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治疗组血糖
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  blood sugar in the treating group
     Blood sugar in the treating group dropped obviously compared with it in the diabetes model group(P<0.01),total antioxidation ability of serum increased(P<0.01),MDA level of serum,liver and pancreas decreased(P<0.01).
     与糖尿病模型组相比,治疗组血糖明显下降(P<0.01),血清总抗氧化能力显著升高(P<0.01),血清、肝胰组织MDA水平显著降低(P<0.01)。
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  “治疗组血糖”译为未确定词的双语例句
     Results: Blood glucose of intensive therapy group decreased more dramatically than that of routine therapy group, which were 7.78±1.29mmol/L and 9.03±1.94mmol/L respectively(P<0.01);
     结果强化治疗组血糖较常规组明显下降,分别为(7.78±1.29)mmol/L和(9.03±1.94)mmol/L(P<0.01);
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     the blood sugar in patients of hyperbaric oxygen treatment group was ( 5.46 ±0.84)mmol/L, while it was (6.04±0.86)mmol/L of control group ,the change was also significant( P <0.01).
     治疗后治疗组血糖为 (5 .46± 0 .84)mmol/L ,对照组为(6.0 4± 0 .86)mmol/L ,差异有非常显著性意义 (P <0 .0 1) ,治疗组血糖明显低于对照组。
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     the blood sugar in patients of the hyperoxialiquid treatment group was (5.46±0.84)mmol/L, while itwas(6.04±0.86) mmol/L of control group,there was significant difference between the two groups(P<0.01).
     治疗后治疗组血糖为(5.44±0.82)mmol/L,对照组为(6.02±0.84)mmol/L,两组比较差异有非常显著性意义(P<0.01),治疗组血糖明显低于对照组。
短句来源
     Results The blood glucose levels in medium-dose and high-dose group declined,the differences were significant as compared with the diabetic control group(F=29.55;q=5.13,7.03;P<0.01).
     结果壳聚糖中剂量、高剂量治疗组血糖降低,与糖尿病对照组相比差异有显著性(F=29.55,q=5.13、7.03,P<0.01);
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     Results:In group with insulin pump,the time achieved satisfactory blood glucose was 3.71±1.15 days,the time retrieved ketosis was 3.75±1.24 days. The daily dosage of insulin markedly decreased.
     结果:用胰岛素泵治疗组血糖达满意控制的天数为3.71±1.15d,酮症纠正时间3.75±1.24d,每日使用胰岛素的量明显减少。
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     ③Therapeutic group.
     (3)治疗
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     and no therapy was given to Group D;
     C为FST治疗 ;
短句来源
     In all patients,the plasma glucose level was measured before and after 10-day treatment.
     测定两患者治疗10d前后血糖浓度。
短句来源
     The SV and CO of the treatment group B increased in 5 minutes, which wa
     B
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     Group B: SD to Wistar rat heart tarnsplantation;
     B
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In this study, 38 experimental subjects of diabetes mellitus were given pectin 25g extra per day orally under carefully controlled dietary plan for six weeks. Another 38 cases were selected at random as control group.Pre- and posttreatrnent, blood and urine sugar and serum insulin of each subject were measured and compared. The results were: 1) the blood sugar dropped markedly as compared with that of pretreatment (p<0,01), but not significantly different from control group (p>05). 2) sugar in urine decreased...

In this study, 38 experimental subjects of diabetes mellitus were given pectin 25g extra per day orally under carefully controlled dietary plan for six weeks. Another 38 cases were selected at random as control group.Pre- and posttreatrnent, blood and urine sugar and serum insulin of each subject were measured and compared. The results were: 1) the blood sugar dropped markedly as compared with that of pretreatment (p<0,01), but not significantly different from control group (p>05). 2) sugar in urine decreased evidently in contrast with control group, but no difference was seen in the volume of urine between the two groups. 3) serum insulin levels dropped significantly when compared with control group (p<0.0l). Thus, pectin was considered to be the simple useful measures in the management of diabetes. The cause of its effect in lowering blood and urine sugar was discussed bri-efly.

为了观察植物纤维——果胶的降低血糖、尿糖、改善糖耐量和提高周围组织对胰岛素的敏感性等效果,对门诊38例糖尿病患者,在饮食控制条件下,加服果胶治疗,并随机另选38例作为对照观察。观察期两个半月,前两周为准备期,第3~8周为果胶治疗期,每人每日服用25g,六周为一疗程,第9、10两周为复查期。结果表明果胶治疗组血糖值明显下降(P<0.01),但与对照组比较,差别不明显。24小时尿糖及血清胰岛素水平较对照组均有明显下降(P<0.01)。因此果胶可以作为糖尿病的一种简单而易行的辅助治疗措施。

This study examined the effects of dexamethasone on binary sepsis-induced hypoglycemia and hepatic gluconeogenesis depression in fasting rabbits. Dexamethasone was administered intraveneously with a dose of 10mg/kg body weight at 3h and 24h following septic challenge. Binary sepsis caused severe hypoglycemia in the rat since 24h after septic challenge. Dexamethasone delayed the development of hypoglycemia in the rabbit till 48h after septic challenge. Hepatic gluconeongenesis was suppressed at 48h after sepsis...

This study examined the effects of dexamethasone on binary sepsis-induced hypoglycemia and hepatic gluconeogenesis depression in fasting rabbits. Dexamethasone was administered intraveneously with a dose of 10mg/kg body weight at 3h and 24h following septic challenge. Binary sepsis caused severe hypoglycemia in the rat since 24h after septic challenge. Dexamethasone delayed the development of hypoglycemia in the rabbit till 48h after septic challenge. Hepatic gluconeongenesis was suppressed at 48h after sepsis as evaluated in isolated and cultured hepatocytes. Dexamethasone ameliorated in the non-infected liver lobes,but did not in the infected liver lobes. It is concluded that dexamethasone protection in binary sepsis is associated with support of gluconeogenesis, and as a result,amelioration of hypoglycemia.

将40只日本大耳白兔随机分为胆源性脓毒症组(n=15,以下简称非治疗组)、地塞米松治疗组(n=15)和正常对照组(n=10),并将前两组复制成胆源性脓毒症模型。治疗组于伤后3和24小时按10mg/kg剂量分两次腹腔注射地塞米松,动态检测和比较三组动物血糖水平的变化,用原代肝细胞培养法分析伤后48小时感染肝叶和非感染肝叶肝细胞糖异生功能。结果显示;伤后24小时起,非治疗组血糖水平持续显著降低,而治疗组至伤后48小时才出现血糖降低;伤后48小时,治疗组感染肝叶肝细胞糖异生的基础速率及激素刺激速率与非治疗组无显著性差异,但非感染肝叶肾上腺素和胰高糖素刺激的肝细胞糖异生速率分别较非治疗组提高了25.0%和46.0%,然而两者仍低于正常对照组水平。本实验结果表明,地塞米松对胆源性脓毒症时宿主糖代谢的稳态具有保护效应。

In this paper, the experimental diabetic rats induced by streptozotocin (STZ, i. p. 50 mg/kg)were divided into three groups, electro-acupuncture (EA group, n = 8), transcutaneous electric nervestimulation (TENS group, n = 8), at bilateral Shenshu and Zusanli points for 20 minutes once every 2- 3 days for five weeks, and without any treatment (DM group, n = 6) respectively. As compared withthe DM group, the increased plasma glucose levels was lowered significantly in EA group (P< 0. 05)and slightly in TENS group(P...

In this paper, the experimental diabetic rats induced by streptozotocin (STZ, i. p. 50 mg/kg)were divided into three groups, electro-acupuncture (EA group, n = 8), transcutaneous electric nervestimulation (TENS group, n = 8), at bilateral Shenshu and Zusanli points for 20 minutes once every 2- 3 days for five weeks, and without any treatment (DM group, n = 6) respectively. As compared withthe DM group, the increased plasma glucose levels was lowered significantly in EA group (P< 0. 05)and slightly in TENS group(P >0. 05) by the end of the sixth week. And the symtoms of polyphagia,polydipsia and polyuria were attenuated in EA group. The motor nerve conduction velocity slowing wasprevented or corrected after a course of four and six weeks treatment in EA and TENS group respec-tively. In the DM and TENS groups the pain threshold was lowered at 6 - 20 days after injection ofSTZ, but elevated in EA group, their differences were of significance (P< 0. 05). In general, the effi-ciency of EA treatment on experimental diabetes and its neuropathy was better than that of TENStherapy.

本文用腹腔注射链脲佐菌素(50mg/kg)诱发大鼠实验性糖尿病,48小时后血糖升高(11.1-33.3mmol/L),出现多饮、多食和多尿现象。随机分为三组,分别给以电针(EA)和经皮电刺激(TENS)治疗,取穴双侧肾俞和足三里,每次20分,每周三次,共五周;DM组未给治疗,另设ND组。到六周末治疗组血糖下降,和DM组相比,EA组差异显著(P<0.05),三多症状也有明显改善;但TENS组没有显著差异。在四周和六周末,DM组运动神经传导速度较正常动物明显减慢(P<0.05),而EA组和TENS组分别于四周和六周时恢复正常,与DM组比较差异显著(P<0.05)。DM组和TENS组在造模后基础痛阈一度出现降低(痛敏),而EA组痛阈不但不降反而升高,与DM组和TENS组相比,差异显著(P<0.05)。本实验肯定了电针对实验性糖尿病的治疗作用,为推广针灸治疗糖尿病及其神经病变提供了实验依据。

 
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