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纤维化病变
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  fibrotic lesions
     The protein expression of TSP 1 and uPA in areas of remnant renal tubulointerstitium,the correlation between both proteins and TSP 1 with tubulointerstitial fibrotic lesions were estimated by means of immunohistochemical technique.
     免疫组织化学染色观察残肾组织TSP 1与uPA蛋白表达变化 ,并比较二者表达的相关性及TSP 1与间质纤维化病变的相关性。
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     An observation on focal myocardial fibrotic lesions was made in hypertensive Wistar rats with one clipped kidney in two.
     观察了两肾一夹型高血压Wistar大鼠心肌局灶性纤维化病变的病理形态。
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  “纤维化病变”译为未确定词的双语例句
     The expression of CTGF was found to have a close correlation with the expression of FN(r = 0.6855, P < 0.001), collagen Ⅳ (r = 0.5964, P < 0. 01), and the degree of interstitial fibrosis (r = 0.4941, P < 0. 05) .
     (3)肾间质内CTGF表达分别与FN及IV型胶原沉积正相关(r=0.6855和0.5964,P<0.01)、与纤维化病变程度正相关(r=0.4941,P<0.05)。
短句来源
     TGF-β family members play an important role in fibrotic diseases. TGF-βs have three types of receptors: TGF-βRⅠ, TGF-βRⅡ, TGF-βRⅢ.
     TGF-βs在纤维化病变的发生发展中起重要作用,其受体主要分三型:TGF-βRⅠ、TGF-βRⅡ和TGF-βRⅢ。
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     Hepatic fibrosis histopathological changes were remarkable alleviated in three dosage groups of CK comparing with those of model group(P<0.01).
     抗纤软肝方高、中、低剂量组肝组织纤维化病变明显减轻,与模型组比较,差异均有显著性意义(P<0.01)。
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     CONCLUSION:Hyperbaric oxygen with free radical antagonists can decrease the expression of MMP-2 in rat liver damaged by CCl4,delaying the development of liver fibrosis.
     结论:高压氧结合自由基拮抗剂可以降低 CCl_4损伤大鼠肝脏 MMP-2表达,可以迟滞肝纤维化病变的发展.
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     And there was a positive correlation between the expression of Collagen type Ⅳ and the degree of tubulo interstitial fibrosis(r=0.631 4,P<0.05).
     Ⅳ型胶原与间质纤维化病变程度呈正相关 (r=0 .6 31 4 ,P <0 .0 5 )。
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  相似匹配句对
     ~1H Nuclear Magnetic Resonance Applied to Hepatic Fibrosis of Rat Liver Tissues
     大鼠肝脏纤维化病变的核磁共振研究
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     Effects of Myocardial Fibroblasts on Fibrosis in Diabetic Cardiomyopathy
     成纤维细胞在糖尿病心肌纤维化病变中的作用
短句来源
     Pancreatic fibrosis
     胰腺纤维化
短句来源
     interstitial fibrosis.
     间质纤维化
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     Space-occuping Lesions of brainstem
     脑干占位病变
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  fibrotic lesions
Light microscopy revealed patchy fibrotic lesions predominately associated with the subendocardial half of of the collateral dependent myocardial wall.
      
Fibrocytes can further differentiate, and they are a likely source of the contractile myofibroblast that appears in many fibrotic lesions.
      
Less severe fibrotic lesions were also observed in the BM+E group.
      
In the histopathologic assesment of lung sections in the bleomycin+vitamin E group, partial fibrotic lesions were observed, but the histopathologic changes were much less severe compared to the bleomycin-treated group.
      
In the course of CVB3 myocarditis, CTGF upregulation coincided with increased cardiac TGF-β and procollagen type I mRNA expression, preceding the formation of fibrotic lesions.
      
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In order to explore the pathogenetic capacity of the diatomoceous dust. We did a experiment of the pulmonary fibrotic effect of the treated and untreated diatomoceous dust was studied in rat. Lungs and lymph nodes were observed histo-pathologically, and the wet and dry weight as well as the total collagen content of the lung were also measured. The results indicated that the severity of fibrosis was shown to be higher in the treated group than that in the untreated group. In addition, similar tendency was found...

In order to explore the pathogenetic capacity of the diatomoceous dust. We did a experiment of the pulmonary fibrotic effect of the treated and untreated diatomoceous dust was studied in rat. Lungs and lymph nodes were observed histo-pathologically, and the wet and dry weight as well as the total collagen content of the lung were also measured. The results indicated that the severity of fibrosis was shown to be higher in the treated group than that in the untreated group. In addition, similar tendency was found in the change of organ weight and collagen content. The differences between those two groups were proved to be highly significant statistically.

本研究对大白鼠肺脏注入生、熟硅藻土粉尘,经病理组织学及其它指标的观察,认为硅藻土粉尘可以引起肺内的纤维化病变,但生硅藻土引起的病变轻,熟硅藻土引起的病变重。

Pathological effects on rat lungsby three organic dusts(silk,featherand cotton dusts) were studied.Atotal of 192 rats were administratedintratracheally at a dosage of 50mg/ml/rat of each dust suspension,andtwo additional groups were given thesame dosage of distilled water andquartz dust suspension as controls.Duration of the study lasted for awhole year Results of the study showed thatsilk and feather dust induced similarchanges in rat lungs:no spots,lumpsor rigid tubercles can be found in thelung.Microscopically,with...

Pathological effects on rat lungsby three organic dusts(silk,featherand cotton dusts) were studied.Atotal of 192 rats were administratedintratracheally at a dosage of 50mg/ml/rat of each dust suspension,andtwo additional groups were given thesame dosage of distilled water andquartz dust suspension as controls.Duration of the study lasted for awhole year Results of the study showed thatsilk and feather dust induced similarchanges in rat lungs:no spots,lumpsor rigid tubercles can be found in thelung.Microscopically,with slightlymphadenia was found around the br-onchi and blood vessels,with hyperp-lasia on some parts of the interalveolar septa,and organic dust fibreswere found both in the alveoli andinteralveolar septa.Inflammatorycells from bronchi cavity and simplegranuloma may occasionally be found1 month after dust exposure. Bathological changes of lung atearlier stage of cotton dust exposurewere mainly of foreign body granu-loma,which gradually disappears la-tter.These granuloma were mainly oflocalized emphysema and hyperplasiaof interalveolar septa in nature. Through the present experimentalperiod only simple node of lymphocy-tes and phagocytes were occasionallyfound,but no typical fibrotic lesionwas caused by the three organic dustson the rat lung.

本文报道了三种有机粉尘(丝尘、羽毛尘、棉尘)对大鼠肺脏影响的病理观察。实验用192只大鼠从气管分别注入三种有机尘混悬液50mg/ml/只。另外以注入1ml/只蒸馏水作为对照组,以注入石英粉尘混悬液50mg/ml/只作为石英对照组,观察一年。结果是羽毛尘和丝尘引起肺脏的改变基本相仿:肺脏无点块状病灶,无硬性结节。镜下见支气管、血管旁淋巴组织轻度增生,部份肺泡间隔增厚,肺泡腔和间隔中有聚集的或散在的有机尘纤维,支气管腔有炎性细胞渗出,染尘一个月内偶见单个肉芽肿。棉尘引起的改变主要为早期肉芽肿,后期逐渐消失,以局限性肺气肿及肺泡间隔增厚为主。三种粉尘全年偶见个别以淋巴细胞和吞噬细胞为主的小结节,是否为有机纤维尘性细胞反应,未见肺脏有典型的纤维化病变

Pathological analysis of twelve cases of Primary cardiomyopathy has been made.of them 6cases are of hypertrophic type.2 are of dilated type and 4 are with endocardioal fibroelastosis. The hypertrophic type is characterized by hypertrophy of Ventricular septum,bizarre arrengment of various sized muscle fibers and severe fi- brosis and other changes including prominert proliforetion of hypertrop- hied smooth muscle cells and marked endo-fibroelactosis of small coron- ary arteries.The pathological changes of 2cases...

Pathological analysis of twelve cases of Primary cardiomyopathy has been made.of them 6cases are of hypertrophic type.2 are of dilated type and 4 are with endocardioal fibroelastosis. The hypertrophic type is characterized by hypertrophy of Ventricular septum,bizarre arrengment of various sized muscle fibers and severe fi- brosis and other changes including prominert proliforetion of hypertrop- hied smooth muscle cells and marked endo-fibroelactosis of small coron- ary arteries.The pathological changes of 2cases of dilated type is similar to that seen in the hypertrophic type.The one of 4 cases of endocardical fibroelactosis amony whom has unexpectedly survived for 49 years.The pathological changs in interstitial small arteries are similar to these wh- ich hadbeen seen in the endo-cardium.

本文对原发性心肌病猝死12例进行了病理分析。其中肥厚型心肌病6例,扩张型心肌病2例,心内膜弹性纤维增生症4例。肥厚型:室间隔明显增厚,镜检可见排列紊乱、形态奇特的心肌纤维和明显的纤维化病变,间质中的小动脉平滑肌肥厚,内膜增厚;扩张型:病变与肥厚型类似;心内膜弹性纤维增生症:其中1例活到49岁,较为罕见,其病理改变除各室腔内膜明显弹性纤维与胶原纤维增生外,心肌间质中小动脉内膜也具有同样病变。

 
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