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细胞阻滞
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  cell block
     ConclusionObesity in puberty may gradually decrease spermatogenic cells percentage in S phase,make cell block in G2 phaseand delay the mitosis of testicle cells.
     结论青春期时的肥胖可以引起睾丸生精细胞S期细胞百分数逐渐减少,出现G2期细胞阻滞,细胞有丝分裂延迟。
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     Conclusion: MeHg may decrease cell percentage in S phase, inhibit the DNA synthesis and make cell block in G 2 phase.
     结论 :不同剂量的甲基汞可以引起 S时相细胞百分数减少 ,DNA合成受到抑制 ,并出现 G2 期细胞阻滞
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     PTEN is a recently identified tumor suppress gene (TSG), has complexful functions including participating in the embryonic-growth , apoptosis and G1 phase cell block, inhibiting the migration and adherence.
     PTEN为新近发现的一种肿瘤抑制基因,功能十分复杂,具有参与胚胎正常发育,诱导细胞凋亡,引起G_1期细胞阻滞,抑制细胞迁移,铺展和局部粘附功能。
短句来源
     Methods: M16 and CZB media containing 15% FCS or BSA were used to culture the 2-cell embryos from Kunmin mice to study their enhancement to the development of early embryos. Then, 1-cell embryos fertilized in vitro were cultured in M16 and CZB media supplemented with 15%FCS to study the ability of the medium to help 1-cell embryos from Kunmin mice to prevent the 2 cell block.
     方法 :用添加 15%FCS或BSA的M16和CZB培养液培养昆明小鼠 2 -细胞胚胎和体外受精卵 ,以观察这两种培养液对昆明小鼠早胚发育的影响及在克服昆明小鼠 2 -细胞阻滞中的作用 ,并通过CZB培养液中葡萄糖成份的增减 ,了解其作用及效应时间 .
短句来源
     This is the first report that M16 medium added with taurine and EDTA can be used in both IVF and culture medium to overcome the 2 cell block of embryo development in Kunming strain mouse.
     本研究进一步证明了牛磺酸和EDTA在昆明小鼠早期胚胎发育和克服2-细胞阻滞中起关键作用。
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  cell arrest
     Cell cycle analysis indicated that AraC blocked HL 60 cells in G 1, inhibited cells in S, while EGCG had no effect on cell cycle at the current concentration, but could enhance the cell arrest by AraC.
     细胞周期研究结果表明AraC可使HL 6 0细胞阻滞于G1期 ,S期细胞减少 ,低浓度的EGCG对细胞周期几无影响 ,但增强了AraC的细胞周期阻滞作用及细胞凋亡。
短句来源
     Exposed to different dose of VK 2 for 72 h, G 0/G 1 cell arrest, but the expression of CD 14 was not notablely enhanced than control group.
     G0 G1 期细胞阻滞 ,CD1 4的表达与对照组相比无差异性。
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     Exhibiting a Mistake of Cell Cycle Analysis in Flowcytom etry through Analyzing the Cell Arrest Model Induced by Camptothecin
     从细胞阻滞模型来看流式细胞术中细胞周期分析的误区
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     Results 1. celebrex inhibited cell survival of NSCLC in a time-dependent and concentration-depended manner, and the effect of celebrex were more pronounced in NCI-H520 than in A549.2.Flow cytometry show that celebrex induced a G0/G1 cell arrest in NSCLC.
     而且,在相同条件下塞来昔布对NCIH520的抑制率明显高于对A549的抑制率。 流式细胞学试验证明,塞来昔布作用后G0/G1细胞比例上升,细胞阻滞于G0/G1期。
短句来源
  cell blocking
     Flow cytometric analysis revealed G 0/G 1 stage cell blocking phenomena, the hypodiploid apoptotic cell increased, displayed typical peak of apoptosis, the rate of cell apoptosis and YQJP serum concentration were in a dose dependent manner.
     流式细胞仪分析显示G0 /G1期细胞阻滞现象 ,亚二倍体凋亡细胞增多 ,出现典型的凋亡峰 ,细胞凋亡率与中药血清浓度具有量效关系。
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  “细胞阻滞”译为未确定词的双语例句
     BxPC-3 cells treated with TSA was arrested in G0/G1 phase. Compared with the blank control group (42.5±2.2)% and ethanol group (47.3±3.4)%,TSA group (61.8±2.5)% was significantly increased (P=0.004).
     TSA可将BxPC-3细胞阻滞于G0/G1期,TSA组G0/G1期细胞达(61.8±2.5)%,较空白对照组(42.5±2.2)%和乙醇对照组(47.3±3.4)%明显增多(P=0.004);
短句来源
     Results: Reversing effect of P-gp expression was found in SGC7901/ADM cell line when treated with different concentrations of As2O3, ranging from 0.1μmol/L to 0.75μmol/L, and cell cycle was blocked at the period of Go-G1, augmented with the increasing concentration of As2O3 (P<0.01).
     结果:As2O3在0.10~0.75μmol/L浓度范围内能够逆转SGC7901/ADM细胞内P-gp的表达,并将细胞阻滞于G0~G1期、诱导细胞凋亡(P<0.01),其作用随As2O3剂量增加而增大。
短句来源
     50μmol/L OAG can arrest the cell cycle in G2/M if it was added in the early of G2 in NIH3T3 cells.
     在G2早期加入 OAG50μmol/L可以引起NIH3T3细胞阻滞在G2/M期交界。
短句来源
     AMSA induced Molt 4 cells to to arrested at G 2/M phase;
     胺苯吖啶诱导Molt 4细胞阻滞于G2 /M期 ;
短句来源
     HL-60 cells in S phase decreased from (56.1±4.7)% to (17.8±3.2) % and cells were arrested in G0/G1 phase.
     S期细胞比值由(56.1±4.7)%降至(17.8±3.2)%,细胞阻滞在G0/G1期。
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  cell block
Fifty-one of 84 cases were diagnosed as malignant or suggestive of malignancy in cytological smears and/or cell block sections.
      
In this study, pronuclear embryos from Kunming mice, characterized by an obvious two-cell block in vitro, were co-cultured with mouse, goat, and chick OECs.
      
Aspiration with an 18-gauge needle and fixing the aspirate to form a cell block was found to be a safe and satisfactory way of obtaining material from cysts or nodules too small to be safely and easily sampled by a Tru-cut? technique.
      
Both specimens were assessed for malignancy using four techniques: filtration process (ThinPrep?), smear, cell block, and immunochemistry using Ber-EP4.
      
These were fixed in formalin, suspended in agar, and embedded in paraffin to produce a cell block.
      
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  cell arrest
Furthermore, the research on the molecular mechanism ofp15 in regulation of cell proliferation shows thatp15 can inhibit the growth of some kinds of tumor cells, andp15 is the mediator of TGF-β-induced cell arrest.
      
Taxol induced significant cell arrest at G2-M phase in monolayer cells (P=0.001), but abrogation of G2-M arrest was observed in MCA cells (P=0.002).
      
The E2 protein can also promote cell arrest and apoptosis in HeLa cells.
      
Whereas Bcl-2 had no effect on etoposide-induced cell arrest, it interrupted all aspects of apoptosis, including activation of caspases, poly(ADP-ribose) polymerase degradation, DNA fragmentation and loss of plasma membrane integrity.
      
Tumour cell arrest in the vascular system and latent phases are important for the occurrence of late metastases.
      
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  cell blocking
In the treatment of food allergy specific elimination diets as well as pharmacotherapy with the use of mast cell blocking agents are recommended.
      
Discharge patterns of single motoneurones were compared before and after opening of the recurrent inhibitory pathways by Renshaw cell blocking agents.
      
These CCH's were compared before and after opening of the recurrent inhibitory loop by Renshaw cell blocking agents.
      
The state probabilities and the corresponding cell blocking probability are determined by means of an exact analysis.
      
For cases (2) and (3), where we deal with more complex input traffic, the compound state process and the overall cell blocking probability are analyzed using a quasi-stationary approximation technique.
      
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There are 16 amiuo acids found so far from the extraction of S-O2-I bacterial cell wall skeleton.The experimental results showed that phagocytosis and the YC-rosette forming activity of macrophages from S-CWS immunized C57BL/6 mice were enchancod.The peritoneal exudate cells (PBC) inducsd by S-CWS in C57BL/6 mice had a cytost-atic activity against S180 tumor cells.

本实验提取的S-O_2-1细菌细胞壁骨架(Cell Wall Skeleton,S-CWS)测出有16种氨基酸。 实验证明,经过S-CWS激活后的小鼠巨噬细胞的吞噬百分率和吞噬指数均有提高,酵母(YC)花环形成率达对照组的3.3倍。 巨噬细胞对肿瘤细胞的体外细胞阻滞实验结果证明,经S-CWS激活后的巨噬细胞对肿瘤细胞的阻滞效应(Cytostasis effect)显著提高。

In this paper the authors studied quantitatively the micronucleus formation by chemical mutagens at various phases of interphase in liuman lymphocytes induced by means of control of cell culture intervals, autoradiography and block of meta- phase cells etc. The results show that mutagenic chemotherapy drugs : bimolane (treatment in vitro)and thio tepa etc.(treatment in vivo)can induce micronucleus formation at various phases of interphase in lymphocyte cell cycle.The frepuency of micronucleus (MNF) induced at...

In this paper the authors studied quantitatively the micronucleus formation by chemical mutagens at various phases of interphase in liuman lymphocytes induced by means of control of cell culture intervals, autoradiography and block of meta- phase cells etc. The results show that mutagenic chemotherapy drugs : bimolane (treatment in vitro)and thio tepa etc.(treatment in vivo)can induce micronucleus formation at various phases of interphase in lymphocyte cell cycle.The frepuency of micronucleus (MNF) induced at G1 phase is signifcantly higher than at G0 and G2 phase.The MNF at S phase of cells is significantly lower than at G1. This result suggests that most of micronucleated cells at G1 phase do not enter S phase and micronucleus could be formed rarely at S phase of cells.

本实验应用具有诱变作用的化疗药:噻地哌、长春新碱、乙双吗啉等,体内或体外处理诱发人体外周血淋巴细胞微核,通过控制细胞培养时间,放射性自显影及中期细胞阻滞等方法,定量地分析了细胞间期各阶段的微核率。结果表明,间期各阶段均可有不同程度的微核形成,其中最多的是G_1期,其次是G_2期和G_0期。S期细胞的微核率(MNF)较G_1期有极显著的下降,这提示大部分G_1期的微核细胞不能进入S期,使细胞增殖中止,这可能是抗癌药物杀伤肿瘤细胞的机制之一。

In order to surmount the embryonic development block of Kunming white mice andmake the zygotes develop to the blastocysts stage, the animals were divided into two experi-mental groups and a control group. The medium of the first experimental group consisted ofBrinster's medium, oviduct epithelial cells and oviductal fluid. The medium of the secondgroup was modificatory Brinster's medium. The medium of the control group was Brinster'smedium. The embryos were cultured in a humidified atmosphere of 5% CO_2 and...

In order to surmount the embryonic development block of Kunming white mice andmake the zygotes develop to the blastocysts stage, the animals were divided into two experi-mental groups and a control group. The medium of the first experimental group consisted ofBrinster's medium, oviduct epithelial cells and oviductal fluid. The medium of the secondgroup was modificatory Brinster's medium. The medium of the control group was Brinster'smedium. The embryos were cultured in a humidified atmosphere of 5% CO_2 and 95% air at 37℃. The experimental result was as follows: The average percentage of embryonic develop-ment from the zygotes to the blastocysts stage in the first group was 43.7±7.76%. In the sec-ond group it was 17.1±5.00%. In the control group it was 15.3±3.06%. The study testified that Brinster's medium with oviduct epithelial cells and oviductal fluidcould surmount the block of mouse embryonic development to a certain degree.

为克服昆明白小鼠胚胎的2—细胞阻滞并使受精卵发育到囊胚期,本研究设两个实验组和一个对照组。第1实验组所用培养液为Brinster氏化学合成培养液与输卵管上皮和输卵管液混合的协同培养液;第2实验组用改良Brinster氏化学合成培养液;对照组用经典的Brinster培养液。培养是在5%CO_2和95%空气,37℃的CO_2箱内进行。3次体外培养试验结果是:第1实验组小鼠胚胎从受精卵发育到囊胚期平均发育率为43.7±7.76%;第2实验组的平均发育率为17.1±5.06%;对照组的平均发育率是15.3±3.06%。统计学分析表明,添加输卵管上皮和输卵管液的Brinster培养液对克服昆明白小鼠胚胎2—细胞阻滞具有一定作用。

 
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