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肝细胞脂肪变性
相关语句
  hepatocyte fatty degeneration
    Morphological Investigation on the Model of the Hepatocyte Fatty Degeneration
    肝细胞脂肪变性模型形态研究
短句来源
    Morphological studies on effect of alcohol,serum,CCl_4 on the hepatocyte fatty degeneration
    乙醇、血清、四氯化碳对肝细胞脂肪变性形态影响的研究
短句来源
    AIM: To study the model of the drug-induced hepatocyte fatty degeneration.
    目的 :对药物导致肝细胞脂肪变性模型进行研究。
短句来源
    Morphology of the model of the hepatocyte fatty degeneration was studied by the scanning electron microscope(SEM.)
    肝细胞脂肪变性形态学的模型研究,国内外罕有报道,也没有成熟的模型可用。
短句来源
  “肝细胞脂肪变性”译为未确定词的双语例句
    Levels of serum ALT,AST,MDA were much lower in GSH group than in NASH group and levels of liver SOD,GSH were much higher in GSH group than NASH group (P﹤0.05).
    肝脏广泛肝细胞脂肪变性,仅有轻微的炎性细胞浸润,未见坏死灶及纤维组织增生。 (4)GSH组大鼠血清TG、FFA及肝组织TG、FFA、ATP、TNFα与NASH组相比无明显变化,血清AST、ALT及MDA显著低于NASH组(P﹤0.05),肝组织GSH、SOD显著高于病理对照组(P﹤0.05);
短句来源
    To establish a model of hepatocyte steatosis, we incubate the normal human L-02 hepatocyte in RPMI-1640 complete medium with 50% fetal bovine serum for 24 hours.
    用50%胎牛血清的RPMI-1640完全培养基孵育人肝L-02细胞24小时,制备肝细胞脂肪变性模型。
短句来源
    Results Plasma PAI 1 activity increased in model group at 6 and 12 weeks, and was significantly higher than normal group at 12 weeks. PAI 1 mRNA relative values of hepatic tissue were also higher than normal group (3.474±0.051 vs 1.210±0.031, P <0.01).
    结果 与正常组相比 ,模型组家兔血浆脂质和PAI 1活性在实验 6周时即显著增高 ,实验结束时模型组肝脏呈重度肝细胞脂肪变性 ,肝组织PAI 1mRNA表达较正常组显著增强。
短句来源
    ①Lobules of liver in the rats of model group was disorder with a pile of deposition of fibrous tissue, inflammatory cell infiltration and fatty degeneration of hepatocytes.
    ①模型组大鼠肝小叶结构紊乱,有大量纤维组织沉积,炎症细胞浸润,肝细胞脂肪变性
短句来源
    Histopathologically, betaine significantly decreased the degree of steatosis (P<0.05 or P<0.01),and betaine 0.8 g/kg had a tendency to normalize basically the structure of hepatocyte.
    组织病理学检查结果显示,甜菜碱可使肝细胞脂肪变性程度明显减轻(P<0.05或P<0.01),0.8g/kg组的肝细胞结构趋于正常。
短句来源
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IM To study the protective effect of the traditional Chinese medicine, Ganyanping(GYP) on chronic liver injury induced by CCl_4.METHODS Wistar rats (n=25) were divided into control (n=5), model (n=10, NaCl+CCl_4) and experimental groups (n=10, GYP+CCl4). Liver histopathology was observed under light microscope. Serum cytokines including IL6, IL8 and TNFα, were detected.RESULTS Serum levels of IL6 (μg/L), IL8 (μg/L) and TNF α (μg/L), were 223±054, 445±186, 608±182 in model group whereas...

IM To study the protective effect of the traditional Chinese medicine, Ganyanping(GYP) on chronic liver injury induced by CCl_4.METHODS Wistar rats (n=25) were divided into control (n=5), model (n=10, NaCl+CCl_4) and experimental groups (n=10, GYP+CCl4). Liver histopathology was observed under light microscope. Serum cytokines including IL6, IL8 and TNFα, were detected.RESULTS Serum levels of IL6 (μg/L), IL8 (μg/L) and TNF α (μg/L), were 223±054, 445±186, 608±182 in model group whereas 064±011, 179±052 and 082±034 in experimental group, the difference being significant (P<001).CONCLUSION GYP can significantly lower the serum levels of TNFα, and IL6 and IL8, and inhibit liver fiberosis.

目的探讨中药肝炎平对肝纤维化的防治作用及其对各种细胞因子的影响.方法用CCl4制备大鼠肝纤维化的动物模型,对病理形态学及血清中各种细胞因子包括:TNFα,IL6,IL8浓度的变化进行观察.结果模型组(n=10),IL6(μg/L),IL8(μg/L)及TNFα分别是223,445及608,而肝炎平组(n=10),仅为064,179及082,两组相比有显著性差异(P<001).病理组织提示,模型组正常肝小叶结构被破坏,有假小叶形成,肝细胞肿胀,呈水变性,部分肝细胞脂肪变性可见肝细胞的点状坏死.而肝炎平组的肝细胞的脂肪变性和水变性明显减轻,未见明显的假小叶形成.结论肝炎平可显著降低血清中TNFα,IL6和IL8的水平,减轻肝组织纤维化的形成.

Objective To study the PAI 1 gene expression of hepatic tissue and plasma PAI 1 activity in hyperlipidemia and fatty liver. Methods Rabbit model of hyperlipidemia and fatty liver were induced by high fat diet for 12 weeks, the plasma PAI 1 activity and hepatic PAI 1 mRNA expression were measured ( n =10) with colorimetry and RT PCR, another 7 rabbits were taken as normal control. Results Plasma PAI 1 activity increased in model group at 6 and 12 weeks, and was significantly higher than normal...

Objective To study the PAI 1 gene expression of hepatic tissue and plasma PAI 1 activity in hyperlipidemia and fatty liver. Methods Rabbit model of hyperlipidemia and fatty liver were induced by high fat diet for 12 weeks, the plasma PAI 1 activity and hepatic PAI 1 mRNA expression were measured ( n =10) with colorimetry and RT PCR, another 7 rabbits were taken as normal control. Results Plasma PAI 1 activity increased in model group at 6 and 12 weeks, and was significantly higher than normal group at 12 weeks. PAI 1 mRNA relative values of hepatic tissue were also higher than normal group (3.474±0.051 vs 1.210±0.031, P <0.01). Conclusion Hepatic PAI 1 gene expression of rabbit with hyperlipidemia and fatty liver was increased, and could be the main cause of high plasma PAI 1.

目的 探讨高脂血症脂肪肝肝组织纤溶酶原激活物抑制物 1型 (PAI 1)mRNA的表达以及血浆PAI 1活性变化。方法 通过持续 12周的高脂饮食建立家兔高脂血症脂肪肝模型 ,分别利用发色底物法和RT PCR法测定模型组 (n =10 )血浆PAI 1活性及肝脏PAI 1mRNA的表达 ,并设正常饮食组 (n =7)作对照。结果 与正常组相比 ,模型组家兔血浆脂质和PAI 1活性在实验 6周时即显著增高 ,实验结束时模型组肝脏呈重度肝细胞脂肪变性 ,肝组织PAI 1mRNA表达较正常组显著增强。结论 高脂血症脂肪肝家兔肝脏PAI 1基因表达增强 ,且可能是高PAI 1血症的主要来源

OBJECTIVE To investigate the effects of tetrandrine on hepatic fibrosis in rats. METHODS The rats were randomly divided into some groups.Liver fibrosis was induced in rats by subcutaneous injection of 50% carbone tetrachloride oil solution for a period of 14 weeks. The treatment groups were treated with tetrandrine twice a week for 14 weeks. Histopathological examination was made in 4?th ,8?th,14?th week after experiment.A quantity of collagenous fibers were determined by using color image analysis system....

OBJECTIVE To investigate the effects of tetrandrine on hepatic fibrosis in rats. METHODS The rats were randomly divided into some groups.Liver fibrosis was induced in rats by subcutaneous injection of 50% carbone tetrachloride oil solution for a period of 14 weeks. The treatment groups were treated with tetrandrine twice a week for 14 weeks. Histopathological examination was made in 4?th ,8?th,14?th week after experiment.A quantity of collagenous fibers were determined by using color image analysis system. RESULTS Hepatic steatosis gradually increased from 4?th week and decrease in 14?th week. Hepatic fibrosis gradually increase from 4th week and pseudo lobules are formed in 14th week. Hepatic steatosis and fibrosis in treatment groups are slighter than those in toxic group.A quantity of collagenous fibers in liver tissue are significantly lower in treatment groups than in toxic group. The effects of tetrandrine at 50?mg/kg dose are better than those at 30?mg/kg. CONCLUSION Tetrandrine has good effects in the treatment of hepatic steatosis and fibrosis induced by carbone tetrachloride oil in rats.

[目的 ]观察汉防己甲素的抗肝纤维化的作用 .[方法 ]将 72只大鼠随机分组 .用 5 0 %四氯化碳油剂皮下注射 14周制作大鼠肝纤维化模型 .治疗组用汉防己甲素灌胃 ,连续 14周 ,第 4,8,14周取肝组织作切片 ,着重观察肝细胞脂肪变性、纤维化程度 ,并利用彩色图象分析系统测量胶元纤维的面积 .[结果 ]中毒组肝细胞脂肪变性从第 4周开始逐渐加重 ,第 14周有所减轻 ,肝纤维化从第 4周开始逐渐加重 ,第 14周形成假小叶 ;治疗组肝细胞脂肪变性、肝纤维化程度比中毒组轻 ,肝组织内胶原含量明显减少 ,与中毒组比较有显著性差异 .汉防己甲素的疗效每kg体重 5 0mg组优于 30mg组 .[结论 ]汉防己甲素有较好的抗肝细胞脂肪变性及抗肝纤维化的作用

 
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