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细胞膜型
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  membrane type matrix
     Methods The effect of hCG on membrane type matrix metalloproteinases (MT MMPs) and tissue inhibitor of metalloproteinase (TIMP) mRNA expression in choriocarcinoma cell line JEG 3 was investigated by applying the method of reverse transcription polymerase chain reaction (RT PCR).
     方法 以绒癌组织来源的JEG 3细胞系为研究对象 ,采用逆转录多聚酶链反应 (RT PCR)方法 ,观察了hCG对JEG 3细胞膜型金属蛋白酶 (MT MMP)和金属蛋白酶组织抑制因子 (TIMP)mRNA表达的影响。
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  “细胞膜型”译为未确定词的双语例句
     Expression and its significance of the membrane CD28 in T cells and the serum level of soluble CD28 in elders
     老年人T细胞膜型CD28及血清中可溶性CD28的表达及其意义
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     Objective To characterize the effects of parathyroid hormone (PTH) on the expression of membrane-type 1 matrix metalloproteinase-1 (MT1-MMP) and receptor activator of NF-κB ligand (RANKL) in human osteoblast-like cell line MG-63 in order to elucidate the mechanism by which PTH acts on bone.
     目的研究甲状旁腺激素(PTH)对人成骨样MG-63细胞膜型基质金属蛋白酶-1(MT1-MMP)表达、INF-κB受体活化素配体(receptor activator of NF-κB ligand,RANKL)信号通路的影响,探讨PTH调节骨代谢作用机制。
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     The membrane form of CD28 in T cells and the serum level of soluble CD28 from peripheral blood of elders(beyond age of 60) were analysed in order to clarify its significance in the changes of immune functions in elders.
     分析老年人(年龄在60岁以上)外周血T细胞膜型CD28(mCD28)及血清中可溶性CD28(sCD28)的表达水平,探讨该分子在老年人免疫功能改变中的意义。
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     The regulation of the expression of membrane type-1 matrix metalloproteinase and gelatinase-A activation by progesterone in human osteoblast-like cells cultures
     孕激素对人成骨样细胞膜型基质金属蛋白酶-1表达和明胶酶-A激活的影响
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     Effect of 17β-estradiol on the expression of membrane type 1 matrix metalloproteinase in human osteoblast-like cells
     雌二醇对人成骨样细胞膜型基质金属蛋白酶1的影响
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  相似匹配句对
     Cellular Spectro-Analysis
     细胞光谱分析
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     SPLINTER CELL
     分裂细胞
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     Therefore electromicroscopy was a useful aid in examination for the diagnosis of acute leukemia. Under the SEM there were 48-90% ridge-like profile-type cells in AGL whereas the surface features of 70-81% leukemic cells in AML were ruffied membranes.
     急单中的皱膜型细胞为70~81%;
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     Comparison of the cytocidal effect induced by transmembrane and secreted TNF alpha
     跨膜型与分泌型TNF-α细胞毒效应的比较
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     Membranous IgA nephropathy
     膜型IgA肾病
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HLA-class Ⅰ and Ⅱ antigens, T cell subsets, HBsAg, HBcAg of the liver were srudied by the APAAP and PAP methods of immunohistochemical technique in 113 cases of acute, chronic and severe types of hepatitis B. Serum HBV DNA and β2-microglobulin were simultaneously assayed by dot blot hybridization and radial immunoassay respectively. The expressions of HLA- class Ⅰ and Ⅱ antigens on the hepatocyte membrane were increased in acute, chronic active and severe hepatitis B, especially for the HLA-class Ⅰ antigens....

HLA-class Ⅰ and Ⅱ antigens, T cell subsets, HBsAg, HBcAg of the liver were srudied by the APAAP and PAP methods of immunohistochemical technique in 113 cases of acute, chronic and severe types of hepatitis B. Serum HBV DNA and β2-microglobulin were simultaneously assayed by dot blot hybridization and radial immunoassay respectively. The expressions of HLA- class Ⅰ and Ⅱ antigens on the hepatocyte membrane were increased in acute, chronic active and severe hepatitis B, especially for the HLA-class Ⅰ antigens. Whereas there was almost no expression in chronic persistent hepatitis B. There existed close relationship between the hepatocyte expression of HLA-class Ⅰ antigens and the inflammatory activity as well as the membrane expressions of HBsAg and HBcAg. It was found that the major infiltrating cells in the hepatic damage area were CD2+ lymphocytes, of which CD8+ cells constituted the major subpopulations. The attack and lysis of hepato-cytes by the antigen-dependent, HLA-restricted cytotoxic T lymphocyte is considered as the main mechanism of the immunopathologic damage in hepatitis B.

用免疫组织化学桥联酶标APAAP法和PAP法对各型乙型肝炎113例检测了肝内的HLA-Ⅰ、Ⅱ类抗原、T细胞亚群、HBsAg和HBcAg,同时测定血清HBV DNA(斑点杂交)和β_2微球蛋白(RIA)。结果表明;急性肝炎、慢性活动性肝炎、重症肝炎患者的肝细胞HLA-Ⅰ、Ⅱ类抗原表达增强,尤以Ⅰ类抗原明显,慢性迁延性肝炎则几乎无表达。肝细胞HLA-Ⅰ类抗原表达与肝内炎症反应、肝细胞膜型HBSAg和浆膜型HBcAg关系密切,肝内活动性病变主要浸润细胞为CD_2~+细胞,CD_0~+细胞构成主要的细胞亚群。认为抗原依赖的、HLA限制的细胞毒性T细胞效应是构成乙型肝炎免疫损害的主要机制。

The purpose of this study is to explore a method by which an

应用硝酸醛复红法观察了鸭肝组织的DHB_sAg及肝组织病变,结果是,1.DHB_sAg要对硝酸醛复红着色迅速,着色后DHB_sAg呈紫红色,背景为蓝绿色。2.DHB_sAg在肝细胞内的存在形式为(1)胞浆型,包括均浆型和包涵体型。(2)细胞膜型。(3)胞浆混合型,即均浆型和包涵体型混合存在。其中胞浆型占优势,细胞膜型少见。这与人类HB_sAg的表现形式及分布状态近似。3.鸭肝组织内DHB_sAg的存在与鸭肝病变关系不大。4.硝酸醛复红着色原理可能与DHB_sAg分子多肽内的二硫键被硝酸氧化有关。5.与斑点分子杂交法比较,灵敏度较差,阳性率为22.1%。

Objective: To study significance of changes of complement receptor I on erythrocyte membrance(RBC-CR1) of hyperlipemia patients. Method: RBC -CR1 of hyperlipemia patients was determined by complement sensitizing yeast polysaccharide and compared with that of normal subjects. Results: The flower - ring forming rate of patients' RBC - CR1decreased significantly (13. 9±2. 2% vs. 10. 5±2. 6%). Conclusion: Erythrocyte immune adhesion of hyperlipemia patients decreases, which is induced by immunocomplex accumulation...

Objective: To study significance of changes of complement receptor I on erythrocyte membrance(RBC-CR1) of hyperlipemia patients. Method: RBC -CR1 of hyperlipemia patients was determined by complement sensitizing yeast polysaccharide and compared with that of normal subjects. Results: The flower - ring forming rate of patients' RBC - CR1decreased significantly (13. 9±2. 2% vs. 10. 5±2. 6%). Conclusion: Erythrocyte immune adhesion of hyperlipemia patients decreases, which is induced by immunocomplex accumulation resulted from lipodystrophy and RBC - CR1 was blocked. Therefore, an amount of sedimented lipid can not be eliminated and atherosclerosis occurs.

目的:探讨高脂血症中红细胞膜Ⅰ型补体受体(RBC—CR_1)的变化。方法:采用补体致敏酵母多糖,测定高脂血症患者RBC—CR_1的变化。结果:血浆中载脂蛋白B(apoB),甘油三酯(TG),总胆固醇(TC)和红细胞膜(RBC—m)TC,载脂蛋白A(ApoA),ApoA/ApoB升高者RBC—CR_1花环率降低,与对照组比较,差异显著,分别为对照组13.9±2.2%,高脂血症组10.5±2.6%(P<0.01)。结论:高脂血症患者红细胞免疫粘附功能调节降低,脂类代谢失衡造成免疫复合物的堆积,封闭了红细胞膜上CR_1的位点,阻碍了免疫复合物充分粘附在红细胞膜上,使其不能及时清除大量的沉积脂质,促进了动脉粥样硬化形成。

 
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