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多重缺失
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  multiple deletion
     With PCR amplification,the authors analyzed the 7.436kb mtDNA deletion in fifty individiuals,an age-dependent increase of mtDNA deletion was found in myocardial cells and blood lymphocytes and multiple deletion was found within lymphocyte mtDNA. The amount of deleted mtDNA was about one tenth that of normal mtDNA.
     淋巴细胞和心肌细胞mtDNA均存在包含有7.436kb缺失的片断,而前者有多重缺失,发生缺失的mtDNA片断约占正常mtDNA的10.3%。
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  “多重缺失”译为未确定词的双语例句
     Association of novel POLG mutations and multiple mitochondrial DNA deletions with variable clinical phenotypes in a Spanish population
     西班牙人群中POLG新突变与不同临床表型线粒体DNA多重缺失之间的联系
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     Results ①The gene deletion accounted for 56 7% (17 cases) of total patients,in which monogene deletion held 47% (8),multigene deletion 52 9% (9);
     结果 ① 30例患儿中基因缺失者 17例 (5 6 7% ) ,单一缺失者 8例 (47 1% ) ,多重缺失 9例(5 2 9% )。
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     CONCLUSION: ①Higher multiple mtDNA deletions were detected in wet AMD patients.
     结论 :①湿性AMD患者体内可能存在较高频率的mtDNA多重缺失 ;
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     Methods By using long polymerase chain reaction techniques, we studied multiple deletions of sperm mitochondrial DNA(mtDNA) in 100 infertile patients(60 with normal sperm mobility and 40 asthenospermia).
     方法 用长链 PCR技术 ,对 6 0例精子活力正常和 40例精子活力异常不育患者的精子线粒体 DNA(mt DNA)进行了多重缺失的分析。
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     Conclusion There is no relationship between multiple deletions in mtDNA and human sperm mobility.
     结论 精子活力与 mt DNA的多重缺失间无相关性。
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  相似匹配句对
     Multiple Imputation in the Missing Data
     缺失数据的多重估算
短句来源
     Analysis of Deletion in Dystrophin Gene by Multiplex Polymerase Chain Reaction
     多重聚合酶链反应分析Dystrophin基因缺失突变
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     On Multiple Questions
     多重疑问句
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     On Multiple Copyrights
     试论多重著作权
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     Malposition and Shortage
     错位与缺失
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  multiple deletion
Using PCR-based gene replacement, a series of multiple deletion strains was constructed by multiple disruption of a maximum of seven protease genes.
      
Effects of the resultant multiple deletion strains on heterologous expression were then measured by practical expression of a proteolytically sensitive model protein, the human growth hormone (hGH).
      
Stochastic systems of Brownian motions with multiple deletion of particles are introduced to model a chemical reaction with diffusion.
      
Multiple deletion mutants of CYP2D6 were constructed from a human liver cDNA library and five recombinant fusion proteins expressed.
      
In addition, multiple deletion mutations have occurred at this locus.
      
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Abstract Besides nuclear DNA,mitochondrial DNA(mt DNA) is also a genetic substance in human body and may be damaged more often than nuclear DNA.With PCR amplification,the authors analyzed the 7.436kb mtDNA deletion in fifty individiuals,an age-dependent increase of mtDNA deletion was found in myocardial cells and blood lymphocytes and multiple deletion was found within lymphocyte mtDNA.The amount of deleted mtDNA was about one tenth that of normal mtDNA.It is suggested that mitochondrial DNA deletion might be...

Abstract Besides nuclear DNA,mitochondrial DNA(mt DNA) is also a genetic substance in human body and may be damaged more often than nuclear DNA.With PCR amplification,the authors analyzed the 7.436kb mtDNA deletion in fifty individiuals,an age-dependent increase of mtDNA deletion was found in myocardial cells and blood lymphocytes and multiple deletion was found within lymphocyte mtDNA.The amount of deleted mtDNA was about one tenth that of normal mtDNA.It is suggested that mitochondrial DNA deletion might be a contributory factor to aging and presbycardia.

用聚合酶链反应(PCR)技术对50例心脏病患者及健康人线粒体DNA(mtDNA)中遗传物质缺失的研究发现,随年龄增长,发生缺失的mtDNA比例增加。淋巴细胞和心肌细胞mtDNA均存在包含有7.436kb缺失的片断,而前者有多重缺失,发生缺失的mtDNA片断约占正常mtDNA的10.3%。提示mtDNA的缺失可能在衰老和老年人心脏病的发生机制中有一定意义。

mplification of one of the frequently deleted re-gions of myocardial and

应用聚合酶链反应(PCR)扩增扩张型心肌病(DCM)及急性心肌梗塞(AMI)病人活检或尸检心肌及淋巴细胞线粒体DNA(mtDNA)缺失常发区之一,并与正常对照比较。结果发现病人及正常对照mtDNA8530~600位之间(相距8.7kb)均存在不同程度的多重缺失,2例AMI病人心肌及10例DCM病人心内膜心肌活检标本均扩增出1.lkb片段,而28例DCM病人和14例正常对照淋巴细胞内则多可扩增出4个片段,对应缺失长度在7/7~8.8kb之间,心肌内mtDNA缺失几乎均可在淋巴细胞mtDNA反映,1.lkb片段阳性率有随年龄增大而升高的趋势,含缺失区的mtDNA所占比例与年龄成正比;占总mtDNA的比例小于10%,SSCP分析发现L853/H933扩增片段存在有点突变。

Purposes: To investigate mitochondrial DNA mutation associated with oxidative phosphorylation defect due to aging and to inspect it whether to play a role in the pathogenesis of age-related macular degeneration(ARMD).

目的:研究与衰老和氧化磷酸化功能缺陷有关的细胞线粒体DNA(mtDNA)突变,从基因水平探讨老年黄斑变性(ARMD)的发病机理。方法:应用聚合酶链反应(PCR)对20例老年黄斑变性(ARMD)病人之血细胞线粒体DNA(mtDNA)缺失进行了初步研究。结果:有6例湿性ARMD扩增出2条异常DNA片段,提示在mtDNA位点7901~13650之间存在有2种mtDNA缺失,其缺失片段大小分别为5.0kb和5.2kb。另有5例湿性ARMD扩增出1条1.2kb的异常片段,提示在mtDNA位点8531~13400之间还存在1种长度为3.67kb的缺失片段,10例对照均未扩增出异常mtDNA片段。结论:提示在湿性ARMD病人血细胞mtDNA存在有包括与年龄相关的长度为5.0kb在内的多重缺失,mtDNA突变与ARMD发病机制的关系还需进一步深入研究。眼科学报 1997;13:52

 
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