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损伤对照组
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  injured control group
     To explore the effect of Ligustrazine on the expression of adherent molecule of bone marrow cells in acute radiation injured mice, 27 Kunmin mice were randomly divided into 3 groups: Normal group, acute radiation injured control group, Ligustrazine group.
     为探讨急性放射损伤小鼠骨髓细胞粘附分子表达及川芎嗪对其影响,将27只昆明小鼠平均分为3组:正常组,急性放射损伤对照组,川芎嗪组。
短句来源
     The rats in the hyperbaric oxygen treatment group and injured control group were killed at hour 48 after injury.
     高压氧治疗组、损伤对照组大鼠均在受伤后48h处死。
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     ②Head injury models were established in the hyperbaric oxygen treatment group and injured control group. After opening bone window,consistent contusion and laceration of brain at right parietal lobe was established with free fall method,with the impact of 50 g×13 cm.
     ②高压氧治疗组、损伤对照组大鼠建立颅脑损伤模型,开放骨窗后采用自由落体法造成右顶叶较为一致的脑挫裂伤,冲击力为50g×13cm。
短句来源
     METHODS: The experiment was performed at the Department of Pathology,Norman Bethune University of Medical Sciences from June 2004 to June 2005. ①A total of 60 adult SD male rats were selected and divided into 3 groups randomly: hyperbaric oxygen treatment group,injured control group and sham operation group with 20 in each group.
     方法:实验于2004-06/2005-06在白求恩医科大学病理科完成。 ①选择成年SD雄性大鼠60只,随机数字表法分为高压氧治疗组、损伤对照组、假手术组,20只/组。
短句来源
     METHODS: The experiment was done in the Animal Experiment Center, Southern Medical University from April to May 2003. Forty clean grade male SD rats were selected and divided randomly into injured control group, aminoguanidin group, 7-nitroindazole (7-NI) group and normal control group with 10 in each group.
     方法:实验于2003-04/05在南方医科大学动物实验中心完成。 选取清洁级雄性SD大鼠40只,随机分为损伤对照组、氨基胍组、7-硝基吲唑组和正常对照组,每组10只。
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  injury control group
     ④Change of nerve cell apoptosis at different time points after transplantation of BMSCs apoptosis rate of nerve cells of cortex at the injured side of the cell transplantation was significantly decreased on day 7 14 and 21 days after transplantation as compared with injury control group (17.26±2.71),(11.78±2.52);
     ④骨髓基质细胞移植后不同时间点伤侧皮质神经细胞凋亡的变化:与损伤对照组比较,细胞移植组伤侧大脑皮质神经细胞凋亡率在移植后7,14,21d明显降低犤(17.26±2.71),(11.78±2.52);
短句来源
     Methods Rats were randomized into three groups: Group S (sham operation group), Group C (balloon injury control group) and Group L (balloon injury + L-arginine group).
     方法 21只大鼠随机分为S组(假手术组),C组(球囊损伤对照组)及L组(球囊损伤+L-精氨酸组)各组 n= 7。
短句来源
     ②Totally 30 adult SD rats were divided into 3 groups at random: injury control group, early transplantation group and postpone transplantation group, 10 in each group.
     ②将30只成年SD大鼠按随机数字表法分为3组,即损伤对照组、早期移植组和延期移植组,每组10只。
短句来源
     Injury control group was give n no irradiation.
     损伤对照组5只不做照射。
短句来源
     Meanwhile,the Bcl-2 protein expression of the Microcapsule grafted group reached its peak 7 d after SCIand maintains its high level until 2 weeks later, however, the Bcl-2expression of injury control group decreased dramatically 3 d after injury.
     免疫组织化学法发现治疗组对损伤脊髓细胞Bcl-2蛋白表达具有显著的促进作用,7d高度表达持续时间达2周以上; 损伤对照组、空囊组神经元Bcl-2蛋白表达的高峰在第3d,随后持续下降。
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  sci control group
     The amplitude in the SCI treatment group was significantly lower than that in the SCI control group[(3.78±1.32)μV vs (9.85±0.84) μV, t=-11.194,P < 0.01].
     损伤治疗组波幅降低度明显低于损伤对照组犤(3.78±1.32)μV,(9.85±0.84)μV,t=-11.194,P<0.01犦。
短句来源
     Having been induced to SCI, the rhesuses in SCI treatment group were injected with phosphate buffered saline(PBS) suspension 200 μL, consisting of neuron-like cells(3.0×106 cells/kg) and GDNF(2 μg) enwrapped by methoxy polyethylene glycol-polylactic acid(mPEG-b-PLA) block copolymer, and those in SCI control group were injected with PBS with the same volume as above.
     制作猴脊髓损伤模型后损伤治疗组植入3.0×106个/kg神经元样细胞与含2μg胶质细胞源性神经营养因子的单甲氧基聚乙二醇聚乳酸嵌共聚体用200μL磷酸盐缓冲液制成悬液,损伤对照组给予等体积磷酸盐缓冲液;
短句来源
     ②Shown by pathological results, the colloid was slightly hyperplastic in the SCI treatment group, and neurons of posterior horn was found few accompanied by infiltration of colloid in the SCI control group.
     ②病理结果表明,损伤治疗组后角轻度胶质增生; 损伤对照组后角神经元少见,胶质浸润。
短句来源
     ④True blue positive cells could been found in cerebral somatic sensory cortex in the SCI treatment group under fluorescence microscope, but not found in the SCI control group.
     ④荧光显微镜下,损伤治疗组大脑皮质躯体感觉区(对侧)存在蓝色荧光阳性细胞,损伤对照组无此现象。
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  “损伤对照组”译为未确定词的双语例句
     The HeLa cells were randomly divided into six groups:control group,UVB radiated groups(radiated control,0.5%PCF,1%PCF,2%PCF,1%Vit C).
     HeLa上皮细胞随机分为6组,未辐射对照组、辐射损伤组犤损伤对照组、0.5%扇贝多肽(PCF)组、1%PCF组、2%PCF组、1%维生素C组犦。
短句来源
     The AChE activity in the experimental group and the control group was reduced 1,3 day after nerve injury (P>0 05).
     结果表明术后1,3天实验组(指损伤给药组)和对照组(指损伤对照组)AChE活性均下降(P>005);
短句来源
     The experiment was carriedout in 4 groups: Undamaged Group, UVA damaged Control Group, UVA+10 g/L Biological Membrane Group, and UVA+10 g/L Vitamin C Group.
     Hela上皮细胞实验设计分为 4组 ,未损伤组、UVA损伤对照组、UVA +10 g/L生物膜组、UVA +10 g/L维生素C组。
短句来源
     The expression of GAP-43 mRNA and ET-1 mRNA was higher in brain tissue during 6~72 h after diffuse brain lesion compared with sham injury group(P<0.01).
     伤后6~72h脑组织GAP-43mRNA及ET-1mRNA均较假损伤对照组增高(P<0.01)。
短句来源
     Methods 20 adult female Sprague-Dawley rats were divided randomly into the spinal cord injury group (n=10) and the control group (n=10).
     方法20只雌性成年Sprague-Dawley大鼠,分为脊髓损伤组(n=10)和损伤对照组(n=10)。
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Effects of intraventricular administration of nerve growth factor (NGF), ganglioside mixture (GM) or monosialoganglioside (GMl) on changes of acetylcholine (ACh), choline acetyltransferase (ChAT) and cholinesterase (ChE) in the hippocampus of rats with unilateral partial septo-hippocampal transection were studied. The neurotrophic factors were injected at 0 and 30 minutes after lesion. On the 7th day after the lesion, biochemical assays confirmed that all the three neurotrophic factors did not increase ChE activity...

Effects of intraventricular administration of nerve growth factor (NGF), ganglioside mixture (GM) or monosialoganglioside (GMl) on changes of acetylcholine (ACh), choline acetyltransferase (ChAT) and cholinesterase (ChE) in the hippocampus of rats with unilateral partial septo-hippocampal transection were studied. The neurotrophic factors were injected at 0 and 30 minutes after lesion. On the 7th day after the lesion, biochemical assays confirmed that all the three neurotrophic factors did not increase ChE activity in the denervated hippocampus. ACh contents in the ipsilateral hippocampus retained 34.9%, 35.3% and 47.7% respectively in NGF, GM and GMl treated rats, whereas it was 20.3% in lesioned controls. ChAT activity retained 77.4%, 78.4% and 69.2% respectively, whereas it was 50% in lesioned controls.All the three neurotrophic factors induced increases in ACh content and ChAT activity in the hippocampus of the lesioned side, suggesting that treatments with NGF, GM and GM 1 have protective effects at early stage of rat septo-hippocampal lesion.

为评价神经生长因子(NGF)、混合型神经节苷脂(GM)和单唾液酸神经节苷脂(GM1)对中枢胆碱能神经损伤早期的影响,在大鼠单侧隔-海马通路部分损伤后即时经脑室分别注入上述三种神经元营养因子,7d后取两侧海马分别测定乙酰胆碱(ACh)、胆碱乙酰基转移酶(ChAT)和胆碱酯酶(ChE)。损伤对照组(脑室注入盐水)术侧海马ACh含量保留率为对侧的20.3%,ChAT活力为50%,ChE活力为48.3%。给予NGF、GM或GM1的实验组,ACh含量保留率分别为34.9%,35.3%和47.7%;ChAT活力为77.4%,78.4%和69.2%;而ChE活力的保留率未见明显改变。这些神经元营养因子显著增加了大鼠隔-海马通路损伤后海马内ACh含量和ChAT活力,说明它们减轻了损伤侧海马胆碱能神经纤维的破坏,具有明显的损伤早期保护作用。

Using method of intracellular recording miniature endplate potentials (MEPP) and resting membrane potentials (RMP), the axonal regeneration after a crush lesion, the testing lesion (TL), and the effect of conditioning lesion (CL) 16 days prior to TL were observed in preparation of peroneus nerve-extensor digitorum longus muscle of rats. The results showed that 1-10 days after TL, the presynaptic spontaneous transmitter release disappeared completely and RMP of the muscle fibers was reduced continuously. 14 days...

Using method of intracellular recording miniature endplate potentials (MEPP) and resting membrane potentials (RMP), the axonal regeneration after a crush lesion, the testing lesion (TL), and the effect of conditioning lesion (CL) 16 days prior to TL were observed in preparation of peroneus nerve-extensor digitorum longus muscle of rats. The results showed that 1-10 days after TL, the presynaptic spontaneous transmitter release disappeared completely and RMP of the muscle fibers was reduced continuously. 14 days after TL, MEPP of low frequency and prolonged duration reappeared. RMP was partially restored. By 30 days after TL, the frequency and duration of MEPP still not returned to normal, being 60% and 169% of control respectively, and RMP was only 84% of control values. In CL group, MEPP reappeared on 10th day after TL, being earlier than TL alone. In the following days its frequency and duration and RMP recovered quickly, being about 78%, 120% and 97% of control respectively on 30th day after TL. It is considered that CL can accelerate spontaneous MEPP reappearance and RMP restoration during axonal regeneration.

在大鼠腓神经伸趾长肌标本上,用微电极记录小终板电位(MEPP)和静息膜电位(RMP),观察腓神经损伤后的再生过程和条件损伤效应。结果表明:损伤后1~10d突触前递质自发释放完全消失,RMP幅度变小。到14d才出现频率慢波幅变宽的MEPP,30d仍未恢复正常,其频率和时程分别为正常的60%和169%。而RMP幅度仅为正常的84%。条件损伤组在损伤后30d MEPP的频率、时程和RMP幅度分别为正常时的78%、120%和97%,均比损伤对照组恢复快。说明条件损伤在运动神经的再生过程中可促进自发MEPP的出现和RMP的恢复。

In this study, carbontetrachloride was administered orally to male Wistarrats at a dose of 2.5ml/kg body weight. The protective effects of RSM were comparedwith that of verapamil (a well-known calcium antagonist), and regitine (a vasodilator),to study the mechanisms of RSM action. The results showed that a significant correlation between CCl_4-induced liver tissuenecrosis and liver tissue calcium content(r=0.887, P<0.01). In both RSM group andverapamil group the liver tissue calcium content, plasma GPT activity,...

In this study, carbontetrachloride was administered orally to male Wistarrats at a dose of 2.5ml/kg body weight. The protective effects of RSM were comparedwith that of verapamil (a well-known calcium antagonist), and regitine (a vasodilator),to study the mechanisms of RSM action. The results showed that a significant correlation between CCl_4-induced liver tissuenecrosis and liver tissue calcium content(r=0.887, P<0.01). In both RSM group andverapamil group the liver tissue calcium content, plasma GPT activity, liver tissue MDAcontent were all much lower and liver cytochrome P-150 was higher than that in CCl_4injured group (P<0.01). RSM was more effective than verapamil in protecting liver cellfrom CCl_4-induced hepatocellular necrosis. Morphological and biochemical changs of regi-tine group were similar to those of CCl_4 injured group. The above results suggested that protective effects of RSM on CCl_4-induced liver cellnecrosis were not via vasodilation but by calcium antagonism of RSM.

本工作在大鼠口服CCL_4所致肝细胞坏死模型上,比较了丹参与钙拮抗剂异搏定和扩血管药酚妥拉明对肝坏死的保护作用。结果发现,CCl_4引起的肝组织坏死与肝组织钙含量呈正相关(r=0.887,P<0.01)。丹参和异搏定使肝组织坏死程度减轻,肝组织钙含量减少,血浆GPT活性下降,肝组织匀浆MDA含量降低,细胞色素P-450活性升高。酚妥拉明组与损伤对照组比较,各项指标无明显差异。上述结果提示,丹参的作用效果优于异搏定。丹参抗CCl_4所致的肝细胞坏死井非通过扩血管作用,而是发挥其钙拮抗作用实现的。

 
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