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实验性脑震荡
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  concussive
     The expression of 5-hydroxytryptamine (S-HT) in rat concussive brain tissue
     5-羟色胺在大鼠实验性脑震荡脑组织中的表达
短句来源
     Conclusions Experimental concussive brain injury has impaired effects on learning and memory in rats in short and mid term post concussion.
     结论 实验性脑震荡的近、中期大鼠有学习记忆能力损害的表现
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  “实验性脑震荡”译为未确定词的双语例句
     Expression and significance of enkephalin and dopamine in experimental rat cerebral concussion tissue
     脑啡肽和多巴胺在大鼠实验性脑震荡脑组织中的表达及意义
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     Objective:To study the significance of changes in tumor necrosis factorα(TNFα) and intercellular adhesion molecule1(ICAM1) in rat cerebral concussion.
     目的 :探讨大鼠实验性脑震荡脑组织中肿瘤坏死因子 α(TNFα)和细胞间黏附分子 1(ICAM 1)的表达及其意义。
短句来源
     Expression of neuron nitric oxide synthase (nNOS) gene in rat cerebral concussion
     nNOS基因在大鼠实验性脑震荡中的表达研究
短句来源
     DYNAMIC MORPHOLOGICAL OBSERVATIONS OF BRAIN TISSUES IN EXPERIMENTAL CEREBRAL CONCUSSION
     实验性脑震荡脑组织形态学改变的动态观察
短句来源
     Expressions of enkephalin and dopamine in brain tissue and their significance in cerebral concussional rats
     脑啡肽和多巴胺在实验性脑震荡大鼠脑组织中的表达及意义(英文)
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  相似匹配句对
     DYNAMIC MORPHOLOGICAL OBSERVATIONS OF BRAIN TISSUES IN EXPERIMENTAL CEREBRAL CONCUSSION
     实验性脑震荡脑组织形态学改变的动态观察
短句来源
     Expression of c-myc Protein on Rats′ Brains after Brain Concussion
     实验性大鼠脑震荡后c-myc癌基因的表达
短句来源
     the practice is experiment;
     实践的实验性;
短句来源
     Reconstruction of the Collateral Circulation in the Experimental Fraction
     实验性骨折愈合的血运重建
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     Pathological mechanism of cerebral concussion
     脑震荡病理机制探讨
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  concussive
Chronic post-traumatic headache: Associations with mild traumatic brain injury, concussion, and post-concussive disorder
      
The second portion of this article considers the persistence of the other symptoms associated with chronic PTH, which constitute the post-concussive disorder.
      
PTH is the most common symptom after a head injury, but post-concussive symptoms often complicate the clinical picture and must be considered in the overall management of the chronic PTH patient.
      
The advent of neuropsychologic (NP) testing batteries designed to assess concussive injury has improved the assessment of cognitive dysfunction that occurs in the absence of structural brain abnormalities.
      
A death due to concussive brain injury augmented by alcohol (case report)
      
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Objective:To study the expression of nNOS gene in rat cerebral concussion. Methods:Eighty male Wistar rats were used for animal model of cerebral concussion.They were sacrificed on 1,3,7,14 and 30 days after injury and the brain tissue was taken. The expression of nNOS gene was studied in the course of cerebral concussion by means of immunohistochemistry and in situ hybridization.Results:Clinical manifesfation of typical cerebral concussion was seen in rats of 100?g(the weight causing concussion) group....

Objective:To study the expression of nNOS gene in rat cerebral concussion. Methods:Eighty male Wistar rats were used for animal model of cerebral concussion.They were sacrificed on 1,3,7,14 and 30 days after injury and the brain tissue was taken. The expression of nNOS gene was studied in the course of cerebral concussion by means of immunohistochemistry and in situ hybridization.Results:Clinical manifesfation of typical cerebral concussion was seen in rats of 100?g(the weight causing concussion) group. The pathologic changes were the cerebral vascular dilatation, congestion,edema of cerebral tissue,neuronal degeneration, necrosis,obvious decrease and even disappearance of the Nissl bodies. The protein and mRNA of nNOS were increased on day 3,peaked on day 7, decreased on day 14 and were still positive on day 30. The positive area was seen in the cytoplasm of neurons in cerebral cortex,hippocampus, thalamus and cerebellum. Conclusions:The main pathologic changes of cerebral concussion were blood circulatory disorder and nervous cell degeneration and necrosis. The expression of nNOS gene participated in the course of cerebral concussion and may play an important role in the nervous cell degeneration and necrosis.

目的 :探讨nNOS在大鼠实验性脑震荡中的表达及意义。方法 :采用Wistar雄性二级大鼠复制脑震荡动物模型 ,于伤后 1,3,7,14及 30d活杀取脑组织 ,经免疫组化和原位杂交等技术 ,研究nNOS在脑震荡中的变化规律。结果 :10 0g(致脑震荡砝码 )组见典型脑震荡的临床表现 ,其病理改变为脑血管扩张 ,脑组织淤血、水肿 ,神经元变性、坏死 ,尼氏体减少甚至消失。nNOS蛋白和mRNA于伤后 3d表达增强 ,7d达高峰 ,14d后开始减少 ,30d仍呈阳性表达。阳性部位见于大脑皮层、海马、丘脑和小脑神经元胞浆内。结论 :脑震荡以血液循环障碍和实质细胞变性、坏死为主要病理改变 ;nNOS基因表达参与脑震荡发生时脑组织损伤的病理过程 ,可能对神经细胞变性、坏死起重要调节作用。

Objective To investigate the changes and significance of 5-hydroxytryptamine (5-HT) in rat cerebral concussion. Methods Eighty Wistar male rats were used for the animal model of cerebral concussion, and were sacrificed on 1,3,7,14 and 30 d after injury and the brain tissues were taken off. The expression of 5-HT in the course of cerebral concussion was examined by means of immunohistochemistry. Results The symptomatic appearances of typical brain concussion were observed in 100 g group. The pathologic changes...

Objective To investigate the changes and significance of 5-hydroxytryptamine (5-HT) in rat cerebral concussion. Methods Eighty Wistar male rats were used for the animal model of cerebral concussion, and were sacrificed on 1,3,7,14 and 30 d after injury and the brain tissues were taken off. The expression of 5-HT in the course of cerebral concussion was examined by means of immunohistochemistry. Results The symptomatic appearances of typical brain concussion were observed in 100 g group. The pathologic changes were the cerebral vascular constriction and dilatation, brain congestion and edema. In addition, neuronal degeneration and necrosis were also observed. Nissl's bodies were dramatically diminished, even disappeared. The 5-HT expression was increased at 1 d, peaked at 3 d, turned down at 7 d and regressed to normal level at 14 d. The positive staining was seen in the cytoplasm of neurons in cortex, hippocampus, thalamus and cerebellum. Conclusion The main pathologic changes of cerebral concussion are blood circulatory disorder and neuronal degeneration and necrosis. The data suggest that 5-HT is involved in the process of brain concussion and may contribute to brain injury.

目的 探讨5-羟色胺在大鼠实验性脑震荡脑组织中的表达及其意义。方法 采用Wistar雄性二级大鼠复制脑震荡动物模型,于伤后1、3、7、14及30 d取脑组织,经免疫组化等技术研究5-羟色胺在脑震荡发生发展中的变化规律。结果 100g组见典型脑震荡的临床表现,其病理改变为脑血管收缩与扩张,脑组织淤血、水肿,神经元变性、坏死,尼氏体减少甚至消失。5-羟色胺于伤后1d表达增强,3d达高峰,7d后开始减少,而于14d后逐渐恢复至正常水平。阳性染色见于大脑、海马、丘脑及小脑神经元胞浆内。结论 脑震荡以血液循环障碍和实质细胞变性、坏死为主要病理改变;5-羟色胺参与脑震荡发生时脑组织损伤的病理过程,可能是脑震荡时脑损伤的重要原因之一。

Objective:To study the significance of changes in tumor necrosis factorα(TNFα) and intercellular adhesion molecule1(ICAM1) in rat cerebral concussion.Methods:Eighty Wistar male rats were used for animal model of cerebral concussion,and animals were sacrificed on days 1,3,7,14 and 30 after injury and the brain tissue were taken off.The expressions of TNFα and ICAM1 were determined at various intervals of cerebral concussion by immunohistochemistry staining.Results:Rats in 100 g group were seen the clinical...

Objective:To study the significance of changes in tumor necrosis factorα(TNFα) and intercellular adhesion molecule1(ICAM1) in rat cerebral concussion.Methods:Eighty Wistar male rats were used for animal model of cerebral concussion,and animals were sacrificed on days 1,3,7,14 and 30 after injury and the brain tissue were taken off.The expressions of TNFα and ICAM1 were determined at various intervals of cerebral concussion by immunohistochemistry staining.Results:Rats in 100 g group were seen the clinical situation for typical cerebral concussion.The pathologic changes included the cerebral vascular constriction and dilatation,congestion and edema of cerebral tissue,and neuronal degeneration as well as necrosis. TNFα was increased on day 1,and the positive area was found in the cytoplasm of nervous cells in cerebral cortex and hippocampus,peaking on day 3,decreasing on day 7 and tending to normal range on day 14. ICAM1 was increased on day 1,and the positive area was observed in the cytoplasm of endothelial cells and nervous cells in hippocampus,cerebral cortex and cerebellum,peaking on day 7,decreasing on day 14 and maintaining on day 30.Conclusions:The main pathologic changes of cerebral concussion are circulatory disorder and nervous cells degeneration,apoptosis as well as necrosis.TNFα may participate in the development of early inflammatory,blood vessel damage and regulation of bloodbrain barrier in cerebral concussion. ICAM1 may play an important role in the pathogenesis of inflammatory response,endothelial cell adhesion of blood vessel and the damage of nervous cells.

目的 :探讨大鼠实验性脑震荡脑组织中肿瘤坏死因子 α(TNFα)和细胞间黏附分子 1(ICAM 1)的表达及其意义。方法 :采用 Wistar雄性二级大鼠复制脑震荡动物模型 ,于伤后 1、3、7、14及 30日活杀取脑组织 ,经免疫组织化学等技术研究脑震荡发生、发展中 TNFα和 ICAM 1的变化规律。结果 :砝码 10 0 g组见典型脑震荡的表现 ,其病理改变为脑血管收缩与扩张 ,脑组织淤血与水肿 ,神经元变性、凋亡与坏死。 TNFα于伤后 1日表达增强 ,阳性部位见于大脑皮质、海马神经细胞胞浆中 ,伤后 3日达高峰 ,7日逐渐减少 ,14日后基本恢复至正常水平。 ICAM 1于伤后 1日 ,见海马、大脑及小脑的血管内皮细胞和血管壁明显增多 ;此外海马及大脑皮质神经元亦见阳性 ,于 7日达高峰 ,14日后开始减少 ,30日仍高于正常水平。结论 :脑震荡以血液循环障碍及实质细胞变性和坏死为主要病理改变 ;TNFα参与脑震荡发生时早期炎症反应、血管损伤及血脑脊液屏障功能的调节 ;ICAM 1参与脑震荡发生时炎症反应、血管内皮细胞黏附及神经细胞损害的过程。

 
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