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   大鼠出现 的翻译结果: 查询用时:0.424秒
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大鼠出现
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  rats displayed
     The rats in VV groups showed breathlessness,fervescence,and swelling in extremitas inferior at 4h after being injected VV.These symptoms were worse and worse,and the rats displayed cyanosis and convulsion at 12h;
     VV组染菌4 h,大鼠出现呼吸急促,皮温升高,下肢肿胀,且症状进行性加重,染菌12 h后大鼠出现紫绀、间断抽搐等;
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     fter immunization with AChR plus CFA, Lewis rats displayed the typical signs of EAMG, while Wistar Furth(WF) rats did not present any signs of EAMG.
     用AChR加CFA免疫大鼠后,Lewis大鼠出现典型的临床肌无力,而WistarFurth(W.F)大鼠不出现任何症状。
短句来源
     Results The rats showed breathlessness, fervescence, and swelling in extremitas inferior at 4 h after being injected VV. These symptoms were worse and worse , and the rats displayed cyanosis and convulsion after 12 h.
     结果染菌后4h,大鼠开始出现活动减少,呼吸急促,皮温升高,下肢肿胀,后症状进行性加重,12h后染毒大鼠出现紫绀、间断抽搐等。
短句来源
     After immunization with ACHR Plus CFA, Lewis rats displayed the typical signs of EAMG,while Wistar Furth(WF) rats did nor present any signs of EAMG.
     用AChR加CFA免疫大鼠后,Lewis大鼠出现典型的临床肌无力,而Wister,Furth(W.F)大鼠不出现任何症状。
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     Results Rats displayed category Ⅳ or Ⅴ seizures (according to Racince) and atatus epilepticus (SE) 21-39 min after pilocarpine injection. After SE, the rats went into a relatively unresponsive and akinetic periold (latent period) for 5-22 days. At last, the rats went into chronic period, in which category Ⅰ-Ⅲ spontaneous recurrent seizures(SRSs) could be observed.
     结果 大鼠在腹腔注射匹罗卡品后21~39 min(平均32 min)出现RacineⅣ级或Ⅴ级发作及癫痫持续状态(SE),SE后大鼠出现5~22 d(平均16.5 d)的静默期,其后大鼠进入慢性癫痫发作期,表现为Racine Ⅰ~Ⅲ级的自发反复发作(SRS)。
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  “大鼠出现”译为未确定词的双语例句
     The results showed that at the end of the administration the serum levels of T 3,T 4 were 0.81±0.11,34.70±0.91 nmol/L respectively,and were higher than those in control animals.
     结果:实验组大鼠出现甲亢体征,T3、T4分别为0.81±0.11和34.70±0.91nmol/L,均高于对照组(P<0.05);
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     The frequency before stimulation was 12.64±1. 92 beats/9s and 3.67±2.73 beats/9s while stimulating on the Hb(P<0.05).
     1 电刺激内侧缰核(MHB)内侧部,出现呼吸明显减慢甚至呼吸暂停,刺激前呼吸频率为12.64±1.92 beats/9s,刺激时呼吸频率减慢,为 3.67±2.73 beats/9s,(P<0.05)刺激结束后,大鼠出现代偿性呼吸加快 。
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     Results A varied degree of hepatic steatosis were induced for 3~5 weeks,the total score in pathology of rat model were significant higher than that of the control rats(F=29.72,P<0.0001).
     结果3~5周后,模型组大鼠出现了程度不等的肝脂肪变性,肝细胞病理学总计分显著高于对照组(F=29.72,P<0.0001)。
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     Results (1) Different degrees of ALI was induced in the rats by different doses of LPS. ARDS was produced in the rats by LPS of 6mg/kg.
     结果  (1)梯度剂量LPS可以引发不同程度的ALI,当LPS≥ 6mg/kg时 ,大鼠出现急性呼吸窘迫综合征 (ARDS)。
短句来源
     Results:Compared with normal group,the normal hepatic lobules in model group were disappeared and fibroplasias formed nodus and false folial. The content of IL-1β and IL-6 in serum and the content of IL-6 in liver tissue were increased obviously(P<0.01 or P<0.05).
     结果:与正常组比较,模型组大鼠出现肝小叶损害,纤维组织增生,假小叶形成,血清IL-1β、IL-6含量和肝组织IL-6含量均明显升高(P<0.01或P<0.05);
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  相似匹配句对
     THE APPEARANCE AND CHANGES OF ACETYLCHOLINESTERASE IN THE BRAINS OF RAT
     大鼠脑内乙酰胆碱酯酶的出现和变化
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     Meanwhile,(hyperinsulinemia) and hypertriglyceridemia were observed in the rats with high-fat diet.
     同时,高脂膳食大鼠出现高脂血症。
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     All rats were cut their head and killed .
     大鼠断头处死。
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     KEY WORDS: newborn rat;
     关键词: 新生大鼠;
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     There was no complications.
     无并发症出现
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  rats displayed
Hyperlipidemic rats displayed a reduction of the intestinal glutathione level and an increase in intestinal lipid peroxidation levels, and in serum cholesterol, total lipid and phospholipids levels.
      
Control rats displayed a reduced daytime heart rate from the beginning of the protocol, whereas exposed rats maintained a significantly elevated heart rate throughout the exposure.
      
When injected iv with GLP-1, BBdp rats displayed lower second-phase insulin response (and insulin/glucose ratios) compared with BBc rats.
      
Thus, young BBdp rats displayed decreased concentrations of bioactive GLP-1 in jejunum and ileum, reduced GLP-1R in islets, and lower second-phase insulin response to iv GLP-1 than controls.
      
The largest ovarian cross-sections from FSH?+?DHT-treated HYPOXD rats displayed 18.3?±?4.1 small follicles with a mean diameter of ~0.437?mm which possessed few granulosa cells.
      
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Through an experiment of adding chromium(Ⅲ)to the hypercholesterolemic ane hyperglycemic diet of rats,it has been proved that chromium has the effect of lowering serum glucose and cholesterol concentrations But this effect seems to demonstrate itself only when slight insufficiency of insulin is present.However,no effect of lowering serum triglyceride was found in this experiment。It was aiso found that after the addition of sucrose the serum cholesterol concentrations of rats fed on the hypercholesterolomic diet...

Through an experiment of adding chromium(Ⅲ)to the hypercholesterolemic ane hyperglycemic diet of rats,it has been proved that chromium has the effect of lowering serum glucose and cholesterol concentrations But this effect seems to demonstrate itself only when slight insufficiency of insulin is present.However,no effect of lowering serum triglyceride was found in this experiment。It was aiso found that after the addition of sucrose the serum cholesterol concentrations of rats fed on the hypercholesterolomic diet was obviously higher than those with chromic additive.The mechanism of this change remained an clear.Presumably chromium acted in coordination with insulin.Pathological examination showed that not over one case of cerebrovascular disease was found in rats fed with chromium,whereas it does occur in the group fed on hypercholesterolemic and hyperglycemic diet,particularly in that with the highest serum cholesterol.Whether this had anything to do with serum cholesterol still remains for further research.Therefore chromium may be used as one of the protective agent in the preven-tion and treatment of cerebrovascular disease due to its effect of lowering serum glucose and cholesterol concentrations.

实验研究表明铬具有降血糖作用,从而对高糖饮食所致血胆固醇升高有明显降低血脂的作用。在病理解剖方面初步观察到服铬大鼠未见脑血管病变,而不服铬的高脂高糖大鼠出现了脑出血现象。因而提供了铬预防脑血管病的理论根据,有进一步研究的价值。

In the present paper in order to interpret the secret of Parkinsin's disease, the models of this disease were reestablished with two different reurotoxines 6-hydroxydopamine(6-OH-DA)and kainic acid(KA)in rat. The results showed that the unilateral intranigral administration of 6-OH-DA selectively injured the DA cell bodies of the SN and made the rat turn and rotate towards the lesioned side. The unilateral intranigral injection of KA selectively destructed nigral perikarya of all kinds of neurons and produced...

In the present paper in order to interpret the secret of Parkinsin's disease, the models of this disease were reestablished with two different reurotoxines 6-hydroxydopamine(6-OH-DA)and kainic acid(KA)in rat. The results showed that the unilateral intranigral administration of 6-OH-DA selectively injured the DA cell bodies of the SN and made the rat turn and rotate towards the lesioned side. The unilateral intranigral injection of KA selectively destructed nigral perikarya of all kinds of neurons and produced a sequelae of contralateral and ipsilateral turning and rotation in the early stage. And then bilateral rotation occurred alternatively. About 24h. after administration of KA, the animal became an asymmetric posture turning contralaterally to the lesioned side. On the basis of these two models, treatment with apomorphine or haloperidol changed the ipsilateral into contralateral turning in unilateral nigral 6-OH-DA lesioned animals. Apomorphine changed the contralatcral into ipsilateral turning while haloperidol significantly potentiated the contralateral turning in unilateral nigral KA lesioned rats. The results suggested that there would be a neuronal system antagonistic to DAergic neurons of SNC. When these neurons were damaged by KA, the asymmetries of posture and behaviour were opposite to the results of unilateral lesion of nigral-striatal DAergic system.

为了进一步探索震颤麻痹的奥秘,本文应用6—羟多巴胺(6—Hydroxydo(?)amine,6—OH—DA)和海人酸(Kainic Acid,KA)两种神经毒在大鼠身上建立了此症的模型。结果当向一侧黑质内注射6—OH—DA选择性损毁多巴胺神经元时,大鼠头和躯干歪向手术侧,向同侧旋转;而向一侧黑质注射海人酸破坏黑质包括DA神经元在内的各类神经元的核周质时,注射后的短时间内顺序出现向手术对侧和同侧歪和旋转的现象,继而两侧旋转交替进行,大约在注药的24小时后,则持续向注药对侧偏转,不再改变方向。在上述动物模型的基础上,皮下注射DA受体激动剂去水吗啡或阻断剂氟哌啶醇,均使一侧SN内注射6—OH—DA的大鼠由向注射的同侧偏转改为向手术对侧偏转。而去水吗啡使一侧SN被KA损毁的大鼠由向损伤的对侧偏转改为同侧偏转;氟哌啶醇使一测SN被KA损毁的大鼠向手术对侧偏转的现象加强。结果提示在黑质网状部可能存在一种神经元系统对SN致密部的DA神经元起抑制作用。当用KA将这类神经也损毁后,大鼠出现的姿势和行为的改变与SN纹体DA系统被损毁后的结果相反。中脑黑质(Substantia Nigra,SN)属于锥体外系的一部分,...

为了进一步探索震颤麻痹的奥秘,本文应用6—羟多巴胺(6—Hydroxydo(?)amine,6—OH—DA)和海人酸(Kainic Acid,KA)两种神经毒在大鼠身上建立了此症的模型。结果当向一侧黑质内注射6—OH—DA选择性损毁多巴胺神经元时,大鼠头和躯干歪向手术侧,向同侧旋转;而向一侧黑质注射海人酸破坏黑质包括DA神经元在内的各类神经元的核周质时,注射后的短时间内顺序出现向手术对侧和同侧歪和旋转的现象,继而两侧旋转交替进行,大约在注药的24小时后,则持续向注药对侧偏转,不再改变方向。在上述动物模型的基础上,皮下注射DA受体激动剂去水吗啡或阻断剂氟哌啶醇,均使一侧SN内注射6—OH—DA的大鼠由向注射的同侧偏转改为向手术对侧偏转。而去水吗啡使一侧SN被KA损毁的大鼠由向损伤的对侧偏转改为同侧偏转;氟哌啶醇使一测SN被KA损毁的大鼠向手术对侧偏转的现象加强。结果提示在黑质网状部可能存在一种神经元系统对SN致密部的DA神经元起抑制作用。当用KA将这类神经也损毁后,大鼠出现的姿势和行为的改变与SN纹体DA系统被损毁后的结果相反。中脑黑质(Substantia Nigra,SN)属于锥体外系的一部分,其主要功能是调节躯体运动。黑质致密部的多巴胺神经元(Dopaminergic Neuron,DAergic Neuron)的变性是震颤麻痹的重要病因。本文应用6—羟多巴胺和海人酸两种神经毒在大鼠身上复制震颤麻痹的动物模型,为进一步探索其病因和治疗奠定基础。

In this experiment four groups have been designed. In the first group, Ap + NaNO_2 were administered to Wistar male rats for nearly 20 weeks through drinking. In the second and third group, besides Ap + NaNO_2 Rosa roxburghii Tratt juice and vitamin C were given to the same animals respectively. In the forth group the materials mentioned above haven't been administered at all. Within 22 weeks, in the first group the incidences of neoplastic nodule, hepatocellular carcinoma and bile ductule adenoma (cystic adenoma)...

In this experiment four groups have been designed. In the first group, Ap + NaNO_2 were administered to Wistar male rats for nearly 20 weeks through drinking. In the second and third group, besides Ap + NaNO_2 Rosa roxburghii Tratt juice and vitamin C were given to the same animals respectively. In the forth group the materials mentioned above haven't been administered at all. Within 22 weeks, in the first group the incidences of neoplastic nodule, hepatocellular carcinoma and bile ductule adenoma (cystic adenoma) were 27.3% (12/44) ,11.4% (5/44), and 25% (11/44) respectively.Meanwhile in the second, third and forth group there were no the same changes to be observed.In respect to the incidences of above changes, the differences between the first group and another 3 groups were most significant (P<0.01) The incidences of proliferative foci of liver cells observed in the second and third group were 16.7% (4/24) and 78.3% (18/23) respectively.The difference between them was also most significant (P<0.01). The results of this experiment showed that both Rosa roxburghii Tratt juice and vitamin C had remarkable effects in preventing liver tumors caused by Ap +NaNO_2, and Rosa roxburghii Tratt juice had an advantage over vitamin C.

实验组大鼠随饮水摄入二甲基亚硝胺前体物氨基比林和亚硝酸钠近20周(137天),至22周实验结束时,27.3%(12/44)的大鼠出现肝细胞增生结节(肝细胞性肝癌癌前病变),25%(11/44)的大鼠发生肝脏肿瘤(肝细胞性肝癌5例,发生率为11.4%,小胆管(囊)腺瘤11例,发生率为25%)。刺梨汁组(摄入氨基林,亚硝酸纳和刺梨汁),维生素C组(摄入氨基比林、亚硝酸纳和与刺梨汁含量相同的维生素C)和对照组均无一例发生上述病变。实验组与对照组、刺梨汁组、维生素C组之间肝脏肿瘤、肝细胞增生结节的发生率均有极显著差异(P<0.01),刺梨汁组和维生素C组肝细胞增生灶的发生率分别为16.7%(4/24)和78.3(18/23),二者之间有极显著差异(P<0.01)。

 
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