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细胞核固缩
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  “细胞核固缩”译为未确定词的双语例句
     in the 7d model, some ganglion cells shrinked and disappeared, with the number of cells 230.8333±4.8751(P<0.05), and nuclei were pyknotic, with average sectional area 48.4917±3.6944μm2 (P<0.05);
     7d组部分视网膜神经节细胞缩小、死亡,视网膜神经节细胞数为230.8333±4.8751(P<0.05),细胞核固缩、平均截面面积为48.4917±3.6944μm2 (P<0.05);
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     Results (1)When the cells were treated with As2O3 at a concentration of 4.0 μmol/L lasting for 72 h,karyopyknosis and karyorrhexis were observed under transmission electron microscope;
     结果(1)4.0μmol/LAs2O3作用72h,在电子显微镜下可见细胞核固缩、核碎裂、染色质边集等改变;
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     However,Nicorandil at 25 μmol/L~100 μmol/L the cell number decreased significantly(P<0.01) ,FCM indicated a peak of apoptosis obviously,stained by Hoechst33258 showed that the nucleus of the cells have atypical features of apoptosis.
     25μmol/L~100μmol/L/Nicorandil显著减少前列腺上皮细胞数(P<0.01),且FCM显示前列腺上皮细胞有明显的凋亡峰。 Hoechst33258染色法显示细胞核固缩、裂解,呈现明显的凋亡形态特征。
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     PC prevented the PC12 cells nuclei from shrinkage, condensation, and cleavage induced by Aβ_ 25-35 . PC decreased the expression of par-4 mRNA and protein, and increased the expression of bcl-2 mRNA and protein.
     结果不同剂量(5、10、20、30mg/L)PC预处理PC12细胞1h可剂量依赖性对抗Aβ25-35引起的细胞凋亡,提高PC12细胞的存活率,减少Aβ25-35引起的PC12细胞核固缩、凝聚和碎裂,降低par-4基因mRNA表达及蛋白表达,增加bcl-2基因mRNA表达及蛋白表达。
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     RESULTS:Indomethacin induced apoptosis in wtp53/SW480 cells.
     结果:吲哚美辛诱导野生型 p53转染的 wtp53/SW480细胞凋亡,出现形态学改变:即细胞核固缩、裂解,核碎片及凋亡小体形成.
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     Its cells can multiply by division.
     基层细胞核固缩,破裂成碎块;
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     the nuclei underwent pyknosis.
     30d时,细胞核固缩细胞消失;
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     Tom Clancys Splinter Cell
     细胞分裂
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     The phenotype of those cells was NKH1+ CD16-.
     _-的细胞
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The present paper embodies a comprehensive investigation of all tissues of the tadpole tail of Kaloula borealis and their degeneration during metamorphosis. The following are the important findings.The structural characteristics of the tailThe tadpole tail of Anura is a temporary organ functioning in its embryonic aquatic life. In this ephemeral organ a number of primitive characters are retained. For example, the epidermis consists of only two layers of cells. A cuticula is present on the superficial layer....

The present paper embodies a comprehensive investigation of all tissues of the tadpole tail of Kaloula borealis and their degeneration during metamorphosis. The following are the important findings.The structural characteristics of the tailThe tadpole tail of Anura is a temporary organ functioning in its embryonic aquatic life. In this ephemeral organ a number of primitive characters are retained. For example, the epidermis consists of only two layers of cells. A cuticula is present on the superficial layer. Its cells can multiply by division. The dermis is extremely thin and consists of fibers only. Muscles exist in the form of myotomes,in which many young myofibers are retained. The connective tissue matrix is not completely differentiated and looks similar to the mesenchyme. These primitive conditions are also present in the trunk portion of the young tadpole, but at a later stage the above mentioned structures gradually assume aspects obtaining in the young frog. The tail however, retains these primitive conditions unaltered to the very end. In the metamorphosis it undergoes regressive degeneration.Maintenance of unity by the tail in degenerationIn the course of degeneration, activities are concerted. An axial gradient is manifest. Degeneration activity is most pronounced at the tip of the tail, less in the middle portion and least at the base. As a result, the tip quickly vanished, the other parts follow in an orderly sequence. Finally the whole organ disappears.The rate of degeneration of various tissues also varies. Epidermis and notochord lag behind. In order to accommondate themselves in the shrinking space the epidermal cells pile on top of each other, and the epidermis thus becomes multilayered and the notochord becomes undulated. The modifications are in accord with the role these tissues play.The general rule of the degeneration of the tail tissueIn a given tissue, elements that are advanced in differentiation also lead in degeneration. E. g. muscle fiber in myqtome, vacuolar cell in notochord, ganglion cell in spinal ganglion. The less differentiated elements lag behind, e. g. endomysium, noto-chordal epithelium and amphicyte.In degeneration, all fibral tissues including connective tissue-, nerve- and muscle fibers, become swollen and losening up, and gradually become dissolved. Cytoplasm of all cells lose their tight and compact nature and takes deeper stain with eosin. Hereafter vacuoles and pigment granules often appear in it. Unless compressed, the nuclei keep their shape but lose their contents. Degenerating epidermal cells of superficial layer undergo keratinization and finally are cast off. Cells of basal layer first fragment and then vanish.The nucleoli show greater persistence. This is especially pronounced in the nucleoli of ganglion cells and muscle fibers.Pigment granules are slow to disintegrate. They frequently collect in masses, and the tail become black during metamorphosis.Mitosis and amitosisAmitosis occurs in both layers of the epidermal cells, in the nuclei of muscle fibers and in the large cells of the connective tissue matrix. In the formative period of muscle fibers, multiplication of nuclei is soly by means of mitosis. At the end of differentiation mitosis ceases. Hereafter multiplication of nuclei is soly by means of amitosis. In their upgrade development, epidermal cells and the large cells of connective tissue matrix too, cells multiply by means of mitosis. But at the completion of differentiation amitosis makes its appearance and goes on side by side until metamorphosis.Before beginning of metamorphosis, all kinds of the cells in the tail tissue cease to multiply. Only the cells of ependyma and wandering cells of the connective tissue matrix mitosis goes on as usual during degeneration.The significance of mitosis and amitosis is discussed.Degeneration and developmentIn the development of the embryo, there is growth and differentiation of cells and tissues, but coupled with it, there is senescence and death. These two phases of life contradict and yet complement each other. This i

本工作较全面地观察了北方狭口蛙成长期蝌蚪尾组织的显微结构及在变态期的退化,主要的结果简述如下。 1.成长期表皮两层细胞都进行无丝及有丝分裂,无丝分裂出现比有丝分裂晚,出现时表皮细胞郎有生理的退化。 表皮退化时由两层改组为多层,表面细胞角质化,组成一到二层角质层,角质细胞不断脱落。基层细胞核固缩,破裂成碎块;细胞质收缩,与膜脱离,破裂成碎块,与核一同消失。 内膜与肌纤维的连接是肌内膜的原纤维伸入肌原生质中与之紧密连接。 绝大多数肌纤维退化时肌原纤维断裂成肌解小体,以后溶解;少数不断裂成肌解小体,直接溶解。 3.脊索退化时泡细胞先退化,脊索上皮稍后,脊索鞘最后。泡细胞先失去液体,细胞膜膨胀加厚,与细胞质脱离,以后共同溶解。脊索鞘纤维膨胀,松散,然后溶解。 4.神经细胞退化时所合成分渐次溶解,但核仁耐力大,在退化的神经节细胞中有时其他成分已经溶解但核仁仍在。神经纤维膨胀,松散,溶解。 5.侧线的感觉蕾退化时与支配它的神经纤维首先失去连接,向表皮表面移位,细胞溶解,残留的少数细胞随表皮角质细胞脱落。 6.血管与淋巴管壁都很薄,退化时由于纤维膨胀,譬加厚,结构才显得清楚。纤维膨胀,松散,与细胞一同溶解。 7...

本工作较全面地观察了北方狭口蛙成长期蝌蚪尾组织的显微结构及在变态期的退化,主要的结果简述如下。 1.成长期表皮两层细胞都进行无丝及有丝分裂,无丝分裂出现比有丝分裂晚,出现时表皮细胞郎有生理的退化。 表皮退化时由两层改组为多层,表面细胞角质化,组成一到二层角质层,角质细胞不断脱落。基层细胞核固缩,破裂成碎块;细胞质收缩,与膜脱离,破裂成碎块,与核一同消失。 内膜与肌纤维的连接是肌内膜的原纤维伸入肌原生质中与之紧密连接。 绝大多数肌纤维退化时肌原纤维断裂成肌解小体,以后溶解;少数不断裂成肌解小体,直接溶解。 3.脊索退化时泡细胞先退化,脊索上皮稍后,脊索鞘最后。泡细胞先失去液体,细胞膜膨胀加厚,与细胞质脱离,以后共同溶解。脊索鞘纤维膨胀,松散,然后溶解。 4.神经细胞退化时所合成分渐次溶解,但核仁耐力大,在退化的神经节细胞中有时其他成分已经溶解但核仁仍在。神经纤维膨胀,松散,溶解。 5.侧线的感觉蕾退化时与支配它的神经纤维首先失去连接,向表皮表面移位,细胞溶解,残留的少数细胞随表皮角质细胞脱落。 6.血管与淋巴管壁都很薄,退化时由于纤维膨胀,譬加厚,结构才显得清楚。纤维膨胀,松散,与细胞一同溶解。 7.结缔组织基质合纤维与三种细胞:小型细胞,大型细胞及游走细胞。后两?

Differences in the injurious effect on the rabbit retina upon exposure to in-tense brom-tungsten source with(DS_6Ⅱ lamp)and without(DS_4 lamp)filteringtheir infra-red contents were examined electron microscopically.Retinas sampled 24 hrs after exposure to DS_6Ⅱ lamp for 15 min show frag-mentation of apical disc membranes phagocytosed by pigment epithelium whichby itself shows little morphological alteration.These changes are aggravated byprolonging the exposure to 60 min and further changes in the proximal structuresuch...

Differences in the injurious effect on the rabbit retina upon exposure to in-tense brom-tungsten source with(DS_6Ⅱ lamp)and without(DS_4 lamp)filteringtheir infra-red contents were examined electron microscopically.Retinas sampled 24 hrs after exposure to DS_6Ⅱ lamp for 15 min show frag-mentation of apical disc membranes phagocytosed by pigment epithelium whichby itself shows little morphological alteration.These changes are aggravated byprolonging the exposure to 60 min and further changes in the proximal structuresuch as pyknosis of nuclei or shrinkage of synaptic terminals can be observed insome of the receptors.While changes in the outer segment by 1 min exposure to DS_4 are comparableto those caused by 15 min exposure to DS_6Ⅱ lamp,the former gives rise to farmore wide-spread changes in other cellular structures such as pyknosis of nucleivacuolization of synapses and swelling of the pigment epithelium.After 15 minexposure,some cells become condensed and shrunken entirely from outer segmentdown to synapse.30 rain exposure causes necrosis and fusion of the receptor andthe pigment epithelium layer.Following the thermal model of Clarence and Clarke,calculation showed thetemperature elevations in the area of image due to DS_6Ⅱ and DS_4 lamp are res-pectivdy 2℃ and 10.4℃.The latter figure corresponds to a temperature riseat which some protein would begin to coagulate.Consequently the much more disastrous effects of DS_4 lamp are mainly at-tributed to its thermal effect,while those due to DS_6Ⅱ lamp are primarily photiceffects.

用电镜观察两种不同光源(一种滤去红外的 DS_6 Ⅱ型溴钨灯,另一种保留红外的 DS_4型溴钨灯)造成的兔网膜损伤,二者在损伤速度、程度、范围和性质上均有不同:DS_6 Ⅱ型灯光照15分钟,色素上皮变化甚微,感光细胞层主要是外段顶端和少数内段线粒体的变化;光照60分钟,除了外段变化更显著外,少数细胞核和突触体发生皱缩。用 DS_4型灯光照网膜1分钟,外段的变化程度已与 DS_6 Ⅱ型灯光照15分钟者相似,此外还有若干细胞核固缩和突触体空泡化以及色素上皮肿胀等改变。DS_4型灯光照15分钟时网膜的损伤远比 DS_6Ⅱ型灯光照15分钟者显著,某些细胞整个出现“浓缩”,光照30分钟的网膜,色素上皮和感光细胞已严重坏死,甚至融合为一。这些结果提示,DS_6 Ⅱ型灯和 DS_4型灯照射造成的网膜损伤,前者主要是由于光效应,后者主要是由于热效应所致。

The bacterial gill-rot disease is a common disease of grass carp, it makes serious harmfulness to the fish. There are two types of histopathological changes: chronicand acuteness. The gill tissue of the chronic generally undergoes three worseningsteps, i.e. inflammatory edema, hyperplasia and necrosis; but the acute one onlypasses through two steps: edema and necrosis, because the cause diseased is so shortthat the edema passes directly to necrosis and there is no hyperplasia step. The liver and kidney of the...

The bacterial gill-rot disease is a common disease of grass carp, it makes serious harmfulness to the fish. There are two types of histopathological changes: chronicand acuteness. The gill tissue of the chronic generally undergoes three worseningsteps, i.e. inflammatory edema, hyperplasia and necrosis; but the acute one onlypasses through two steps: edema and necrosis, because the cause diseased is so shortthat the edema passes directly to necrosis and there is no hyperplasia step. The liver and kidney of the diseased fish also show histopathological changes,the granular denaturation and vecuolation were occured, glycogen was disappeared,up until the necrosis in liver cells. The epithelial cells in the proximal convolutedtubules of the kidney also make same change. A number of nuclei around necrotictubules of kidney appeared condensated. A few of glomeruli of kidney atrophied. The quantity of leukocytes of artificially infected fish is much less than thecontrolled one, but the erythrocytes and hymoglobin of experimental group wereslightly less than the control.

草鱼细菌性烂鳃病是一种危害严重的常见鱼病。其组织病理变化可以分为慢性和急性两种类型。鳃组织病理变化,经过炎性水肿、细胞增生和坏死三个进程。慢性型以增生为主;急性型的鳃组织病理变化,由于病程短,炎性水肿迅速转入坏死,增生不严重或几乎不出现,以炎性水肿和坏死为主。 患细菌性烂鳃病的草鱼的肝脏、肾脏也发生病理变化。肝细胞发生颗粒变性、水样变性、醣元颗粒消失,以至坏死;其肾组织主要发生近曲小管上皮细胞颗粒变性、水样变性、坏死,在坏死的肾小管周围的造血组织中的一些细胞核固缩,个别肾小球萎缩。 人工感染患细菌性烂鳃病的当年草鱼的白细胞数显著地低于对照组,而红细胞数和血红蛋白量虽低于对照组,但没有显著性差异。

 
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