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肝细胞脂肪变性
相关语句
  hepatocyte fatty degeneration
     Morphological Investigation on the Model of the Hepatocyte Fatty Degeneration
     肝细胞脂肪变性模型形态研究
短句来源
     Morphological studies on effect of alcohol,serum,CCl_4 on the hepatocyte fatty degeneration
     乙醇、血清、四氯化碳对肝细胞脂肪变性形态影响的研究
短句来源
     AIM: To study the model of the drug-induced hepatocyte fatty degeneration.
     目的 :对药物导致肝细胞脂肪变性模型进行研究。
短句来源
     Morphology of the model of the hepatocyte fatty degeneration was studied by the scanning electron microscope(SEM.)
     肝细胞脂肪变性形态学的模型研究,国内外罕有报道,也没有成熟的模型可用。
短句来源
  “肝细胞脂肪变性”译为未确定词的双语例句
     ⑤The hepatic steatosis of the model group was more severe compared with the control group (P < 0.05),the training group was obviously lighter than the model group (P < 0.05),but still more serious than the control group (P < 0.05).
     ⑤模型组肝细胞脂肪变性程度显著重于对照组(P<0.05),运动组显著轻于模型组(P<0.05),但仍然显著比对照组严重(P<0.05)。
短句来源
     Histopathologically, betaine significantly decreased the degree of steatosis (P<0.05 or P<0.01),and betaine 0.8 g/kg had a tendency to normalize basically the structure of hepatocyte.
     组织病理学检查结果显示,甜菜碱可使肝细胞脂肪变性程度明显减轻(P<0.05或P<0.01),0.8g/kg组的肝细胞结构趋于正常。
短句来源
     However, TP and EGCG treatment improved histological changes, and significantly decreased the levels of plasma endotoxin and serum TNF-α, IL-1, IL-18, as well as the expression of CD14, LBP, TNF-α, IL-1 and IL-18 mRNA (P < 0.05 or P < 0.01). Fatty degeneration was still observed in TP and EGCG treatment group, but focal necrosis and inflammatory cell infiltration disappeared.
     茶多酚、EGCG高低剂量分别同时处理显著降低了酒精性肝损伤大鼠血清ALT、TNF-α、IL-1、IL-18与血浆内毒素水平及肝组织CD14、LBP、TNF-α、IL-1、IL-18mRNA的表达(P<0.05或P<0.01),肝组织仍可见肝细胞脂肪变性,但未见坏死及炎性细胞浸润.
短句来源
     In rats treated with 45 and 150 mg/kg/day, sperm counts were decreased and there was a greater incidence of sperm morphological abnormalities.
     氟吗啉使45mg/kg/day和150 mg/kg/day剂量组的雄鼠、雌鼠发生肝脏毒性,引起肝细胞脂肪变性
短句来源
     After administerd repeatedly for 180 days,in the 9.0 g/kg dose groups,the TP and Crea increased(P<(0.01)orP<0.05);
     2只大鼠肝细胞脂肪变性。 给药180 d时,9.0 g/kg和4.5 g/kg剂量组肌酐升高(P<0.01或P<0.001);
短句来源
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  相似匹配句对
     2. Decreasing fatty degeneration of liver cell;
     2、减轻肝细胞脂肪变性
短句来源
     2. Alleviate the fat denaturatiDn of the liver cells.
     2、减轻肝细胞脂肪变性
短句来源
     Liver Cell Adenoma
     肝细胞腺瘤
短句来源
     (4) hepatocyte injury was ameliorated.
     (4)肝细胞损伤减轻。
短句来源
     7.obviousl fat degeneration of liver;
     ⑦肝脏显著脂肪变性;
短句来源
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Guinea pigs were treated with chloro-butadiene,99.9% pure as determined bygas chromatography,by exposure to theconcentration of 37. 52±11. 44, 94.05±26.58,and 340. 22±83. 30mg/m~3, 2h per day for1, 3 and 6 weeks. The results were: 1. Histochemistry study of the livertissue:The amount of glycogen and theactivity of LAP,SDH, G-6PD, HKG,G-6P,5'-N,ICD, MAO decreased;the activity ofLDH and the amount of lipid increased. 2. Study of ultrastructure of the livercells: Changes were mainly hyperplasiaand dilatation of...

Guinea pigs were treated with chloro-butadiene,99.9% pure as determined bygas chromatography,by exposure to theconcentration of 37. 52±11. 44, 94.05±26.58,and 340. 22±83. 30mg/m~3, 2h per day for1, 3 and 6 weeks. The results were: 1. Histochemistry study of the livertissue:The amount of glycogen and theactivity of LAP,SDH, G-6PD, HKG,G-6P,5'-N,ICD, MAO decreased;the activity ofLDH and the amount of lipid increased. 2. Study of ultrastructure of the livercells: Changes were mainly hyperplasiaand dilatation of smooth endoplasmicreticulum of liver cells,and rough endo-plasmic reticulum and mitochondria wereinvolved. 3. Histopathology study: The mainchange was massive necrosis. 4. Serum biochemistry study: The ac-tivity of OCT, ICD, GOT, GPT and LDHwas not different from that of the control.In the individual group,albumin wassignificantly different from that in thecontrol group,but the dose-responserelation was not present. The sensitivity of the four types ofstudied marker decreased according to theabove order. The essence of liver toxicity by chloro-butadiene could be summarized as follows:injury to endoplasmic reticulum andmitochondria,disturbance of glycogenand lipid metabolism and protein synthesis,interference of mitochondrial oxidativephosphorylation and citric cycle,anda final induction of liver cell fattydegeneration.

作者对氯丁二烯所致豚鼠肝脏的损害进行了组织化学观察;肝细胞超微结构观察;组织病理学检查及血清生物化学检查。结果提示氯丁二烯所致肝损害的实质是对内质网、线粒体的损害,并影响糖、脂代谢和蛋白质合成,最终导致肝细胞脂肪变性

Six cases of suicidal sedative-soporific drug poisoning is reported.The main clinical features were coma,

本文分析6例镇静催眠药急性中毒死亡的临床症状,法医学尸检和毒物分析资料。镇静催眠药中毒临床表现为昏迷、呼吸抑制、低血压、反射消失、周围循环衰竭等。病理变化以肺、肝、肾、脑等脏器较明显,肺淤血、水肿、肝细胞脂肪变性、肾近曲小管上皮细胞变性、脑水肿等。病变程度与服药至死亡时间长短有关,中毒死亡与药物剂量、个体差异、所患疾病及中毒后抢救所致的临床并发症等因素有关。

The paper reports a study of the anti-hypercholesterolemic and antisteatotic ef-fects of konjac-polysaccharide. The Konnya-ku powder(KP)used was natively producedand refined from the tubers of Amor-phophallus konjac K Koch, and contained84.8% of glucomannan. One hundred andtwenty Sprague-Dawley rats were dividedinto 5 groups: a basal diet group, a highcholesterol diet group and 3 test groupsfed a diet similar to that of the high chol-esterol diet group with addition of KP ata dosage of 2. 5%, 5%, or 10%, respectively.Eight...

The paper reports a study of the anti-hypercholesterolemic and antisteatotic ef-fects of konjac-polysaccharide. The Konnya-ku powder(KP)used was natively producedand refined from the tubers of Amor-phophallus konjac K Koch, and contained84.8% of glucomannan. One hundred andtwenty Sprague-Dawley rats were dividedinto 5 groups: a basal diet group, a highcholesterol diet group and 3 test groupsfed a diet similar to that of the high chol-esterol diet group with addition of KP ata dosage of 2. 5%, 5%, or 10%, respectively.Eight animals of each group were killedat 4, 8, 12 weeks after the treatment. Theresults showed that KP could markedlylower the cholesterol levels in the seraand in the livers of rats fed high choles-terol diets. At the end of the 4th week,the serum cholesterol levels of the 5% andthe 10% KP groups and the liver cho-lesterol levels of the 10% KP group wereshown to be lowered more significantlythan those of the high cholesterol dietgroup. At the end of the 12th week, serumcholesterol levels of all the three KP groupswere found to be lowered to the normallevel, and so did the liver cholesterol levelof the 10% KP group. Steatotic effect ofKP was also confirmed by the histopatho-logic examination of the livers. Fattydegeneration of the livers of all the 3 KPgroups was found to be ameliorated invarying degrees, and at the end of the12th week, no significant difference wasnoticed between the basal and the 5%,10% KP groups.

本文报告以2.5%,5%及10%三种魔芋精粉分别喂饲SD大鼠12周,同时以高胆固醇组和基础饲料组为对照。实验结果表明:4周末,5%和10%的魔芋精粉组血清胆固醇及10%魔芋精粉组肝胆固醇水平均显著低于高胆固醇组;12周末,所有魔芋精粉组血清胆固醇及10%魔芋精粉组肝胆固醇水平降至正常。病理组织学检查证实,三个魔芋精粉组的肝细胞脂肪变性均有不同程度的减轻。12周末,5%和10%魔芋精粉组肝细胞脂肪变性近于消失。

 
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