It was lower in the NGF experimental group and citicoline natrium drug control group after treatment than that in saline control group [ (0.63±0.40), (1.04±0.44), (2.61±0.38) points, (t=10.18 and 7.64, P < 0.05)].
① Percentages of infarct volume at 1 hour of ischemia and 6, 24 hours of reperfusion in N-acetylcysteine group were (8.39±2.54)%,(24.54±6.02)% respectively, and that of corresponding saline control group were (15.50±4.18)%,(32.22±3.99)%.
① Comparison of leukotriene B4 in the lung tissue of mice It was obviously lower in the saline control group than in the ovalbumin induced asthmatic model group 0.151±0.055 0.293±0.058 mg/L P < 0.01.
As compared with that in normal saline control group Fas expression in bFGF group was significantly decreased at ischemia 1 hour and reperfusion 1 6 12 24 48 and 72 hours especially at the 6th 12th and 24th hours after reperfusion t=3.954-9.327 P < 0.05.
② Comparison of nerve ectal membrane collagen fiber thickness through Masson staining at different time points after operation nerve ectal membrane collagen fiber thickness was significantly decreased at 4 and 12 weeks after operation in the hyaluronic acid treatment group as compared with normal saline control group (16.967±4.806),(10.903±3.245) μm ;
The cultured cells were divided into 8 groups as follows: the NS control group, the H2O2 group, the catechin groups (the final concentrations were 10.0,15.0, and 20.0 mg/L respectively) and the various catechin microcapsulation groups (the final concentrations were 10.0, 15.0, and 20.0 mg/L respectively).
METHODS:Ninety-eight male Sprague Dawley rats were randomly divided into normal saline group,ulcer model group,EA group,and AVP groups(injected with 100,200,300 ng AVP,re- spectively,into nucleus tractus solitarius),and AVP-V_1 receptor antagonist group.
The results showed that the recombinant plasmid pcDNA3.1-flaA was constructed, in which the bacterial colony of Legionella pneumophila in the pcDNA3.1-flaA group were significantly lower than that of the normal saline group and the pcDNA3.1(+) group(P<0.05), the pathological changes of lungs in the pcDNA3.1-flaA group showed little blank pcNDA3.1(+) group and normal saline group.
The viability of Caco-2 cells exposed to EC (1 mM), ECg (1 mM) or EGC (1mM) respectively, for 30 min, was comparable to that of the saline control group, while EGCg (1 mM) apparently enhanced cellular activity.
To study the influence of a gram-positive sepsis on the metabolism of circulating lipids, fasted rats were injected with saline (control group) or with a suspension of heat-killed or liveStaphylococcus aureus.
An intravesical catheter was inserted and 1 ml saline (control group) or the drugs 2% ultracaine in group 2, 2% lidocaine in group 3 and 0.5% bupivacaine in group 4 were administered intravesically.
Our results showed that lung injury parameters, including the wet/dry weight ratio and protein content in BALF, were significantly higher in the LPS alone group than in the saline control group (P>amp;lt;0.01).
In the LPS alone group, a larger number of neutrophils and greater MPO activity in cell-free BAL and lung homogenates were observed when compared with the saline control group (P>amp;lt;0.01).
Forty minutes after middle cerebral artery (MCA) occlusion, patches of 10?nM ET-1 (low-endothelin group), 100?nM ET-1 (high-endothelin group), or normal saline (control group) were placed on the IC of rats for a 20-min period.
Control rats without SAH also received intracisternal infusion of normal saline (control group, N?=?6).
Left wounds were treated with Zn-7 gel (test group) and right wounds were treated with normal saline (control group) twice daily.
The values were all compared statistically with the normal saline control group.