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冷缺血损伤
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  cold ischemia injury
     Conclusion Cold ischemia injury can induce pancreatic cell apoptosis.
     结论 胰腺冷缺血损伤可引起胰腺腺泡细胞凋亡。
短句来源
     Role of activation of hepatic stellate cells in grafts cold ischemia injury in rat liver transplantation model
     肝星状细胞的激活在大鼠供肝冷缺血损伤中的作用
短句来源
     AIM: [JP4][HJ*4]To investigate the activation of hepatic stellate cells in rat liver grafts after cold ischemia injury.
     目的:在大鼠全肝移植模型的基础上,检测大鼠全肝移植中,肝星状细胞的激活在供肝冷缺血损伤中所起的作用.
短句来源
     CONCLUSION: Significant activation of hepatic stellate cells might play an important role in graft cold ischemia injury in rat liver transplantation model.
     结论:在大鼠全肝移植中,肝星状细胞的激活与供肝冷缺血损伤密切相关.
短句来源
  cold ischemic injury
     Effect of pentoxifylline pretreatment on donor liver cold ischemic injury in the rats
     己酮可可碱预处理对大鼠供肝冷缺血损伤影响的实验研究
短句来源
  prolonged cold ischemia
     Conclusion The transplantation of abdominal aorta grafts used SD/Wistar as donors and recipients with prolonged cold ischemia time is sufficient to promote the development of transplant arteriosclerosis.
     结论 以SD/Wistar作为供 /受体行腹主动脉移植 ,强化冷缺血损伤 ,可加快移植物动脉硬化 ,可望成为新型慢排模型。
短句来源
  “冷缺血损伤”译为未确定词的双语例句
     [Objective] To explore the effect of different cold ischemia times on the patterns of intragraft cytokines of tumor necrosis factor-α(TNF-α) and interleukin-6(IL-6) and their correlation with liver regeneration after rat liver transplantation(LT).
     【目的】研究不同冷缺血损伤条件下,肿瘤坏死因子(TNF-α)和白介素-6(IL-6)等细胞因子在大鼠移植肝内的变化规律与肝脏再生的关系。
短句来源
     Pattern of Intragraft Cytokines after Cold Ischemia Reperfusion Following Rat Liver Transplantation
     冷缺血损伤后大鼠移植肝内细胞因子的变化
短句来源
     [Results] LT was successfully performed in experimental groups. Survival in all groups was 100%(>14 d).
     【结果】冷缺血损伤1h、16h肝移植组和假手术组存活率均为100%(>14d)。
短句来源
     [Conclusion] Severe cold ischemia initiated liver regeneration and graft recovery after rat LT.
     【结论】重度冷缺血损伤启动大鼠肝移植后的肝脏再生,修复组织损伤。
短句来源
     Objective To investigate the effect of microtubule in heart hypothermal preservation.
     目的研究心肌细胞微管在心肌低温冷缺血损伤中的作用。
短句来源
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  cold ischemia injury
The data reported here confirmed that steatotic livers were more susceptible to cold ischemia injury than nonsteatotic livers.
      
  prolonged cold ischemia
Effects of cytomegalovirus infection and prolonged cold ischemia on chronic rejection of rat renal allografts
      
Previous studies have demonstrated that both cytomegalovirus (CMV) infection and prolonged cold ischemia of the allograft (CI) are associated with chronic rejection of renal transplants.
      
Deletrious effect of prolonged cold ischemia on renal function
      
The detrimental effect of prolonged cold ischemia (CI) on posttransplant renal function has long been recognized.
      
As a control, one group of animals was transplanted with aortic isografts exposed to prolonged cold ischemia.
      


Using isolated perfusion rat liver model(IPRL),we studied the protective effects of drugs Hand X on rat’s liver cold ischemic injurv. After therat's liver was preserved for 12 hours in Collins Ⅱ so-lution with the addition of drugs H and X,the necroticrate of sinusoidal lining cells was significantly lowerthan that of control group.Within certain range of thedosage of drugs H and X, the necrotic rate of sinu-soidal lining cells was in the inverse ratio of the dosagein the Collins Ⅱ solution. We also found that...

Using isolated perfusion rat liver model(IPRL),we studied the protective effects of drugs Hand X on rat’s liver cold ischemic injurv. After therat's liver was preserved for 12 hours in Collins Ⅱ so-lution with the addition of drugs H and X,the necroticrate of sinusoidal lining cells was significantly lowerthan that of control group.Within certain range of thedosage of drugs H and X, the necrotic rate of sinu-soidal lining cells was in the inverse ratio of the dosagein the Collins Ⅱ solution. We also found that drugs Hand X had obvious protective efect on liver cell redoxfunction,But these drugs could not inhibit tissue fromswelling after Collins Ⅱ solution preservation. Theprotective mechanisms of the drugs maybe relate to theremoral of free radicals and suppression of Kupffer’scells activity.

采用大鼠肝脏离体灌注模型观察药物H、X对大鼠肝脏冷缺血损伤的保护作用。发现鼠肝经加入药物H、X的CollinsⅡ液低温保存12小时后,其肝窦内皮细胞死亡率明显低于对照组 ,且肝窦内皮细胞死亡率与CollinsⅡ液内药物H、X剂量在一定范围内呈负相关关系,药物H、X对肝细胞氧化磷酸化功能具有一定保护作用,但对CollinsⅡ液保存肝脏过程中产生的组织水肿无抑制作用。作者认为药物H、X保护鼠肝免受冷缺血损伤的作用机理可能与清除氧自由基、抑制Kupffer细胞活化有关。

Objective To investigate the efficacy of donor and liver pretreatment with pentoxifylline (PTX) simultaneously on donor liver graft cold ischemic injury. Methods Sixty four Wistar rats were divided into two groups (donors and recipients). Then the donors were divided four groups at random: experimental group 1 (E1),experimental group 2 (E2),experimental group 3 (E3) and control group (C). The donors in the group E1 were injected intraperitonealy with PTX 50?mg/kg body weight before 1?h ...

Objective To investigate the efficacy of donor and liver pretreatment with pentoxifylline (PTX) simultaneously on donor liver graft cold ischemic injury. Methods Sixty four Wistar rats were divided into two groups (donors and recipients). Then the donors were divided four groups at random: experimental group 1 (E1),experimental group 2 (E2),experimental group 3 (E3) and control group (C). The donors in the group E1 were injected intraperitonealy with PTX 50?mg/kg body weight before 1?h of laparotomy. In the group E2,livers were flushed with 10?ml Euro Collins solution containing 25?mg/kg PTX body weight.The donors in the group E3 were pretreated with methods of group E1 plus group E2. After livers were transplanted after preserved in 4?℃ Euro Collins solution for 6?h . Results At 30?min and 3?h ,the levels of TNF α in the experimental groups were significantly lower than in the control group (RIA),and the levels in the group E3 were significantly lower than those in the group E1,E2 ( P < 0.05 ).At 30?min ,GST levels in the experimental groups were significantly lower than those in the control group,and the levels in the group E3 were significantly lower than in the group E1 and E2 ( P < 0.05 ).At 3?h ,the levels in the experimental groups were significantly lower than those in the control group,and the levels in the group E3 were significantly lower than those in the group E2 ( P < 0.05 ).At 30?min and 3?h ,the levels of ALT in the experimental groups were significantly lower than those in the control group,levels of ALT in the group E3 were significantly lower than those in the groups E1 and E2 ( P < 0.05 ).At 24?h ,levels of ALT in the group E3 were significantly lower than those in the control group and group E2 ( P < 0.05 );Levels of AST in the experimental groups were significantly lower than those in the control group,levels in the group E3 were significantly lower than those in the groups E1 and E2 at 30?min ( P < 0.05 ).Conclusion PTX pretreatment can reduce liver injury through suppressing Kupffer cells activation and TNF α production.

目的 探讨已酮可可碱预处理供者及肝脏对供肝冷缺血损伤的影响。方法 Wistar大鼠按处理方法的不同分为 4组 :对照组、供者预处理组 (实验 1组 )、供肝预处理组 (实验 2组 )、供者及供肝联合预处理组 (实验 3组 ) ,预处理使用己酮可可碱。各组动物均在供肝冷保存 6h后行原位肝移植 ,门静脉血流恢复后第 30min、3h及 2 4h取门静脉血测定肿瘤坏死因子 (TNF α)、丙氨酸转氨酶(ALT)、天冬氨酸转氨酶 (AST)及谷胱甘肽S转移酶 (GST)的水平。结果 门静脉复流后 30min、3h时血清TNF α水平 ,各实验组明显低于对照组 ,实验 3组显著低于实验 1组及实验 2组 (P <0 .0 5) ;30min及 3h时血清GST水平 ,各实验组明显低于对照组 (P <0 .0 5) ,实验 3组显著低于实验 1组及实验 2组 (P <0 .0 5) ;30min及 3h时血清ALT水平 ,各实验组明显低于对照组 ,实验 3组显著低于实验1组及实验 2组 (P <0 .0 5) ;2 4h时前述各指标各实验组明显低于对照组 ,实验 3组显著低于实验 2组 (P <0 .0 5...

目的 探讨已酮可可碱预处理供者及肝脏对供肝冷缺血损伤的影响。方法 Wistar大鼠按处理方法的不同分为 4组 :对照组、供者预处理组 (实验 1组 )、供肝预处理组 (实验 2组 )、供者及供肝联合预处理组 (实验 3组 ) ,预处理使用己酮可可碱。各组动物均在供肝冷保存 6h后行原位肝移植 ,门静脉血流恢复后第 30min、3h及 2 4h取门静脉血测定肿瘤坏死因子 (TNF α)、丙氨酸转氨酶(ALT)、天冬氨酸转氨酶 (AST)及谷胱甘肽S转移酶 (GST)的水平。结果 门静脉复流后 30min、3h时血清TNF α水平 ,各实验组明显低于对照组 ,实验 3组显著低于实验 1组及实验 2组 (P <0 .0 5) ;30min及 3h时血清GST水平 ,各实验组明显低于对照组 (P <0 .0 5) ,实验 3组显著低于实验 1组及实验 2组 (P <0 .0 5) ;30min及 3h时血清ALT水平 ,各实验组明显低于对照组 ,实验 3组显著低于实验1组及实验 2组 (P <0 .0 5) ;2 4h时前述各指标各实验组明显低于对照组 ,实验 3组显著低于实验 2组 (P <0 .0 5) ;30min时血清AST水平 ,各实验组明显低于对照组 ,实验 3组显著低于实验 1组及实验 2组 (P <0 .0 5)。结论 己酮可可碱预处理对供肝的冷缺血再灌注损伤有一定的保护作用

Objective To establish a novel,representative and steady transplant arteriosclerosis model.Methods Abdominal aorta grafts from SD rats were cold stored for 1,24,or 48 h before being orthopically transplanted to SD or Wistar recipients.The pathohistological changes and the expression of TGF-β1 of the grafts were observed.The changes of serum lipid peroxides pre- and post-transplantation were also measured.Results The intimal thickness of SD→SD and SD→Wistar ischemia for 1 h was significantly greated than that...

Objective To establish a novel,representative and steady transplant arteriosclerosis model.Methods Abdominal aorta grafts from SD rats were cold stored for 1,24,or 48 h before being orthopically transplanted to SD or Wistar recipients.The pathohistological changes and the expression of TGF-β1 of the grafts were observed.The changes of serum lipid peroxides pre- and post-transplantation were also measured.Results The intimal thickness of SD→SD and SD→Wistar ischemia for 1 h was significantly greated than that of pre-transplantation in 10 weeks and 6 weeks respectively,whereas grafts with 24 h of ischemia developed pronounced thickening in 2 weeks.Serum levels of lipid peroxides were significantly higher in 2 h post-transplantation in all groups than pre-transplantation,but there was no significant different pre- and 4 or 24 h post-transplantation.The staining intensity of TGF-β1 in 24 and 48 h of ischemia was stronger one week after transplantation regardless of syngeneic or allogeneic transplants.Conclusion The transplantation of abdominal aorta grafts used SD/Wistar as donors and recipients with prolonged cold ischemia time is sufficient to promote the development of transplant arteriosclerosis.

目的 建立一种简捷 ,有代表性且稳定的移植物动脉硬化模型。方法 将SD大鼠的腹主动脉分别冷缺血 1、2 4、48h行SD→SD及SD→Wistar的原位腹主动脉移植 ,观察术后植入段血管病理改变、TGF β1表达及手术前后过氧化脂质的变化。结果 SD→SD及SD→Wistar缺血1h组分别于术后 10周及 6周见内膜明显增厚 ,而缺血 2 4h组只需 2周 ;各组移植后 2h过氧化脂质均明显高于术前 ,术后 4、2 4h与术前比差异无显著性 (P >0 .0 5 ) ;强化缺血组TGF β1不论是SD→SD还是SD→Wistar均于术后 1周即出现高表达。结论 以SD/Wistar作为供 /受体行腹主动脉移植 ,强化冷缺血损伤 ,可加快移植物动脉硬化 ,可望成为新型慢排模型。

 
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