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扁桃体疝的
相关语句
  tonsillar herniation
     Clinical practice of cerebellar tonsillar herniation
     急性小脑扁桃体疝的临床实践
短句来源
  “扁桃体疝的”译为未确定词的双语例句
     Conclusion:Patients with masses in the fourth ventricle, cerebellum and CPA were prone to tonsillar hernia more than those with masses in other areas of posterior fossa and pineal region and the bigger the mass,the higher possibility of tonsillar hernia.
     而位于松果体区和脑干的占位性病变对小脑扁桃体位置影响较小 (P>0.01)。 结论:颅后窝占位性病变的部位和大小与小脑扁桃体的位置有明显相关性,根据占位性病变的大小可以预测 小脑扁桃体疝的发生。
短句来源
  相似匹配句对
     Clinical practice of cerebellar tonsillar herniation
     急性小脑扁桃体疝的临床实践
短句来源
     Mycosis of the tonsil
     扁桃体真菌病
短句来源
     The classification of inguinal hernias.
     腹股沟疝的分型
短句来源
     The length of the herniated tonsils was unrelated with degree of the expansion of the spinal cord and ventrical.
     小脑扁桃体疝的长度与脊髓增粗和脑室扩大无相关性。
短句来源
     Anesthetic management of tonsillectomy
     扁桃体摘除术的麻醉处理
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  tonsillar herniation
Patients with grade 4 or 5 SEPs were found to have severe brain oedema giving rise to transtentorial herniation, which was combined with secondary midbrain haemorrhage and tonsillar herniation in all patients with bilateral grade-5 SEPs.
      
Tonsillar herniation was not found to influence supine or upright blood pressure, heart rate or plasma norepinephrine levels in the patients.
      
This, in turn, may produce subdural hygroma or hematoma, brainstem compression, focal cranial nerve palsies, or cerebellar tonsillar herniation.
      
The Chiari I malformation is a cerebellar anomaly of uncertain origin, defined in part as tonsillar herniation of at least 3 to 5 mm below the foramen magnum; it is associated with crowding of the craniocervical junction.
      
Hydrocephalus and low intracranial pressure syndrome should be ruled out in patients showing tonsillar herniation in an MRI study and consulting due to daily headache.
      
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The report dealt with the pathological changes in the extrahepatic organs from 30 autopsied cases with clinical manifestations of severe viral hepatitis (SAH) . The main findings were epitomized as follows.(1 ) Brain edema with herniation of tonsil cerebelli in 24 (71%) cases among the 27 whose brains were examined. (2) Bilateral pulmonary edema in 20 (43%) cases among the 28 whose lungs were examined. 45% cases of pulmonary edema were complicated by bronchopneumonia, 10% by fungal infection, and 15% by pulmonary...

The report dealt with the pathological changes in the extrahepatic organs from 30 autopsied cases with clinical manifestations of severe viral hepatitis (SAH) . The main findings were epitomized as follows.(1 ) Brain edema with herniation of tonsil cerebelli in 24 (71%) cases among the 27 whose brains were examined. (2) Bilateral pulmonary edema in 20 (43%) cases among the 28 whose lungs were examined. 45% cases of pulmonary edema were complicated by bronchopneumonia, 10% by fungal infection, and 15% by pulmonary hemorrhage. ( 3 ) Cardiac hypertrophy in ( 43% ) cases among the 28 whose hearts were examined. (4) Biliary nephrosis in 22 (92%) cases among the 24 whose kidneys wers examined. ( 5 ) All the 30 cases showed intestinal congestion and edema throughout the 3 laminae of intestinal wall. (6) of 30 cases, 8 (27%) showed esophageal varicosis. Early hepatic cirrhosis was established in half of the cases. (7) of 29 cases, 25 (85%) had ascitis with various amount of transudate ranging from 2000-8000 ml in adults and 300-1500ml in children. (8) of 30 cases, 4 (13%) had subacute phlegmonous colitis. (9) of 30 cases, 3 (10%) were complicated by the generalizad aspergillosis and/or candiasis involving the lungs, brain, heart, kidneys, intestines, et al.It was considered that the alterations in extrahepatic organs should aggravate the primary disease and thus be prone to increase its motality rate. Some of them, such as brain edema might be the direct cause of death. It was thought that two factors might be responsible for the initiation of extrahepatic alterations. ( 1 ) increase in intrahepatic resistance to the portal blood flow which is presumably due to reduction of hepatic microcirculatory beds, and ( 2 ) hypoxemia which is probably due to intrapulmonary vascular shunts in case of hepatic failure. These factors, once established, might in turn trigger off a sequence of pathological events.

本文对30例重型肝炎时肝外脏器病变作了详细的形态学观察和分析,结果表明:27/30例(90%)有脑水肿,其中24例伴发小脑扁桃体疝;20/28例(约71%)有肺水肿,其中9例伴发支气管肺炎、3例伴发霉菌感染、2例伴发肺出血;12/28例(约43%)有心脏肥大;30/30例(100%)有肠道充血、水肿;8/30例(约27%)有食道下端静脉曲张,其中2例破裂出血;25/29例(约86%)有腹腔积液;22/24例(约92%)有胆汁性肾病;对17例脾脏作了观察,都有脾窦扩张、充血,其中6例重量在210~750g之间;在30例中有亚急性蜂窝织性结肠炎者4例;全身性霉菌病3例。 对这些肝外脏器病变的发生和它们之间相互关系作了分析和讨论,认为重型肝炎时肝内毛细血管床容量缩小所导致的门脉血流受阻和肺内循环短路引起的低血氧症,是一系列肝外并发症发生的两个始动因素。

This paper reports 25 cases with otogenous cerebral hernia, in which, 13 were tentorial hernia, (10 inferior tentorid notch hernia, 3superior tentorid notch hernia), 12 tonsillar hernia. We found that the predisposing factors of cerebral hernia were mastaidotomy Inmbal puncture, postural change and drainage of cerebral puneture, etc.Whenever the cerebral pressure of a patient with cerebral abscess is increased, one should avoid aforesaid factors in order to avoid the emergence of cerebral hernia incarceration.It...

This paper reports 25 cases with otogenous cerebral hernia, in which, 13 were tentorial hernia, (10 inferior tentorid notch hernia, 3superior tentorid notch hernia), 12 tonsillar hernia. We found that the predisposing factors of cerebral hernia were mastaidotomy Inmbal puncture, postural change and drainage of cerebral puneture, etc.Whenever the cerebral pressure of a patient with cerebral abscess is increased, one should avoid aforesaid factors in order to avoid the emergence of cerebral hernia incarceration.It is emphasized that once cerebral hernia emerged in patient. a measure of dehydrant should be taken as a preparatory step before opration

本文报道耳源性脑疝25例,其中小脑幕疝13例(小脑幕切迹下疝10例,小脑幕切迹上疝3例),枕骨大孔疝(小脑扁桃体疝)12例.脑疝的诱因有乳突凿开术、腰穿、体位改变和脑穿刺引流等.当脑脓肿病人有颅内压增高时.应避免以上诱发脑疝的因素.一旦出现脑疝,作者强调应使用脱水剂作为术前的准备措施

16 cases of acute cerebellartonsillar herniation(ACTH)are reported.There were 11 cases ofsuccessful rescue,1 vegetable status and 4 deaths.The clinical manifestation,mechanism of ACTHand rescue of respiratory arrest are discussed.Toimprove the effect of treatment,the diagnosisshould be made timely at prodromal stage orcompensatory stage of the herniation.Noteworthi-ness were 5 cases which stopped breathing 1 to12 hours after lumbar puncture.So lumbar punctureis,generally speaking,not recommendable forintracranranial...

16 cases of acute cerebellartonsillar herniation(ACTH)are reported.There were 11 cases ofsuccessful rescue,1 vegetable status and 4 deaths.The clinical manifestation,mechanism of ACTHand rescue of respiratory arrest are discussed.Toimprove the effect of treatment,the diagnosisshould be made timely at prodromal stage orcompensatory stage of the herniation.Noteworthi-ness were 5 cases which stopped breathing 1 to12 hours after lumbar puncture.So lumbar punctureis,generally speaking,not recommendable forintracranranial space occupying lessions,especiallyin posterior fossa accompanied with advancedpapilledema.Once respiration stops,tracheal in-tubation should be performed and ventricular drain-age,and effective dehydration drugs.When naturalrespiration restores,primary lesions should be removedquickly for relieving the direct compression uponbrain stem.

本文报告16例急性小脑扁桃体疝,抢救成活11例,植物生存1例,死亡4例。对其临床表现、发生机制、呼吸停止的抢救等问题进行探讨。为提高疗效,在脑疝前驱期或脑疝代偿期即应作出诊断。一旦呼吸停止,采取迅速的气管插管人工呼吸、准确的颅骨钻孔脑室引流、有效的脱水药物。自主呼吸恢复,及时将原发病灶清除,解除对脑干的直接压迫。

 
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