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皮染
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  skin administration
     The MLD by skin administration was in excess of 4800.0 mg/kg in mice and 3840 mg/kg in rats.
     小鼠经皮染毒最小致死量大于4800mg/kg,大鼠为大于3840mg/kg。
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  “皮染”译为未确定词的双语例句
     Methods Wistar rats were exposed to CCT(215,86 and 43 mg/kg respectively)by skin contamination.
     方法 Wistar大鼠经皮染毒 ,剂量为 2 15 ,86 ,4 3mg/kg。
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     Different concentrations of hydroquinone(50.0%,33.3%,20.0%,11.1%,0.0%) applied topically to the skin of 16 guinea-pigs and 3 New Zealand rabbits once per day for 14 days.
     另取豚鼠16只,新西兰白兔3只,动物背部用50%、33.3%、20%、11.1%、0%氢醌涂皮染毒,每天1次,连续14天。
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     [Methods] 45 male Wistar rats were divided into control group,3 different exposure level groups,which exposed to gasoline on 2cm×3cm skin area with the dose of 250mg/cm 2 for 1 (group A),4 (group B) and 8 days (group C),respectively,and an intervention group exposed to gasoline of 250mg/cm 2 for 8 days after the application of protective agent on skin.
     [方法 ]45只 Wistar雄性大鼠分为对照组、实验组和干预组 ,染毒方式为经皮染毒。 实验组染毒剂量分别为 :2 5 0 m g/ cm2 × 1 d,2 5 0 mg/ cm2 × 4d,2 5 0 m g/ cm2 × 8d,染毒面积为 2× 3cm2 。
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     Frost top" foxskin in black and brown
     狐狸皮染草上霜黑色、棕色拔微霜工艺
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     Results:All the carcinogenic and mutagenic tests are negative,except the micronucleus rate(4.6‰) in the chronic bronchus contaminated rats was significantly higher than that of control rats(0.81‰).
     在致癌及致突变试验中,仅见大鼠气管注入慢性试验染毒组的微核率(4.6‰)较对照组有所升高。 大鼠经支气管注入染毒1年及兔经皮染毒1年半后,均未诱导产生肿瘤。
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     OBSERVATION OF INFECTING ABILITY OF MARMOTA FUR INFECTED WITH YERSIN A PESTIS
     疫旱獭再感能力的观察
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     Dyeing and Finishing of Peach-skin Viscose Crepe
     人棉桃整工艺探讨
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     Sculpture on Skin
     雕画
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     THE ANGIOARCHITECTURE OF SKIN FLAP
     瓣的血管构筑
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     the dyeing dynamic is first order when m=1 and α exhaustion rate.
     α为上率。
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Objective To investigate the expression of CD44v3 and CD44v6 in oral premalignant epithelia and its relationship to the carcinogenesis of oral mucosa Methods 85 normal, hyperplastic, dysplastic and malignant oral epithelia were studied by immunohistochemistry Results Normal and hyperplastic epithelia showed strong CD44v3, v6 staining on cell membrane Cells in the deep layers of moderate and severe dysplastic epithelia showed remarkably reduced staining intensity or no positive staining as compared...

Objective To investigate the expression of CD44v3 and CD44v6 in oral premalignant epithelia and its relationship to the carcinogenesis of oral mucosa Methods 85 normal, hyperplastic, dysplastic and malignant oral epithelia were studied by immunohistochemistry Results Normal and hyperplastic epithelia showed strong CD44v3, v6 staining on cell membrane Cells in the deep layers of moderate and severe dysplastic epithelia showed remarkably reduced staining intensity or no positive staining as compared with normal and hyperplastic epithelia This down regulation of CD44v3, v6 expression was noted in all 12 cases of severe dysplasia In invasive front of squamous cell carcinoma and some metastases to lymphnode CD44v3 and v6 staining were also weak or negative Conclusion Low level expression of CD44v3 and v6 exists in moderate and severe epithelial dysplasia This may be related to the reduction of cellular adhesion and thereby facilitates the infiltration of basement membrane by dysplastic cells

目的 研究细胞粘附分子CD44v3和CD44v6在口腔癌前病变中的表达特点及其与癌变的关系。方法 用SP免疫组织化学染色法检查 85例正常、单纯及异常增生的口腔上皮和口腔鳞癌中CD44v3和CD44v6的表达。结果 正常及单纯增生口腔上皮细胞膜有CD44v3和v6的强表达 ,上皮染成网状。上皮轻度异常增生变化不明显。随上皮异常增生程度加重 ,上皮深层细胞出现CD44v3和v6低表达 ,染色变浅或无染色 ,12例重度异常增生者均出现低表达。同一标本中既有上皮单纯增生又有上皮中、重度异常增生的 9例中 ,8例有CD44v3和v6的低表达。鳞状细胞癌的浸润缘有上述低表达 ,部分转移灶较原发灶染色浅。结论 上皮中、重度异常增生时 ,CD44v3和v6表达下降。这种低表达可能与异常增生时细胞粘附力下降及癌变时发生基底膜浸润有关。

 
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