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移行
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  migration
    Low Concentration of H_2O_2 Promoted-Cell Adhesion and Migration in Cultured Rabbit Corneal Epithelial Cells
    低浓度H_2O_2促进兔角膜培养上皮细胞黏附及移行的离体实验研究
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    Study of Molecular Mechanism of UVB & Oxygen Stress-induced Collagen Type I Degradation and EGF-Induced Cell Migration in Cultured Human Lens Epithelial Cells
    中波紫外线和氧化应激诱导晶体上皮细胞中I型胶原降解及EGF诱导人晶体上皮细胞移行的分子机制研究
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    an in vitro wound healing model was used to detect the migration of HLECs treated with HGF(10,20,50μg/L) after 24 hours.
    HLECs损伤愈合模型,观察不同浓度HGF(10、20、50μg/L)处理24h后HLECs的移行情况。
短句来源
    HGF showed effects on migration of 22.1%, 57.1%, 208.6% at the concentration of 10μg/L,20μg/L,50μg/L, respectively.
    HGF可明显促进HLECs的移行,其移行能力分别为22.1%(10μg/L)、57.7%(20μg/L)和208.6%(50μg/L)。
短句来源
    Conclusion HGF can induce the proliferation and obvious migration of HLECs, consequently HGF was a mitogen and potent migratory factor for HLECs.
    结论HGF可促进HLECs的增生和移行,是HLECs的有丝分裂原和强有力的促移行因子。
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    EGFR and AKT phosphorylation induced by EGF were inhibited by PD153035 and LY294002,respectively. Furthermore,EGF could increase the activity of MMP-2 in a time dependent manner in HLECs,which peaked between 24 hours to 48 hours after treatment.
    EGF可显著提高细胞中MMP-2的活性,并随时间延长活性增强,24~48h达到较高水平(1.4倍),PD153035、LY294002可抑制MMP-2的活性以及细胞的移行
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    Effects of Zhaoke defibrase and anti-α_vβ_3 mAb on the adhesion and immigration of bovine retinal vascular endothelial cells
    兆科降纤酶和抗α_vβ_3整合素抗体对牛视网膜血管内皮细胞粘附、移行的影响
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    The changes in adhesion and transmigration of cells are important in the formation, infiltration and spreading of tumor.
    细胞粘附移行的变化在肿瘤的形成、浸润和播散中起重要作用。 肿瘤细胞之间、肿瘤细胞与细胞外基质(ECM),肿瘤细胞与内皮细胞的粘附是由不同的粘附分子来介导的。
短句来源
    (3) The residual lens epithelial cells(RLECs) of control group and EM DDS implanted group migrated obviously than the two atRA DDS implanted groups by electron microscopy examination.
    3.电镜观察可见75ug atRA DDS植入组和150ug atRA DDS植入组与EM DDS植入组及对照组相比,RLECs移行不明显;
短句来源
    Conclusion: The metabolize of lens epithelial cell is most active in all parts of len.
    结论:晶状体上皮细胞是晶状体代谢最活跃的部位,终身进行有丝分裂,并移行至赤道部分化成晶状体纤维。
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  migration
PHENYLPYRUVIC ACID DERIVATIVES AS ENZYME INHIBITORS: THERAPEUTIC POTENTIAL ON MACROPHAGE MIGRATION INHIBITORY FACTOR
      
Moreover, we suppose that migration is not allowed.
      
Relative standard deviations of the relative migration times of DNA segments were >amp;lt;3.6%.
      
A highly cross-linked structure was formed in both the cores and the shells by using a cross-linking agent, which could prevent the migration of hydrophobic PS shells to the inside of particles.
      
By compensating the range migration in wideband airborne mechanic scanning radar, the proposed DBS imaging algorithm can efficiently improve the resolution of a DBS image.
      
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Passive hemagglutination test for humoral immunity and leukocyte inhibition migration test for cellular immunity to lens protein were performed on 72 patients with mature senile cataract before and after cataract extraction.32 persons without cataract and ocular inflammation were served as controls.Tab.I.showed that after intracapsular cataract extraction 100% of the humoral immune responses to lens protein did not change and 40% of

本文用被动血凝集试验和白细胞移行抑制试验,检测了72例老年性白内障成熟期患者手术前、后对晶体蛋白的体液免疫和细胞免疫反应,并以32例无白内障无眼部炎症者作为对照组。结果说明白内障囊内摘出术后,对晶体蛋白的体液免疫反应100%无改变,而细胞免疫反应则有40%从阳性变为阴性。白内障囊外摘出术后,对晶体蛋白的体液免疫反应95.23%无改变,而细胞免疫反应则有21.43%从阳性变为阴性,有7.14%从阴性变为阳性。从这初步的结果,我们建议在白内障囊外摘出术前检查患者对晶体蛋白的免疫反应,手术后如果有炎症反应产生,应重复此项检查,以便考虑使用免疫抑制剂治疗或手术清除晶体残留物。

[ABSTRACT]In 427 eyes which had undergone

在427眼穿透角膜移植手术中,通过寿命表格法计算的植片累加透明率在头2年下降很快,然后减慢:随访12年之后的最终透明率为65%。109眼圆锥角膜术后12年的透明率为90.5%,而49眼大疱性角膜病变术后4年的透明率只有29%。应用内皮显微照相法对252只术眼植片内皮细胞作测量,术后2年内皮细胞平均面积增加较快然后减慢。84只圆锥角膜术眼术后4-5年内皮细胞面积大大增加,但此后即趋稳定且其透明率仍高。然而33只大疱角膜病变术眼的内皮细胞面积明显增加并且与透明率的下降呈进行性线性相关。在实验性大疱角膜病变的兔眼中用正常兔眼植片作移植:植片内皮细胞呈明显进行性增大并可向原缺乏内皮细胞的植床移行。大疱性角膜病变时植床角膜缺乏内皮似乎可引起植片内皮细胞向植床扩展,植片内皮细胞进行性减少及穿透角膜移植术的预后差与此有关。在角膜移植手术中务必保护植片内皮,应用粘性的透明质酸钠可达到此目的。

In order to find out the relationship between the development of complicated cataract inuveitis and immune responses to specific antigen inpatients with complicated cataract of uveitis,the passive hemagglutination assay for humoral immunityand the leukocyte migration inhibition test for cellu-lar immunity to lens and uveal antigen were perfomedon 54 patients suffering from uveitis with com-plicated cataract,52 patients who had uveitis butwithout complicated cataract and 34 persons withoutocular inflammation...

In order to find out the relationship between the development of complicated cataract inuveitis and immune responses to specific antigen inpatients with complicated cataract of uveitis,the passive hemagglutination assay for humoral immunityand the leukocyte migration inhibition test for cellu-lar immunity to lens and uveal antigen were perfomedon 54 patients suffering from uveitis with com-plicated cataract,52 patients who had uveitis butwithout complicated cataract and 34 persons withoutocular inflammation and cataract served as controls.The results showed that immune responses tolens and uveal antigen in the patients suffering fromuveitis with complicated cataract were cellularimmune responses to lens and uveal antign,but inthe patients who had uveitis but without complicatedcataract they were cellular immune responses to uvealantigen.It was suggested that during the development ofcomplicated cataract in uveitis,the lens and uvealantigen entered the systemic circulation,sensitizinglmyphocytes and roducing cellular mediated immune responses.The causality of uveitis and complicated cata-ract in uveitis was discussed.

为了了解葡萄膜炎并发性白内障的发生与患者对特异性抗原免疫反应的关系,本文用间接血凝集试验和白细胞移行抑制试验同时检测了54例葡萄膜炎并发性白内障患者对晶体和葡萄膜抗原的体液和细胞免疫反应。并以52例葡萄膜炎(无并发性白内障)患者和34例无白内障无眼部炎症的健康人作对照。结果可见:葡萄膜炎并发性白内障患者对以上抗原的免疫反应主要表现为对晶体和葡萄膜抗原的细胞免疫反应;而葡萄膜炎(无并发性白内障)患者则主要表现为对葡萄膜抗原的细胞免疫反应。提示在葡萄膜炎并发性白内障形成过程中,晶体和葡萄膜抗原进入血循环,致敏了淋巴细胞,产生细胞介导免疫反应。讨论中指出葡萄膜炎与其并发性白内障互为因果关系。

 
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