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   损伤对照组 在 外科学 分类中 的翻译结果: 查询用时:0.118秒
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损伤对照组
相关语句
  injured control group
    The rats in the hyperbaric oxygen treatment group and injured control group were killed at hour 48 after injury.
    高压氧治疗组、损伤对照组大鼠均在受伤后48h处死。
短句来源
    Results Compared to the spinal cord injured control group, the expression of NOS in the anterior horn of the transected spinal cord was increased in embryonic transplanted group (P<0.01), the locomotor functions of the transplated rats improved as well compared to the injured control.
    结果 胚胎大鼠脊髓移植后可以使全横断脊髓前角内NOS表达增加 ,与损伤对照组比较有显著性差异 (P <0 .0 1) ; 大鼠后肢的自主运动功能恢复好于损伤对照组
短句来源
    METHODS: The experiment was done in the Animal Experiment Center, Southern Medical University from April to May 2003. Forty clean grade male SD rats were selected and divided randomly into injured control group, aminoguanidin group, 7-nitroindazole (7-NI) group and normal control group with 10 in each group.
    方法:实验于2003-04/05在南方医科大学动物实验中心完成。 选取清洁级雄性SD大鼠40只,随机分为损伤对照组、氨基胍组、7-硝基吲唑组和正常对照组,每组10只。
短句来源
    ① In the normal tissue there was the content of nitrogen monoxide and NOS, which showed the trend of increasing after injury. That in the injured control group was the most significant, and the difference was significant compared with other 3 groups (P < 0.05).
    ①正常组织中即存在一氧化氮含量和一氧化氮合酶,损伤后呈升高趋势,以损伤对照组最明显,与其他3组相比差异明显(P<0.05)。
短句来源
    ② The action potential in the 7-NI group and aminoguanidin group had significant recovery than that in the injured control group, while there was little clear wave form appeared in the injured control group;
    ②7-硝基吲唑组和氨基胍组动作电位较损伤对照组有明显恢复,而损伤对照组则基本没有明确的波形出现;
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  injury control group
    ②Totally 30 adult SD rats were divided into 3 groups at random: injury control group, early transplantation group and postpone transplantation group, 10 in each group.
    ②将30只成年SD大鼠按随机数字表法分为3组,即损伤对照组、早期移植组和延期移植组,每组10只。
短句来源
    Injury control group was give n no irradiation.
    损伤对照组5只不做照射。
短句来源
    ④Change of nerve cell apoptosis at different time points after transplantation of BMSCs apoptosis rate of nerve cells of cortex at the injured side of the cell transplantation was significantly decreased on day 7 14 and 21 days after transplantation as compared with injury control group (17.26±2.71),(11.78±2.52);
    ④骨髓基质细胞移植后不同时间点伤侧皮质神经细胞凋亡的变化:与损伤对照组比较,细胞移植组伤侧大脑皮质神经细胞凋亡率在移植后7,14,21d明显降低犤(17.26±2.71),(11.78±2.52);
短句来源
    Meanwhile,the Bcl-2 protein expression of the Microcapsule grafted group reached its peak 7 d after SCIand maintains its high level until 2 weeks later, however, the Bcl-2expression of injury control group decreased dramatically 3 d after injury.
    免疫组织化学法发现治疗组对损伤脊髓细胞Bcl-2蛋白表达具有显著的促进作用,7d高度表达持续时间达2周以上; 损伤对照组、空囊组神经元Bcl-2蛋白表达的高峰在第3d,随后持续下降。
短句来源
    ②The rats of cell transplantation group and injury control group were injured by free-falling instrument of Feeney to establish the animal models. There was no management in the normal control group.
    ②细胞移植组和损伤对照组采用改进Feeney’s自由落体装置建立大鼠创伤性脑损伤模型,正常对照组不做处理,假伤组仅开骨窗不至伤。
短句来源
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  sci control group
    The amplitude in the SCI treatment group was significantly lower than that in the SCI control group[(3.78±1.32)μV vs (9.85±0.84) μV, t=-11.194,P < 0.01].
    损伤治疗组波幅降低度明显低于损伤对照组犤(3.78±1.32)μV,(9.85±0.84)μV,t=-11.194,P<0.01犦。
短句来源
    Having been induced to SCI, the rhesuses in SCI treatment group were injected with phosphate buffered saline(PBS) suspension 200 μL, consisting of neuron-like cells(3.0×106 cells/kg) and GDNF(2 μg) enwrapped by methoxy polyethylene glycol-polylactic acid(mPEG-b-PLA) block copolymer, and those in SCI control group were injected with PBS with the same volume as above.
    制作猴脊髓损伤模型后损伤治疗组植入3.0×106个/kg神经元样细胞与含2μg胶质细胞源性神经营养因子的单甲氧基聚乙二醇聚乳酸嵌共聚体用200μL磷酸盐缓冲液制成悬液,损伤对照组给予等体积磷酸盐缓冲液;
短句来源
    ②Shown by pathological results, the colloid was slightly hyperplastic in the SCI treatment group, and neurons of posterior horn was found few accompanied by infiltration of colloid in the SCI control group.
    ②病理结果表明,损伤治疗组后角轻度胶质增生; 损伤对照组后角神经元少见,胶质浸润。
短句来源
    ④True blue positive cells could been found in cerebral somatic sensory cortex in the SCI treatment group under fluorescence microscope, but not found in the SCI control group.
    ④荧光显微镜下,损伤治疗组大脑皮质躯体感觉区(对侧)存在蓝色荧光阳性细胞,损伤对照组无此现象。
短句来源
  “损伤对照组”译为未确定词的双语例句
    Method:Seventy-two SD rats were randomly divided into sham group(A),normal saline group(B) and ChABC group(C) with each group of twenty-four.
    方法:SD大鼠72只,雌雄不限,随机分为假手术组(A组)、损伤对照组(B组)和ChABC治疗组(C组),每组24只。
短句来源
    Group 3(n=24) was divided into 3 subgroups: non-injured group(n=8), vehicle-treated group (n=8)to whom Saline was applied and CsA - treated group(n=8).
    第3组大鼠(n=24)随机分为正常对照组(n=8)、损伤对照组(n=8)和CsA治疗组(n=8),以Morris水迷宫实验和避暗实验测定伤后不同时间点 DA大鼠学习记忆能力的变化,研究 CsA的治疗作用。
短句来源
    3. Behavior rehab of experimental group of implanted HHK was well than that of no implanted HHK.
    3 证实了植入HHK实验组的恢复早于单一损伤对照组,其恢复程度也比同一时期单一损伤对照组好。
短句来源
    3. Compared with the ACI control, ET concentration in plasma and injuried cortex at each time group is decreased after HBO therapy(p<0.05).
    3.动物急性颅脑损伤经高压氧治疗后6h,24h,48h各时间组血浆及损伤皮层组织内皮素含量均明显低于相应急性颅脑损伤对照组(p<0.05);
短句来源
    Control group(C);
    损伤对照组(C);
短句来源
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Objective:To study the effects of Kupffer cells on the lipid peroxide reaction in rat liver,induced by galactosamine(GalN),with silica(SiO 2) as depressor and triglyceride(TG) as stimulator.Methods:The rats were divided at random into control,GalN,SiO 2 and TG goups.All animals were killed 30 hours after administration of GalN.The lipid peroxide(MDA),superoxide dismutase(SOD),glutathione(GSH) in liver and endotoxin(ET) in plasma were deteremined.Results:MDA content in the liver injuried group was much higher...

Objective:To study the effects of Kupffer cells on the lipid peroxide reaction in rat liver,induced by galactosamine(GalN),with silica(SiO 2) as depressor and triglyceride(TG) as stimulator.Methods:The rats were divided at random into control,GalN,SiO 2 and TG goups.All animals were killed 30 hours after administration of GalN.The lipid peroxide(MDA),superoxide dismutase(SOD),glutathione(GSH) in liver and endotoxin(ET) in plasma were deteremined.Results:MDA content in the liver injuried group was much higher than that of the control group( P <0.05),whereas SOD and GSH were lower.In SiO 2 group,MDA content was decreased and SOD,GSH content increased,and in the TG group,the changes in MDA,SOD and GSH content were not significant,but decreased in level of plasma ET.Conclusions:The results indicated that Kupffer cells could both engulf endotoxin and aggravate lipid peroxide reaction,therefore,the clinical treatment with either Kupffer cell depressor or stimulator should be used according to the difference in hepatic injury.

目的:探讨Kupfer细胞对实验性肝损伤脂质过氧化反应的影响。方法:用半乳糖胺(GalN)诱发大鼠肝损伤,分别给予Kupfer细胞吞噬功能促进剂甘油三酯(TG)或抑制剂二氧化硅(SiO2),并设正常和损伤对照组。给GalN30h后处死动物,测定血浆及肝组织有关指标。统计学处理,用F和H检验。结果:损伤组肝组织/g脂质过氧化物(MDA)含量明显高于正常组(P<0.05),超氧化物歧化酶(SOD)和还原型谷胱甘肽(GSH)含量低于正常组(P<0.005,P<0.001),血浆转氨酶(ALT)含量明显高于正常组(P<0.05);与损伤组比较,SiO2组MDA含量明显下降(P<0.05),SOD和GSH含量增加(P<0.01,P<0.05);ALT含量下降(P<0.05);TG组动物肝组织/gMDA、SOD、GSH及血浆ALT含量与损伤组无差异;但内毒素血症明显减轻(P<0.01)。结论:Kupfer细胞激活剂或抑制剂有益于治疗不同程度的肝损伤。

Sprague-Dawley rats were divided into three groups; normal group, 0. 9% sodium chloride solution-treated control group and GM1 treated group. The results showed that mito-chondrial respiratory function significantly decreased in control group on 8th and 16th hour after injury, while markedly increased in GMj treated group, which were similar to those in normal group. Cortical neurons and their mitochondrial ultrastructure of control group were severely damaged, but the pathological changes in GMj treated group...

Sprague-Dawley rats were divided into three groups; normal group, 0. 9% sodium chloride solution-treated control group and GM1 treated group. The results showed that mito-chondrial respiratory function significantly decreased in control group on 8th and 16th hour after injury, while markedly increased in GMj treated group, which were similar to those in normal group. Cortical neurons and their mitochondrial ultrastructure of control group were severely damaged, but the pathological changes in GMj treated group were much milder. The possible mechanism is that GM1 protects changes in the plasma membrane hpids, and membrane enzyme activity and reduces ionic imbalance, edema and free radical formation.

把SD大鼠分成正常组、生理盐水治疗的损伤对照组及GM_1治疗的损伤治疗组。结果发现对照组伤后8h和16h。脑线粒体呼吸功能明显降低,治疗组则明显好转。对照组皮层神经元细胞和线粒体超微结构有明显损害,治疗组损害明显减轻。其可能机制与GM_1保护膜脂和膜酶活性,维持膜内外离子平衡,减轻水肿及减少自由基形成等有关。

Aim To study the effect of microgene pSVPoMcat implanted to genetically modify Schwann cells (SC) on apoptosis after spinal cord injury (SCI). Methods Experimental animals were divided into three groups: the group of microgene pSVPoMcat implanted to genetically modify SC (group A), SC implanted group (group B) and the control group (group C). The apoptosis of spinal slice from the injured spinal cord were examined by the methods of methye green, terminal deoxynucleotidyl and the dUTP nick end labelling technique,...

Aim To study the effect of microgene pSVPoMcat implanted to genetically modify Schwann cells (SC) on apoptosis after spinal cord injury (SCI). Methods Experimental animals were divided into three groups: the group of microgene pSVPoMcat implanted to genetically modify SC (group A), SC implanted group (group B) and the control group (group C). The apoptosis of spinal slice from the injured spinal cord were examined by the methods of methye green, terminal deoxynucleotidyl and the dUTP nick end labelling technique, and the expression of Bc1 2 and Fas protein were measured immunohistochemically 12 weeks post operation. Result The rate of apoptosis sequence, the strong expression sequence of Bc1 2 and Fas protein expression were found in all three groups, but C>B>A ( P <0.05), A>B>C ( P <0.05), and C>B>A ( P <0.05), respectively. Conclusion It indicates that the pSVPoMcat implanted to genetically modify SC can inhibit the apoptosis, and trauma can induce apoptosis after SCI.

目的探讨pSVPoMcat微基因修饰雪旺氏细胞(SC)移植对脊髓损伤(SCI)后细胞凋亡的作用。方法将实验动物分为pSVPoMcat微基因修饰SC组(A组),SC移植组(B组),损伤对照组(C组)。移植后12周,对SCI区的脊髓组织切片进行细胞凋亡的检测(甲基绿-派诺宁染色法和荧光原位标记法)以及Bc1~2和Fas蛋白表达的测定(免疫组化法)。结果在A、B、C组中,均发现凋亡细胞及Bc1~2和Fas蛋白阳性表达。体视学计量发现,细胞凋亡率为C>B>A;Bc1~2蛋白阳性表达顺序为A>B>C;Fas蛋白阳性表达顺序为C>B>A。结论pSVPoMcat微基因修饰SC移植能抑制SCI后的细胞凋亡,而创伤则可诱发细胞凋亡

 
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