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抑制肝纤维化     
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  inhibiting liver fibrosis
     CONCLUSION: Since CMJP could play the role of protecting the liver cells, inhibiting liver fibrosis, decreasing AFP level and reducing the enlargement of spleen, it had good curative effects to chronic hepatitis B patients.
     结论:复方木鸡冲剂具有保护肝细胞、抑制肝纤维化、降低AFP水平和减轻脾肿大的作用,对慢性乙肝有良好疗效。
短句来源
  anti-fibrosis
     Conclusion DHC could inhibit TIMP-1 expression in liver fibrosis tissues,thus attenuate the inhibition of matrix metalloproteinase-1,promote the degradation of extracellular matrix,which might be the possible mech- anism of DHC in anti-fibrosis of liver.
     结论丹芍化纤胶囊能够抑制肝纤维化组织中,TIMP-1的表达,从而减弱了其对基质金属蛋白酶-1 (matrix metalloproteinase-1,MMP-1)的抑制作用,促进了细胞外基质的降解,可能是丹芍化纤胶囊抗肝纤维化作用的机制之一。
短句来源
     As an animal experiment, a fibrosis model of rats with carbon tetrachloride and bovine serum albumin was made. The effect on anti-fibrosis was determined by pathologic cytology, immune histochemistry and image analysis. KANGXIANRUANJIAN could inhibit HSCs, TGF β1 and the receptors of TGF β1 in the fibrosis model of rats.
     采用四氯化碳和小牛血清白蛋白建立大鼠肝纤维化模型 ,通过病理细胞学、免疫组化及图像分析等方法 ,证明抗纤软坚冲剂能明显抑制肝纤维化大鼠的肝星状细胞与转化生长因子 β1及其受体。
短句来源
     The anti-fibrosis mechanism of glycyrrhizin may be through increasing the expression of Smad7.
     甘草酸抑制肝纤维化的作用机制之一是通过增强Smad7的表达实现的。
短句来源
     Conclusion Estradiol has anti-fibrosis function. This mayberesults from inhibiting the following factors:①they inhibit the proliferation and actiation of activatedHSCs;
     结论雌二醇具有抑制肝纤维化作用是因为:①可以抑制HSC的增殖、活化;
短句来源
  anti hepatic injury
     Conclusion: RGG can decrease the serum levels of TGFβ 1, Ⅰ and Ⅲ collagen and the liver content of Hyp ,which might contribute to its effects of anti hepatic injury, protecting hepatocytes, improving liver function ,and protecting against liver fibrosis.
     结论 :软肝冲剂能明显降低免疫性肝纤维化大鼠血清TGFβ1和Ⅰ、Ⅲ型胶原水平 ,以促进肝细胞再生 ,抑制肝纤维化、肝硬化的形成
短句来源
  anti-fibrogenesis
     Objective To evaluate the relationship between serum testosterone (T) and anti-fibrogenesis of 17β-estradiol (β-Est).
     目的探讨体内睾酮(T)和雌激素(β-Est)抑制肝纤维化的关系。
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      anti-fibrosis
    The amelioration effect of HGF on postinfarction heart failure could not be limited to stimulating angiogenesis, anti-apoptosis, anti-fibrosis, but was also involved in the recruitment of stem cells into myocardium.
          
    Chronic pancreatitis is a progressive pancreatic fibrotic process, and anti-fibrosis is the key therapy for liver fibrosis.
          
    Recently, anti-fibrosis therapy has attracted attention as a new treatment strategy.
          


    The inhibitory effect of vitamin A on experimental hepatic fibrosis in rats induced by ad-ministration of carbon tetrachloride was studied with light,electron microscope,immunohistochemical ??and morphologic quantitative technique.The results of investigation indicated that vitamin A could de-crease the deposit of fibronectin,type Ⅰ and type Ⅲ collagen in liver,inhibit transformation of thefat storing cell to fibroblast and alleviate hepatic fibrosis in rats induced by administration of carbontertrachloride.In...

    The inhibitory effect of vitamin A on experimental hepatic fibrosis in rats induced by ad-ministration of carbon tetrachloride was studied with light,electron microscope,immunohistochemical ??and morphologic quantitative technique.The results of investigation indicated that vitamin A could de-crease the deposit of fibronectin,type Ⅰ and type Ⅲ collagen in liver,inhibit transformation of thefat storing cell to fibroblast and alleviate hepatic fibrosis in rats induced by administration of carbontertrachloride.In addition,the mechanism and significance of vitamin A inhibition to the hepatic fibro-sis were analysed.

    利用光镜、电镜、免疫组化和形态学定量技术动态研究维生素A对大鼠四氯化碳肝纤维化的抑制作用。结果表明,维生素A可减少四氯化碳中毒大鼠肝内纤维连接蛋白和Ⅰ、Ⅲ型胶原沉积,抑制贮脂细胞向成纤维细胞转化,并可明显地减轻肝纤维化程度。本文还对维生素A抑制肝纤维化的机理及意义作了初步探讨。

    Fifty-six patients with chronic hepatitis B were treated with Fuzheng Huayu 319 Recipe (319 Recipe) for supplementing body resistance and removing blood stasis, the efficacy was compared with that of 19 cases treated with Dahuang Zhechong Pill as control. The effective rate of the two groups were 87.5o/o and 68.4o/o respectively. Results showed that the 319 Recipe could not only improve the clinical symptoms and liver functions, but also reduce the serum levels of monoamine oxidase B activity, tissue inhibitor...

    Fifty-six patients with chronic hepatitis B were treated with Fuzheng Huayu 319 Recipe (319 Recipe) for supplementing body resistance and removing blood stasis, the efficacy was compared with that of 19 cases treated with Dahuang Zhechong Pill as control. The effective rate of the two groups were 87.5o/o and 68.4o/o respectively. Results showed that the 319 Recipe could not only improve the clinical symptoms and liver functions, but also reduce the serum levels of monoamine oxidase B activity, tissue inhibitor of metalloproteinase I (TIMP), procollagen typeⅢN-terminal peptide, collagen type Ⅳ, hyaluronic acid and laminin, as well as increase the excretion of droxyproline markedly. As compared with control group, the difference between the two groups were significant except the change of TIMP,the improvement of 319 Recipe group were more obvious than that of control group (P <0 . 001 ̄ 0 . 05). These results displayed the good effect of 319 Recipe in treating chronic hepatitis B and preventing the development of hepatic fibrosis.

    扶正化瘀319方治疗慢性乙型肝炎56例,并以大黄虫丸治疗的19例患者为对照,临床有效率分别为87.5%和68.4%。该方不但能有效地改善肝功能及临床症状,且能降低患者显著增高的血清单胺氧化酶B(MAO-B)活性,金属蛋白酶组织抑制因子-1(TIMP)、Ⅲ型前胶原肽、Ⅳ型胶原、透明质酸及层粘蛋白(LN)的含量,提高尿羟脯氨酸(Hyp)排泄量(P<0.001~0.05)。与对照组相比,除TIMP外.改善程度均以观察组为著。表明该方对慢性乙型肝炎具有较好的治疗效果,在一定程度上抑制了肝纤维化的进展。

    The rats hepatic impairment caused from CCL 4 was studied. CCL 4 was accepted in the study on new drugs. By using soft liver pill,the result of the experiment indicated that the pill had good effect to prevent the hepatocyte from necrisis,fibrosis and promote regeneration of the hepatocyte. The pharmacodynamic studies also indicated that the pill could lower the level of ALT in the serum and hydroxyphotine in the hepatic tissue.

    本实验中采用了新药物研究中认可的CCL4诱导大白鼠慢性肝损害的模型作为研究对象.病理结果说明使用软肝丸有良好的抗细胞坏死,能明显抑制肝纤维化过程,促进肝细胞再生作用.药效学的研究,也提示软肝丸可以降低血清ALT及肝组织中的羟脯氨酸的含量.

     
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