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hdl
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  hdl
     Also it could increase the levels of serum high density lipoprotrin subgroup2 cholesterol(HDL 2 C), HDL C/TC, HDL C/LDL C, HDL 2 C/HDL C and HDL 2 C/HDL 3 C.
     可明显提高血清高密度脂蛋白亚组分2胆固醇(HDL2C)的含量、提高HDLC/TC、HDLC/LDLC、HDL2C/HDLC及HDL2C/HDL3C。
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     whereas the levels of HDLC,HDL2C,apoA1 and the ratio of HDL2C/HDL3C,apoA1/apoB were markedly reduced,showing significant difference between both groups (t=1.996~2.762,P<0.05,0.01).
     而HDLC,HDL2C,apoAI水平及HDL2C/HDL3C,apoAI/apoB比值比对照组明显降低,两组间比较差异均有显著意义(t=1.996~2.762,P<0.05,0.01);
短句来源
     to detect high density lipoprotein C(HDLC)and its subcomponents (HDL2C and HDL3C) by sodium phosphotungstateMg2+ one step precipitation method;
     用磷钨酸钠Mg2+一步沉淀法检测高密度脂蛋白(HDLC)及其亚组分(HDL2C和HDL3C);
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     LDL C was calculated. RESULTS:After 4 and 8 weeks treatment,TC was decreased by 26.2% and 25.0%( P <0.001)respectively,LDL C was decreased by 32.1% and 34.2%( P <0.001) and HDL C was decreased by 13.9%( P <0.05)and 6.4% ( P >0.05).
     结果:服用普罗布考片4周和8周后,患者TC下降26.2%和25.0%(P均<0.001),LDLC下降32.1%和34.2%(P均<0.001),HDLC下降13.9%(P<0.05)和6.4%(P>0.05)。
短句来源
     RESULTS:At the end of the study,the serum level of TC,LDL C,TG,and TC/HDL C ratio in the active group were lowered by 12.7%,10.1%,34 7%,and 15.9%( P <0.05~0.001)respectively and the HDL C was elevated by 11.7%( P <0.001)in comparison with baseline data.
     结果:与服药前相比,8周后,决明子散剂组平均血清TC、LDLC、TG水平和TC/HDLC比值分别下降12.7%、10.1%、34.7%、及15.9%,HDLC升高11.7%(P<0.05~0.001)。
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  “hdl”译为未确定词的双语例句
     It was found that total cholesterol, triglyceride, very low density lipoprotein, low density lipoprotein cholesterol and atherosclerosis index were significantly increased( P <0.05, and P <0.01), while the level of high density lipoprotein was decreased( P <0.05), in obese children than in healthy children.
     发现肥胖儿童较健康儿童胆固醇(TC)、甘油三酯(TG)、极低密度脂蛋白胆固醇(VLDLC)及低密度脂蛋白胆固醇(LDLC)、动脉硬化指数(AI)明显增高(P<0.001、0.01及0.05),高密度脂蛋白胆固醇(HDLC)及其与TC之比值明显降低(P<0.05及0.001)。
短句来源
     Lowdensity liporotein cholesterol (LDLc) were measured by enayme.
     并用同一标本采用酶法测定HDLc,LDLc。
短句来源
     Results BMI of PIH group was significantly higher than that in control group ( P <0.01); newborn's body weight in PIH group was significantly lower than that in control group ( P <0.01); TG,ApoCⅡ,ApoCⅢ and ApoCⅡ/CⅢ in PIH group were significantly higher than that in control group ( P <0.01);
     结果 妊高征组体质指数高于对照组( P<0.01),新生儿出生体重低于对照组(P< 0.01) ,妊高征组TG、ApoCⅡ、ApoCⅢ和ApoCⅡ/CⅢ高于对照组( P< 0.01) ,HDLC、LDLC低于对照组( P<0 .01);
短句来源
     There were also no statistical differences in GHbA1C, fasting and postpradial glucose, C peptide levels between group A and group B. Group A had a raised albeit nonsignificantly so, mean fasting serum insulin concentration as compared with group B. But group A had increased postprandial 1 h, 2 h and 3 h insulin concentration when compared with group B (P<0.05).
     更存在ApoA1、ApoA2、ApoA1/ApoB、HDLc/TC比值(A组)等反映保护因子水平的显著下降,P均<0.01。
短句来源
     Results After three months , in the treatment group seru m cholesterol, triglyceride ,low densitylipoprotein cholesterol and fasting glucose decreased obviously , high density lipoprotein cholesterol increasedslightly , glucose tolerance improved and insulin sensitivity index increased .
     结果 3 个月以后发现治疗组血胆固醇、甘油三酯, L D L C,空腹血糖有较明显下降, H D L C 轻度上升,糖耐量改善,胰岛素敏感指数上升。
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  相似匹配句对
     PAI 1 higher correlated with ISI, tPA, WHR, HDL C PAI 1 independently correlated with ISR.
     以BMI、WHR、ISI、HDLC、LDL?
短句来源
     HDL-C, LDL-C;
     HDL:a.
短句来源
     Gly.
     Gly。
短句来源
     HDL C was significantly lower in severe OSAS than in control.
     重度OSAS组HDLC较正常组明显降低。
短句来源
     functions and serum lipid, HDL-C/T.
     血脂HDL-C/T.
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  hdl
CETP thus plays a potential pro-atherogenic role by moving CE from HDL into pro-atherogenic VLDL and LDL particles, thereby lowering atheroprotective HDL cholesterol.
      
VEGF, microalbuminuria, HbA1c, creatinine, triglycerides, total cholesterol, fasting serum glucose, high-density lipoprotein (HDL), and low-density lipoprotein (LDL) were measured.
      
Serum VEGF was found to be negatively correlated with serum HDL.
      
Mean serum high-density lipoprotein cholesterol (HDL-c) levels were decreased significantly from 1982-1983 to 1997-1999, with the changes from -13.2% to -38.6% in males and from -9.8% to -21.7% in females.
      
The ratio of total cholesterol to high-density lipoprotein cholesterol (TC/HDL-c) increased dramatically during this period because of a concurrent decline in the levels of HDL-c and the increase in the levels of TC.
      
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A method of using dextran-Sulfate ( D.S. ) and Catt in combination with poly-aerylamide gel preparative electrophoresis to isolate serum high density lipoprotein ( HDL ) in a pure form was developed. In this method HDL was precipitated by D.S, and Catt from serum freed previously of very low density lipoprotein (VLDL ) and low density lipoprotein ( LDL ) in a form of complex, which can be redissolved in 12% NaCl and reprecipitated by dilution and addition of D.S. and Catt. The above process was repeated...

A method of using dextran-Sulfate ( D.S. ) and Catt in combination with poly-aerylamide gel preparative electrophoresis to isolate serum high density lipoprotein ( HDL ) in a pure form was developed. In this method HDL was precipitated by D.S, and Catt from serum freed previously of very low density lipoprotein (VLDL ) and low density lipoprotein ( LDL ) in a form of complex, which can be redissolved in 12% NaCl and reprecipitated by dilution and addition of D.S. and Catt. The above process was repeated twice. The precipitate was finally dissolved in 0.5M.K2G2O4 solution. After centrifugation an orange coloured supernatant which contained mainly HDL was obtained.In order to prepare HDL in pure form the supernatant was, further separated and isolated by preparative gel electrophoresis and eluted with Tris-glycine buffer.One main peak appeared in the elution profile. The eluates were analysed with agarose gel, polyacrylamide gel and immuno-electrophoresis.Pure HDL was present in the eluent corresponding to the accending side of the peak. Further characterization of the pure HDL solution with electron microscope:the HDL particles were spherical in shape with a diameter of 80 A to 130A.The present paper offers a method of isolation and purification of HDL from serum without ultracentrifugation.

作者以多聚阴离子—硫酸右旋糖酐沉淀分离,再以聚丙烯酰胺凝胶制备电泳进一步纯化,可获得较纯净的血清高密度脂蛋白。为不具备超速离心机的实验室提供一个可以较大量分离提纯血清高密度脂蛋白的方法。

Insulin secretion and carbohydrate andlipid metabolic changes were studied in31 normal subjects, 11 patients withmaturaty onset diabetes mellitus (MO-DM) and 59 persons of various degreeglucose intolerance. The plasma glucosetriglycerides, cholesterol, HDL-chole-sterol, free fatty acid (FFA), cAMP,lecithin cholesterol acyl transferase(LCAT) and immuno-reactive insulin (IRI) response to an oral load of100 gm of glucose were evaluated. The results of this study showed thatthe plasma glucose area in the subjectsof...

Insulin secretion and carbohydrate andlipid metabolic changes were studied in31 normal subjects, 11 patients withmaturaty onset diabetes mellitus (MO-DM) and 59 persons of various degreeglucose intolerance. The plasma glucosetriglycerides, cholesterol, HDL-chole-sterol, free fatty acid (FFA), cAMP,lecithin cholesterol acyl transferase(LCAT) and immuno-reactive insulin (IRI) response to an oral load of100 gm of glucose were evaluated. The results of this study showed thatthe plasma glucose area in the subjectsof slight, moderate and severe degree ofglucose intolerance and MODM wereincreased than that of the normal groupby 33.8%, 52.9%, 82.2% and 154.7%respectively after oral glucose. Whilethe plasma IRI area were increasedthan that of normal group by 67.9%92. 1 %, 90. 2% and 21. 4% respectiyely.The plasma IRI area/glucose area ratioin various glucose intolerance groupwere larger than that of normal groupby 25. 5%, 25. 5% and 4% respectively,but, the ratio in diabetic group weresmaller than that of normal subjects by52. 3%. Significant hyperglucemia andhyperinsulinemia were observed in thepatients with MODM and the subjectswith glucose intolerance after oral glu-cose. The plasma triglycerides, cholesterol,and FFA levels in the diabetic groupand glucose intolerance group were sig-nificantly higher than those in the nor-mal group (P<0. 001), and the levelsof triglycerides increased to the greatestextent, amounting to 189%, 264%,265% and 269% respectively. The pla-sma HDL-cholesterol levels and theHDL-cholesterol/cholesterol ratio inpatients with MODM and glucose in-tolerance were lower than those in thenormal group (P<0. 05 and P<0. 001). The plasma triglycerides levels of thesubjects with glucose intolerance andpatients with diabetes were higher thanthe fasting levels 30 minutes after theintake of glucose meal by 19.0%,17. 9%, 21. 7%and 10. 0% respectively. FFA levels were significantly decreased(P<0.01 and P<0.001) after oralglucose load. While the plasma LCATtended to reduce 2 hours after the in-take of 100 gm of glucose. The average levels of fasting plasmacAMP in the moderate and severe glu-cose intolerance groups were loweredby 30. 3% and 38.9% respectively ascompared with the fasting level in thenormal group (P<0.05), while thefasting plasma cAMP level in MODMgroup was the same as that in the nor-mal group (20. 1±1. 8 pmol/ml).The plasma cAMP levels in the subjectsof various glucose intolerance werelowered 60-120 minutes after the intakeof glucose, while that of the diabeticgroup was insignificantly decreased butstill significantly increased by 63. 4%as compared with the normal group(P<0. 05) 3 hours after oral glucose.Significant insulin resistance wasobserved in the subjects with variousdegree of glucose intolerance and pa-tients with MODM.The results are dis-cussed.

对31例正常人,11例成年型糖尿病患者及59例不同程度(轻、中、重度)耐糖量减低者负荷糖后胰岛素的分泌及血糖、血脂以及血浆cAMP的改变进行了分析。结果发现,耐糖量降低者,胰岛素的分泌有不同程度增加,峰值有不同程度之延后,胰岛素面积/血糖面积比值有不同程度之增加,而糖尿病组则未见明显之峰值,比值反较正常降低52.3%。血脂除HDL-胆固醇下降外,均较正常明显升高(P<0.001)。中度及重度耐糖降低组空腹血浆cAMP含量分别较正常降低30.3%及38.9%(P<0.05),而糖尿病组与正常组比较无差异。负荷糖后,糖尿病组cAMP含量不仅不下降,3小时后反较正常组升高63.4%(P<0.05)。对所得结果进行了讨论。

A study on the metabolic changesassociated with obesity and on the roleof insulin in obesogenesis was made in31 normal subjects whose body weightswere ≤15% of the desirable weightof an average 94. 4±15. 0% (range71-115%) and 38 cases with obesitywhose weights were≥20% of the desi-rable weight of an average of 131.5±10. 0% (range 120-154%). The results of this study showed thatsignificant impairment of carbohydrateand lipid metabolism were observed inthe obese patients. Significant hyper-glycemia and hyperinsulinemia...

A study on the metabolic changesassociated with obesity and on the roleof insulin in obesogenesis was made in31 normal subjects whose body weightswere ≤15% of the desirable weightof an average 94. 4±15. 0% (range71-115%) and 38 cases with obesitywhose weights were≥20% of the desi-rable weight of an average of 131.5±10. 0% (range 120-154%). The results of this study showed thatsignificant impairment of carbohydrateand lipid metabolism were observed inthe obese patients. Significant hyper-glycemia and hyperinsulinemia wereobserved in the obese subjects afteroral glucose. The insulin secretion inresponse to an oral glucose load inobesity was significantly excessive ascompared with that of the normal group(P<0.001). A higher peak insulin res-ponse and a slower decline in plasmainsulin levels were present in the obe-sity group than in normal group. Plasmainsulin area and insulin area/ glucosearea ratio after glucose loading in obe-sity were significant higher than inthose of the normal group. The meanplasma free fatty acid (FFA) concen-tration in obesity decreased to lowerlevel than in the controls and failed toreach the fasting level three hours af-ter taking 100gm of glucose. Theobservations in this study lend strongsupport to the view that the adipose andhepatic tissue in obese subjects arenormal with regard to its sensitivityto circulating insulin, whereas themuscle and connective tissue are resis-tance to the circulating insulin. The plasma triglycerides, cholesteroland FFA levels in obesity were signi-ficantly higher than in normal subject.(P<0.001). The plasma HDL-chole- sterol level and HDL -cholesterol /cholesterol ratio were significantlyreduced in obesity (P<0. 05, P<0. 001). The average levels of fasting plasmacAMP in obesity were lower than inthe normal group by 39.8% (P<0. 01).The decline peak of plasma cAMP le-vels in obesity after glucose loadingdelayed to 120 minutes after takingglucose. The role of insulin in the pathogen-esis of obesity is discussed.

对31例正常人及38例肥胖者的糖、脂代谢及胰岛素在形成肥胖中的作用进行了研究。结果发现,肥胖者有糖、脂代谢的显著异常。负荷糖后胰岛素的分泌增强,峰值延后,分泌面积增加,产生高胰岛素血症,但其耐糖量显著下降,说明肥胖者外周靶组织对胰岛素发生抵抗。空腹血浆甘油三酯及胆固醇含量显著增加(P<0.001),HDL-胆固醇及HDL-胆固醇/胆固醇比值显著下降(P<0.05,P<0.001);负荷糖后,血浆游离脂酸迅速显著下降,甘油三酯在30分钟内明显升高,显示肥胖者脂肪及肝细胞对胰岛素仍敏感。空腹血浆cAMP含量较正常降低39.8%(P<0.01),负荷糖后下降峰值延后,可能系胰岛素分泌增加对靶细胞作用的结果。本文对胰岛素在形成肥胖中的作用及其与代谢异常的关系进行了讨论。

 
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