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大鼠移植肝脏
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  rat liver graft
     The effect of Shenfu injection on ischemia reperfusion injury of rat liver graft
     参附注射液对大鼠移植肝脏缺血再灌注损伤的影响
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     Objective To observe the effect of ischemic postconditioning on apoptosis of hepatocytes and expression of caspase-3 gene protein of rat liver graft,and to elucidate the possible mechanisms.
     目的:观察缺血后处理(ischemic postconditioning,Post-con)对大鼠移植肝细胞凋亡及 Caspase-3蛋白表 达的影响,探讨在体内条件下,缺血后处理对大鼠移植肝脏凋亡的保护机制。
短句来源
     Objective To investigate the protective e ffect of nitric oxide (NO) induced by ischemic preconditioning (IP) on ischemic reperfusion injury of rat liver graft.
     目的 探讨一氧化氮 (NO)在缺血预处理 (IP)保护大鼠移植肝脏缺血再灌注损伤中的作用。
短句来源
     Conclusion IP could protect the microcirculation of rat liver graft from injury during the early phase of reperfusion.
     结论 IP对大鼠移植肝脏微循环的早期再灌注损伤有保护作用
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     ObjectiveTo investigate the effect and mechanism of ischemic preconditioning (IP)on preservation/reperfusion injury of rat liver graft.
     目的探讨缺血预处理 (ischemicpreconditioning ,IP)对大鼠移植肝脏保存再灌注损伤的保护作用及机理。
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  “大鼠移植肝脏”译为未确定词的双语例句
     Experimental Study on Immune Privilege Induced by Homologous Sertoli Cells with Fas Ligand in Transplanted Liver of Rat
     同源FasL~+Sertoli细胞诱导大鼠移植肝脏免疫豁免的研究
短句来源
     Objective To explore the feasibility of interleukin 1 receptor associated kinase-4 (IRAK-4) as gene therapy target for liver ischemia/reperfusion injury (I/RI) and effective approach in vivo for short hairpin RNA (shRNA) interference used to gene therapy in liver graft hqappened.
     目的探讨以白细胞介素-1受体相关激酶-4(IRAK-4)为靶点,阻断内毒素胞内信号转导后对大鼠移植肝脏再灌注损伤(I/RI)的影响并探索肝移植时可行的RNA干扰(RNAi)治疗途径。
短句来源
     The Influence with Block the Endotoxin Signal Transduction for Ischemia/Reperfusion Injury of Graft Liver in Rats.
     阻断内毒素胞内信号转导通路对大鼠移植肝脏再灌注损伤的影响
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     Effects of IRAK-4-shRNA perfused through portal vein in vivo or in vitro on ischemia/reperfusion injury of liver graft in rats
     活体/离体门静脉灌注转染IRAK-4-shRNA对大鼠移植肝脏再灌注损伤的影响
短句来源
     The Study on anti oxygen derived free radical injury of rewarm in grafted liver of rats
     复温对大鼠移植肝脏抗氧自由基损伤的研究
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  相似匹配句对
     Indications of liver transplanation.
     肝脏移植的适应证
短句来源
     Results E.
     结果 大鼠肝脏E.
短句来源
     Protective effect of ischemic preconditioning on microcirculation of rat liver graft
     缺血预处理对大鼠移植肝脏微循环的保护作用
短句来源
     Combined intestine-auxiliary liver transplantation in rats
     大鼠小肠—辅助性肝脏联合移植的实验研究
短句来源
     The Study on anti oxygen derived free radical injury of rewarm in grafted liver of rats
     复温对大鼠移植肝脏抗氧自由基损伤的研究
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  rat liver graft
Time-dependent changes in the viability of rat liver graft during cold preservation with Euro-Collins solution were evaluated with NADH fluorometry.
      
High-Na + low-K + UW cold storage solution reduces reperfusion injuries of the rat liver graft
      
The effects of vascular bed expansion in steatotic rat liver graft viability
      
The beneficial effect of superoxide dismutase on the rat liver graft
      
These results indicate the beneficial effect of SOD on the rat liver graft and may implicate oxygen free radicals in the pathogenesis of ischemia/reperfusion injury in liver grafts.
      


ObjectiveTo investigate the effect and mechanism of ischemic preconditioning (IP)on preservation/reperfusion injury of rat liver graft.MethodsOne hundred and twenty eight male Sprague Dawley rats undergoing orthotopic liver transplantation were randomly divided into 4 groups: group A (control group), group B (IP group), group C (adenosine,Ado group), and group D (inhibitor of NO synthesis,NAME group).ResultsPosttransplantation one week survival rate, 2 hrs reperfusion serum NO, and hepatic tissue adenosine in...

ObjectiveTo investigate the effect and mechanism of ischemic preconditioning (IP)on preservation/reperfusion injury of rat liver graft.MethodsOne hundred and twenty eight male Sprague Dawley rats undergoing orthotopic liver transplantation were randomly divided into 4 groups: group A (control group), group B (IP group), group C (adenosine,Ado group), and group D (inhibitor of NO synthesis,NAME group).ResultsPosttransplantation one week survival rate, 2 hrs reperfusion serum NO, and hepatic tissue adenosine in IP group and Ado group were 88%(7/8) and 88%(7/8), (33 0±6 1)?μmol/l and (29 1±6 5)?μmol/l, ( 7 2± 1 8)?μmol/g and (5 7±1 3)?μmol/g, respectively, while in control group they were 38%(3/8),( 15 4± 3 0)?mol/l, and (3 69±0 54)?μmol/g, respectively(all P <0 05).Serum ALT and TNF in IP group and Ado group were (287±82)?IU/L and (357±93)?IU/L,(1 15±0 23)?ng/ml and (1 14±0 27)?ng/ml, respectively,which were lower than (588±58)?IU/L, and (1 59±0 35)?ng/ml, respectively in control group(all P <0 05).Histology in IP group and Ado group showed less injury than those in control group. One week survival rate,serum NO and ALT in NAME group were 25%(2/8)?(13 74±3 11)?μmol/l and (634±65)?IU/L, respectively, similar to those in control group( P >0 05). However, hepatic tissue adenosine level was (5 56±1 19)?μmol/g, higher than that in control group( P <0 05).ConclusionIP protects rat liver graft from preservation/reperfusion injury, possibly by increasing adenosine in hepatic tissue, inducing the generation of NO.

目的探讨缺血预处理 (ischemicpreconditioning ,IP)对大鼠移植肝脏保存再灌注损伤的保护作用及机理。方法采用SD大鼠原位肝移植动物模型 ,12 8只大鼠随机分成A(对照组 )、B(IP组 )、C(腺苷 ,Ado组 )、D(NO合成抑制剂 ,NAME组 )组 ,每组 32只。其中各组的半数用于观察存活率 ,另一半用于移植肝脏再灌注 2h后取血及肝脏检测。结果IP组和Ado组的 1周存活率、血清NO水平及肝组织腺苷含量分别为 88% (7/ 8)和 88% (7/ 8) ,(33 0± 6 1) μmol/l和 (2 9 1± 6 5 ) μmol/l,(7 2± 1 8) μmol/g和 (5 7± 1 3) μmol/g ,均高于对照组的 38% (3/ 8) ,(15 4± 3 0 )mol/L和 (3 6 9±0 5 4 ) μmol/g (P <0 0 5 ) ,血清ALT及TNF含量分别为 (2 87± 82 )IU/L和 (35 7± 93)IU/L ,(1 15± 0 2 3)ng/ml和 (1 14± 0 2 7)ng/ml,均低于对照组的 (...

目的探讨缺血预处理 (ischemicpreconditioning ,IP)对大鼠移植肝脏保存再灌注损伤的保护作用及机理。方法采用SD大鼠原位肝移植动物模型 ,12 8只大鼠随机分成A(对照组 )、B(IP组 )、C(腺苷 ,Ado组 )、D(NO合成抑制剂 ,NAME组 )组 ,每组 32只。其中各组的半数用于观察存活率 ,另一半用于移植肝脏再灌注 2h后取血及肝脏检测。结果IP组和Ado组的 1周存活率、血清NO水平及肝组织腺苷含量分别为 88% (7/ 8)和 88% (7/ 8) ,(33 0± 6 1) μmol/l和 (2 9 1± 6 5 ) μmol/l,(7 2± 1 8) μmol/g和 (5 7± 1 3) μmol/g ,均高于对照组的 38% (3/ 8) ,(15 4± 3 0 )mol/L和 (3 6 9±0 5 4 ) μmol/g (P <0 0 5 ) ,血清ALT及TNF含量分别为 (2 87± 82 )IU/L和 (35 7± 93)IU/L ,(1 15± 0 2 3)ng/ml和 (1 14± 0 2 7)ng/ml,均低于对照组的 (5 88± 5 8)IU/L及 (1 5 9± 0 35 )ng/ml(P <0 0 5 ) ,组织的病理学改变也轻于对照组 ;NAME组的 1周存活率、血清NO及ALT含量等分别为 2 5 % (2 / 8)、(13 74± 3 11) μmol/l及 (6 34± 6 5 )IU/L ,与对照组相近 (P >0 0 5 ) ,而肝组织腺苷含量为 (5 5 6± 1 19)μmol/g ,与对照组差异有显著意义 (P <0 0 5 )。 结论IP对大鼠移植肝脏的保存再灌注损伤具有保护

Objective To investigate the protective effect of ischemic preconditioning on microcirculation of rat liver graft during the early phase of reperfusion injury. Methods Male Sprague Dawley (SD) rats were used as donors and recipients of orthotopic liver transplantation. The period of cold preservation and anhepatic phase were 100 min and 25?min respectively. 32 rats were randomly divided into 2 groups (n=16 in each). In the control group, donor livers were flushed through the portal veins with physiological...

Objective To investigate the protective effect of ischemic preconditioning on microcirculation of rat liver graft during the early phase of reperfusion injury. Methods Male Sprague Dawley (SD) rats were used as donors and recipients of orthotopic liver transplantation. The period of cold preservation and anhepatic phase were 100 min and 25?min respectively. 32 rats were randomly divided into 2 groups (n=16 in each). In the control group, donor livers were flushed through the portal veins with physiological saline solution containing heparin only before harvested; In the IP group, before donor livers were harvested, the portal veins and hepatic arteries of them were interrupted for 10?min, and reflow was initiated for another 10?min, then did as control group. The sample of blood and hepatic tissue of both groups were taken after 2?h of reperfusing liver graft. Results Activity of anti-oxidase in hepatic tissue was higher in IP group than in control group (P

目的 探讨早期再灌注损伤中缺血预处理 (IP)对大鼠移植肝脏微循环的保护作用。方法 采用SD大鼠原位肝移植动物模型 ,供肝冷保存时间 10 0min ,无肝期 2 5min。 3 2只SD大鼠随机平均分成两组 ,每组 16只。对照组 :获取供肝前仅以肝素生理盐水经门静脉灌注 ;IP组 :获取供肝前阻断肝门血供 10min ,再灌注 10min ,然后再以肝素生理盐水经门静脉灌注。移植肝脏再灌注 2h后取血及肝脏检测。结果 IP组的肝脏抗氧化酶活力明显高于对照组 (P <0 .0 1) ,血清丙氨酸转氨酶(ALT)、门冬氨酸转氨酶 (AST)、乳酸脱氢酶 (LDH)及肝组织中的过氧化产物丙二醛 (MDA)含量均明显低于对照组 (P <0 .0 0 1) ;肝组织损伤以窦状内皮细胞为主 ,并且是以凋亡的方式发生死亡 ,IP组窦状内皮细胞损伤明显轻于对照组 (P <0 .0 0 1)。结论 IP对大鼠移植肝脏微循环的早期再灌注损伤有保护作用

Objective To investigate the protective effect of ischemic preconditioning (IP) on ischemic reperfusion injury of rat liver graft. Methods Male Sprague Dawley rats were used as donors and recipients of orthotopic liver transplantation,the period of cold preservation and anhepatic phase were 100 min and 25 min respectively.Sixty four rats were randomly divided into 2 groups ( n =32),control group: donor livers were flushed through the portal veins with physiological saline solution containing heparin...

Objective To investigate the protective effect of ischemic preconditioning (IP) on ischemic reperfusion injury of rat liver graft. Methods Male Sprague Dawley rats were used as donors and recipients of orthotopic liver transplantation,the period of cold preservation and anhepatic phase were 100 min and 25 min respectively.Sixty four rats were randomly divided into 2 groups ( n =32),control group: donor livers were flushed through the portal veins with physiological saline solution containing heparin only before harvested; IP group: before donor livers were harvested,the portal veins and hepatic arteries of them were interrupted for 10 min,and reflow was initiated for another 10 min,then did as control group.One half of each group were used to investigate 1 week survival rate of recipients,and another half of each group were used to take sample of blood and hepatic tissue after 2 hours of reperfusion of liver graft. Results One week survival rate,amount of bile,serum NO and activity of anti oxidase were higher in IP group than those in control group( P <0.05),meanwhile,serum ALT,AST,LDH,TNF and superoxide in hepatic tissue were lower in IP group than those in control group ( P <0.05),and histological findings in IP group showed less injury than those in control group. Conclusion IP could increase production of serum NO,reduce the level of serum TNF and protect rat liver graft from ischemic reperfusion injury.

目的 探讨缺血预处理 (ischemicpreconditioning ,IP)对大鼠移植肝脏缺血再灌注损伤的保护作用。 方法 采用SD大鼠原位肝移植动物模型 ,供肝冷保存时间 10 0min ,无肝期 2 5min。 64只SD大鼠随机均分成两组 :对照组 ,获取供肝前仅以肝素生理盐水经门静脉灌注 ;IP组 ,获取供肝前阻断肝门血供 10min ,再灌注 10min ,然后再以肝素生理盐水经门静脉灌注。每组受体的一半 (n =8)用于观察存活率 ,另一半 (n =8)用于移植肝脏再灌注 2h后取血及肝脏检测。结果 IP组的 1w存活率、胆汁分泌量、抗氧化酶活力、血清NO水平均明显高于对照组 (P<0 .0 5 ) ,血清ALT、AST、LDH、TNF及肝组织中的过氧化产物含量均明显低于对照组 (P<0 .0 5 ) ,组织的病理改变也轻于对照组。结论 IP能够提高血清NO水平 ,降低血清TNF含量 ,对大鼠移植肝脏的缺血再灌注损伤具有保护作用

 
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