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重型
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  severe
    Role of Th1 Cell in the Pathogenesis of Severe Aplastic Anemia
    1. Th1细胞在重型再生障碍性贫血病理机制中意义的研究 2. 与免疫相关的全血细胞减少症
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    SEVERE APLASTIC ANEMIA: CLINICAL ANALYSIS OF 31 CASES
    31例重型再生障碍性贫血临床分析
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    Clinical Therapy with Cyclosporin A in Nine Patient with Adult Severe Aplastic Anemia
    环孢菌素A治疗成人重型再障9例
短句来源
    STUDY ON INVERSION OF INTRON 22 OF FACTORVIII GENE IN SEVERE HEMOPHILIA A PATIENTS
    重型血友病甲凝血因子Ⅷ基因内含子22倒位的研究
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    Cyclosporin A in Combination with Androgens Treatment for Patients with Severe Aplastic Anemia
    环孢霉素A合并雄激素治疗成人重型再生障碍性贫血
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  “重型”译为未确定词的双语例句
    CYCLOSPORIN A IN TREATMENT OF CHILDREN SEVEREAPLASTIC ANEMIA
    环孢菌素A治疗小儿重型再生障碍性贫血
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    CLINICAL OBSERVATION AND GENE ANALYSIS OF β-THALASSEMIA MUTATIONS (A REPORT OF 13 CASES)
    13例重型β地中海贫血的临床观察和基因分析
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    Umbilical cord blood transplantation for a patient with β thalassemia major
    脐血造血干细胞移植治疗重型β地中海贫血
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    Hematopoietic stem cell transplantation for β-thalassemia major
    造血干细胞移植治疗重型β地中海贫血
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    Bone marrow transplantation for β-thalassemia major: report of 2 cases
    骨髓移植治疗重型β地中海贫血(附2例报告)
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  severe
Podophyllotoxin and related analogs present numerous challenges associated with optimal antitumor activity and severe unpredictable toxicity.
      
There appear many Internet-scale worm incidents in recent years, which have caused severe damage to the society.
      
Many recently proposed subspace clustering methods suffer from two severe problems.
      
It was due to stomatal limitation and osmotic organic molecules accumulation that would affect the photosynthetic shoots to resist severe drought stress.
      
Osmosis of organic molecules was the most important factor to adjust leaves to severe water stress at this period.
      
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Blood platelets aggregation function was studied in 45 cases of epidemic hemorrhagic fever, and specific Ag-Ab complexes were examined in 22 cases. During the early febrile stage, platelets count decreased to<50, 000/cmm in 22 cases (48.3%), and percentage of aggregation was <10% (normal 53±7.9%)in 31 cases (69%). The decrease was significant in seriously ill patients. This might explain the severe bleeding in febrile stage. The aggregation function was further impaired as the disease progressed to shock and...

Blood platelets aggregation function was studied in 45 cases of epidemic hemorrhagic fever, and specific Ag-Ab complexes were examined in 22 cases. During the early febrile stage, platelets count decreased to<50, 000/cmm in 22 cases (48.3%), and percentage of aggregation was <10% (normal 53±7.9%)in 31 cases (69%). The decrease was significant in seriously ill patients. This might explain the severe bleeding in febrile stage. The aggregation function was further impaired as the disease progressed to shock and oliguric stages. In polyuric and recovery stages, the function tended towards normal. Specific Ag-Ab complexes were demonstrated on the surface of platelets in platelet smears in 10 out of 22 cases.

对45例流行性出血热患者,检测血小板计数及聚集功能(比浊法)。发热期患者中重型19例、中型13例、轻型13例,血小板计数小于5万/mm~3者各有13例、3例、6例;聚集功能低于10%者各为16例、9例、6例,占69%。血小板功能障碍,随病程进展而加重,且与肾功能损害有关。随疾病的好转,血小板计数与功能也逐渐恢复正常。22例发热期患者作血小板涂片,用间接荧光染色检测表面免疫复合物,有10例荧光染色阳性。用封闭试验证实其有特异性。

A Chinese couple whose two children died of β-thalassemia major requestedprenatal diagnosis during the third pregnancy.On the basis of hematological andhemoglobin examinations,the clinical diagnosis of 21 relatives in this family weredone.Then,by the RFLP haplotype analyses of DNA from the 21 relatives and fromamniotic fluid cells,the haplotype chromosomes with β-thalassemia determinant wereidentified.The fetus was found to have both haplotype chromosomes with β-thalassemia determinant from the ancestors,i.e.one...

A Chinese couple whose two children died of β-thalassemia major requestedprenatal diagnosis during the third pregnancy.On the basis of hematological andhemoglobin examinations,the clinical diagnosis of 21 relatives in this family weredone.Then,by the RFLP haplotype analyses of DNA from the 21 relatives and fromamniotic fluid cells,the haplotype chromosomes with β-thalassemia determinant wereidentified.The fetus was found to have both haplotype chromosomes with β-thalassemia determinant from the ancestors,i.e.one chromosome carrying β-thalassemia determinant was transmitted via great-grandmother→father→fetus,and another one via grandmother→mother→fetus.The fetus was thus sufferedfrom β-thalassemia major.

广西柳州一名孕妇曾生育过2例重型β-地中海贫血患儿,已先后死亡,因此要求进行β-地中海贫血的产前诊断。本文应用 DNA 限制酶酶谱技术,通过对这个家系各成员的β-珠蛋白基因簇单体型分析,鉴定出携带β-地中海贫血基因的染色体的单体型,诊断出胎儿患重型β-地中海贫血,即胎儿细胞携带两个β-地中海贫血基因,其中一个是从曾外祖母,通过祖母、父亲传下来,另一个是从外祖母通过母亲传给胎儿。这是国内对β-地中海贫血产前诊断的首例报道。

This paper reports local observation of the myocardial change in 16autopsy cases with Keshan disease under light and electron microscopes.Thesevere degeneration and deterioration changes were similar to the lesionsof 9 subacute and 1 acute case with Keshan disease.This result suggeststhat most of myocardial necroses of Keshan disease belong to the type ofcoagulated myocytolysis.The above facts indicate that the fresh lesionsoccurred repeatedly in transitional,absorbable and repairable subacutestage after extensive...

This paper reports local observation of the myocardial change in 16autopsy cases with Keshan disease under light and electron microscopes.Thesevere degeneration and deterioration changes were similar to the lesionsof 9 subacute and 1 acute case with Keshan disease.This result suggeststhat most of myocardial necroses of Keshan disease belong to the type ofcoagulated myocytolysis.The above facts indicate that the fresh lesionsoccurred repeatedly in transitional,absorbable and repairable subacutestage after extensive necrosis in Keshan disease,which may promoteprimary lesion of myocardium.This pathological changes draw attention toto the importance of epidemiology,prevention,treatment and etiology ofKeshan disease.

本文分析了16例小儿克山病,用光学显微镜及电镜对心肌病变进行定位观察发现,9例亚急型克山病和1例急型克山病均有不同程度的类似北方急重型克山病严重变性、变质改变,对病变特点及其意义进行了简要讨论。其中6例慢型克山病均未见上述急性心肌损伤病变。

 
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