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机制是
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  mechanism for
     Conclusion Methylation of the CDKN2/p16 gene 5′ CpG island may be an important mechanism for gene inactivation in lung cancer progression.
     结论 CDKN2 /p16基因 5′端CpG岛甲基化是该基因失活的重要机制 ,是肺癌细胞区别于正常细胞的分子事件之一 ,可能参与肺癌的发生发展过程
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     For N-type Hg_(1-x)Cd_xTe MIS devices, it is revealed that the dominating dark current mechanism for T<130K is the indirect tunneling current via band-gap states and that for T>130K is the generation-ecombination current.
     对于N型Hg_(1-x)Cd_xTeMIS器件,当温度T<130K时,占优势的暗电流机制是通过禁带态的间接隧道电流;
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     TEM and SEM analysis show that α-Si_3N_4 nanowires have the diameters in the range of 100-150nm and with the lengths extending to several tens of microns. A growth mechanism for nanowires is attributed to a solid-gas process.
     TEM和SEM分析显示,合成的-αSi3N4纳米线直径为100~150 nm,长几十μm,-αSi3N4纳米线的生长机制是气固(VS)生长机制.
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     Ontology provides semantic expression mechanism for Web information and is the key technology of Semantic Web.
     而Ontology为Web信息提供了语义表示机制,是实现语义Web的关键技术。
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     It is essential to design QoS supported MAC mechanism for supporting QoS in wireless LAN.
     支持QoS的MAC机制是WLAN支持QoS的关键所在.
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  “机制是”译为未确定词的双语例句
     Results from the investigation indicate that physical mechanisms of drain current collapse and maximum transconductance reduction are different underdifferent bias stresses.
     研究表明,不同电应力条件下,导致漏电流崩塌和最大跨导下降的物理机制不同,在大电场应力下,主要物理机制是栅隧穿电子填充表面态;
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     Based on the analysis of the current problems of library resource sharing, the paper points out that market mechanism is the reasonable and confirmative driving force , and puts forward the new library resource sharing pattern leaded by market mechanism.
     文章在分析目前图书馆资源共享存在的问题之基础上,指出市场机制是实现图书馆资源共享合理的、巩固的驱动力,并构建了市场机制引导下的图书馆资源共享新模式。
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     CONCLUSION: Etoposide-induced Jurkat cell apoptosis is initiated through mitochondria signaling pathway with cytochrome c release into cytoplasm and caspase is the ultimate executioner of cell apoptosis.
     结论:依托泊苷诱导Jurkat白血病细胞株凋亡的机制是降低线粒体膜电位和释放细胞色素c到细胞浆启动线粒体信号转导通路,最终激活caspase而导致细胞凋亡。
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     Conclusions GS affords protection against IL-1βinduced inflammatory mediators PGE_2 production in HOC by regulating the expression of their respective enzymes.
     结论GS抑制HOC在IL-1β诱导下分泌炎性介质PGE_2,其机制是下调调控它们表达的COX-2 mRNA和蛋白的表达;
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     Conclusion Ultrasound combined with HP has definite killing effect on human lung cancer lines NCI-446,PLA-801C and PLA-801D in vitro through inducting apoptosis.
     结论:超声激活HP对体外培养的人肺癌细胞NCI-446、PLA-801C和PLA-801D有明确的杀伤效应,其发生机制是通过诱导细胞凋亡。
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     P and mechanism of increased INa.
     P 的机制
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     Developing Mechanism
     发展机制
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     What is the physiological mechanism of the changes?
     其生理学机制什么?
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     How is Occurred mechanism?
     发生机制什么?
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  mechanism for
It was suggested that the mechanism for the eruption of heat was that La3+ ion damages the outer cell membrane and increases its permeability and the proton-electron potential energy across the cell membrane was reduced or couldn't even be initiated.
      
The implementation mechanism for a question answering system is presented in this paper and is based on concepts and statistics.
      
The mechanism for polarized variability is so simple that only mechanical rotation is needed.
      
The difficulty is how to clarify the underlying mechanism for fluctuations of Q10 values for one specific habitat and element (e.g.
      
On the basis of the requirements presented for the integration design, measuring mechanism for the robotic end trephine's force and cutting depth are studied.
      
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The observation of Koshtoyantz that urea has beneficial effect on muscle fatigued by indirect stimulation, has been confirmed. This"defatigue" effect really only represents a removal of conduction block at the neuromuscular junction. On fatigue of the muscle fibres itself urea has no ameliorating effect. To obtain the above mentioned"defatigue" effect of urea requires rather strict experimental conditions; in particular, the Ringer used to soak the muscle and to prepare the urea solution must contain a small...

The observation of Koshtoyantz that urea has beneficial effect on muscle fatigued by indirect stimulation, has been confirmed. This"defatigue" effect really only represents a removal of conduction block at the neuromuscular junction. On fatigue of the muscle fibres itself urea has no ameliorating effect. To obtain the above mentioned"defatigue" effect of urea requires rather strict experimental conditions; in particular, the Ringer used to soak the muscle and to prepare the urea solution must contain a small amount of phosphate and bicarbonate and a relatively low concentration of calcium. Cysteine not only has no similar"defatigue" effect, but when present together with urea even tends to suppress the"defatigue" effect of the latter. Another contrast between urea and cysteine is that while cysteine can remove the neuromuscular block due to cadmium chloride, urea can not. Neuromuscular block resulting from lack of sodium ions or a certain ionic inbalance in the medium can also be partially removed by urea. Soaking in urea solution causes the muscle to respond repetitively to single stimulus, whether indirect or direct. The hypothesis that the"defatigue" effect of urea on the muscle results from the release of the SH groups of some proteins, has been examined and found to be at present very uncertain. The authors are indebted to Prof. T. P. Feng for guidance and encouragement throughout this work.

對於肌肉在接連的間接刺激中所發展的疲乏,脲有解除作用,半胱氨酸沒有。這實際上只是對神經肌肉接頭的傳導阻遏的解除,對於肌纖維本身的疲乏,脲並不能減輕。要得到脲對於神經肌肉接頭的疲乏的解除作用,試驗須具備一定的條件,特别是用以浸潤肌肉和配製脲溶液的任氏溶液,必須含有少量的重碳酸根或磷酸根離子並有適宜的鈣離子濃度。脲的處理能使肌肉纖維對單個刺激作重複反應,不論刺激是間接的或直接的。 對於由於電解質缺乏或不平衡而起的神經肌肉接頭的傳導阻遏,脲亦有解除作用。但氯化鎘引起的傳導阻遏,脲不能解除,而半胱氨酸則能。 關於脲作用的機制,本文有所討論,指出釋放硫氫基的假說能否成立,尚属疑問。

In chronical experiments on dogs provided with gastric pouches, we showed that the gastric secretion stimulated by histamine can be reflexly inhibited by electrical stimulation on the skin. This inhibition can be shown with Heidenhain pouches as well as with Pavlov pouches, indicating that the phenomenon does not depend on an intact vagus innervation. Furthermore it can still appear in Heidenhain pouches after bilateral subdiaphragmatic section of the splanchnic nerves and bilateral extirpation of the solar...

In chronical experiments on dogs provided with gastric pouches, we showed that the gastric secretion stimulated by histamine can be reflexly inhibited by electrical stimulation on the skin. This inhibition can be shown with Heidenhain pouches as well as with Pavlov pouches, indicating that the phenomenon does not depend on an intact vagus innervation. Furthermore it can still appear in Heidenhain pouches after bilateral subdiaphragmatic section of the splanchnic nerves and bilateral extirpation of the solar plexuses. In such dogs the adrenal glands are of course also severed from their external nerve supplies. It thus follows that the sympathetic innervation to the stomach and the adrenal glands are not indispensable either. Apparently there is some humoral factor as yet unknown, which inhibits gastric secretion and which can be reflexly mobilized by such means as electrical stimulation of the skin. On the other hand, we were able to show that both direct electrical stimulation of the splanchnic nerve and intravenous injection of adrenaline can inhibit the gastric secretion stimulated by histamine. This suggests that when the sympathetico-adrenal system is intact it is likely to be also involved in the inhibitory phenomenon under consideration. The phenomenon is evidently a complex one involving both nervous and humoral factors, and we had also shown that it requires the participation of higher nervous centres, being not elicitable in anesthetized animals. We wish to express our deep gratitude to Prof. T. P. Feng for suggesting this problem and for constant guidance throughout this work.

(一)在慢性實驗中用備有小胃的狗,我們证明組織胺引起的胃分泌能反射地为皮膚電刺激及一些其他外界刺激所抑制。 (二)這抑制現象在除去胃的迷走神經支配相交或神經支配,並隔離腎上腺的神經聯系時,仍能出現。它似有某種可以反射地形成的抑制性體液變化為基礎。 (三)但在急性實驗中我們又證明大內臟神經和注射腎上腺素均能抑制組織胺引起的胃分泌,這指示在正常動物胃分泌的反射抑制仍可能通過交感神經-腎上腺系統而得實現。 (四)本文所述的胃分泌的反射抑制現象顯然包含複雜的神經-體液機制,並且是在高級神經中樞參加下產生的。

Observations were made to compare the effect of HCl introduced into the small intestine of dogs on pancreatic secretion in the following three conditions: (1) Acute experiments with barbiturates(pentothal sodium or pentobarbital sodium) as an(?)sthesia; (2) Chronic experimental dogs with Thomas pancreatic fistula; (3) Chronic dogs in(2) an(?)sthetized by means of the same barbiturates as in the acute experiments. The results indicated that in the acute experiments, the latent period of pancreatic secretion was...

Observations were made to compare the effect of HCl introduced into the small intestine of dogs on pancreatic secretion in the following three conditions: (1) Acute experiments with barbiturates(pentothal sodium or pentobarbital sodium) as an(?)sthesia; (2) Chronic experimental dogs with Thomas pancreatic fistula; (3) Chronic dogs in(2) an(?)sthetized by means of the same barbiturates as in the acute experiments. The results indicated that in the acute experiments, the latent period of pancreatic secretion was about 3-5 minutes and the amount secreted was usually below 20 drops in the first 30 minutes after the introduction of the acid. But in the case of chronic experiments, the results were quite different. The latent period was shorter, usually less than 2 minutes and the amount of pancreatic secretion was usually 10 times as much as in the acute experiments. Moreover, the pancreatic secretion of the chronic fistula dogs was not changed either in latent period or in amount when under the barbiturate an(?)sthesia. From the above results, it is quite natural to assume that the influence of the barbiturate an(?)sthetics, pentothal sodium and pentobarbital sodium on pancreatic secretion induced by HCl in the intestine of dogs is negligible. The great difference in the pancreatic secretion between the acute and the chronic experiments might, therefore, be due to the operative trauma which is unavoidable in acute experiments. Another observation was made to determine the effect of atropine on pancreatic secretion induced by HCl in chronic fistula dogs. The result showed that atropine distinctly reduced the response of pancreas to the acid. The latent period was increased and the amount of pancreatic secretion was decreased as compared with the result obtained in normal chronic fistula dogs. Hence it is quite suggestive that there might exist a nervous component in the mechanism of pancreatic secretion induced by HCl in the small intestine. We wish to express our gratitude to Comrade for his valuable advice and to professors T. C. Shen and T. F. Liu for their encouragement throughout this work.

本實驗比較急性實驗狗、慢性胰瘻狗和經過麻醉的慢性胰屢狗對於鹽酸注入小腸所引起的胰液分泌量和潛伏期,結果證明: (1)在急性實驗情况下,狗胰腺對鹽酸刺激小腸所引起的胰液分泌量遠較在慢性實驗時為少,且潛伏期較長。 (2)巴比妥類麻醉劑:硫賁妥鈉(sodium pentothal)和戊烷巴比妥鈉(sodiumpentobarbital)對鹽酸所引起的胰液分泌量及潛伏期影響極微。 (3)在急性實驗情况下,由鹽酸所引起的胰液分泌量的減少和潛伏期的加長,似乎不是由於巴比妥類麻醉劑的作用,而可能是由於手術創傷的影響。 (4)注射阿托平後,胰腺對於鹽酸刺激小腸所引起的反應顯著减小,故推测在鹽酸引起胰液分泌的機制中可能有神經反射作用的參與。本工作在进行過程中,承蘇聯專家同志親切地給予指導,并承沈(?)淇、劉曾復二教授关懷和支持,(?)此誌謝。

 
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