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肝细胞脂肪
相关语句
  liver cell fat
     Conclusions A like of diagram not typical model but like fat liver, we can try to make the " liver cell fat gradually a like of diagram" it hint, toing guide the clinical attention, the earlier period examines a patient, treatment.
     结论声像图不典型而又疑似脂肪肝者,我们可以尝试作“肝细胞脂肪浸润声像图”之提示,以指导临床注意,早期诊断、治疗。
短句来源
  “肝细胞脂肪”译为未确定词的双语例句
     TG and FIns were positive correlation with the degree of hepatocyte steatosis.
     TG,FIns与肝细胞脂肪变程度成正相关,ISI,HDL-C与肝细胞脂肪变程度成负相关。
短句来源
     In comparison with the control group,there was difference in FBG,FIns,TG,insulin sensitivity index(ISI),HDL-C and the degree of fatty liver in model group.
     与正常对照组相比,模型组大鼠的FBG,FIns,TG,高密度脂蛋白胆固醇(HDL-C),胰岛素敏感指数(ISI),肝细胞脂肪变程度与对照组相比均有差异。
短句来源
     Results The incidence of fatty degeneration among all the 3000 cases liver biopsied specimens was 12.7%, and the incidence of nonalcoholic fatty liver disease was about 6.1%.
     结果在所有3000例行肝活检的病例中,出现肝细胞脂肪变的病例约占12.7%,而NAFLD的发生率约6.1%。
短句来源
     2. promoting the convalescence of hepatocellular function;
     2、减轻肝细胞脂肪变牲;
短句来源
     Results Electroacupuncture at “Zusanli” (ST 36) could significantly reduce fatty degeneration of hepatic cells, and increase serum Chol level and decrease serum TG level as compared with the natural recovery group.
     结果 :与自然恢复组比较 ,电针“足三里”穴可显著减轻肝细胞脂肪变 ,提高血清Chol,降低血清TG。
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     2. promoting the convalescence of hepatocellular function;
     2、减轻肝细胞脂肪变牲;
短句来源
     Liver Cell Adenoma
     肝细胞腺瘤
短句来源
     3. Promoting the convalescence of hepatocellular function;
     3、促进肝细胞功能恢复,有效治疗脂肪性肝炎;
短句来源
     FAT: Extrinsic Proposition
     脂肪:外部陈述
短句来源
     ②fat;
     ②脂肪成份 ;
短句来源
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Guinea pigs were treated with chloro-butadiene,99.9% pure as determined bygas chromatography,by exposure to theconcentration of 37. 52±11. 44, 94.05±26.58,and 340. 22±83. 30mg/m~3, 2h per day for1, 3 and 6 weeks. The results were: 1. Histochemistry study of the livertissue:The amount of glycogen and theactivity of LAP,SDH, G-6PD, HKG,G-6P,5'-N,ICD, MAO decreased;the activity ofLDH and the amount of lipid increased. 2. Study of ultrastructure of the livercells: Changes were mainly hyperplasiaand dilatation of...

Guinea pigs were treated with chloro-butadiene,99.9% pure as determined bygas chromatography,by exposure to theconcentration of 37. 52±11. 44, 94.05±26.58,and 340. 22±83. 30mg/m~3, 2h per day for1, 3 and 6 weeks. The results were: 1. Histochemistry study of the livertissue:The amount of glycogen and theactivity of LAP,SDH, G-6PD, HKG,G-6P,5'-N,ICD, MAO decreased;the activity ofLDH and the amount of lipid increased. 2. Study of ultrastructure of the livercells: Changes were mainly hyperplasiaand dilatation of smooth endoplasmicreticulum of liver cells,and rough endo-plasmic reticulum and mitochondria wereinvolved. 3. Histopathology study: The mainchange was massive necrosis. 4. Serum biochemistry study: The ac-tivity of OCT, ICD, GOT, GPT and LDHwas not different from that of the control.In the individual group,albumin wassignificantly different from that in thecontrol group,but the dose-responserelation was not present. The sensitivity of the four types ofstudied marker decreased according to theabove order. The essence of liver toxicity by chloro-butadiene could be summarized as follows:injury to endoplasmic reticulum andmitochondria,disturbance of glycogenand lipid metabolism and protein synthesis,interference of mitochondrial oxidativephosphorylation and citric cycle,anda final induction of liver cell fattydegeneration.

作者对氯丁二烯所致豚鼠肝脏的损害进行了组织化学观察;肝细胞超微结构观察;组织病理学检查及血清生物化学检查。结果提示氯丁二烯所致肝损害的实质是对内质网、线粒体的损害,并影响糖、脂代谢和蛋白质合成,最终导致肝细胞脂肪变性。

Six cases of suicidal sedative-soporific drug poisoning is reported.The main clinical features were coma,

本文分析6例镇静催眠药急性中毒死亡的临床症状,法医学尸检和毒物分析资料。镇静催眠药中毒临床表现为昏迷、呼吸抑制、低血压、反射消失、周围循环衰竭等。病理变化以肺、肝、肾、脑等脏器较明显,肺淤血、水肿、肝细胞脂肪变性、肾近曲小管上皮细胞变性、脑水肿等。病变程度与服药至死亡时间长短有关,中毒死亡与药物剂量、个体差异、所患疾病及中毒后抢救所致的临床并发症等因素有关。

The paper reports a study of the anti-hypercholesterolemic and antisteatotic ef-fects of konjac-polysaccharide. The Konnya-ku powder(KP)used was natively producedand refined from the tubers of Amor-phophallus konjac K Koch, and contained84.8% of glucomannan. One hundred andtwenty Sprague-Dawley rats were dividedinto 5 groups: a basal diet group, a highcholesterol diet group and 3 test groupsfed a diet similar to that of the high chol-esterol diet group with addition of KP ata dosage of 2. 5%, 5%, or 10%, respectively.Eight...

The paper reports a study of the anti-hypercholesterolemic and antisteatotic ef-fects of konjac-polysaccharide. The Konnya-ku powder(KP)used was natively producedand refined from the tubers of Amor-phophallus konjac K Koch, and contained84.8% of glucomannan. One hundred andtwenty Sprague-Dawley rats were dividedinto 5 groups: a basal diet group, a highcholesterol diet group and 3 test groupsfed a diet similar to that of the high chol-esterol diet group with addition of KP ata dosage of 2. 5%, 5%, or 10%, respectively.Eight animals of each group were killedat 4, 8, 12 weeks after the treatment. Theresults showed that KP could markedlylower the cholesterol levels in the seraand in the livers of rats fed high choles-terol diets. At the end of the 4th week,the serum cholesterol levels of the 5% andthe 10% KP groups and the liver cho-lesterol levels of the 10% KP group wereshown to be lowered more significantlythan those of the high cholesterol dietgroup. At the end of the 12th week, serumcholesterol levels of all the three KP groupswere found to be lowered to the normallevel, and so did the liver cholesterol levelof the 10% KP group. Steatotic effect ofKP was also confirmed by the histopatho-logic examination of the livers. Fattydegeneration of the livers of all the 3 KPgroups was found to be ameliorated invarying degrees, and at the end of the12th week, no significant difference wasnoticed between the basal and the 5%,10% KP groups.

本文报告以2.5%,5%及10%三种魔芋精粉分别喂饲SD大鼠12周,同时以高胆固醇组和基础饲料组为对照。实验结果表明:4周末,5%和10%的魔芋精粉组血清胆固醇及10%魔芋精粉组肝胆固醇水平均显著低于高胆固醇组;12周末,所有魔芋精粉组血清胆固醇及10%魔芋精粉组肝胆固醇水平降至正常。病理组织学检查证实,三个魔芋精粉组的肝细胞脂肪变性均有不同程度的减轻。12周末,5%和10%魔芋精粉组肝细胞脂肪变性近于消失。

 
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