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icr小鼠
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  icr mice
Twenty-five tiptoe-walking Yoshimura (twy) mice with calcified mass lesions compressing the spinal cord posterolaterally at the C1-C2 vertebral levels were compared with five Institute of Cancer Research (ICR) mice that served as controls.
      
albicans) in mice, the fungal vaginitis model was established in female ICR mice by intravaginal inoculation of suspension of C.
      
To study the effect of itraconazole on the vaginal candidiasis caused by Candida under different immunity conditions, the fungal vaginitis model was established in female ICR mice by intravaginal inoculation of suspension of C.
      
The relationship between Ni-induced hepatic lipid peroxidation (LPO) and the concentrations of Ni and trace elements was investigated in male ICR mice.
      
The l-SeMet α,γ-elimination enzyme was ubiquitous in tissues from ICR mice and the activity was relatively high in the large intestine, brain, and muscle, as well as the liver.
      
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  icr mouse
ICR mouse embryos at the two-cell stage obtained by flushing the oviducts were frozen by two cryopreservation procedures, slow freezing using a programmable freezer or vitrification by direct plunging into liquid nitrogen.
      
The tail was flexed towards the left side of the body in 5.2 and 6.2% of ICR mouse embryos at 10.5 and 11.5 days of gestation respectively.
      
In the present study to analyze the role of ACTH in fetal tissues and organs, we observed the expression of melanocortin type 2 (MC2) and 5 (MC5) receptors in ICR mouse embryos from E11.5 to E18.5 by immunohistochemistry.
      
These recoveries were all higher than those in a previous report of 22 (7.3%) worms recovered from a single Institute of Cancer Research (ICR) mouse exposed to 300 cysts 22?days prior to necropsy.
      
The duodenum and ileum of 15 to 18-day old Swiss ICR mouse embryos were studied with the electron microscope to follow the differentiation of endocrine cells.
      
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  the icr mice
We then assessed the acute toxicity of the small molecules on the ICR mice by administrating TGG and luteolin intraperitoneally.
      


60 inbred ICR mice were fed with CCl4 once a week for two weeks to produce toxic hepatic nec-rosis. They were randomly divided into three groups according to the treatmert: One group of animals were fed with colchicine 0.5μg(CT), Second group with prednisone 10 μg(PT) and a control group fed with distilled water(DW) daily for 14 days. Another 14 mice were fed with neither CCl4 nor drugs as blank control. The death rate in CT group was 25%, PT 50% and DW 35%. The death rate of the female mice was almost equal...

60 inbred ICR mice were fed with CCl4 once a week for two weeks to produce toxic hepatic nec-rosis. They were randomly divided into three groups according to the treatmert: One group of animals were fed with colchicine 0.5μg(CT), Second group with prednisone 10 μg(PT) and a control group fed with distilled water(DW) daily for 14 days. Another 14 mice were fed with neither CCl4 nor drugs as blank control. The death rate in CT group was 25%, PT 50% and DW 35%. The death rate of the female mice was almost equal to that of the males in PT and DW groups. No male mice died in CT group. Microscopic examination of the liver slices showed that the degree of steatosis and hepatic necrosis in CT group was milder than that in PT and DW groups, and no fibrosis was found in CT group but there were two and three mice with fibrosis in PT and DW groups respectively. The results showed that colchicine might lower the death rate of the male mice with toxic hepatic necrosis and also prevent liver fibrosis, while prednixone did not have such effect.

60只纯种ICR小鼠喂服CCI_4每周1次,共2次,致中毒性肝坏死。随机分为3组,分别喂以秋水仙碱0.5μg、泼尼松10μg和蒸溜水(对照组),每日1次,共14次。另以ICR小鼠14只作空白对照。3个实验组死亡率各为25%、50%和35%。泼尼松组和对照组雌、雄鼠死亡数相近,但秋水仙碱组雄鼠无死亡。各鼠肝剖验镜检以秋水仙碱组脂肪变性及坏死少且程度轻,未发现纤维组织增生。泼尼松组和对照组除肝脏病变明显外,各有2只和3只鼠肝见纤维组织增生。示秋水仙碱治疗似可降低中毒性肝坏死的雄鼠死亡率,减轻小鼠肝脏病理改变,防止肝脏纤维化。泼尼松则否。

ICR mice, irradiated lethally by 60Co r-ray, were administered orally and daily with varying doses of organic germanium for 14 days. The 30-day survival rate of those mice administered with a dose of 500mg/kg was increased by 37.5% as compared with the control group ( P<0.0l) . Significant stimulating effects of Ge-132 on internal colony forming units in spleen (CFU-S) of mice were also found. The results showed that organic germanium compound has certain protective effect on lethally irradiated mice and stimulating...

ICR mice, irradiated lethally by 60Co r-ray, were administered orally and daily with varying doses of organic germanium for 14 days. The 30-day survival rate of those mice administered with a dose of 500mg/kg was increased by 37.5% as compared with the control group ( P<0.0l) . Significant stimulating effects of Ge-132 on internal colony forming units in spleen (CFU-S) of mice were also found. The results showed that organic germanium compound has certain protective effect on lethally irradiated mice and stimulating effects on the proliferation and differentiation of hematopoietic stem cells.

ICR小鼠受致死量射线照射以后,接受有机锗治疗14天.剂量为500mg/kg的小鼠30天活存率比对照组提高37.5%;当Ce—132剂量为500mg/kg和1000mg/kg时,其内源性脾结节的数量明显地高于对照组。实验结果表明,Ce—132具有一定的治疗急性骨髓型放射病的作用和刺激内源性造血干细胞的增殖与分化作用。

Comparison was made in different periods after unilateral nephrectomy in mice about the weights ard the morphological changes between the remaining kidney and kidney of sham-operated controls. Results revealed that after unilateral nephrectomy in ICR mice a series of changes occurred in the remaining kidney : there were ircrease in weight, ircrease in thicknesses of renal cortex and medulla, inccase in size of renal corpuscle, increase in numar of cells in the glomerulus, hyperplasia of epithelial cells in the...

Comparison was made in different periods after unilateral nephrectomy in mice about the weights ard the morphological changes between the remaining kidney and kidney of sham-operated controls. Results revealed that after unilateral nephrectomy in ICR mice a series of changes occurred in the remaining kidney : there were ircrease in weight, ircrease in thicknesses of renal cortex and medulla, inccase in size of renal corpuscle, increase in numar of cells in the glomerulus, hyperplasia of epithelial cells in the rennal tubule, dilatation of renal vessels and stromal hyperplasia. Administration of chemical carcinogens PBNA to the hypertrophic and hyperplastic kidney may aggravate cell hyperplasia and degeneration in the remaining kidney, and promote the development of tumor. The shortening period of tumors after unilateral nephrectomy might help to provide a better model for chronic animal experiment.

实验比较了切除一侧肾后不同时期的残留肾和假切肾的肾脏重量、解剖外观、皮质部与髓质部的厚度、肾盂的大小、肾小球体积大小、肾小球最大切面丝球体细胞数、肾小管增生、肾小管扩张、肾实质内纤维组织增生以及给致癌物后肾脏肿瘤发生的情况。实验结果表明,ICR小鼠在切除一侧肾后,另一侧残留肾发生了一系列的变化,包括在不同时期的形态组织学和细胞的改变。在残留肾肥大增生的基础上给化学致癌物PBNA,能促进残留肾的细胞增生和变性,并有肿瘤的形成。因为这一方法可以明显缩短引癌的周期,有可能作为整体慢性诱癌实验一个改良的模型。

 
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