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   肺腺癌细胞的 的翻译结果: 查询用时:1.461秒
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肺腺癌细胞的     
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  lung adenocarcinoma cells
     CONCLUSION: The growth inhibitory effect of 9-cis-RA on lung adenocarcinoma cells may be tightly correlated with its mediating the transcription of cyclinD1 and cdk4.
     结论:9-顺-维甲酸对肺腺癌细胞的生长抑制作用,与其调控细胞周期因子cyclinD1、cdk4的表达密切相关。
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     Objective To investigate the effect of synthesized Her-2/neu specific siRNA on the cell cycle and apoptosis of Her-2/neu upregulating human lung adenocarcinoma cells.
     目的研究人类表皮生长因子受体2(Her2/neu)小分子干扰RNA(siRNA)对Her2/neu过表达的肺腺癌细胞的细胞周期和凋亡机制的影响。
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     Objective: We studied the effect of DBcAMP on human lung adenocarcinoma cells and on the expression of lectin receptors in the cells.
     目的:研究双丁酰环腺苷酸(DBcAMP)对人肺腺癌细胞的诱导分化作用,及其与癌细胞表达凝集素受体的关系。
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     FasL gene transfer may increase caspase-3 in lung adenocarcinoma cells and enhance their apoptosis.
     FasL基因转染的肺腺癌细胞的凋亡与caspase 3的活性增加密切相关
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     The Killing Effect of Vinorelbine Combined with Radiation on Human Lung Adenocarcinoma Cells in Vitro
     诺维本联合放射对人肺腺癌细胞的杀伤作用
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  lung cancer cells
     Conclusion Liposomal transfection of cyclin D1 ASON can inhibit in vitro the proliferation of lung cancer cells, which might provide a new target for gene therapy.
     结论脂质体转染cyclinD1ASON能在体外明显抑制肺腺癌细胞的生长,且有诱导细胞凋亡、抑制细胞内cyclin D1合成的作用,可能会成为基因治疗的一个新靶点。
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     The Biological Characteristics in vitro and Anti-Tumor Effect in vivo of Lung Cancer Cells Infected with Recombinant Adenovirus Expressing hTNF-α
     hTNF-α重组腺病毒转染人肺腺癌细胞的体外生物学特性及体内抗肿瘤研究
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     The biological characteristics in vitro and anti-tumor effect in vivo of lung cancer cells infected with recombinant adenovirus expressing hIL-2
     人IL-2重组腺病毒转染人肺腺癌细胞的体外生物学特性及体内抗肿瘤研究
短句来源
     The in-vitro biological characteristics of lung cancer cells infected with recombinant adenovirus expressing hTNF-α
     人TNF-α重组腺病毒转染人肺腺癌细胞的体外生物学特性研究(英文)
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     The in-vitro biological characteristics of lung cancer cells infected with recombinant adenovirus expressing hIL-2
     人IL-2重组腺病毒转染人肺腺癌细胞的体外生物学特性的研究(英文)
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  pulmonary adenocarcinoma cells
     Killing Effect of Human Pulmonary Adenocarcinoma Cells with TK+ CD/5-Fc+ GCV Coexpression Suicide Gene Systems
     自杀基因CD、TK的共表达对人肺腺癌细胞的杀伤作用
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  lung adenocarcinoma cell lines
     Conclusion Ad-p53 (Gendicine) can inhibit the growth of human lung adenocarcinoma cell lines irrespective of the status of endogenous p53 gene.
     结论重组人p53腺病毒注射液能抑制肺腺癌细胞的生长,并不受内源性p53状态的影响。
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     Conclusion Ad-p53(Gendicine) combined with docetaxel shows significantly synergistic inhibition effects on the growth of human lung adenocarcinoma cell lines.
     结论Ad-p53与化疗药物Docetaxel联用能显著协同抑制肺腺癌细胞的生长,增加化疗敏感性。
短句来源
     Effect of hyperthermia combined with anti-cancer drugs for human lung adenocarcinoma cell lines
     高温与治癌药物联合应用对人肺腺癌细胞的杀伤作用
短句来源
     Objective To observe the kill effect of hyperthermia combined with anti-cancer drugs(mitomycin,vindesine,cisplatin) on human lung adenocarcinoma cell lines(D6).
     目的 观察高温和丝裂霉素 (MMC)、长春酰胺 (VDS)及顺铂 (DDP)联合应用对人肺腺癌细胞的杀伤作用。
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      lung adenocarcinoma cells
    Arachidonic Acid Metabolism in Growth Control of A549 Human Lung Adenocarcinoma Cells
          
    The role of individual eicosanoids of the arachidonic acid (AA) cascade in the growth control of A549 human lung adenocarcinoma cells has been studied.
          
    Selective reversal of drug resistance in drug-resistant lung adenocarcinoma cells by tumor-specific expression of mdrl ribozyme
          
    The effect of RNAi-mediated gene silencing on her-2/neu gene expression in lung adenocarcinoma cells
          
    The objective of this paper is to investigate the effect of chemically synthesized siRNA targeting her-2/neu on her-2/neu dysregulated human lung adenocarcinoma cells.
          
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      lung cancer cells
    Increasing drug resistance in human lung cancer cells by mutant-type p53 gene mediated by retrovirus
          
    The retroviral vector pD53M of the mt-p53 was constructed and introduced into the drug-sensitive human lung cancer cells GAO in which p53 gene did not mutate.
          
    Reversal of drug resistance of multidrug-resistant human lung cancer cells by an MDR1 ribozyme
          
    To characterize the genes associated with differentiation/apoptosis induced by all-trans retinoic acid (ATRA) in human lung cancer cells, mRNA differential display was employed.
          
    This study suggests that combined treatment with TNP-470 plus gemcitabine may augment the antiangiogenic and antineoplastic effects in lung cancer cells in vitro.
          
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      pulmonary adenocarcinoma cells
    Some differentiating effects of selenium on the cultured human hepatoma cells and human pulmonary adenocarcinoma cells in vitro
          
    Differential Effects of selenium on the proliferation of human pulmonary adenocarcinoma cells and human embryonic lung diploid c
          
    The effect of different concentrations of Na2SeO3 on human pulmonary adenocarcinoma cells and human embryonic lung diploid cells in vitro was investigated.
          
    For human pulmonary adenocarcinoma cells, mitotic index and cell count decreased with increasing selenium concentrations.
          
    Kodate and col29 also showed that MMP-2 and MMP-9 are expressed in pulmonary adenocarcinoma cells.
          
      lung adenocarcinoma cell lines
    Identification of drug resistant related cDNA in lung adenocarcinoma cell lines
          
    Objective: To clone multidrug resistance (MDR) related genes in lung adenocarcinoma cell lines.
          
    Etoposide-resistant human colon and lung adenocarcinoma cell lines exhibit sensitivity to homoharringtonine
          
    Restoration of the p16 gene is related to increased radiosensitivity of p16-deficient lung adenocarcinoma cell lines
          
    The rapid exit of the p16-transfected cells from the G2/M phase in the cell cycle, both before and after irradiation, possibly contributes to the increased radiosensitivity of our experimental p16-transfected lung adenocarcinoma cell lines.
          
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    Electron microscopy is of advan-tage for diagnosis and identification of the histogenetic classification of lung tumors.Using this techniques,an ultrastructural observation on the cha-racteristics and its dynamic changes during cultivation from 1~(st) to 5~(th) day of human lung adenocarcinoma cell line (SPC-A-1) was made. The cultivated cells of 3~(rd)-4~(th) day were more complicated in structure than that of 1~(st)-2~(nd) and 5~(th) day.They showed higher activity of secreting func-tion and more increased...

    Electron microscopy is of advan-tage for diagnosis and identification of the histogenetic classification of lung tumors.Using this techniques,an ultrastructural observation on the cha-racteristics and its dynamic changes during cultivation from 1~(st) to 5~(th) day of human lung adenocarcinoma cell line (SPC-A-1) was made. The cultivated cells of 3~(rd)-4~(th) day were more complicated in structure than that of 1~(st)-2~(nd) and 5~(th) day.They showed higher activity of secreting func-tion and more increased in cytoplas-mic constituents.At this stage,the nuclei of cells appeared more hetero-morphic.The majority of nuclei were irregular in contour.Only a small ??amount of short rough endoplasmic reticulum dispersed in cytoplasm,some were extended with very few riboso-mes forming flat empty spaces.Mi-tochondria were swollen,showing dis-solution of cristae and vacuolation.The amount of mucous granules in-creased gradually with the increase of days of subculture.They often fused with each other and some were connected with the intracellular spa-ces.Some mucus accumulated peculiar-ly in the degenerated mitochondria.In peripheral region,many microvilli and short cytoplasmic processes existed,a number of pinocytotic vesicles,coated vesicles and many intracellular cannules could be seen,extending deeply into cytoplasm.All these indicated the en-hanced activity of cellular metabo-lism.However,the cells of 1~(st)-2~(nd) and 5~(th) day were seen simplified in structures.Mucous granules were very few at these stages. It should be pointed out,the in-creased production of mucous gra-nules closely related to cellular activity and their unusual accumulation in degenerated mitochondria and rough endoplasmic reticulum appeared in quite different pan from normal mucin-secreting cells. Besides,mucous granules and va-rious forms and sizes of lamellar bo-dies similar to that of Type II alveolar epithelial cells indicated that this cell line was probably originated from human bronchiolar epithelial cells with mucous metaplasia during carcino-genesis.

    本文对体外培养的人肺腺癌细胞(SP-C-A-l)进行了传代周期1—5天的超微结构观察。从第49、61、68和84代细胞的观察结果表明,传代培养5天过程中,细胞的超微结构发生明显变化。主要表现在胞质内粘液颗粒的数量随着细胞进入对数生长期(3—4天)逐渐增多,并有粘液在异常的部位(变性的线粒体)积聚;与此同时,线粒体的数量相应增加,内部结构出现明显改变。当细胞生长活动处于相对稳定阶段(1—2天和5天),粘液颗粒和变性的线粒体明显减少。细胞的高尔基复合体不发达以及正常形态的粗面内质网少的情况,在传代5天内变化不大,提示该系腺癌细胞粘液颗粒的形成与正常的粘液腺细胞不尽相同。结合细胞内有肺泡Ⅱ型上皮细胞特有的板层体的存在,文中进一步讨论了SP-C-A-l细胞系可能来源于人的细支气管上皮细胞在细胞癌变过程中发生粘液化生,而使细胞具有合成和分泌粘液的功能。

    Three hybridoma lines secreting McAbs against the lung cancer cell line (LTEP-78) have been established. One of them, BLTI-01, has been subcloned six times and cultured more than eight months in vitro.The experimental data showed that the BLTI-01 has no crossreaction with human lymphocyte surface Ags (within 140 samples) , human erythrocyte surface Ags (100 samples) , marrow cell surface Ags (10 samples) , CEA and AFP. Further experiments with 24 human normal and tumour cell lines indicated that the McAb from...

    Three hybridoma lines secreting McAbs against the lung cancer cell line (LTEP-78) have been established. One of them, BLTI-01, has been subcloned six times and cultured more than eight months in vitro.The experimental data showed that the BLTI-01 has no crossreaction with human lymphocyte surface Ags (within 140 samples) , human erythrocyte surface Ags (100 samples) , marrow cell surface Ags (10 samples) , CEA and AFP. Further experiments with 24 human normal and tumour cell lines indicated that the McAb from BLTI-01 has strong positive reaction with three lung cell lines, one liver cancer, one osteoblastic sarcoma and one breast cancer lines, on the contrary, it hardly reacts with four normal tissue cell lines and the ther human caneer lines.We suggest that the corresponding Ag of McAb from BLTI-01 is a tumour associated Ag.

    用人肺鳞癌细胞LTEP-78细胞系免疫Blab/c小鼠获得3株抗人肺癌细胞的单克隆抗体杂交瘤系。其中BLTI-01株经六次克隆化培养,体外传代8个月以上。BLTI-01与白细胞抗原及血型抗原基本上无交叉反应;与骨髓细胞无交叉反应;与癌胚抗原和胎甲球蛋白不相关;与肺鳞癌、肺腺癌细胞系及部分其它肿瘤细胞呈阳性反应。

    Antibodies were prepared against epidermal growth factor receptor isolated from human placental villus tissue. This antibody is capable of competing with tumor transforming growth factor for receptor binding and inhibits proliferation of human pulmonary adenocarcinoma cells in culture. This result demonstrates that antibody against growth factor receptors may be useful in cancer immunotherapy.

    首次报道从人胎盘绒毛组织提取表皮生长因子受体,并制备受体抗体。此抗体可与肿瘤转化生长因子竞争人肺腺癌细胞表面受体结合点,从而抑制肿瘤细胞的增殖,生长。提示该抗体可能作为肿瘤免疫治疗的一种手段。

     
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