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电迷宫
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  y maze
    Methods Nitric acid redactase and radioimmunity method are taken to detect the content of NO and SS in rat hippocampus after N ω nitro L arginine (L NA),a nitric oxide synthase (NOS) inhibitor,microinjected into hippocampus,and Y maze is used to observe the changes of capability of learning and memory,and a correlative analysis is made.
    方法 大鼠海马微量注射一氧化氮合酶 (NOS)抑制剂 N-ω-硝基 - L -精氨酸 (N -ω- Nitro- L - Arginine,L - NA )后采用 Y型电迷宫观察大鼠学习记忆能力的改变 ,以硝酸还原酶法测定海马 NO含量 ,以放射免疫法测定海马 SS含量 ,并进行学习记忆能力、NO含量和 SS含量相关性分析。
短句来源
    Results NO and SS of hippocampus decreased(P<0.01) and Y maze test times increased(P<0.01) significantly after the microinjection of L NA. The level of NO was positively correlated with that of SS,and the content of NO and SS were negtively correlated with test times of learning and memory respectively.
    结果 海马微量注射 L - NA组大鼠与生理盐水对照组和正常对照组比较 ,海马中 NO、SS含量明显下降 (P<0 .0 1) ,Y型电迷宫测试次数明显增加 (P <0 .0 1) ,L - NA组大鼠海马 NO水平和SS水平呈显著正相关 ,NO、SS水平均与 Y型电迷宫测试次数呈显著负相关。
短句来源
    To establish the model of memory obtain induced by scopolamine and memory strengthen induced by sub sodium nitrite, Y maze test and step down test were used to determine the same indexes.
    建立东莨菪碱记忆获得障碍模型及亚硝酸钠记忆巩固障碍模型 ,采用一次性训练的空间辨别反应实验 -电迷宫实验和一次性学习的回避性条件反射试验 -跳台试验测定小鼠记忆功能 ,观察Hup-ATDDS对其影响。
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  “电迷宫”译为未确定词的双语例句
    Methods: Y-electronic maze was used to study the behavior of dementia rats induced by D-galactose and QA.
    方法:采用D-半乳糖、喹啉酸致痴呆大鼠模型,使用大鼠Y型电迷宫法,研究药物对大鼠学习记忆的影响;
短句来源
    The toxicity of CTX was measured in acute toxicity test. The analgesic effect of CTX in mice was measured by hot plate method and acetic aicd writhing test. The ameliorating effect of CTX on learning and memory impairment induced by scopolamine in mice was studied by Y-Maze, Shuttle-box, Step-down and Water Maze methods respectively.
    通过热板法及醋酸扭体法观察中华眼镜蛇心脏毒素(CTX)对成年昆明种小鼠疼痛的影响,通过Y-型电迷宫及穿梭箱法研究CTX对东莨菪碱诱发成年昆明种小鼠学习记忆障碍的影响,利用跳台法研究CTX对东莨菪碱诱发成年BABL/C小鼠学习记忆障碍的影响,利用Morris水迷宫法研究CTX对对东莨菪碱诱发成年C57BL/6J小鼠学习记忆障碍的影响。
短句来源
    ② Y-maze test: Forty Kunming mice were randomly divided into saline group, ethanol group, ALEC high, mid-dle and low dosage groups with 8 mice in each group.
    ②Y型电迷宫法:取昆明种小鼠40只,随机分为生理盐水组、乙醇组、亚力克高、中和低剂量组5组,每组8只。
短句来源
    Methods: MG-2 type Y-electronic maze and morris water maze were used to study the behaviors of senile model rats and dementia model rats induced by scopolamine and D-galactose.
    方法:采用东莨菪碱、D-半乳糖和老年大鼠三种痴呆动物模型,使用Y型电迷宫和Morris水迷宫法,从整体动物水平研究药物对大鼠的学习记忆的影响;
短句来源
    In the Y-maze test,there were normal saline group,Glu group,GABA group, GABA+Glu group,diazepam+Glu group,piracetam+GABA group,diazepam group and dia zepam+piracetam group. The times og mistakes of mistates and latent period recor ded during lesrning and testing.
    在跳台法中,在上述分组的基础上两个Pir组再各分为低剂量组和高剂量组; 在Y电迷宫法中,小鼠分成生理盐水组,Glu组、GABA组、GABA+Glu组、Dia+Glu组、Pir+GABA、Dia组、Dia+Pir组。
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  y maze
Rats given posttrial injections of pentylenetetrazole and strychnine showed improved learning of a brightness discrimination in a simple Y maze.
      
Rats were trained to shuttle between two selected ("ON") arms of a Y maze, to obtain electrical stimulation of the medial forebrain bundle.
      
Rats were trained in a Y maze on a two-choice simultaneous brightness discrimination with light as S+ and dark as S- (position irrelevant).
      
The mice received, 20 min after training, an IP injection of either diazepam (2 mg/kg) or saline; half of the mice in each treatment group were exposed, 40 min after avoidance training (and 20 min after the injections) to a Y maze.
      
Exposure to the Y maze disrupted retention of the avoidance task in the saline-treated animals, and enhanced it in the diazepam-treated mice.
      
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Dipfluzine {1-diphenyl methyl-4-[4-(p-fluorophenyl)-4-oxobutyl]-piperazine, Dip} is a new derivative of cinnarizine (Cin). The improving effects of Dip on the amnesia induced by sodium nitrite and sodium pentobarbital in mice were studied using a step-down test and an electric maze method respectively. Dip improved sodium nitrite-induced impairment of the retention of memory in the step-down test, which was similar to Cin, and improved sodium pentobarbital-induced impairment of the orientation in the electric...

Dipfluzine {1-diphenyl methyl-4-[4-(p-fluorophenyl)-4-oxobutyl]-piperazine, Dip} is a new derivative of cinnarizine (Cin). The improving effects of Dip on the amnesia induced by sodium nitrite and sodium pentobarbital in mice were studied using a step-down test and an electric maze method respectively. Dip improved sodium nitrite-induced impairment of the retention of memory in the step-down test, which was similar to Cin, and improved sodium pentobarbital-induced impairment of the orientation in the electric maze test in mice significantly. In the doses of 20 and 40 mg·kg-1, both Dip and Cin significantly decreased the error times of mice and the percentage of the animals showing errors (P<0.05 and P<0.01 respectively, compared with the solvent control) in the step-down test, but 10 mg·kg-1 of both drugs did not. In the electric maze test 20 mg ·kg-1 of Dip increased the times of the correct response of mice significantly in d 1 and d 2 of training (P<0.05, compared with the solvent control), but 10 mg·kg-1 of Dip did not. At the effective dose (20 mg·kg-1) of influencing memory, Dip made the duration (32.2± s 6.6 s) of breathing movement with the mouth opened after the decapitation longer than that of the solvent control (20.2± s 3.8 s) significantly (P<0.01), and thus protected mice against the acute cerebral hypoxia.

用跳台法和电迷宫法,观察双苯氟嗪(dipfluzine)对亚硝酸钠和戊巴比妥钠造成的小鼠记忆障碍的改善作用,双苯氟嗪可明显改善亚硝酸钠引起的小鼠记忆障碍和戊巴比妥钠造成的小鼠方向辨别的获得障碍,且有剂量关系,20和40mg·kg~(-1)双苯氟嗪使小鼠跳台错误次数和出现跳台错误动物的百分率均显著低于溶剂对照组.20 mg·kg~(-1)双苯氟嗪使小鼠在训练的d1和d2受到电刺激后的正确逃避反应数均显著高于溶剂对照组(P<0.05).双苯氟嗪改善亚硝酸钠引起的小鼠记忆障碍的作用与桂利嗪(cinnarizine)相似,在改善记忆的有效剂量(20mg·kg~(-1))下,双苯氟嗪还有抗小鼠急性脑缺氧作用,使断头后小鼠张口呼吸动作持续时间(32±s7s)显著长于溶剂对照组(20±s4s,P<0.01).

Objective To determine the threshold of millimeter wave irradiation leading to fetal damage in mice and to clarify the mechanism of decrease in their learning and memory function. Methods Pregnant mice were irradiated by millimeter wave with frequencies of 37.4, 42.2, 53.0 and 60.0 GHz at power densities of 1 to 8 mW/cm 2 for two hours daily from the 6th to 15th day of their gestation. Learning and memory functions of their offsprings were tested by a Y type electric maze. M cholinergic receptor (M R),...

Objective To determine the threshold of millimeter wave irradiation leading to fetal damage in mice and to clarify the mechanism of decrease in their learning and memory function. Methods Pregnant mice were irradiated by millimeter wave with frequencies of 37.4, 42.2, 53.0 and 60.0 GHz at power densities of 1 to 8 mW/cm 2 for two hours daily from the 6th to 15th day of their gestation. Learning and memory functions of their offsprings were tested by a Y type electric maze. M cholinergic receptor (M R), monoamine transmitter and arginine vasopressor (AVP) in the brain were determined for their offsprings by receptor radiation binding analysis (RBA), high performance liquid chromatography electrochemical detection (HPLC ECD) and radioimmunoassay (RIA). Results The minimal power density of millimeter wave of 37.4, 42.2, and 53.0, 60.0 GHz causing the decrease in learning and memory function, increase of Bmax in the hippocampus and decrease in dopamine (DA) in the brain of the adult offspring mice was five and three mW/cm 2 and that causing the decrease in AVP in the hypothalamus was eight and five mW/cm 2. Severity of damage in fetal offspring mice caused by irradiation increased with the power density of millimeter wave. Millimeter wave could not cause the increase of their body temperature in pregnant mice. Conclusion Threshold of millimeter wave with frequencies of 37.4, 42.2 and 53.0, 60.0 GHz causing fetal damage in mice was three to five mW/cm 2 and one to three mW/cm 2, respectively. The decrease in learning and memory functions in offspring mice related with lowering of AVP and DA in the brain and of activity in cholinergic nerve. All these were caused by the non thermal effects of millimeter wave without a frequency specific pattern.

目的确定毫米波导致胚胎损伤阈值及仔鼠学习记忆功能降低的机理。方法用374、42.2、53.0和60.0GHz,功率密度1~8mW/cm2毫米波,在小鼠怀孕6~15天时每天进行2小时辐射,用电迷宫对仔鼠进行学习记忆功能测试,用受体放射配基分析、高效液相色谱电化学检测和放射免疫法对仔鼠脑M胆碱能受体、单胺递质和精氨酸加压素(AVP)进行测定。结果37.4、42.2GHz和53.0、60.0GHz毫米波导致仔鼠学习记忆功能降低、海马区最大受体结合数增加、脑多巴胺(DA)下降的最小功率密度分别为5和3mW/cm2,导致成年子鼠下丘脑内AVP降低的最小功率密度分别为8和5mW/cm2。随功率密度增加,辐射导致胎仔损伤程度加重。毫米波未使孕鼠体温升高。结论37.4、42.2GHz和53.0、60.0GHz毫米波导致胚胎损伤阈值分别为3~5mW/cm2和1~3mW/cm2,其导致仔鼠学习记忆功能降低与其脑中AVP、DA含量下降及海马区胆碱能神经活性降低相关,此效应由毫米波非热效应所致,未见毫米波的频率特异性

Objective To study the action and interaction of Nitric Oxide(NO) and somatostatin(SS) in the process of learning and memory. Methods Nitric acid redactase and radioimmunity method are taken to detect the content of NO and SS in rat hippocampus after N ω nitro L arginine (L NA),a nitric oxide synthase (NOS) inhibitor,microinjected into hippocampus,and Y maze is used to observe the changes of capability of learning and memory,and a correlative analysis is made. Results NO and SS of hippocampus decreased(P<0.01)...

Objective To study the action and interaction of Nitric Oxide(NO) and somatostatin(SS) in the process of learning and memory. Methods Nitric acid redactase and radioimmunity method are taken to detect the content of NO and SS in rat hippocampus after N ω nitro L arginine (L NA),a nitric oxide synthase (NOS) inhibitor,microinjected into hippocampus,and Y maze is used to observe the changes of capability of learning and memory,and a correlative analysis is made. Results NO and SS of hippocampus decreased(P<0.01) and Y maze test times increased(P<0.01) significantly after the microinjection of L NA. The level of NO was positively correlated with that of SS,and the content of NO and SS were negtively correlated with test times of learning and memory respectively. Conclusion A normal level of NO and SS in hippocampus is foundermental for normal function of learning and memory. NO regulates the level of SS to promote the process of learning and memory.

目的 探讨一氧化氮 (NO)和生长抑素 (SS)在学习记忆过程中的作用及相互作用。方法 大鼠海马微量注射一氧化氮合酶 (NOS)抑制剂 N-ω-硝基 - L -精氨酸 (N -ω- Nitro- L - Arginine,L - NA )后采用 Y型电迷宫观察大鼠学习记忆能力的改变 ,以硝酸还原酶法测定海马 NO含量 ,以放射免疫法测定海马 SS含量 ,并进行学习记忆能力、NO含量和 SS含量相关性分析。结果 海马微量注射 L - NA组大鼠与生理盐水对照组和正常对照组比较 ,海马中 NO、SS含量明显下降 (P<0 .0 1) ,Y型电迷宫测试次数明显增加 (P <0 .0 1) ,L - NA组大鼠海马 NO水平和SS水平呈显著正相关 ,NO、SS水平均与 Y型电迷宫测试次数呈显著负相关。结论 海马中 NO、SS水平的正常是机体实现正常学习和记忆过程的重要因素 ;NO可能通过调节 SS的合成和释放 ,共同促进学习和记忆过程

 
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