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在受体
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  at receptor
     SMS obviously inhibited the synthesis of DNA and protein, and prohibited the SW480 cell shifting from phase G 0/G 1 in phase S, G 2M, which suggests that somatostatin (SS) possessed an inhibitive effect on large intestinal at cancer cell, it is achieved at receptor by inhibiting the synthesis of DNA and protein and prohibiting cell cycle of cancer.
     SMS明显抑制了大肠癌细胞DNA及蛋白质合成,抑制SW480细胞从G0/G1期过渡到S期和G2M期,提示生长抑素在受体后通过抑制DNA及蛋白质合成,抑制细胞周期循环,从而抑制大肠癌细胞的生长。
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     CONCLUSION: The mechanism of growth hormone insensitivity induced by endotoxin was associated with down-regulated GHR mRNA expression at receptor level and up-regulated SOCS-3 mRNA expression at post-receptor level.
     结论:内毒素诱导的生长激素不敏感的机制在受体水平与GHR mRNA的表达下调有关,在受体后水平与SOCS-3 mRNA的表达上调有关.
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     Conclusions:The growth hormone insensitivity could be induced by LPS injection, which was associated with down regulated GHR mRNA expression at receptor level and up regulated SOCS 3 mRNA expression at post receptor level.
     结论 :静注LPS可以诱导生长激素的不敏感 ,在受体水平与GHRmRNA的表达下调有关 ,在受体后水平与SOCS 3mRNA的表达上调有关。
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     Aim To investigate the role of angiotensin Ⅱ(AngⅡ) 1 type recepter antagonist and aldosterone (Ald) receptor antagonist in reversing myocardial remodeling in hypertensive rats by blocking renin angiotensin system at receptor level and the mechanism of hypertensive myocordial remodeling.
     在受体水平阻断肾素—血管紧张素系统 ,比较血管紧张素Ⅱ 1型受体拮抗剂与醛固酮受体拮抗剂对逆转高血压心肌重塑的作用 ,来探讨高血压性心肌重塑的可能机理。
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     Conclusion TGFβ-1 may act as a mediator of the CNV process, its down-regulation may play an important role in the development of CNV. And the expression of TβRⅠdown-regulated in the same way to TGFβ-1 implies that TGFβ-1 plays roles at receptor level in CNV.
     结论光凝区内TGF-β1合成、分泌不足可能是CNV形成和增殖的主要原因之一; TGF-β1在受体水平表现出调节作用,存在由TβRⅠ介导的生物学效应。
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  “在受体”译为未确定词的双语例句
     ③In 1~5 months period, stable and effective expression of mdr1 gene could be detected in hematopoietic cells of the recipient mouse, the percentage of mdr1 gene expression cells in recipient's hematopoietic cells decreased monthly to 8.0%,8.0%,7.5%,4.0% and 3.0%.
     ③mdr1在移植小鼠细胞内稳定、有效地表达 ,观察 1~ 5个月mdr1转染的细胞在受体鼠骨髓单个核细胞中所占比例分别为 8.0 % ,8.0 % ,7.5 % ,4 .0 % ,3.0 % ;
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     but blood velocity of renal vein of receptors was decreased in all three groups (7.33±0.42),(5.79±0.38),(4.46±0.43) cm/s;
     单纯肾移植组、肾移植+丙泊酚组供肾在受体时肾静脉血流速度均显著下降犤(7.33±0.42),(5.79±0.38),(4.46±0.43)cm/s;
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     Results The engrafted Flk1+ CD34-cells carrying Bcr/Abl fusion gene from the bone marrow of CML patients can differentiate to the leukemia cells, it can be detected the Bcr/Abl fusion gene positive cells from the bone marrow of receptor mice.
     结果植入的CML患者骨髓源BCR/ABL+、Flk1+CD34-细胞在受体小鼠体内分化为白血病细胞,受体小鼠骨髓细胞中存在Bcr/Abl融合基因阳性细胞。
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     Furthermore, the activation domain 1 (AD1) of SRC-1 was required for the coactivation of hB1F, and that was synergied by the general coactivator CREB-binding protein (CBP).
     SRC-1对hB1F的共激活作用需要其激活结构域1(activation domain 1,AD1)的参与,并与通用辅激活子CBP(CREB-binding protein)协同,提示组蛋白乙酰化在受体转录激活中的重要作用。
短句来源
     In order to determine the integration and localization of hDAF transgene on transgenic pig chromosomes, nonisotope in situ hybridization technique was applied. The results indicate that exogene hDAF was limitedly distributed on chromosomes of transgenic pigs and localized on 2P, 6P14, 7P11--p13, 7q26, 11q12, 13q44--q48, 14q26, respectively.
     应用非同位素原位杂交技术研究转基因猪外源hDAF基因在受体基因组染色体上的整合及其定位.结果表明,外源基因在转基因猪中呈随机多位点整合,转基因分别定位在2P、6P14、7P11-P13、7q26、11q12、13q44-q48、14q26.
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  相似匹配句对
     Auxin Receptor in Plants
     植物生长素受体
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     Melatonin receptor
     褪黑素受体
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     neuron and receptor.
     神经元普遍存突触前受体
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  at receptor
In ventricular preparations from normal rat and pig hearts, the density of AT1 receptors is higher than that of AT2 receptors, whereas data on the AT receptor subtype density and its distribution in human hearts remain controversial.
      
However, the antiatherosclerotic potential of ACE inhibitors and AT receptor antagonists in humans is still unknown.
      
The isolation of macromolecules that bind drugs and neurohumoral mediators at receptor sites will require analytical methods adequate to detect these substances in homogenates and crude mixtures.
      
Such findings are difficult to reconcile with the widely accepted theory that antidepressant drugs counteract the symptoms of depression by increasing the concentration of noradrenaline and/or serotonin at receptor sites within the brain.
      
had no significant effects even at receptor occupancies approaching 100%).
      
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Inactive mutants (non-rifamycin-producing) of Nocardia mediterranei wereisolated by mutagenic treatment. Appling the spot-method of our laboratoryand the agar-strip method described by Delic et al., forty-eight inactive mutantswere tested for cosynthetic ability, and eight pairs of inactive mutants capableof biochemical complementation were obtained. The cosynthesis action of these pairs was further confirmed in liquid culture,and the rifamycin yields in the mixed culture ranged from 10 to 100 μg/ml. Five of...

Inactive mutants (non-rifamycin-producing) of Nocardia mediterranei wereisolated by mutagenic treatment. Appling the spot-method of our laboratoryand the agar-strip method described by Delic et al., forty-eight inactive mutantswere tested for cosynthetic ability, and eight pairs of inactive mutants capableof biochemical complementation were obtained. The cosynthesis action of these pairs was further confirmed in liquid culture,and the rifamycin yields in the mixed culture ranged from 10 to 100 μg/ml. Five of the products of the complementing pairs were identified to be therifamycin group by their biological activity and characteristic absorption spectraof UV and visible light. These results shown that each complementing pair must consisted of a secre-tor and a convertor. The secretor strain produced an intermediate of rifamycinbiosynthetic pathway, and the convertor converted it into rifamycin. Obviously,the blocked step on the biosynthetic pathway of rifamycin of the convertorshould be earlier than that of the secretor.

本文报导了由力复霉素产生菌,地中海诺卡氏菌诱变得到的非活性突变株,经共合成方法由6株非活性突变株配成8对有共合成能力的生化互补菌株,并证明互补菌株共合成产生力复霉素并不是由于酶的作用,而是由于一株非活性突变株产生一种力复霉素合成途径的中间产物,经另一非活性突变株转化为力复霉素。前者为供体菌,后者为受体菌或转化菌,供体菌在力复霉素生物合成途径上经突变而引起的阻断部位必然在受体菌的后面。 五株突变株生化互补的产物具有生物活性,并经紫外和可见光吸收光谱的测定,证明是属于力复霉素一类环桥抗菌素。

The mechanism of how a sweetener is combined with its receptor by segmental fitting of a definite polar sequence is discussed and the possibilities of the influence of its various functional groups in the course of reaction explored.Thus it can be explained in a rational way why a compound is sweet or nonsweet(a fitting of the ABC sequence is a necessary but iusufficient condition),the rapidity of taste onset (solubility partition and conformational adjustment),the intensity of sweetness (related to the neural...

The mechanism of how a sweetener is combined with its receptor by segmental fitting of a definite polar sequence is discussed and the possibilities of the influence of its various functional groups in the course of reaction explored.Thus it can be explained in a rational way why a compound is sweet or nonsweet(a fitting of the ABC sequence is a necessary but iusufficient condition),the rapidity of taste onset (solubility partition and conformational adjustment),the intensity of sweetness (related to the neural response R_m),the difference between intensity and magnitude of sweetness,i.e,multiples to a standard solution of sucrose by adjusting the concentration of the tested swectener with many times the volume of water until its sweetness equals that of the former solution(related to its adsorption constant K),taste changes from sweet to bitter or tasteless(the magnitude of the ratio r), the inhibition or elimination of a taste,etc..Before a complete elucidation of the receptor structure from primary sequence to the quartenary structure has been achieved,this paper ventures to employ the probe mapping method to develop a sweet taste theory to improve the predictive ability of structure-activity relationships so as to stimulate or to design researches on new sweeteners.

本文以统一的诱导适应受体模型来总结甜剂与受体分段顺序结合的反应机制,探讨了了甜剂分子中各类基团对味感的影响趋势.因而对甜剂的甜与不甜(以 ABC 等顺序为必需条件,但非充分条件),甜感快慢(构象繁简和水溶性大小)、甜味深度(Rm)、其深度与倍数(与 K 有关)的区别、甜苦与甜淡的转变(比值 r 的大小)、甜讯息的抑制以致消失等一系列现象作出了合乎实验事实的理性解释.在受体蛋白的一级至高级结构未测定以前,就如何用探针的方法发展甜味理论,提高其“构效”关系的预见性以及设计新的糖精研究,希望此文能起抛砖引玉的作用.

A plasmid vector pMM15 containing a lac UV5 promoter has been constructed. The plasmid vector has a unique EcoRI restriction site downstream to the lac UV5 promoter which controls the expression of a galactokinase gene in suitable host. Cloning of a DNA segment in the EcoRI site can be easily detected by the appearance of white recombinant colonies in a background of red vector colonies on MacConkey gala-ctose indicator plates.

以pLJ3和pKO-6质粒DNA为材料,经体外重组构建了质粒pMM15。该质粒带有一个lac UV5启动子片段和一个半乳糖激酶基因。二者之间有单一的EcoRI位点。此外,还有一个Amp~1药物抗性标记。分子量约为3.8kb。该质粒在受体细胞内能够产生有活性的半乳糖激酶。在EcoRI位点插入外源DNA后,则半乳糖激酶基因表达受阻。因而在用于研究外源基因的表达及无性繁殖试验中,可通过遗传互补试验,在麦康凯半乳糖指示剂平板上,根据菌落颜色的改变来检出重组转化子。

 
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