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效应t细胞
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  effector t cell
     The strongest E749-57 peptide-specific, IFN-γsecreting effector T cell response can be generated by this kind of prime-boost strategy.
     ELISPOT可以检测到高水平的E749-57肽特异性,分泌IFN-γ的效应T细胞
短句来源
     IL-21 is a new member of the IL-2 family,mainly produced by the activated CD4+ T cells. It has extensive biological activities,especially enhancing the functions of the effector T cell and activated NK cell,including the enhancement of cytoxity and the production of IFN-γ,which can effectively strengthen the innate immunity,specific immunity and promote the tumor-bear animal to produce anti-tumor immunity.
     IL-21是IL-2家族中的新成员,主要是由活化的CD4+T细胞分泌的,具有广泛的生物学功能,主要表现为免疫调节功能,其中最显著的特征是能增强效应T细胞及活化的NK细胞的功能,包括增强胞毒活性及促使分泌IFNγ,从而有效增强机体的天然免疫以及特异性免疫,促使荷瘤动物产生较强的抗肿瘤免疫效应。
短句来源
  t cells in vitro induced
     Conclusion: NY-ESO-1 protein can be processed by HCC cells, and that HLA-A2 restricted NY-ESO-1b peptide (157-165) can also be presented on the surfaces of cells and recognized by NY-ESO-1b specific CD8+ T cells in vitro induced.
     结论HepG2细胞中表达的NY-ESO-1蛋白可被此肝癌细胞有效地加工处理,而且HLA-A2限制性的抗原肽NY-ESO-1157-165也可被有效地提呈到细胞表面,从而被效应T细胞所识别。
短句来源
  “效应t细胞”译为未确定词的双语例句
     The number of developing IFN-γ+T stimulated by HSPgp96-peptide complex was maximum (287.50± 22.34),followed by the number of developing IFN-γ+T stimulated by peptide (135.25± 12.80),PHA (102.25± 14.76) and blank control (35.50± 11.10).
     γ-干扰素ELISPOT检测,HSPgp96肽复合物组产生γ-干扰素效应T细胞频数最高(287.50±22.34),以后依次为单纯多肽组(135.25±12.80)、阳性对照组组(102.25±14.76)、阴性对照组(35.50±11.10),各组与阴性对照组比较差异有统计学意义(P<0.05);
短句来源
     The frequency of IFN-γ +T cells stimulated by HSPgp96-peptide complex and polypeptide derived from two different renal tissues had significant difference (P<0.05).
     两组中HSPgp96 肽复合物与单纯多肽产生γ 干扰素效应T细胞频数存在明显差异(P<0.05)。
短句来源
     Results: HSPgp96-peptide complex from renal carcinoma produced maximal frequency of IFN-γ +T cells (287.6±22.3 per hole). The HSPgp96-peptide complex and polypeptide from renal carcinoma produced more frequency of IFN-γ +T cells than that from normal renal tissues (P<0.01).
     结果:肿瘤组中 HSPgp96 肽复合物产生γ 干扰素效应 T细胞频数最高[每孔(287.6±22.3)个],肿瘤组中HSPgp96 肽复合物,单纯多肽产生γ 干扰素效应 T细胞频数明显高于正常肾组织组(P<0.01)。
短句来源
     The influx of cytotoxic T cells into A/WSN influenza virus-infected mouse lungs was investigated by adoptive transfer with 1251-5-iodo-2'-deoxyuridine (125IUdR) labelled syngeneic cells.
     流感病毒A/WSN株免疫小鼠的次代效应T细胞,经~(125)I-UdR标记,转输给同种病毒感染的同系鼠,T细胞进入感染肺的数量为未感染肺的2.5~5倍;
短句来源
     Enzyme-linked immunospot assay (Elispot) was used to detect the frequency of interferon(IFN)-γ +T cell produced by peripheral blood mononuclear cells(PMBCs), which stimulated by HSPgp96-peptide complex and polypeptide derived from human carcinoma and normal renal tissues.
     应用酶联免疫斑点(ELISPOT)法检测肾癌患者肿瘤组织,正常肾组织中提取的HSPgp96 肽复合物、单纯多肽刺激人外周血单核细胞(PMBC)产生γ 干扰素效应T细胞频数。
短句来源
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  相似匹配句对
     the effector cell of RTE is T cells.
     RTE的效应细胞T细胞 ;
短句来源
     Effect of Single α-Particle on Cells
     单个α-粒子的细胞效应
短句来源
     Central Memory T Cells and Effector Memory T Cells
     中枢记忆性T细胞效应记忆性T细胞
短句来源
     Electric Field Effect of Cell Calcium
     细胞中钙离子的电场效应
短句来源
     T lymphocyte is the main effector cell of cellular immunity.
     T淋巴细胞细胞免疫的主要效应细胞
短句来源
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  effector t cell
Galectin-1 contributes to the suppression of intestinal inflammation by the induction of effector T cell apoptosis.
      
"Variant A2" responder cells cocultivated with "common A2"-bearing stimulators gave rise to effector T cell lines which recognized only the "common A2"-bearing subgroup of targets.
      
Although such a tumor-specific tolerant state was long-lasting, the recovery of anti-X5563 effector T cell responses was observed when the above ordinary immunization procedure was performed 6 months after the tolerance induction.
      
The role in uveitis of a recently discovered IL-17-producing effector T cell type, Th17, is being intensively studied.
      
Taken together, naive T cells in asthma have a peculiar function to produce TRAC and MDC, which contribute to local migration of Th2 cells into lung and lymphoid tissues, along with a function as precursor for memory/effector T cell.
      
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The influx of cytotoxic T cells into A/WSN influenza virus-infected mouse lungs was investigated by adoptive transfer with 1251-5-iodo-2'-deoxyuridine (125IUdR) labelled syngeneic cells. More A/WSN virus-immune secondary effe-ctor T cells localized in the A/WSN virus-infected lungs than in the uninfected lungs, the ratios being in the range 2.5-5.0. Non-immune control cells, in contrast, showed no significant difference between localization pattern of infected and uninfected lungs. Virus immune T cells of different...

The influx of cytotoxic T cells into A/WSN influenza virus-infected mouse lungs was investigated by adoptive transfer with 1251-5-iodo-2'-deoxyuridine (125IUdR) labelled syngeneic cells. More A/WSN virus-immune secondary effe-ctor T cells localized in the A/WSN virus-infected lungs than in the uninfected lungs, the ratios being in the range 2.5-5.0. Non-immune control cells, in contrast, showed no significant difference between localization pattern of infected and uninfected lungs. Virus immune T cells of different antigenic specificities, i.e., Sendai or Semliki Forest virus-immune secondary effector T cells, however, also localized more in A/WSN virus-infected than in uninfected lungs, but the heterologous virus-immune T cells were retained in the A/WSN virus-infected lungs for a shorter time than A/WSN virus-immune secondary effector T cells

流感病毒A/WSN株免疫小鼠的次代效应T细胞,经~(125)I-UdR标记,转输给同种病毒感染的同系鼠,T细胞进入感染肺的数量为未感染肺的2.5~5倍;未免疫的对照T细胞进入感染及未感染肺的数量则无明显差别。抗原性与流感无关的仙台病毒及Smliki森林病毒免疫效应T细胞,进入流感感染鼠肺的数量亦远高于未感染鼠肺,表现为非特异性,但异种病毒效应T细胞存留时间较同种病毒效应T细胞短暂。

Objective To investigate the role of T lymphocyte subsets and the adhesion molecules in the pathogenesis of the experimental allergic neuritis (EAN). Methods The animal model was induced in rats by immunization with myelin emulsion of rabbit′s sciatic nerve. The Antibody to intercellular adhesion molecule 1 (ICAM 1) was injected to the rats before inducing of EAN. The manifestations of the animals and the pathological changes were observed and compared with the group which was not injected with the antibody...

Objective To investigate the role of T lymphocyte subsets and the adhesion molecules in the pathogenesis of the experimental allergic neuritis (EAN). Methods The animal model was induced in rats by immunization with myelin emulsion of rabbit′s sciatic nerve. The Antibody to intercellular adhesion molecule 1 (ICAM 1) was injected to the rats before inducing of EAN. The manifestations of the animals and the pathological changes were observed and compared with the group which was not injected with the antibody and with the control group. The distributions of CD4 positive T cells and CD8 positive T cells and the expressions of adhesion molecules on the infiltrating T cells in blood and peripheral nerve tissue.Results (1) The treatment with monoclonal antibody to ICAM 1 suppressed the active EAN. The histological sections of the peripheral nerve tissue showed a pronounced reduction of the inflaminatory infiltrates treated with the antibody to ICAM 1. The semi thin sections also confirmed the same finding. (2) The animals treated with antibody to ICAM 1 showed lower expressions of adhesion molecules on CD4 + cells in the impaired tissue but not in the peripheral blood than the naturally attacked group. (3) There are prevalently higher expressions of CD54 and CD11a on CD4 + cells but not CD8 + cells in the impaired tissue than peripheral blood. (4) The impaired tissue showed higher CD4 +/CD8 + in the naturally attacked animals than in those pretreated with the antibody to ICAM 1, though the peripheral blood showed differeat results. Conclusions (1) CD4 positive T cells acted primarily in the immune response of EAN. (2) The espressions of ICAM in CD4 positive T cells might be remarkably involved in the pathogenesis of EAN. (3) The ICAM 1 antibody to adhesion molecules might be of use in the approach to the autoimmune disease such as EAN.

目的探讨T细胞亚群及粘附分子在实验性变态反应性神经炎(EAN)中的作用。方法用兔坐骨神经匀浆免疫Wistar大鼠,建立EAN模型;同时用抗细胞间粘附分子-1(ICAM-1)单抗注入大鼠后再诱导EAN。观察自然病程组、抗体注射组及对照组动物的临床病理。用双重酶标免疫组化技术检测CD4+、CD8+T细胞分布以及粘附分子CD54、CD11a、CD62在CD4+及CD8+细胞上的表达。结果抗体注射组发病率及发病程度明显低于自然病程组;组织中粘附分子在CD4+细胞上的表达及CD4+/CD8+自然病程组高于抗体注射组;CD54、CD11a在CD4+细胞上的表达高于CD8+细胞。结论CD4+细胞是主要的效应细胞,CD4+细胞上粘附分子的表达对效应T细胞进入病变组织起主导作用;抗ICAM-1抗体能够预防EAN发生。

Objective:To prolong the allograft survival time in immunized mice with immature dendritic cell, and to analyze the mechanism of hypo allo immuno responsiveness induced by immature dendritic cells Methods:Based on mouse cardiac allograft model, the survival of cardiac recipients immunized with mature or immature DC were observed Meanwhile the CTL activity of splenocytes from immature DC immunized recipients was detected Results:The survival time of cardiac allograft was substantially prolonged and the...

Objective:To prolong the allograft survival time in immunized mice with immature dendritic cell, and to analyze the mechanism of hypo allo immuno responsiveness induced by immature dendritic cells Methods:Based on mouse cardiac allograft model, the survival of cardiac recipients immunized with mature or immature DC were observed Meanwhile the CTL activity of splenocytes from immature DC immunized recipients was detected Results:The survival time of cardiac allograft was substantially prolonged and the mean survival time extended from 9 1±0 73 days to 25 4±4 27 days It became more effective if those immunized mice were treated in combination with adriamycin application, the mean survival time of allograft was extended to 30 5±3 98 days It was proved that the CTL activity in spleen cells from the mice immunized with immature DCs was much lower (specific release was 16 32%) than that from the immunized mice with mature DCs (specific release was 39 58%) Conclusion:Immature DCs could induce prolongation of allograft survival time It may be possible that low CTL activity in recipients immunized with immature DCs promised this prolongation

目的 :利用未成熟DC免疫受鼠诱导同种移植物存活延长 ,分析未成熟DC在诱导同种免疫低应答反应中的可能机理。方法 :以小鼠耳后心肌移植为模型 ,观察未成熟DC和成熟DC免疫受鼠同种移植心肌的存活情况。同时检测未成熟DC免疫的小鼠脾细胞对同种细胞的CTL活性。结果 :未成熟DC免疫受鼠的同种移植物存活时间明显延长 ,平均存活期从9 1± 0 73d延长到 2 5 4± 4 2 7d ,联合使用阿霉素效果更好 ,平均存活期为 30 5± 3 98d ;C5 7BL/ 6未成熟DC免疫的Balb/c小鼠脾细胞对C5 7BL/ 6靶细胞的杀伤 (3 H TdR释放率为 16 32 % )明显低于经成熟DC免疫的受鼠的脾细胞 (3 H TdR释放率为39 5 8% )。结论 :未成熟DC免疫可诱导同种移植心肌的存活明显延长 ,可能与受鼠体内的效应T细胞对移植物细胞杀伤低下有关。

 
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