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   白血病组 的翻译结果: 查询用时:0.017秒
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心血管系统疾病
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白血病组
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  leukemia patients
     Method: Telomerase activity and hTERTmRNA were detected in 52 acute leukemia patients and 20 normal controls by TRAP and RT-PCR respectively.
     方法 :采用TRAP和RT PCR法分别检测 5 2例急性白血病患者 (白血病组 )及 2 0例非白血病且骨髓象正常者 (对照组 )的端粒酶活性与hTERTmRNA表达。
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     Stratification with leukemia types,age and gender was made for further comparison. Results The frequencies of GSTT1 0/0 genotype and GSTT1 0/0-GSTM1 0/0 combined genotype were higher in leukemia patients than in controls, and the differences were significant.
     结果 白血病组和正常对照组之间 ,GSTT10 / 0基因型以及GSTT10 / 0 GSTM10 / 0联合基因型的分布频率差异有显著性 ,缺损基因型在白血病组显著升高。
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     Methods:The expression of Survivin and Smac mRNA were measured in 119 adult leukemia patients(including 78 de novo acute leukemia patients,7 replased patients,14 chronic granulocytic leukemia patients in chronic phase and 20 complete remission patients),15 samples of normal controls(NC) and K562,NB4,KG-1α,HL-60 cell lines by semi-quantityreverse transcription polymers chain reaction(RT-PCR).
     方法:本研究的研究对象包括119例白血病患者(包括78例初治急性白血病患者、7例复发患者、14例慢性粒细胞白血病慢性期患者和20例缓解期患者),15名健康人为正常对照和K562、NB4、KG-1α、HL-60细胞株为阳性对照。 将其分为初治急性白血病组、缓解组、复发组、慢性粒细胞白血病组和正常对照组。
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     Results:(1)The results of vectorcardiogram showed that the magitude of maximum QRS vector in Harizantal plane and the R/T in Frontal and Sagittal planes were greater in leukemia patients than in the normal group;
     结果 :VCG检测显示白血病组横面 QRS环最大向量振幅、额面及横面 R/ T均高于正常对照组 ;
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     Results None of 94 leukemia patients developed VOD (0%).
     白血病组94例患者中无一例发生VOD(0%),其中27例为非亲缘供者移植;
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  “白血病组”译为未确定词的双语例句
     ResuIts (1)The positive rates of CD133 in AML,ALL,chronic leukemia and MDS patients were 55.2%,42.9%,22.2% and 55.6% respectively.
     结果 ①AML组CD133表达阳性率为55.2%,ALL组为42.9%,慢性白血病组为22.2%,MDS组为55.6%。
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     Results The levels of IL-15 in ALL group,ANLL group, non-leukemia group and healthy children were (34.37±2.8) ng/L, (29.61±3.2 )ng/L, (117.54±3.9) ng/L, (122.23±4.2) ng/L.
     结果ALL组、ANLL组、非白血病组及正常对照组血清IL-15水平分别为(34.37±2.8)ng/L,(29.61±3.2)ng/L,(117.54±3.9)ng/L,(122.23±4.2)ng/L;
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     Results SHP-2 mRNA was(5.08±2.87) in leukemia mice,(4.59±2.36) in mice free of canceration and(3.54±1.02) in controls.
     结果白血病组、辐射未癌变组及对照组SHP-2 mRNA含量分别为(5.08±2.87)、(4.59±2.36)、(3.54±1.02);
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     ③The expression of RbAp46 protein was lower in refractory leukemia than those in non-refractory leukemia (mean A, 87.1±33.8 vs126.6±21.2, P<0.05).
     ③难治性白血病患者BMMNCRbAp46蛋白表达的平均A值为87.1±33.8,明显低于非难治性白血病组的126.6±21.2(P<0.05)。
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     The rate of DNA-PKcs expression in childhood leukemia was lower than that in the control group(χ2=7.898,P<0.01).
     DNA-PKcs在儿童白血病组的阳性表达率低于对照组(2χ=7.898,P<0.01)。
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     The SV and CO of the treatment group B increased in 5 minutes, which wa
     B
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     ] group.
     ]
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     treatment group which was injected ALF.
     治疗 ,以抑白血病因子治疗。
短句来源
     That of a cute leukemic group was higher than that of chronic leukemic group.
     急性白血病显著高于慢性白血病
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     WHAT IS LEUKEMIA?
     提问白血病
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  leukemia patients
Biochips have been developed for identification of the tuberculosis pathogen and its antibiotic-resistant forms; of orthopoxviruses, including the smallpox virus; of the anthrax pathogen; and chromosomal rearrangements in leukemia patients.
      
With clinical specimens, the method was shown to efficiently identify the chromosome translocations in leukemia patients.
      
In this study, the effects of inhibition of ubiq-uitin-proteasome pathway on human bone marrow (BM) mononuclear cells (MNCs) obtained from 10 normal persons and 8 leukemia patients were examined.
      
Cytostatic therapy and blood antioxidants/prooxidants balance in acute myeloblastic leukemia patients
      
Results: Bone marrow tissues of 20 acute leukemia patients were studied, 11 healthy donors' bone marrows were taken as a control.
      
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This paper reports the evaluation of plasma antithrombin Ⅲ quantity and activity in 98 cases with various leukemia and 100 control cases.Plasma antithrombin Ⅲquantity was evaluated by the rocket immunoelectophorcsis.Plasma antithrombin Ⅲactivity was evaluated by the method of thrombin gel inhibit ring test.The resultsshowed that the quantity of plasma antithrombin Ⅲ in leukemia group was obviouslylower than that in the control group(P<0.05).The groups of acúte pro-myeloeyticleukemia,acute monoeytic leukemia,and...

This paper reports the evaluation of plasma antithrombin Ⅲ quantity and activity in 98 cases with various leukemia and 100 control cases.Plasma antithrombin Ⅲquantity was evaluated by the rocket immunoelectophorcsis.Plasma antithrombin Ⅲactivity was evaluated by the method of thrombin gel inhibit ring test.The resultsshowed that the quantity of plasma antithrombin Ⅲ in leukemia group was obviouslylower than that in the control group(P<0.05).The groups of acúte pro-myeloeyticleukemia,acute monoeytic leukemia,and chronic myeloeytie leukemia were all likewiselower than that of the control group(P<0.001).Possible mechanism and clinicvalue are discussed.

在对98例各种类型白血病血浆 AT-Ⅲ含量及活性测定结果中,发现白血病组血浆AT-Ⅲ含量比正常对照组显著降低(P<0.05)。其中以急性早幼粒细胞性自血病组,急性单核细胞白血病组及慢性粒细胞性白血病组降低非常显著(均 P<0.001)。初步探讨了产生这些结果的部分原因及其临床意义。

Cytogenitic studies indicated that chromosome abnormality of patients withMDS had been associated mith leukemia.The result of the chromosome examinationin 20 cases was analysed,9 MDS and 11 leukemia.The chromosome aberration ratesof the MDS group and the leukemia group were much higher than that of the com-pared group,and then the chromosome abnormality of MDS was multiform and non-random.The abnormal karyotype was found in six of the twenty cases,one MDS casewith 46,XX,t (8;21) (q~(24);q~(22);),and five cases...

Cytogenitic studies indicated that chromosome abnormality of patients withMDS had been associated mith leukemia.The result of the chromosome examinationin 20 cases was analysed,9 MDS and 11 leukemia.The chromosome aberration ratesof the MDS group and the leukemia group were much higher than that of the com-pared group,and then the chromosome abnormality of MDS was multiform and non-random.The abnormal karyotype was found in six of the twenty cases,one MDS casewith 46,XX,t (8;21) (q~(24);q~(22);),and five cases leukemia with 46,XY/45,X,-Y;45,XX,-5 and three of 46,XY,ph.It was shown that the chromosome abnormalityin MDS might be consistent with cancer characteristics and the clonal chromosomeabnormality as well.The chromosome examination of leukemia was of value to thediagnosis,treatment and prediction of the disease,especially much more valuable tothe research in the cause of leukemia,the location of oncogene and their relationship.

报告分析20例血液病染色体检查结果,MDS9例,白血病11例。MDS 组染色体畸变率与白血病组一样明显高于正常人,并有多样化和非随机性的特点。异常核型检出6例,MDS1例46,XX,t(8;21)(q~(24);q~(22)),白血病5例为46,XY/45,X-Y;45,XX,-5及46,XY,ph3例。表明 MDS 染色体改变已具备了恶性肿瘤的特点、还可有克隆性异常改变。

Neuraminidase activity on red cell membrane was determined in 30 normal subjects, 27 patients with noncancerous disease and 83 with various cancers. Among the cancer patients 32 had gastric cancer, 17 leukemia and 34 other cancers, including lung, liver, intestine and bladder cancers, etc. The results showed that the differences of neuraminidase activity on red cell membrane between various cancer groups and control group were significant (P<0.001 or P<0.01). However there was no significant difference(P>0.05)...

Neuraminidase activity on red cell membrane was determined in 30 normal subjects, 27 patients with noncancerous disease and 83 with various cancers. Among the cancer patients 32 had gastric cancer, 17 leukemia and 34 other cancers, including lung, liver, intestine and bladder cancers, etc. The results showed that the differences of neuraminidase activity on red cell membrane between various cancer groups and control group were significant (P<0.001 or P<0.01). However there was no significant difference(P>0.05) between noncancerous disease group and control. Value exceeding the upper limit (mean±2SD) of normal neuraminidase activity was regarded as positive for malignancy. The positive percentage in gastric cancer was 81.3%, leukemia 88% and other cancers 94%.

作者对对照组30例,非癌疾病组27例,各类恶性肿瘤83例(包括胃癌组、白血病组、肺癌、肝癌、肠癌、膀胱癌等)进行红细胞膜神经氨酸苷酶活力测定。结果各类恶性肿瘤组红细胞膜神经氨酸苷酶活力差异展都比对照组高,差异有非常显著性(P<0.001或<0.01);而非癌疾病组与对照组之间差异无显著性(P>0.05)。以对照组神经氨酸苷酶活力最高上限数(+2SD)为参考值,超过此值者判为恶变阳性,胃癌组阳性率为81.3%,白血病组为88%,其它恶性肿瘤组为94%。结果提示红细胞膜神经氨酸苷酶活力检测对恶性肿瘤的诊断有临床意义。

 
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