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肺损伤指标
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  lung injury parameters
     ② A close correlation was shown between MDA content and NO 2 -/NO 3 - content in plasma and lung. Furthermore,the content of NO 2 -/NO 3 - in lung homogenate showed strong positive correlation with the lung injury parameters.
     ②血浆、肺组织中NO2 -/NO3 -含量与MDA含量均呈正相关 ,且肺组织中NO2 -/NO3 -含量和肺损伤指标呈显著正相关。
短句来源
     There were marked upregulation of ICAM-1 mRNA expression in injuried lung tissue compared with the normal lung tissue. Furthermore, the infiltrated number of macrophage and the level of ICAM-1 expression showed strong positive correlation with the lung injury parameters, PaO 2 and LPI.
     油酸组肺组织ICAM - 1表达明显高于对照组 ,且肺组织中巨噬细胞浸润数、ICAM - 1表达水平和肺损伤指标呈显著正相关。
短句来源
     Lung injury parameters,7S portion of type Ⅳ collagen content,and gelatinase activities in BALF were measured.
     检测肺损伤指标、肺泡灌洗液中7S蛋白含量及明胶酶活性。
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  “肺损伤指标”译为未确定词的双语例句
     CONCLUSION: Partial liquid ventilation with perfluorocarbon has anti-inflammatory effect on lung lavage-induced acute lung injury in the piglets.
     结论:部分液体通气改善动物肺损伤指标,提示以氟碳化合物为呼吸媒介的部分液体通气对肺灌洗诱导的急性肺损伤家猪具有抗炎效应。
短句来源
     The content of total protein was significantly related to the γ and γ_ min of BALF, and the correlation coefficient were 0.435 (P<0.01) and -0.419(P<0.01) respectively.
     ②吸入后24hBALF中PC明显低于对照组,PE含量明显高于对照组。 以BALF中总蛋白作为肺损伤指标与PS异常的指标γ和γmin分别做直线相关分析,相关系数分别为0·435(P<0·01)和0·419(P<0·01)。
短句来源
     Hemodynamics,pulmonary mechanics and gas exchange were measured before,during and 2 min,5 min after recruitment.
     分别监测肺复张手法前 ,复张期间及复张后 2min、5min家兔血流动力学、肺力学、肺气体交换和肺损伤指标
短句来源
     Hemodynamics, pulmonary mechanics and gas exchange were observed before, during, and 2 min, 5 min after applying SI.
     在SI前、SI期间及SI后 2min和 5min监测家兔血流动力学、肺力学、肺气体交换和肺损伤指标
短句来源
     The wet dry ratio (W/D) of lung tissue, lactate dehydrogenase (LDH), total protein, surface tension (γ) and minimal surface tension (γ_ min), phosphatidyl choline (PC) and phosphatidyl ethanolamine (PE) in bronchoalveolar lavage fluid (BALF) were measured, and histopathology of lung tissue was observed.
     测定各组大鼠常压下支气管肺泡灌洗液(BALF)的表面张力(γ)、加压下最低表面张力(γmin)、磷脂酰胆碱(PC)和磷脂酰乙醇胺(PE)。 同时测定肺组织湿/干重比(W/D)、BALF中总蛋白和乳酸脱氢酶(LDH)等肺损伤指标,评价病理学改变。
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  相似匹配句对
     Radiation induced lung injury
     放射性损伤
短句来源
     acute lung injury;
     急性损伤;
短句来源
     index;
     指标;
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     and safety.
     安全性指标
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     The main experiment target include: Ca, Ma ,P, AKP, BMD, INS.
     实验指标:
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  lung injury parameters
Our results showed that lung injury parameters, including the wet/dry weight ratio and protein content in BALF, were significantly higher in the LPS alone group than in the saline control group (P>amp;lt;0.01).
      
Our results showed that lung injury parameters, including the wet/dry weight ratio and protein content in BALF, were significantly higher in the L group than in the S group (P>amp;lt;0.01).
      
Lung injury parameters in rats with different forms of aspiration as a function of time.
      


This study observed the relationship between active oxygen radicals and early lung injury after smoke inhalation injury in awake goats (N = I5) with chronic lung lymph fistular. We measured superoxide dismutase (SOD) to demonstrate the active oxygen radicals; Lung lymph flow (QL), lung lymph/plasma total protein and albumin ratio (L/P) , lung lymph total protein clearance (CL) , lung transvascu-lar protein flux, extravascuiar lung water (EVLW) , as well as PaO2 as parameters indicating lung injury.The result...

This study observed the relationship between active oxygen radicals and early lung injury after smoke inhalation injury in awake goats (N = I5) with chronic lung lymph fistular. We measured superoxide dismutase (SOD) to demonstrate the active oxygen radicals; Lung lymph flow (QL), lung lymph/plasma total protein and albumin ratio (L/P) , lung lymph total protein clearance (CL) , lung transvascu-lar protein flux, extravascuiar lung water (EVLW) , as well as PaO2 as parameters indicating lung injury.The result demonstrated that during the earlier postinjury period the injuried animals were suffering from hypoxemia, matabolic acidosis and respiratory alkalosis. EVLW and QL increased sharply during the first hour. Interstitial and alveolar edema were seen under microscope. 'These results indicated that the animals develop lung edema and acute respiratory failure. Changs of QL and EVLW were similar to L/P, CL and lung transvascular protein flux; the correlation between L/P and EVLW was significant. These data showed that the pulmonary edema was mainly a permeability one. Changes of active oxygen radicals of lung lymph fluid and arterial blood were earlier than that of venous blood; and active oxygen radicals of arterial blood was slightly higher than that of venous blood. The active oxygen radicals in pulmonary tissue and fluid of lung lavage and the lung lymph SOD clearance increased immidiatly in postinjury. These data suggested that active oxygen radicals were increased and produced in lungs. The active oxygen radicals changed with EVLW, QL, L/P and lung transvascular protein fluxi the correlation between EVLW and active oxygen radicals in pulmonary tissue, fluid of lung lavage, lung lymph SOD clearance and lung transvascular protein flux were all significant. These observations showed that early after smoke inhalation injury active oxygen radicals played a possible role in the development and progression of lung injury and pulmonary edema.

本文通过慢性肺淋巴瘘,动态观察了清醒山羊(N=15)严重烟雾吸入伤后早期肺损伤与活性氧变化的关系。实验观察6h。检测SOD活性作为反映O_2~-的变化;EVLW、QL、L/P等作为反映肺损伤的指标。 实验结果表明,伤后均较早出现严重低氧血症、代谢性酸中毒合并呼吸性碱中毒,EVLW及QL伤后1h即明显增加,光镜下见肺间质和肺泡水肿。说明实验动物伤后出现了肺水肿和急性呼吸功能不全。在EVLW及QL增加的同时,CL、L/P及肺血管蛋白漏出量也增加,EVLW与L/P的变化密切相关。提示肺水肿主要系血管通透性增加所致。伤后肺淋巴液、动脉血活性氧变化比静脉血早,动脉血活性氧水平高于静脉血;肺组织、肺灌洗液活性氧及肺淋巴SOD清除重伤后均立即增加,表明吸入伤后活性氧立即增加,且主要来自肺部。活性氧的变化高峰与肺损伤程度的加重变化规律一致,且呈线性关系。提示活性氧可能参与了烟雾吸入伤后早期肺损伤、肺水肿发生和发展的作用。

The hydrogen peroxide levels of breath condensale were measured in endotoxin-induced acute lung injury in awake sheep. The result showed that the hydrogen peroxide content in breath was significantly increased base line from 0.48±0.16μ mol/L to 0.68±0.16, 0.71±0.22, 0.86±0.23 and 0.70±0.14μmol/L at 1, 2, 4 and 6 h after endotoxin infusion respectively. We conclude that hydrogen peroxide play an important role in the endotoxin-induced acute lung injury and the hydrogen peroxide level in breath is a sensitive...

The hydrogen peroxide levels of breath condensale were measured in endotoxin-induced acute lung injury in awake sheep. The result showed that the hydrogen peroxide content in breath was significantly increased base line from 0.48±0.16μ mol/L to 0.68±0.16, 0.71±0.22, 0.86±0.23 and 0.70±0.14μmol/L at 1, 2, 4 and 6 h after endotoxin infusion respectively. We conclude that hydrogen peroxide play an important role in the endotoxin-induced acute lung injury and the hydrogen peroxide level in breath is a sensitive maker reflecting acute lung injury.

清醒绵羊内毒素急性肺损伤后,呼出气冷凝液中H_2O_2含量由致伤前0.48±0.16μmol/L升高至注射内毒素后1h 0.68±0.16μmol/L,2h 0.71±0.22μmol/L,4h 0.86±0.23μmol/L,6h 0.70±0.14μmol/L.以上表明,H_2O_2浓度与肺毛细血管通透表面积(PS)显著相关(r=0.8478.P<0.05)。H_2O_2在内毒素肺损伤起重要作用,呼出气H_2O_2检测是灵敏反映急性肺损伤的指标。

In order to identify comprehensively the effects of polymorphonuclearleukocytes(PMN )on the pathogenesis of smoke inhalation injury,a series of animalexperimental studies were performed.Chemiluminescence of PMN,the activities of PMN-NADPH oxidase,-myeloperoxidase,-elastase and the lung Schiff’s base were measured.The in vitro,observation of effects of activated PMN mediating self-body red blood cellcytotoxicity and cultured vascular endothelial cells injury were made. Some indices ofacute lung injury were also...

In order to identify comprehensively the effects of polymorphonuclearleukocytes(PMN )on the pathogenesis of smoke inhalation injury,a series of animalexperimental studies were performed.Chemiluminescence of PMN,the activities of PMN-NADPH oxidase,-myeloperoxidase,-elastase and the lung Schiff’s base were measured.The in vitro,observation of effects of activated PMN mediating self-body red blood cellcytotoxicity and cultured vascular endothelial cells injury were made. Some indices ofacute lung injury were also determined. The results further proved that PMN aggregatedin lungs,by means of releasing oxygen free radicals,proteolytic enzymes,etc,may playan important role in acute lung injury after smoke inhalation.

为全面系统地探讨中性粒细胞(PMN)在吸入性损伤发病机制中的作用,通过一系列动物实验,观察了PMN的化学发光,NADPH氧化酶、髓过氧化物酶、弹性蛋白酶活性,肺希夫氏硷,以及PMN在体外对自体红细胞、培养的血管内皮细胞的损害作用,并同时检测了多项肺损伤指标。进一步证实烟雾吸入伤后,聚集于肺内的PMN通过释放氧自由基、蛋白水解酶等,从而在肺损伤的发生发展中起着重要作用。

 
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