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休克对
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  shock on
     Effect of Cold Shock on the Expression of p21WAF1/cip1 and the Cell Cycle Control in PG and HeLa Cell Lines
     冷休克对PG、HeLa细胞周期调控及p21WAF1/cip1蛋白表达的影响
短句来源
     Effect of Heat Shock on the Expression of UPA in Human Tongue Carcinoma Cell Line (Tca8113)
     热休克对人舌癌Tca8113细胞UPA表达的影响
     Effect of chemotherapeutic drugs and heat shock on the expression of MDR1 gene inhuman hepatoma cell line SMMC-7721
     化疗药和热休克对人肝癌细胞株SMMC—7721中MDR1基因表达的影响
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     BACKGROUND & AIM: To study whether heat shock could induce the formation of γ H2AX foci in CHL cells, and the effect of heat shock on N-methyl-N' -nitro-N-nitrosoguanidine(MNNG)-induced γ H2AX foci formation.
     背景与目的:探讨热休克是否可诱导中国仓鼠肺细胞(CHL)中γH2AX焦点的形成,以及热休克对N-甲基-N'-硝基-N-亚硝基胍(MNNG)诱导γH2AX焦点形成的影响。
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     The effect of heat shock on the expression of urokinase-type plasminogen activator receptor in human tongue squamous cell carcinoma Tca 8113 cells
     热休克对人舌癌Tca8113细胞尿激酶型纤溶酶原激活物受体表达的影响
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  “休克对”译为未确定词的双语例句
     Influence of Heat Shock Treatment upon the Expression of Membrane HSP70 and Its mRNA of H22 Cell
     热休克对H22细胞膜HSP70蛋白及其mRNA水平的影响
短句来源
     Influence of elemene or heat shock treatment upon the expression of membrane HSP70 and HSPs genes in HepG_2 cells
     榄香烯或热休克对人肝癌细胞HepG_2 HSP70膜表达及多种HSP基因表达的影响
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     Objective To investigate the expression of p21WAF1/cip1 induced by cold shock and the cell cycle control in PG and HeLa cell lines.
     目的 研究冷休克对p5 3基因突变的PG细胞、p5 3野生型的HeLa细胞的周期调控及 p2 1WAF1/cip1蛋白表达的影响。
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     Objective To study the dyna mi c expression of heat shock protein (HSP) 70 and HSP27 in lens epithelial cells ( LECs) in contused eyes of rat model and to detect the dynamic change of the expr ession of HSP70 and HSP27 after thermotolerance.
     目的探讨大鼠眼钝挫伤后晶状体上皮细胞HSP70和HSP27的表达,并给予预先的热刺激(热休克),观察热休克对晶状体上皮细胞HSP70和HSP27表达的调节。
短句来源
     Effect of Heat-shock on Synthetic Functions of RNA and HSP 70 mRNA in Different Cell
     热休克对不同细胞RNA和HSP70mRNA合成功能的影响
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  相似匹配句对
     In [1] W.
     W.
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     Calculation for the example given in T.
     T.
短句来源
     Shock
     休克
短句来源
     CARDIAC DAMAGE MANIFESTATIONS OF SEPTIC SHOCK
     感染性休克心脏的影响
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     Effects of pentoxifylline in dogs with endotoxic shock.
     己酮可可碱内毒素休克犬的影响
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  shock on
Study of gas flow in region of incidence of a shock on a cylinder in high supersonic flow
      
Study of gas flow and heat transfer in region of incidence of a compression shock on a cylinder in a high supersonic stream
      
The incidence of a weak shock on a plate moving at supersonic speed is examined theoretically.
      
Effect of a weak shock on a wing of complex planform at supersonic speeds
      
In [4] the effect of such a shock on the mixing layer of flows with Mach numbers M = 3 and 5 was experimentally investigated and it was shown that behind the shock no turbulence is generated.
      
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With the combination effect of heat shock and concanavalin A stimulation on human T lymphocytes,an in vitro model system was established for studying the mechanism of natural immune response to infection accompanied with fever.T cells were known to synthesize many proteins of importance in cell growth and proliferation during con A activation.When further subjected to heat shock,some of the proteins were synthesized synergetically while the others were under depression in these cells.In view of the facts that...

With the combination effect of heat shock and concanavalin A stimulation on human T lymphocytes,an in vitro model system was established for studying the mechanism of natural immune response to infection accompanied with fever.T cells were known to synthesize many proteins of importance in cell growth and proliferation during con A activation.When further subjected to heat shock,some of the proteins were synthesized synergetically while the others were under depression in these cells.In view of the facts that the synthesis of proteins with noticeable cellular function,such as actin.was less affected and the higher potential of inducing heat shock proteins (HSP) in con A activated T cells as compared with resting T cells,we suggested that con A activated cells were more resistant to elevated temperature.This notion may be of significance in both the understanding of the regulation of immune response and the application of hyperthermia treatment in the clinic.

为模拟天然免疫条件下,病毒等抗原对T淋巴细胞激活同时伴有体温升高的环境,我们建立了人外周血T淋巴细胞体外激活和热休克的模型。热休克对ConA激活细胞中多肽的合成既有协同也有抑制作用。ConA激活细胞受热休克的影响较静止细胞小,而其热休克蛋白(HSP)的诱导合成则较强,为了解HSP在淋巴细胞中的作用提供了线索。

The left ventricular myocardial catecholamine(CA), glycogen and pgosphorylase were histochemically studied in 35 rabbits which were divided into 3 groups---normal control, sham operation and hemorrhagic shock.

本文以35只家兔分三组对失血性休克2小时动物心肌进行了三种方法的组织化学观察。结果看到:失血性休克发生后,心肌儿茶酚胺(CA)含量明显减弱以至耗竭(乙醛酸诱发CA荧光方法)、心肌糖原(PAS反应)和磷酸化酶(磷酸化酶改良法)大量脱失,部分心肌细胞明显肿胀。而假手术对上述指标影响很轻。结果说明,失血性休克对心肌组织化学影响很大,导致心脏交感神经功能低下,心肌代谢紊乱,可能是造成本文绝大部分(13/14)休克动物死亡的主要因素。

The influence of shock in varied degrees on pial-microcirculation in rats and the dynamic change of pial-microcirculation in hypotension were observed. It was shown that severe disturbance occurred in pial-microcirculation when blood ressure was lowered to 3.20 kPa.In early stage after severe bleeding the main manifestation of pial-microcirculatory disturbance was hyperemia, i. e., dilatation of arteriole or segmental dilatation-contraction of arteriole, hyperemia of capillary net, increased number of opened...

The influence of shock in varied degrees on pial-microcirculation in rats and the dynamic change of pial-microcirculation in hypotension were observed. It was shown that severe disturbance occurred in pial-microcirculation when blood ressure was lowered to 3.20 kPa.In early stage after severe bleeding the main manifestation of pial-microcirculatory disturbance was hyperemia, i. e., dilatation of arteriole or segmental dilatation-contraction of arteriole, hyperemia of capillary net, increased number of opened capillaries and appearance of 0-I degree of blood flow status (according to the classification of Tia Niu).Ninety minutes after bleeding arterioles and venules became gradually constricted,red blood cells aggregation was increased, ischemia of pial-microcirculation became more severe and blood flow status changed from 1st to 2nd, even 3rd degree.The results demonstrate that autoregulation of cerebral microcirculation is much stronger than that of other organs. Microcirculatory disturbance does not appear until blood pressure drops to the degree beyond the ability of autoregulation.

作者观察了不同程度的失血性休克对大鼠软脑膜微循环的影响,以及在一定低血压下微循环障碍的变化规律。实验结果表明,当血压降至3.2 kPa(24 mmHg)时,可造成微循环的明显障碍。早期以充血为主,表现为微动脉扩张或微动脉的节段性扩张与收缩,毛细血管网充血开放数目增多,流态维持在O~1级。持续90min以后,微血管逐渐收缩,红细胞聚集加重,流态逐渐过渡到2~3级;晚期呈现出微循环的严重缺血状态。

 
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