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氧化应激状态
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  oxidative stress state
     The Effect of Qinghua Xiaoyu on Oxidative Stress State in Type 2 Diabetics
     2型糖尿病合并高脂血症患者氧化应激状态及清化消瘀方干预作用研究
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     The Oxidative Stress State of Mice After Air Simulated Diving
     空气模拟潜水后小鼠的氧化应激状态研究
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     Objective To study the oxidative stress state of mice before and after air simulated diving.
     目的探讨空气模拟潜水前后小鼠氧化应激状态的变化。
短句来源
     Objective To observe the effect of Qinghua Xiaoyu (QHXY) on oxidative stress state in type 2 diabetics.
     目的观察清化消瘀方对2型糖尿病合并高脂血症患者氧化应激状态的影响。
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     Conclusion The oxidative stress state of mice changed obviously in simulated air diving environment and hyperbaric oxygen may play an important role in it.
     结论在空气模拟潜水条件下小鼠氧化应激状态发生显著变化,高分压氧在其中扮演了极为重要的角色。
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  oxidative stress status
     Conclusion These findings suggest that NADPH oxidase subunit gp91 phox homologue nox4 gene over-expression is correlated with the oxidative stress status of SHR.
     结论结果提示SHR体内氧化应激状态与NADPH氧化酶gp91phox类似物nox4mRNA过表达有关;
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     Objective: To investigate the effects of levocarnitine on nutrition,microinflammation and oxidative stress status in maintenance hemodialysis(MHD) patients.
     目的:通过观察静脉注射左卡尼汀对维持性血液透析(maintenance hemodialysis,MHD)患者营养、微炎症和氧化应激指标的影响,探讨左卡尼汀对MHD患者微炎症及氧化应激状态的治疗作用。
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     Objective:To investigate the oxidative stress status in patients with primary nephrotic syndrome(PNS) and the antioxidative effects of Astragalus.
     目的 :探讨原发性肾病综合征 (PNS)患者氧化应激状态以及中药黄芪的抗氧化作用。
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     Analysis of oxidative stress status of pregnant women with premature rupture of membranes
     胎膜早破孕妇血清氧化应激状态分析
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     Conclusions Rat aging might be related with the alteration of oxidative stress status, energy production and protein metabolism.
     结论 小鼠衰老可能与机体氧化应激状态、能量产生和蛋白质代谢等的改变有关。
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  “氧化应激状态”译为未确定词的双语例句
     Objective To determine the changes of advanced oxidation protein products(AOPP) in diabetic nephropathy(DN) patients,as well as its relationship with superoxide dismutase(SOD),glutathione peroxidase(GPx) and neopterin(NPT).
     目的:研究糖尿病肾病患者血清蛋白氧化损伤指标晚期蛋白氧化产物(AOPP)的改变及其与超氧化物歧化酶(SOD)、谷胱甘肽过氧化物酶(GPx)及新喋呤(NPT)的关系,探讨蛋白氧化损伤与糖尿病肾病氧化应激状态和慢性炎症状态的关系。
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     The cells of the third generation are exposed to the medium containing low carbohydrates,0.1mmol/L H_2O_2,10%FBS for 24 hours.
     取第三代细胞,用含0.1mmol几H_2O_2,10%胎牛血清的低糖DMEM培养基处理24h后造成氧化应激状态
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     Oxidative stress can be induced when H2O2 was utilized to stimulate C2C12 at different level, under this condition , F-GC could relieve oxidative damage to some extent and promote glucose uptake, F-GC also could promote proliferation and differentiation of C2C12 . oxidative stress could slow the flowability of C2C12 membrane so that it couldn’t function normally.
     当实验采用不同浓度的H2O2氧化刺激C2C12细胞,使其处于不同程度的氧化应激状态时,发现各剂量组的F-GC在不同程度上可以缓解H2O2诱导的氧化应激对C2C12细胞的氧化损伤,增加糖消耗,并促进C2C12细胞的增殖与分化,增加细胞的活力;
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     Results SF inhibited collagen synthesis in hepatic stellate cells stimulated with TGFβ 1. SF was shown to decrease TGFβ 1 level in the supernatant of HSC-T6 increased by oxidative stress.
     结果 阿魏酸钠对TGFβ1 处理条件下HSC T6细胞中胶原的合成有抑制作用 ; 能够降低HSC T6细胞在氧化应激状态下TGFβ1 的表达水平 ;
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     Conclusion DN patient has enhanced protein oxidation than DM patient,which is related to oxidative stress and chronic inflammation in DN.
     结论:糖尿病肾病患者的血清蛋白氧化较无糖尿病肾病患者增强,并且与糖尿病肾病氧化应激状态和慢性炎症状态有关。
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  oxidative stress state
These changes seemed to be related and might point to an oxidative stress state that exacerbated with prolonged time and/or increased isoproturon dose.
      
Such a model can also account for differences in aging rate or longevity of different animal species by differences that may exist in their innate ability to reduce oxidative stress state.
      
Our laboratory has been testing this oxidative stress state (OSS) hypothesis of aging and longevity by determining if a positive correlation exists between OSS of an animal and its aging rate.
      
  oxidative stress status
However, these studies in which the oxidative stress status were investigated have a small number of patients and they are heterogeneous.
      
Functions and oxidative stress status of leukocytes in patients with nephrotic syndrome
      
This study was conducted to establish the functions and oxidative stress status in leukocytes of adult patients with nephrotic syndrome.
      
High oxidative stress status is known to be one of the most important factors determining cell injury in thalassemic patients and causing other serious medical complications, including a continuous proinflammatory status.
      
Our investigation showed that apart from oxidative burst caused by accumulation of inflammatory cells, hapten properties might also influence the oxidative stress status of the skin.
      
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Occupational investigation in the plant operating trinitrotoluene(TNT) in Henan province showed that the TNT concentrations in workplace air mostly exceeded the national maximum allowable concentration(MAC=1mg/m 3)and the skin of the TNT exposed workers appeared to be severely contaminated The ferro concentration of serum in both TNT exposed group and HBsAg positive group were significantly higher than those of the control group( P <0 0005) The ferro...

Occupational investigation in the plant operating trinitrotoluene(TNT) in Henan province showed that the TNT concentrations in workplace air mostly exceeded the national maximum allowable concentration(MAC=1mg/m 3)and the skin of the TNT exposed workers appeared to be severely contaminated The ferro concentration of serum in both TNT exposed group and HBsAg positive group were significantly higher than those of the control group( P <0 0005) The ferro concentration of serum in HBsAg postitve group was also significantly higher than those of TNT group ( P <0 0025) It seemed that the mechanism of liver damage induced by TNT and HBV would be partly associated with oxidative stress of the body

对河南省某化工厂三硝基甲苯(TNT)作业现场劳动卫生学调查结果发现,车间空气中TNT浓度多数超过国家最高容许浓度(MAC=1mg/m3),TNT作业工人皮肤污染严重。血清铁测定结果表明,TNT接触组与HBsAg阳性组极显著高于对照组(P<00005),HBsAg阳性组显著高于TNT接触组(P<00025)。推测TNT与HBV诱发的肝损害部分机理可能与机体处于氧化应激状态有关。

Objective To study the influence of oxidative stress on the production of nitric oxide in cultured neurons and the protective effects of heat shock on the process.Methods The cultured rat cortical neurons were divided into 5 groups: control, hypoxia, H 2O 2 , heat shock +hypoxia , and heat shock +H 2O 2 .The parameters malondialdehyde (MDA),superoxide dismutase (SOD),nitric oxide (NO) and lactate dehydrogenase (LDH) were determined and analysed.Results After the insult with hypoxia or H 2O 2 ,the contents...

Objective To study the influence of oxidative stress on the production of nitric oxide in cultured neurons and the protective effects of heat shock on the process.Methods The cultured rat cortical neurons were divided into 5 groups: control, hypoxia, H 2O 2 , heat shock +hypoxia , and heat shock +H 2O 2 .The parameters malondialdehyde (MDA),superoxide dismutase (SOD),nitric oxide (NO) and lactate dehydrogenase (LDH) were determined and analysed.Results After the insult with hypoxia or H 2O 2 ,the contents of MDA and NO in cultured neurons increased significantly (P<0.01) ,the activity of SOD decreased (P<0.01) ,and they were accompanied by an increase in the release of LDH (P<0.01) .When the cultures were pretreated by heat shock before the onset of hypoxia or H 2O 2 insult ,the production of MDA ,NO and LDH decreased markedly (P<0.01).Conclusion Excessive production of NO may play an important role in the oxidative stress injury of neurons .Heat shock seems to provide protection for neurons against oxidative stress .The mechanism may be related to the inhibition of excessive release of NO from the neuron.

目的 揭示脑在氧化应激状态下一氧化氮 (NO)产生增多对神经细胞的毒性作用和热休克减少NO的产生对神经细胞的保护作用机制。方法 对培养大鼠脑皮质神经元分别进行缺氧、H2 O2 、热休克 +缺氧和热休克+H2 O2 处理 ;检测丙二醛 (MDA)、超氧化物歧化酶 (SOD)、NO和乳酸脱氢酶 (LDH) ,并分析它们的变化和相互关系。结果 缺氧组和H2 O2 处理组MDA和NO产生明显增加 ,SOD活性降低 ,反映神经细胞受损指标LDH也明显增加 (P 均 <0 .0 1) ,对神经细胞在进行缺氧和H2 O2 作用前先进行热休克处理能明显降低NO含量 ,分别与缺氧组和H2 O2 组比较 ,差异非常显著 (P <0 .0 1) ,神经细胞受损程度也减轻。结论 在氧化应激条件下神经细胞NO产生增加并引起毒性作用 ,热休克能减少氧化应激时神经细胞NO的产生。

Aim: In order to elucidate the mechanism of cardiomyocyte injury in heat stress, the changes of oxidative phosphorylation and calcium metalolism in cardiac mitochondria of heatexposed Wistar rats were observed. The changes of mitochondrial permeability transition and its pathological significance were studied as well. Methods: The respiratory function of mitochondria was measured by Klark oxgenelectrode polarography; Myocardial ATP content and activity of Ca 2+ ATPase in mitochondria were analyzed with bioluminescent...

Aim: In order to elucidate the mechanism of cardiomyocyte injury in heat stress, the changes of oxidative phosphorylation and calcium metalolism in cardiac mitochondria of heatexposed Wistar rats were observed. The changes of mitochondrial permeability transition and its pathological significance were studied as well. Methods: The respiratory function of mitochondria was measured by Klark oxgenelectrode polarography; Myocardial ATP content and activity of Ca 2+ ATPase in mitochondria were analyzed with bioluminescent method; Mitochondrial Ca 2+ content was assayed by ICP; PT was measured using spectrophotometer. Results: The respiratory control rate (RCR) and oxidative phosphorylation efficiency (P/O) were decreased gradually as rectal temperature (Tr) increased. The activity of Ca 2+ ATPase and Ca 2+ content were also reduced. Exposing to the Ca 2+ overload and oxidative stress, mitochondrial PT altered markedly. Ruthenium red and SOD could prevent the changes of mitochondrial PT. Conclusion: Destruction of mitochondrial structure and function may be of great significance in the development of impaired cardiac function in the heat stress.

目的 :观察热应激对大鼠心肌细胞线粒体氧化磷酸化和钙代谢功能的影响、研究线粒体膜渗透性转换 (PT)的变化及其病理学意义、探索热应激心肌细胞损伤发生机制。方法 :用Klark氧电极极谱法测定线粒体呼吸功能 ,用生物发光法测定心肌ATP含量及线粒体Ca2 +。ATP酶活性 ;用电感耦合等离子 原子发射光谱仪测定线粒体内Ca2 +含量 ,用分光光度法测定线粒体膜PT。结果 :热应激大鼠心肌细胞线粒体的呼吸控制率 (RCR)及氧化磷酸化效率 (P/O)均随热应激强度的增强呈显著逐步降低趋势 ;心肌线粒体Ca2 + ATP酶活性和Ca2 +含量亦明显下降 ;暴露于Ca2 +超载和氧化应激状态的线粒体 ,膜PT发生明显变化 ,钌红、抗氧化剂及反应体系酸化状态的纠正能够有效阻抑PT变化。结论 :线粒体结构与功能的破坏在热应激机体心功能损伤的发生中有着极其重要的意义。

 
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