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ns对照组
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  ns group
     1.5 hours following bacterial injection the peak levels of serum TNF α in the BPI group were also markedly lower than that in the NS group (P<0.01).
     注菌后1.5h,BPI组血清TNF_α峰值也明显低于NS对照组(P<0.01);
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     There was definite difference in TNF content between ET and ET + GLY group(P < 0. 01 ), while no clear difference between GIY and NS group(P > 0 .05 ).
     ET组TNF含量与ET+Gly组TNF含量比较也有非常显著性差异(P<0.01),而单纯Gly组与NS对照组比较无显著性差异(P>0.05)。
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     Results:Tumor volumes of mice were significantly smaller in xVEGF group than in mVEGF or NS group(P<005),and survival rate of mice was also significantly higher in xVEGF group than in mVEGF or NS group(P<001).
     结果:xVEGF组肿瘤明显小于mVEGF组或NS对照组(P<0.05),xVEGF组小鼠生存率也明显高于mVEGF组或NS对照组(P<0.01)。
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     Methods:Twenty-seven SD rats were divided into 3 groups randomly,the NS group was instilled saline into trachea for 12 h; and the lung sample were made. The LPS1group and LPS2 group were instilled LPS 1.0 ml/kg(100 μg/ ml)for 12 h and 24 h respectively,and the lung sample were made.
     方法:将27只SD大鼠随机分成3组,其中NS对照组为气管内滴注生理盐水12 h后取材,LPS1组、LPS2组为气管内滴注LPS 1.0 ml/kg(浓度为100μg/ml)后分别于12 h,24 h取材。
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     Results:To compare NS group, there was a significant difference(P<0.001 ) among groups of As_2O_3 and TNP-470, which inhibited the angiogenesis of CAM. There was an obvious difference(P<0.05) between the combination drug group and single drug groups.
     结果与NS对照组比较,As2O3和TNP-470均有显著抑制CAM血管生成作用(P<0·001),且联合组的抑制作用强于单药组(P<0·05)。
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  ns control group
     the content of glutamic acid was (12.3±1.2), (12.0±0.8) , (10.2±0.6), and (11.8 ±1.0) μmol/g in NS control group, DMPS control group, TETS control group, and DMPS protection group, respectively.
     在NS对照组、DMPS对照组、TETS对照组和DMPS保护组中,小鼠脑内游离的谷氨酸(Glu)含量分别是(12.3±1.2),(12.0±0.8),(10.2±0.6)和(11.8±1.0)μmol/g。
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     RESULTS:Compared to NS control group,each composition of Lamiophlomis rotata Kudo can shorten the TT by 18.59%、—3.12%、24.11%and 9.92%respectively,and increase the content of FIB by 25.32%、8.67%、28.29%and 5.36%respectively. There was obvious change in HLRE and P2(P<0.05).
     结果与NS对照组比较,独一味各类成分给药后大鼠TT分别缩短18.59%、—3.12%、24.11%和9.92%,FIB含量分别增加25.32%、8.67%、28.29%和5.36%,HLRE及P2组有显著性差异(P<0.05);
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     RESULTS:Compared with the NS control group,TFP group showed a decreased scoring in pain reaction(15min later),reduced writhing response(50,100mg/kg),prolonged latent phases of foot licking(50,100mg/kg)and tail-shrinking(50mg/kg),significant decrease of the content of malonaldehyde,prostaglandin-2and NO in mice'blood serum and brain tissue.
     结果:与NS对照组比较,TFP组用药后疼痛评分降低(15min后),扭体反应数减少(50、100mg/kg),舔足潜伏期延长(50、100mg/kg),缩尾反应潜伏期延长(50mg/kg),小鼠血清和脑组织中丙二醛、前列腺素-2合成及一氧化氮含量明显降低。
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     After 10 weeks immunization, CD 8 + T cells of immunized group and pBK CMV control were still increased compared with NS control group( P <0.01), but there was no remarkable difference between them( P >0.05).
     免疫后 10周 ,与 NS对照组相比较 ,免疫组和 p BK- CMV对照组 CD8+细胞仍升高 (P<0 .0 1) ,但它们之间的差异无显著性 (P>0 .0 5 )。
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     ②Compared with NS control group,the SOD activities in serum and brain tissue of rat models in HSFZC group were highly increased and the content of MDA decreased after treatment for 7 days (P<0.01),but only an increased SOD activity in brain tissue was showed in the NFK group (P<0.05) and was markedly lower than that in HSFZC group (P<0.05).
     2与 NS对照组比较 ,HSFZC组痴呆模型大鼠治疗 7d后 ,血清及脑组织 SOD活性升高 ,MDA含量降低 (P<0 .0 1) ; 但 NFK组仅显示脑组织SOD活性升高 (P<0 .0 1) ,并且其活性明显低于 HSFZC组。
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  “ns对照组”译为未确定词的双语例句
     Results The survival time of the glycine-treated group was significantly longer than that of the control group [(159.25 ±22.78) min vs (138.75 ± 19.05) min] ( P < 0.01).
     结果甘氨酸组的存活时间(159.25±22.78)min长于NS对照组(138.75±19.05)min,差异有显著性(P<0.01)。
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     The ulcer index in the escipient group is not different from the control group(34.22?.24 vs 36.49?.49 P>0.05).
     赋形剂组的溃疡指数为34.22±2.24,较之NS对照组(36.49±4.49)差异无显著性(P>0.05)。
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     RESULTS: After icv and ip DMPS, the number of mice experiencing convulsions reduced from 20 in control group to 4 and 2 respectively in TETS poisoned mice.
     结果:与NS对照组比较,icv和ip DMPS后小鼠的惊厥数从20只下降至4只和2只;
短句来源
     Method: Sixty healthy age from 25 to 35 days rats were randomly divided into 4 groups(n=15): normal sodium(NS) treated as control group(A),KA treated as model group(B),TPM treated as group C,combination of NGF and TPM handled as group D.
     方法:60只日龄25—35天健康大鼠随机分成4组(n=15只):A组(正常NS对照组),B组(KA致痫模型对照组),C组(TPM治疗组),D组(TPM和NGF联合治疗组)。
短句来源
     The cytotoxic rates of the effector cells on the target cells in group of mice immunized with BCG,pTB30s and the control group of mice with injection of saline were 28.14%,45.18% and 5.13% respectively.
     CTL杀伤实验:BCG、pTB30s及NS对照组杀伤率分别为28.14%、45.18%和5.13%。
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  ns group
The rats were randomly divided into Sham group, HTS group (hypertonic saline resuscitation group) and NS group (normal saline resuscitation group).
      
EAMG mice were divided randomly into 5 groups: MG group, NS group and three antibiotics groups.
      
The positive rates of RNS of three antibiotics groups were 69.23%, 58.82% and 63.16% respectively, which were significantly higher than those of MG group and NS group (40.00%, 40.00%,P>amp;lt;0.05).
      
MVD was significantly lower in cFR-1-immunized group than in mFR-1-immunized group and NS group (16.8±5.6 vs 64.6±1.8 and 59.6±8.7, P>amp;lt;0.01).
      
Comparing the response of the E-ARM between groups, the increase in [La]b of the NS group [Δ: 1.40 (0.4)?mmol/l] was not different than that observed in the ES group [Δ: 2.10 (0.7)?mmol/l; P>amp;gt;0.05).
      
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  ns control group
The mortality was decreased from 33.3% in the NS control group to zero by BQ610, although there was no statistical significance.
      


Four gut hormones, somatostatin ( SS ) , neurotensin ( NT ) , glucagon ( GC ) and TRH have been used to observe their effects on the hyperglycemia induced by streptoZotocin ( STZ ) . The changes of serum glucose concentration were assayed by the method of glucose-oxidase. The results were as follows. ( 1 ) Injection of SS or NT 10 minutes prior to STZ could reduce the hyperglycemic level induced by STZ in varying degrees on the day 6th, 8 th 10th, 15th of experiment and showed a dose-related response. However,...

Four gut hormones, somatostatin ( SS ) , neurotensin ( NT ) , glucagon ( GC ) and TRH have been used to observe their effects on the hyperglycemia induced by streptoZotocin ( STZ ) . The changes of serum glucose concentration were assayed by the method of glucose-oxidase. The results were as follows. ( 1 ) Injection of SS or NT 10 minutes prior to STZ could reduce the hyperglycemic level induced by STZ in varying degrees on the day 6th, 8 th 10th, 15th of experiment and showed a dose-related response. However, no change of hyperglycemia induced by STZ has been found after preventive injection of GC or TRH. ( 2 ) There was no reduction of serum glucose concentration, after therapeutic injections of SS or NT into the hyperglycemic (serum glucose 350 mg% ) mice 15 days after STZ treatment. ( 3 ) The concentration of serum glucose was kept at normal levels on normal animals after subcutaneous injections of NS, SS, NT, GC or TRH. These results demonstrate that the injection of SS or NT can prevent the production of hy-perglycemiait induced by STZ, but they do not have therapeutic effect when the hyperglycemia h-as been produced. This suggests that both of these two gut hormones might play a role in the cytoprotection of islet B cells.

本实验用腹腔注射链佐霉素(Streptozotocin简称STZ)方法破坏小鼠胰岛B细胞以诱发高血糖,观察生长抑素(Somatostatin,SS)、神经降压素(neurotensin,NT)、胰高血糖素(glucagon,GC)和促甲状腺素释放激素(TRH)4种胃肠激素对小鼠高血糖的影响。在每天腹腔注射STZ(60mg/kg)前10分钟分别经皮下注射上述4种胃肠激素,连续注射5天,在实验的第1,6,8,10,15天取血测血清葡萄糖浓度,对照组注射生理盐水(NS)。结果发现:(1)预先注射SS和NT可不同程度地抑制由STZ引起的实验性高血糖,并呈剂量一效应关系,(2)预先注射GC和TRH,血糖浓度仍明显升高,与NS对照组比较无显著差异,(3)取注射STZ后第15天的高血糖小鼠(血糖高于350mg%者)分为8组,分别以SS和NT作治疗性注射,每天一次共5日,并未见对小鼠高血糖有缓解效果;(4)正常小鼠单独皮下注射NS、SS、NT、GC、和TRH,每天一次连续5天,在注射后15天内未见对血糖水平有明显影响。以上结果提示:预防性注射SS和NT可显著抑制由STZ诱发的高血糖的产生。

42 rats were divided into 7 groups(6 rats per each group) and six group of them were treated with different dosages of 1/10 and 1/60 of LD50 (mice i. p.)of two different dosages of ACMA(3, 0.5 mg/kg),ACMB and ADM(1.3, 0.217mg/kg) respectively. The rest one group was treated with normal saline as a control group by daily i. p. injection for 7 days consecutively.

大鼠42只按体重均分 7组,每组 6只,分别给以ACMA、ACMB和 ADM小鼠急性LD50的1/10及1/60剂量,另一组为生理盐水对照组。连续腹腔给药 7天后,检查大鼠的外周血象和骨髓有核细胞的变化。 1/10 LD50剂量组的ACMA和ADM外周血液内白细胞数显著下降,且ACMA组中性分叶核白细胞百分率显著降低。两剂量组的骨髓有核细胞计数明显的减少,其中 ACMA 3mg/kg组的骨髓增生程度为 2例低下、4例极度低下,粒系细胞的抑制尤为显著。而等毒剂量的ACMB组对大鼠外周血象与骨髓有核细胞与NS对照组比较均无显著差异。 因此,虽然ACMB对小鼠的急性毒性比ACMA大一倍多,与ADM相当,但以等毒剂量比较它们对大鼠骨髓造血功能的影响则以ACMB为最轻。

The present study was attempted to observe the protective effects of tungsten(W) on the lung injury induced by endotoxin (ET) in rats and to analyze its' mechanisms. 29 male Wistar rats were randomly divided into three groups: (1) ET group(n = 11), given a bolus injection of disintegrated E. coli (5.4×1010 organisms/kg); (2) ET + W group (n=12) , given an equivalent E. coli after being pretreated with Sod. Tungstate rich food and water for 4-5 weeks and (3) Control group (N.S,n = 6). Theresults showed that the...

The present study was attempted to observe the protective effects of tungsten(W) on the lung injury induced by endotoxin (ET) in rats and to analyze its' mechanisms. 29 male Wistar rats were randomly divided into three groups: (1) ET group(n = 11), given a bolus injection of disintegrated E. coli (5.4×1010 organisms/kg); (2) ET + W group (n=12) , given an equivalent E. coli after being pretreated with Sod. Tungstate rich food and water for 4-5 weeks and (3) Control group (N.S,n = 6). Theresults showed that the following parameters in ET + W group had significant difference at 120 minutes after injection of ET as compared with ET group (P<0.05 or <0.01) , BP (78.2±6.4 vs 31.8±10.0mm Hg) , Total count of WBC (114.8±9.2%) vs 41.6±4.8%) ; PMN% (131.9±6.1 vs 41.6±4.7) ; in the lung tissue, the level of MDA (7.9±1.4 vs 10.4±2.4nMol/mg protein) ; activity of XOD (11.3±7.0 vs 167.0±14.2u/mg protein) ; D W/W W (0.22±0.02 vs 0.15±0.01) ; the ratio of W/Mo (24.89±2.51 vs 0.48±0.02) and survival rate (100% vs 54.5%). Pathological examination also revealed less morpholog ical changes and less infiltration of WBCs in the lung in ET + W group than that in ET group. The results suggested that W could piotect ET induced lung injury in rats.

钨酸钠对iv灭活大肠杆菌所致大白鼠内毒素休克时的肺损伤有明显保护作用,表现在喂钨组血压、外周血白细胞总数与中性粒细胞百分数无明显下降,血浆丙二醛(MDA)无明显升高。肺组织中,MDA含量以及黄嘌呤氧化酶(XOD)活性均比不喂钨组低;肺组织干/湿重比值和钨/钼(W/Mo)比值比不喂钨组高。存活率亦增高。NS对照组各项指标无明显变化。其机制可能与钨抑制XOD,使氧自由基(OFRs)生成减少有关。本实验还提示整体条件下,(即PMN和XOD均存在时),内毒素休克所致肺损伤时,XOD是氧自由基产生的始动因素。

 
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