In the dissertation, the self-diagnosing damage method and optimal sensor placement of smart materials and structures are mainly studied in order to offer theory guide for the design and realization of the fiber smart layer with self-diagnosing function. The main research contents in this dissertation are as follows:At first, On the base of the traditional frequency method for damage detection, an improved frequency damage indexes are given.
② A close correlation was shown between MDA content and NO 2 -/NO 3 - content in plasma and lung. Furthermore,the content of NO 2 -/NO 3 - in lung homogenate showed strong positive correlation with the lung injury parameters.
There were marked upregulation of ICAM-1 mRNA expression in injuried lung tissue compared with the normal lung tissue. Furthermore, the infiltrated number of macrophage and the level of ICAM-1 expression showed strong positive correlation with the lung injury parameters, PaO 2 and LPI.
A damage index, used to identify possible locations and corresponding severities of local damage, is defined and expressed in terms of modal displacements that are obtained numerically from mode shapes of the undamaged and the damaged structures.
Based on a small damage assumption, a damage index equation is revealedn the context of elasticity.
It turns out that the resulting damage index equation inherently suffers from singularities near inflection nodes.
With the aid of the singular value decomposition technique, the solution to the damage index equation is achieved in the Haar wavelet space.
Next, the physical desired solution to the damage index equation is reconstructed from the one in the wavelet space.
Simulations using three crash pulses, soft, moderate, and severe, are conducted to investigate the influence of crash severity on the child dummy head and thorax accelerations, upper neck forces, and injury parameters.
Our results showed that lung injury parameters, including the wet/dry weight ratio and protein content in BALF, were significantly higher in the LPS alone group than in the saline control group (P>amp;lt;0.01).
Our results showed that lung injury parameters, including the wet/dry weight ratio and protein content in BALF, were significantly higher in the L group than in the S group (P>amp;lt;0.01).
ALL treatment did not affect liver injury parameters, as concentrations of lactate dehydrogenase (LDH) and alanine transferase (ALT) increased in plasma after ischemia, both in controls and in the ALL-treated group.
Immunosuppressant pretherapy significantly stimulated BrdU uptake by hepatocytes, in association with increased MDA production, while there were no differences in serum liver injury parameters among the groups given or not given immunosuppressants.