助手标题  
全文文献 工具书 数字 学术定义 翻译助手 学术趋势 更多
查询帮助
意见反馈
   非肿瘤粘膜 的翻译结果: 查询用时:0.016秒
图标索引 在分类学科中查询
所有学科
更多类别查询

图标索引 历史查询
 

非肿瘤粘膜
相关语句
  non-tumor mucosas
     The densities of p53 protein positive cells in malignant areas of adenomas and adenocarcinomas were significantly higher than those in benign areas of adenomas and non-tumor mucosas respectively (all P< 0.01).
     腺癌及腺瘤恶变区p53蛋白阳性细胞密度均高于腺瘤非恶变区(P<0.01)及非肿瘤粘膜(P<0.01)。
短句来源
     Results:The density of the apoptotic cells in benign areas of adenomas was significantly higher than that in non-tumor mucosas,malignant areas of adenomas or adenocarcinomas respectively(all P < 0.01).
     结果:腺瘤非恶变区凋亡细胞密度分别高于腺癌(P<0.01)、腺瘤恶变区(P<0.01)及非肿瘤粘膜(P<0.01)。
短句来源
  “非肿瘤粘膜”译为未确定词的双语例句
     The positive rate and staining intensity of p53 and bcl 2 oncoprotein in adenoma and adenocarcinoma were significantly higher than in non tumor mucosa( P <0.01),their staining intensity in adenocarcinoma was higher than that in adenoma( P <0.01).
     p53和bcl-2蛋白的表达率和表达强度,腺癌和腺瘤高于非肿瘤粘膜(P<0.01),腺癌的表达强度也高于腺瘤(P<0.05)。
短句来源
     Results The density of apoptotic cells in adenoma and adenocarcinoma was significantly higher than that in nontumor mucosa( P <0 01),and their density in adenoma was higher than in adenocarcinoma( P <0.01).
     结果腺瘤和腺癌中的凋亡细胞密度均显著高于非肿瘤粘膜(P<0.01); 腺瘤高于腺癌(P<0.01)。
短句来源
     Results: The densities of apoptotic cells in benign areas of malignantly changed adenomas was significantly higher than those in adenocarcinomas (P<0.01) malignant areas of adenomas (P<0.01) tubular and villous adenomas (P<0.01) and nontumor mucosas (P<0.01), respectively.
     结果 :腺瘤非恶变区凋亡细胞密度分别高于腺癌、管状绒毛状腺瘤及非肿瘤粘膜 (P均<0.01)。 腺癌及腺瘤恶变区p53蛋白阳性细胞密度分别高于管状绒毛状腺瘤 (P均<0.01)和腺瘤非恶变区 (P均<0.01)及非肿瘤粘膜 (P分别<0.01)。
短句来源
     Results The expression of Fhit protein was positive in 23/53 (43.4%) cases of cancer tissue and in 47/53 (88.7%) cases of adjacent non\|tumoral tissue.
     结果 癌组织Fhit表达为 2 3/ 5 3例 (43.4 % ) ,而癌旁非肿瘤粘膜为 4 7/ 5 3例 (88.7% ) ,相差有非常显著性 (P <0 .0 0 1)。
短句来源
     Expression and Significance of Epidermal Growth Factor Receptor in Normal Bladder Mucosa of Patients with Bladder Cancer
     表皮生长因子受体在膀胱癌非肿瘤粘膜的表达及意义
短句来源
更多       
  相似匹配句对
     Non malignant tumor group:I.
     [结果]肿瘤组 :I.
短句来源
     Nontransitional cell tumor of the bladder
     膀胱移行细胞肿瘤
短句来源
     Tumor lymphangiogenesis
     肿瘤淋巴管生成
短句来源
     PARAPHARYNGEAL SPACE TUMORS
     咽旁间隙肿瘤
短句来源
     Expression and Significance of Epidermal Growth Factor Receptor in Normal Bladder Mucosa of Patients with Bladder Cancer
     表皮生长因子受体在膀胱癌肿瘤粘膜的表达及意义
短句来源
查询“非肿瘤粘膜”译词为用户自定义的双语例句

    我想查看译文中含有:的双语例句
例句
没有找到相关例句


Superoxide dismutase (SOD) activity was evaluated in 27 cases of bladder carcinoma and in 10 spectimens of bladder mucosa free from tumor as controls. SOD activity, especially MnSOD,was markedly decreased,the poorer the tumor differentiation the lower the SOD level. Cancer with mucosa invasion had a lower SOD activity than superficial bladder cancer. A significant difference in SOD level in bladder mucosa was noted between bladder cancer patients and noncancer individuals. The decrease of SOD activity might...

Superoxide dismutase (SOD) activity was evaluated in 27 cases of bladder carcinoma and in 10 spectimens of bladder mucosa free from tumor as controls. SOD activity, especially MnSOD,was markedly decreased,the poorer the tumor differentiation the lower the SOD level. Cancer with mucosa invasion had a lower SOD activity than superficial bladder cancer. A significant difference in SOD level in bladder mucosa was noted between bladder cancer patients and noncancer individuals. The decrease of SOD activity might be considered as a marker for the early detection of bladder cancer.

为了解SOD活性与膀胱癌生物学行为的关系,采用肾上腺素自体氧化法分析了27例膀胱移行细胞癌病人肿瘤组织及非肿瘤粘膜,以及10例非肿瘤病人膀胱粘膜的SOD活性。结果显示:肿瘤组织SOD活性低于正常组织,并与分级分期有关;复发及多发膀胱癌病人非瘤粘膜SOD活性低于非肿瘤病人的膀胱粘膜(P<0.01)。SOD活性的降低可能是膀胱癌的早期表现,并与生物学行为有关

Objective To investigate the effects of apoptosis and its regulating genes in different stages of malignant transformation of large instestine epithelium in colorectal adenoma and adenocarinoma.Methods The density and distribution of the apoptotic cells and the positive expression of p53 and bcl 2 oncoprotein were observed in situ in 32 villous adenomas and 33 papillary adenocarinomas of the large intestine,using DNA nick end labelling technique and immunohistochemical staining for p53 and bcl 2 oncoprotein.15...

Objective To investigate the effects of apoptosis and its regulating genes in different stages of malignant transformation of large instestine epithelium in colorectal adenoma and adenocarinoma.Methods The density and distribution of the apoptotic cells and the positive expression of p53 and bcl 2 oncoprotein were observed in situ in 32 villous adenomas and 33 papillary adenocarinomas of the large intestine,using DNA nick end labelling technique and immunohistochemical staining for p53 and bcl 2 oncoprotein.15 nontumor mucosa were used as controls.Results The density of apoptotic cells in adenoma and adenocarcinoma was significantly higher than that in nontumor mucosa( P <0 01),and their density in adenoma was higher than in adenocarcinoma( P <0.01).The positive rate and staining intensity of p53 and bcl 2 oncoprotein in adenoma and adenocarcinoma were significantly higher than in non tumor mucosa( P <0.01),their staining intensity in adenocarcinoma was higher than that in adenoma( P <0.01).In adenoma,the density of apoptotic cells in the bcl 2 oncoprotein positive group was higher than that in the bcl 2 oncoprotein negative group( P <0.01).Conclusion The abnormal regulation of apoptosis may play an important role in the pathogenesis of large intestine carcinoma.The bcl 2 oncoprotein can inhibit apoptosis in adenoma and adenocarcinoma.However,mutational p53 oncoprotein may likely block apoptosis in adenocarcinoma

目的通过观察结、直肠腺癌和腺瘤中的细胞凋亡及其调控基因p53、bcl-2的表达状态,探讨细胞凋亡及其调控基因在大肠上皮恶性转化进程的不同阶段中的作用。方法利用DNA缺口末端标记技术和p53、bcl-2蛋白免疫组化染色,原位观察32例大肠绒毛状腺瘤和33例大肠乳头状腺癌中的凋亡细胞和p53、bcl-2蛋白阳性表达细胞的密度与分布,以15例非肿瘤大肠粘膜做为对照。结果腺瘤和腺癌中的凋亡细胞密度均显著高于非肿瘤粘膜(P<0.01);腺瘤高于腺癌(P<0.01)。p53和bcl-2蛋白的表达率和表达强度,腺癌和腺瘤高于非肿瘤粘膜(P<0.01),腺癌的表达强度也高于腺瘤(P<0.05)。腺瘤中bcl-2蛋白表达阳性组的凋亡细胞密度显著低于bcl-2阴性组(P<0.01)。结论细胞凋亡调控异常在大肠癌发病中可能起重要作用。bcl-2蛋白在腺瘤和腺癌中均可抑制细胞凋亡,而突变型p53蛋白可能仅在腺癌中起抑制细胞凋亡的作用。

To investigate the relationship between p53 and apoptosis in different stages of malignant transformation of large intestine epithelia. Methods:Using DNA nick end labeling technique for apoptotic cells, immunohistochemical staining for p53 protein and double staining for both,the density and distribution of the apoptotic cells and positive expression of p53 protein were observed in situ in 21 cases of malignantly transformed adenomas and 27 cases of adenocarcinomas of the large intestine. Fifteen cases of non-tumor...

To investigate the relationship between p53 and apoptosis in different stages of malignant transformation of large intestine epithelia. Methods:Using DNA nick end labeling technique for apoptotic cells, immunohistochemical staining for p53 protein and double staining for both,the density and distribution of the apoptotic cells and positive expression of p53 protein were observed in situ in 21 cases of malignantly transformed adenomas and 27 cases of adenocarcinomas of the large intestine. Fifteen cases of non-tumor mucosas were used as controls. Results:The density of the apoptotic cells in benign areas of adenomas was significantly higher than that in non-tumor mucosas,malignant areas of adenomas or adenocarcinomas respectively(all P < 0.01). The densities of p53 protein positive cells in malignant areas of adenomas and adenocarcinomas were significantly higher than those in benign areas of adenomas and non-tumor mucosas respectively (all P< 0.01). The distributions of p53 protein and apoptotic cells observed through double staining showed a tendency to decrease of apoptosis in p53 protein positive areas, while showing a tendency to increase in p53 negative areas. Conclusion: Probably through decreasing in apoptosis,p53 mutations play a critical part in the formation and progression of carcinomas of large intestine.

目的:观察大肠腺癌,腺瘤恶变区及非恶变区细胞凋亡及其调控基因p53的表达状态,探讨它们在大肠上皮恶性转化进程中的作用及二者的关系。方法:利用DNA缺口末端标记技术,p53蛋白免疫组化染色及双重染色技术,原位观察27例大肠腺癌及21例腺瘤恶变标本中凋亡细胞和p53阳性表达细胞的密度与分布,以8例非肿瘤大肠粘膜作为对照。结果:腺瘤非恶变区凋亡细胞密度分别高于腺癌(P<0.01)、腺瘤恶变区(P<0.01)及非肿瘤粘膜(P<0.01)。腺癌及腺瘤恶变区p53蛋白阳性细胞密度均高于腺瘤非恶变区(P<0.01)及非肿瘤粘膜(P<0.01)。双染原位观察p53蛋白与凋亡细胞的分布发现:p53阳性区凋亡细胞有明显减少趋势,而p53阴性区凋亡细胞分布则有增加趋势。结论:p53蛋白突变在大肠癌发展中具重要的促恶变形成作用,其作用可能是通过抑制细胞凋亡而实现的。

 
<< 更多相关文摘    
图标索引 相关查询

 


 
CNKI小工具
在英文学术搜索中查有关非肿瘤粘膜的内容
在知识搜索中查有关非肿瘤粘膜的内容
在数字搜索中查有关非肿瘤粘膜的内容
在概念知识元中查有关非肿瘤粘膜的内容
在学术趋势中查有关非肿瘤粘膜的内容
 
 

CNKI主页设CNKI翻译助手为主页 | 收藏CNKI翻译助手 | 广告服务 | 英文学术搜索
版权图标  2008 CNKI-中国知网
京ICP证040431号 互联网出版许可证 新出网证(京)字008号
北京市公安局海淀分局 备案号:110 1081725
版权图标 2008中国知网(cnki) 中国学术期刊(光盘版)电子杂志社