助手标题  
全文文献 工具书 数字 学术定义 翻译助手 学术趋势 更多
查询帮助
意见反馈
   为此作用 的翻译结果: 查询用时:0.355秒
图标索引 在分类学科中查询
所有学科
更多类别查询

图标索引 历史查询
 

为此作用
相关语句
  this effect
     Conclusion:Ketamine can reduce the neurons apoptosis and bax/bclˉ2value after cerebral ischemiaˉreperfusion in gerbils. This effect of it might be an important mechanism which protects the cerebrum from ischemiaˉreperfusion damage.
     结论 :氯胺酮减少沙土鼠脑缺血再灌注损伤后细胞的凋亡 ,bax/bcl 2比值较再灌注组相对降低可能为此作用的机理之一
短句来源
  “为此作用”译为未确定词的双语例句
     Conclusions Ketamine decreased neuron apoptosis after cerebral ischemia/reperfusion injury. This may be related to the suppressed expressions of caspase-3 and c-fos by ketamine.
     结论 氯胺酮减少沙土鼠脑缺血 -再灌流损伤后细胞的凋亡 ,其对caspase - 3激活和c -fos表达的抑制可能为此作用的机制之一
短句来源
  相似匹配句对
     Thus,P.
     为此P.
短句来源
     Affect of Role Intrusion Detection System
     入侵检测系统的作用
短句来源
     Penetration effect
     侵彻作用
短句来源
     So the theories can play more active role in researches and applications generally.
     从而为此技术的研究和推广应用起到了积极的作用
短句来源
     This article gives a comprehensive analyis about the madical effect of copper element.
     为此,对铜元素的药理作用进行了全面分析
短句来源
查询“为此作用”译词为用户自定义的双语例句

    我想查看译文中含有:的双语例句
例句
为了更好的帮助您理解掌握查询词或其译词在地道英语中的实际用法,我们为您准备了出自英文原文的大量英语例句,供您参考。
  this effect
Our theoretical description, which is carried out in contrast with coherent light and two-photon state sources, demonstrates that this effect can be explained in accordance with the Hanbury-Brown and Twiss experiment.
      
This effect is used to investigate the dynamic process of nanocompartment switching and molecular thermosensing, as well as perform molecular recognition.
      
This effect of oxytocin may be mediated through the vagus and sympathetic nerve, and then lead to the reduction of gastric juice output and the depression of gastric acidity.
      
This effect was not associated with protein degradation or modification of the spectrum of secreted proteins.
      
Our studies and data found in the literature suggest that this effect is due to the synthesis of sugars from amino acids by gluconeogenesis.
      
更多          


Objective:To investigate the effect of ketamine on neurons apoptosis after cerebral ischemiˉaˉreperfusion in gerbils.Methods:Thirty gerbils were randomly assigned to3groups:shamˉoperative(SO)group,ischemiaˉreperfusion(IR)group and ketamine treatment(K)group.Apoptosis was detected by termiˉnalˉdeoxynucleotidyl transfarase mediated nick end labeling(TUNEL),and bax,bclˉ2were detected by imˉmunohistochemistry.Results:Compared with SO group,there were more TUNEL and bax positive cells in IR and K groups(P<0.01).There...

Objective:To investigate the effect of ketamine on neurons apoptosis after cerebral ischemiˉaˉreperfusion in gerbils.Methods:Thirty gerbils were randomly assigned to3groups:shamˉoperative(SO)group,ischemiaˉreperfusion(IR)group and ketamine treatment(K)group.Apoptosis was detected by termiˉnalˉdeoxynucleotidyl transfarase mediated nick end labeling(TUNEL),and bax,bclˉ2were detected by imˉmunohistochemistry.Results:Compared with SO group,there were more TUNEL and bax positive cells in IR and K groups(P<0.01).There was no obvious difference of the numbers of bclˉ2positive cells among the3groups(P>0.05).Conclusion:Ketamine can reduce the neurons apoptosis and bax/bclˉ2value after cerebral ischemiaˉreperfusion in gerbils.This effect of it might be an important mechanism which protects the cerebrum from ischemiaˉreperfusion damage.

目的 :研究氯胺酮对沙土鼠脑缺血再灌注损伤后细胞凋亡状态及相关基因bax、bcl 2表达的影响。方法 :沙土鼠全脑缺血模型 ,缺血10min ,再灌注24h。分为假手术组、缺血再灌注组和氯胺酮组。采用TUNEL法原位标记DNA片段检测凋亡细胞 ,采用SP法进行bax、bcl 2的免疫组织化学染色。结果 :缺血再灌注组及氯胺酮组脑组织中TUNEL及bax阳性细胞数明显多于假手术组(P<0 .01) ;氯胺酮组阳性细胞数目明显低于缺血再灌注组(P<0 .01)。bcl 2阳性细胞数3组间比较无明显差异。结论 :氯胺酮减少沙土鼠脑缺血再灌注损伤后细胞的凋亡 ,bax/bcl 2比值较再灌注组相对降低可能为此作用的机理之一

Objective To investigate the effect of ketamine on neurons apoptosis and expression of caspase-3 and c-fos after cerebral ischemia/reperfusion in gerbils.Methods Thirty gerbils were randomly assigned to three groups:sham operated group(SO,n=10),ischemia/reperfusion group(IR,n=10),and ketamine treatment group(K,n=10).The bilateral common carotid arteries of the gerbils were isolated.In group IR,the bilateral common carotid arteries were occluded with aneurysm clips for 10min.The clips were released to...

Objective To investigate the effect of ketamine on neurons apoptosis and expression of caspase-3 and c-fos after cerebral ischemia/reperfusion in gerbils.Methods Thirty gerbils were randomly assigned to three groups:sham operated group(SO,n=10),ischemia/reperfusion group(IR,n=10),and ketamine treatment group(K,n=10).The bilateral common carotid arteries of the gerbils were isolated.In group IR,the bilateral common carotid arteries were occluded with aneurysm clips for 10min.The clips were released to resume global blood flow.In group K,ketamine(10mg/kg) was injected intrapenitoneally 30 minutes before arteries were occluded.And in group SO,gerbils were operated without occluding the arteries.All the gerbils were anesthetized with sodium pentobarbital,and the brain were removed 24h after operation.Apoptosis were detected by terminal-deoxynucleotidyl transferase mediated nick end labeling(TUNEL),and caspase-3 and c-fos were detected by immunohistochemistry.Results Apoptotic neurons and the expressions of caspase-3 and c-fos in groups IR and K increased significantly,compared with those in group SO( P <0 01).Apoptotic neurons and the expression of caspase-3 and c-fos in group K were significantly decreased than those in group IR( P <0 01).Conclusions Ketamine decreased neuron apoptosis after cerebral ischemia/reperfusion injury.This may be related to the suppressed expressions of caspase-3 and c-fos by ketamine.

目的 研究氯胺酮对沙土鼠脑缺血 -再灌流损伤后细胞凋亡状态及相关基因caspase - 3、c-fos表达的影响。方法 沙土鼠随机分为假手术组、缺血 -再灌流组和氯胺酮组 ,各 10例。缺血 -再灌流组 :分离双侧颈总动脉 ,用无创动脉夹阻断脑血流 10min后松开动脉夹恢复血流。假手术组 :仅分离双侧颈总动脉但不予阻断。氯胺酮组 :血流阻断前 30min腹腔内注射氯胺酮 10mg/kg,余同缺血 再灌流组。采用缺口末端标记法 (TUNEL)原位标记DNA片段检测凋亡细胞 ,免疫组化SP法进行caspase - 3、c -fos的免疫组织化学染色。结果 缺血 -再灌流组及氯胺酮组脑组织中TUNEL及caspase- 3、c-fos阳性细胞数明显多于假手术组 (P <0 0 1) ;氯胺酮组 3项指标阳性细胞数目均明显低于缺血 -再灌流组 (P <0 0 1)。结论 氯胺酮减少沙土鼠脑缺血 -再灌流损伤后细胞的凋亡 ,其对caspase - 3激活和c -fos表达的抑制可能为此作用的机制之一

 
图标索引 相关查询

 


 
CNKI小工具
在英文学术搜索中查有关为此作用的内容
在知识搜索中查有关为此作用的内容
在数字搜索中查有关为此作用的内容
在概念知识元中查有关为此作用的内容
在学术趋势中查有关为此作用的内容
 
 

CNKI主页设CNKI翻译助手为主页 | 收藏CNKI翻译助手 | 广告服务 | 英文学术搜索
版权图标  2008 CNKI-中国知网
京ICP证040431号 互联网出版许可证 新出网证(京)字008号
北京市公安局海淀分局 备案号:110 1081725
版权图标 2008中国知网(cnki) 中国学术期刊(光盘版)电子杂志社