLOGICAL INDUCED LIVER FIBROGENESIS IN RATS AND EFFECT OF CORDYCEPS POLYSACCHARIDE ON THEM
     转化生长因子β_1及虫草多糖对大鼠肝纤维化形成的影响
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  “肝纤维化形成”译为未确定词的双语例句
     Dynamic change of MMP-1/TIMP-1 expression in experimental immune hepatic fibrosis
     大鼠免疫性肝纤维化形成中MMP-1和TIMP-1表达的动态变化
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     Effect of Chinese drug compound Ganzhlfu on liver fibrosis in rats
     温阳中药复方肝之福对大鼠肝纤维化形成的影响
短句来源
     Effect of TIMP-1 in hepatic fibrosis
     TIMP-1在肝纤维化形成过程中的作用
短句来源
     Objective:To examine the expression changes of leptin in the process of DMN inducing hepatic fibrosis in rats.
     目的:观察二甲基亚硝胺(DMN)诱导实验性肝纤维化模型大鼠肝纤维化形成过程中瘦素(Leptin)的表达变化。
短句来源
     3) to verify the expression of the genes related with mitogen-activated protein kinase (MAPK) signal pathway in the development of hepatic fibrosis;
     3)有丝分裂原活化的蛋白激酶(mitogen-activated protein kinase,MAPK)信号通路相关基因在肝纤维化形成过程中的表达;
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  hepatic fibrogenesis
Great progress has been made over the past 20 years in elucidating the mechanisms of hepatic fibrogenesis.
      
Great progress has been made over the past 20 years in elucidating the mechanisms of hepatic fibrogenesis.
      
The embryonal origin of hepatic stellate cells (HSCs), the principal cells in hepatic fibrogenesis, is still intriguing.
      
The present results indicate that SPARC is expressed by activated HSCs in chronic hepatitis, suggesting the involvement of SPARC in hepatic fibrogenesis after chronic injuries.
      
mansoni egg antigen-induced granuloma formation and hepatic fibrogenesis, resulting in more severe liver pathology.
      
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  liver fibrogenesis
The liver fibrogenesis was assessed using a digital image analysis instrument of Masson's trichrome stained sections.
      
Exciting new data are expanding our understanding of the mechanisms of steatogenesis in HCV infection and providing potential links between insulin resistance or hyperglycemic states and liver fibrogenesis.
      
The expression of basic fibroblast growth factor (bFGF) in rat liver fibrosis and hepatic stellate cells (HSCs) and the relationship between the expression of bFGF and rat liver fibrogenesis were studied.
      
bFGF possibly regulates liver fibrogenesis through regulating metabolism of extracellular matrix (ECM) by autocrine and paracrine stimulation.
      
At present, liver fibrogenesis is referred to as a dynamic process involving complex cellular and molecular mechanisms, resulting from the chronic activation of the tissue repair mechanisms that follows reiterated liver tissue injury.
      
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Rabbits were infected with Schistosoma japonicum cercariae and the collagen content, collagenase activities in the liver and monoamine oxidase(MAO), α2-M in serum were examined post-infection. The results showed that 8 weeks post-infection (pi), the liver collagen content in infected rabbits was 23.14±5.23 (normal control value: 10.56±1.73 mg/g wet weight), and was 32.89±7.04 11 weeks p.i, 27.29±8.86 18 weeks p.i, but 28.34±11.95mg/g wet weight 14 weeks after pyquiton treatment. Collagenase activities were 32.99±7.34...

Rabbits were infected with Schistosoma japonicum cercariae and the collagen content, collagenase activities in the liver and monoamine oxidase(MAO), α2-M in serum were examined post-infection. The results showed that 8 weeks post-infection (pi), the liver collagen content in infected rabbits was 23.14±5.23 (normal control value: 10.56±1.73 mg/g wet weight), and was 32.89±7.04 11 weeks p.i, 27.29±8.86 18 weeks p.i, but 28.34±11.95mg/g wet weight 14 weeks after pyquiton treatment. Collagenase activities were 32.99±7.34 (compared with normal group, P〈0.05), 16.88 ± 5.99, 11.13±3.35 (P〈0.05) and 14.30±5.41u, respectively. Serum MAO increased from the 4th week on but tended to return to normal after pyquiton treatment. A negative relationship was noticed between collagenese activities and collagen content (P〈0.01). It was suggested that the abnormal collagen metabolism was related to immunological reaction caused by granuloma formed by schistosome eggs and the increase in collagenase activities is an important factor in the catabolism of liver collagen.

家兔感染日本血吸虫尾蚴后第8周,其肝内胶原含量增加至正常值的2倍,11周时为正常值的3倍;杀虫治疗后14周仍为正常值的2.5倍左右。血清MAO活性在第4周已显著增高,杀虫治疗后便回复到正常范围。肝组织胶原活性在第8周时,显著高于正常值(P<0.05),11周时显著下降,18周(杀虫治疗后1周)对进一步低下,显著低于正常值(P<0.01)。肝组织胶原酶活性与肝内胶原含量之间呈显著的负相关(r=-0.647,P<0.01).表明血吸虫病肝纤维化形成过程中肝内胶原代谢的异常,似与血吸虫卵引起的肉芽肿免疫反应密切相关。提示对于血吸虫病肝纤维化,除给予病原治疗外,尚须设法提高肝组织胶原酶活性,促进已沉积于肝内胶原的分解。

Hepatic fibrsosis was induced by carbon tetrachloride in mice C 57.Then the feed containing Dangshen pollen was given to the animals. It was demonstrated under the electron microscope that the Dangshen pollen lowered the activity of the fibroblasts around the central vein and the fatstoring ceils of sinusoids, and lowered the grade of collagenous fibers of Disse's spaces (P<0.01). These retarded the progress and development of hepatic fibrosis;

用四氯化碳造成C_(57)小鼠肝纤維化,然后饲喂含党参花粉的饲料。电镜观察表明,党参花粉降低了中央静脉下成纤维细胞和血窦储脂细胞的活性,降低了Disse 腔胶原纤维的分级(P<0.01),其结果是减缓了肝纤维化的形成和发展。

Sequential immunohistochemical observations were made on fibroneetin (FN), laminin (LN) and collagen type Ⅳ (Ool Ⅳ) from early formation to the subsidence of rat hepatic fibrosis which was induced by carbon tetrachloride, plug drinking water containing low concentration of alcohol and a low choline diet, Plasma FN was determined simultaneously by the single radial diffusion test.We found that FN dramatically increased along the wall of sinusoids which had dilated forming septa after hepatocytic necrosis and...

Sequential immunohistochemical observations were made on fibroneetin (FN), laminin (LN) and collagen type Ⅳ (Ool Ⅳ) from early formation to the subsidence of rat hepatic fibrosis which was induced by carbon tetrachloride, plug drinking water containing low concentration of alcohol and a low choline diet, Plasma FN was determined simultaneously by the single radial diffusion test.We found that FN dramatically increased along the wall of sinusoids which had dilated forming septa after hepatocytic necrosis and in the matrix around it during the initial period of the experiment, Later, LN and Ool Ⅳ deposited on the wall of sinusoids mentioned above and on the basement membrane of blood vessels and newly formed small bile ducts within the septa. However, no noticeable LN and Ool Ⅳ were observed in the matrix there. With progression of the experiment, FN increased continuously forming complete septa before formation of collagen septa. When the liver recovered from fibrosis, FN was the quickest to subside, LN and Ool Ⅳ deposition subsided more slowly.Plasma FN rose markedly during the initial period of the experiment and decreased thereafter, but did not parallel changes of FN in the livers. The rolea and the dynamic changes of FN, LN and Ool Ⅳduring fibrogenesis ware-discussed.

以免疫组化方法对CCl_4加低酒精饮水及低胆碱饲料诱发的大鼠早期肝纤维化形成及消退过程中纤维连接蛋白(FN),层粘连蛋白(LN)和Ⅳ型胶原蛋白(Col Ⅳ)的演变作了观察;何时用免疫单扩散法测定了实验各期大鼠血浆FN。发现实验早期在肝细胞坏死区扩张的血窦壁及周围基质中首先见FN增多,形成早期间隔;以后LN和Col Ⅳ也在此处血窦壁及间隔内血管和新生小胆管的基膜处沉积,但基质中无明显沉积。随实验进展,FN继续增多,并先于胶原纤维在肝内形成完全间隔。肝纤维化消退时,FN消退最快,LN和Col Ⅳ则较慢。血浆FN在实验早期明显上升,以后逐步回降,其变化与肝组织中FN的量并不平行。

 
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