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   前列腺癌风险 的翻译结果: 查询用时:0.494秒
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前列腺癌风险
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  prostate cancer risk
     Polymorphisms of glutathione-S-transferase genes GSTM1 and GSTT1 and prostate cancer risk in Chinese population
     GSTM1、GSTT1基因多态性与中国人前列腺癌风险关系
短句来源
     Relationship between Glutathione S-transferase M1, T1 and P1 Polymorphisms and Prostate Cancer Risk in Chinese Population
     GSTM1、GSTT1、GSTP1 基因多态性与中国人前列腺癌风险关系研究
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  “前列腺癌风险”译为未确定词的双语例句
     Association between A49T polymorphism of SRD5A2 gene and risk of prostate cancer
     SRD5A2基因A49T多态性与前列腺癌风险性关系的研究
短句来源
     Objective To observe the relationship between the glutathione S-transferase genes (GSTM1 and GSTT1) and the risk of prostate cancer (PCa) in Chinese population.
     目的 检测谷胱甘肽巯基转移酶GSTM1、GSTT1基因多态性与中国人前列腺癌风险关系。
短句来源
     Association between single-nucleotide polymorphisms in estrogen receptor beta gene and risk of prostate cancer
     雌激素受体β的单核苷酸多态性与前列腺癌风险的相关性研究
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  相似匹配句对
     Chemoprevention of Prostate Cancer
     前列腺癌的化学预防
短句来源
     Radiotherapy for Prostate Cancer
     前列腺癌放射治疗
短句来源
     Association between single-nucleotide polymorphisms in estrogen receptor beta gene and risk of prostate cancer
     雌激素受体β的单核苷酸多态性与前列腺癌风险的相关性研究
短句来源
     Polymorphisms of Phase Ⅰ or Ⅱ Metabolic Enzyme Genes and Susceptibility to Prostate Cancer
     Ⅰ、Ⅱ相代谢酶基因多态性与前列腺癌发病风险
短句来源
     Polymorphisms of glutathione-S-transferase genes GSTM1 and GSTT1 and prostate cancer risk in Chinese population
     GSTM1、GSTT1基因多态性与中国人前列腺癌风险关系
短句来源
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  prostate cancer risk
Testosterone levels have been shown to modulate prostate cancer risk and progression.
      
A prospective evaluation of prostate cancer risk with testosterone replacement therapy has not been conducted.
      
Testosterone levels have been shown to modulate prostate cancer risk and progression.
      
A prospective evaluation of prostate cancer risk with testosterone replacement therapy has not been conducted.
      
There are multiple etiologic factors that likely contribute to the development of prostate cancer; however, one potential factor that may explain differences of prostate cancer risk among ethnic and racial groups is the androgen receptor (AR) gene.
      
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Objective To clarify the association of CYP3A5 gene polymorphism with the genetic

目的 探讨CYP3A5基因多态性与前列腺癌发生风险和病理特点的关系。方法 采用限制性片段多态性分析法对356例前列腺癌患者和306个男性对照中CYP3A5基因第3内含子多态性进行了研究。结果 在前列腺癌和对照组之间CYP3A5基因型分布差异无显著性(P=0.063),但两组间 CYP3A5~*1等位基因的分布差异存在显著性(P=0.025);与携带 CTh3A5~*3~*3基因型者相比,携带 CYP3A5~*1等位基因的男性患前列腺癌的风险降低了30%(P=0.026)。在不同分期和分级的前列腺癌患者之间 CYP3A5基因型分布差异无显著性(P=0.904和 0.986)。结论 CYP3A5基因中的CYP3A5~*1等位基因可能与前列腺癌的患病风险降低有关。

Objective To observe the relationship between the glutathione S-transferase genes (GSTM1 and GSTT1) and the risk of prostate cancer (PCa) in Chinese population. Methods Blood samples from 81 prostate cancer patients and 90 controls with age above 50 in Chongqing were collected. DNA was extracted from each blood sample, the frequencies of GSTM1 and GSTT1 genotypes were analyzed using polymerase chain reaction (PCR). Results The frequencies of GSTM1 null in PCa was 54.3%, 44.4% in controls, suggesting no significant...

Objective To observe the relationship between the glutathione S-transferase genes (GSTM1 and GSTT1) and the risk of prostate cancer (PCa) in Chinese population. Methods Blood samples from 81 prostate cancer patients and 90 controls with age above 50 in Chongqing were collected. DNA was extracted from each blood sample, the frequencies of GSTM1 and GSTT1 genotypes were analyzed using polymerase chain reaction (PCR). Results The frequencies of GSTM1 null in PCa was 54.3%, 44.4% in controls, suggesting no significant difference between them (P=0.197, OR=1.486,95%CI=0.813-2.718). The frequencies of GSTT1 null in PCa was 54.3%, 44.4% in controls, suggesting no significant difference between them (P=0.974, OR=0.99, 95%CI=0.543-1.807). The combination of GSTM1 null with GSTT1 null gene type was significantly less in PCa than that of the controls (P=0.034, OR=2.314, 95%CI=1.054-5.076). Conclusion Neither the GSTM1 null genotype nor the GSTT1 null genotype was associated with prostate-cancer risk, but the combination of them both had significant relationship with prostate cancer risk. No statistically significant associations were found between polymorphism of GSTM1 or GSTT1 with disease status and Gleason grade of PCa.

目的 检测谷胱甘肽巯基转移酶GSTM1、GSTT1基因多态性与中国人前列腺癌风险关系。方法 收集重庆地区前列腺癌血标本81例,5 0岁以上对照组血标本90例,从外周血提取DNA ,采用聚合酶链反应(PCR)分析GSTM1及GSTT1无效基因型分布频率。结果 GSTM1无效基因型在前列腺癌和对照组分布频率分别为5 4 3 %、44 4% ,两组间无显著统计学差异(P =0 197,OR =1 486,95 %CI为0 813~2 718) ;GSTT1无效基因型在前列腺癌和对照组中分布频率分别为5 3 1%和5 3 3 % ,两组间无显著统计学差异(P =0 974,OR =0 99,95 %CI为0 5 43~1 80 7) ;GSTM1无效基因型合并GSTT1无效基因型在两组中的分布频率为3 0 %。13 3 % ,两组间有显著差异(P =0 0 3 4,OR =2 3 14 ,95 %CI为1 0 5 4~5 0 76)。结论 GSTM1无效基因型合并GSTT1无效基因型与重庆地区前列腺癌发病明显关联。GSTM1、GSTT1无效基因型与前列腺癌发病风险间无明显相关。G...

目的 检测谷胱甘肽巯基转移酶GSTM1、GSTT1基因多态性与中国人前列腺癌风险关系。方法 收集重庆地区前列腺癌血标本81例,5 0岁以上对照组血标本90例,从外周血提取DNA ,采用聚合酶链反应(PCR)分析GSTM1及GSTT1无效基因型分布频率。结果 GSTM1无效基因型在前列腺癌和对照组分布频率分别为5 4 3 %、44 4% ,两组间无显著统计学差异(P =0 197,OR =1 486,95 %CI为0 813~2 718) ;GSTT1无效基因型在前列腺癌和对照组中分布频率分别为5 3 1%和5 3 3 % ,两组间无显著统计学差异(P =0 974,OR =0 99,95 %CI为0 5 43~1 80 7) ;GSTM1无效基因型合并GSTT1无效基因型在两组中的分布频率为3 0 %。13 3 % ,两组间有显著差异(P =0 0 3 4,OR =2 3 14 ,95 %CI为1 0 5 4~5 0 76)。结论 GSTM1无效基因型合并GSTT1无效基因型与重庆地区前列腺癌发病明显关联。GSTM1、GSTT1无效基因型与前列腺癌发病风险间无明显相关。GSTM1、GSTT1基因多态性与前列腺癌分期、分级无关。

 
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