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rg治疗
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  rg 1
     (16.2 ± 1.7)%,(294.7 ± 18.5)%,(15.9 ± 2.8)% in ST + Rg1 group.
     ST+Rg1治疗组为(16.2±1.7)%、(294.7±18.5)%、(15.9±2.8)%。
短句来源
     (9.8 ± 0.9)%, (179.5 ± 11.3)%, (10.4 ± 0.94)% in Rg1 group;
     Rg1治疗组为(9.8±0.9)%,(179.5±11.3),(10.4±0.94)%;
短句来源
     There were statistical differences between I/R group and every other group (P < 0.05), between Rg1 group and control group(P < 0.05), between ST + Rg1 and Rg1 group (P < 0.05).
     3项检测数据I/R组明显高于各组(P<0.05); Rg1治疗组明显低于I/R组(P<0.05)和高于正常对照组(P<0.05);
短句来源
     G-Rg1 treatment group: closing the abdominal cavity after left ureteral ligation, and perfusing stomach with Ginsenoside Rg1(120mg/kg/day).
     G-Rg1治疗组:行左侧输尿管结扎后关腹,并给予人参皂甙Rg1灌胃(120mg/kg/day)。
短句来源
     METHODS The model of hemorrhagic shock in rats were utilized. The RNA was extracted from the intestinal epithelial cells. Reverse transcriptional polymerase chain reaction(RT-PCR) was used to detect the expression of COXⅠ,COXⅡ,COXⅢ and the therapeutic effects of Rg 1 respectively.
     方法 采用失血性休克模型 ,分离肠上皮细胞后进行RNA的提取 ,应用RT PCR检测大鼠失血性休克后及用Rg1治疗后COXⅠ ,COXⅡ ,COXⅢmRNA的表达变化情况。
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  “rg1治疗”译为未确定词的双语例句
     Results SOD,LDH and MMP in hemorrhagic shock group was significantly decreased,while MDA was significantly increased compared with control group(P<0.01).
     Rg1治疗显著提高肠黏膜组织中SOD活性、降低MDA含量、提高LDH活力; 稳定肠上皮细胞线粒体膜电位(P<0.01),显著改善动物存活情况及病理形态改变。
短句来源
     And notoginsenoside-Rg_ 1 can strengthen mice immune function.
     采用三七皂苷Rg1治疗或预先采用三七皂苷Rg1注射均可明显增强小鼠的免疫能力,对环磷酰胺有一定的拮抗作用。
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  相似匹配句对
     The total courses were 24 weeks.
     d-1,治疗24w。
短句来源
     The interval of treatment is 1 year.
     治疗时间1年。
短句来源
     L -1 respectively .
     L-1
短句来源
     L-1;
     L1;
短句来源
     Rg2 ;
     Rg2;
短句来源
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  rg 1
The introgressed gene of spike color was shown to be allelic to Rg1 located on chromosome 1B of common wheat.
      
In all treatments tested, all major ginsenosides (Rb1, Rc, Rb2, Rd, Rf, Rg1, and Re) were found in the culture.
      
In 2,4-D-containing medium, the preferential synthesis of Re ginsenoside was observed while both Rg1 and Re were synthesized in other treatments.
      
The results suggested that with the supply of exogenous energy decreasing during late pregnancy, maternal body weight and net body weight loss in RG2 and RG1 were lower than those of CG (Ps0.01).
      
The glucose concentration of ewes in RG2 and RG1 was decreased throughout the feed restriction period, and the differences were observed between RG1 and CG on d 120 of gestation (P>amp;lt;0.05).
      
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Objective To investigate the effect of nitric oxide(NO) on Tolllike receptors(2/4)((TLR 2/4)) gene expression in the liver in acute hemorrhagic necrotizing pancreatitis(AHNP) in rats.(Methods)(Seventy) SD male rats were randomly divided into shamoperated group(n=10),AHNP group(n=30) and(Larginine)((LArg)) treatment group(n=30).Blood samples and liver(tissues) were obtained at 6 hours in shamoperated group,and 3,6,12 hours respectively in AHNP group and LArgtreated group.Alanine aminotransferase(ALT),aspartate...

Objective To investigate the effect of nitric oxide(NO) on Tolllike receptors(2/4)((TLR 2/4)) gene expression in the liver in acute hemorrhagic necrotizing pancreatitis(AHNP) in rats.(Methods)(Seventy) SD male rats were randomly divided into shamoperated group(n=10),AHNP group(n=30) and(Larginine)((LArg)) treatment group(n=30).Blood samples and liver(tissues) were obtained at 6 hours in shamoperated group,and 3,6,12 hours respectively in AHNP group and LArgtreated group.Alanine aminotransferase(ALT),aspartate aminotransferase(AST),amylase in serum,NO in liver(tissue) were determined.Tumor necrosis factorα(TNFα) and TLR2/4 mRNA expressions in the liver(tissue) were measured by reverse transcription polymerase chain reaction.Results TLR2/4 mRNA could be detected in the liver with low values in shamoperated group ((1.150±0.725)×10~(-6),(11.450±(1.724)×10~(-4))), but they were markedly upregulated at 3 hours in AHNP group ((1.970±0.362)×10~(-3),(175.000±0.111)×10~(-3)),and peaked at 12 hours ((2.940±0.316)×10~(-3),(2 673.000±88.380)×10~(3),(all P<0.01)).At the same time serum levels of amylase,ALT and AST increased,hepatic injuries were(aggravated),levels of TNFα mRNA in the liver were increased and levels of NO in the liver were lowered((P<0.05 or P<0.01)).Treatment with LArg could effectively inhibit TLR2/4 mRNA expression((3 h:(0.351±0.153)×10~(-3)),(135.000±22.310)×10~(-3);6 h:(2.100±0.535)×10~(-3),(187.000±(26.850)×10~(-3);12 h:(2.620±0.208)×10~(-3)),(1 959.000±270.000)×10~(-3),P<0.05 or P<0.01) and(alleviate) hepatic(injuries).The levels of serum amylase,ALT and AST lowered, and the levels of TNFα mRNA in the liver were(lowered) and the levels of NO were markly increased(P<0.05 or P<0.01).(Conclusion) These findings(suggest) that the expression of TLR2/4 mRNA is increased in the liver in AHNP,and the hepatic injuries are aggravated.NO could markly inhibit TLR2/4 mRNA gene expression in the liver in AHNP.

目的探讨一氧化氮(NO)对于急性出血坏死性胰腺炎(AHNP)时肝脏中T o ll样受体2/4(TLR 2/4)mRNA表达的影响。方法采用牛磺胆酸钠(TAC)逆行胰胆管注射制备AHNP肺损伤大鼠模型。动物分为假手术组、胰腺炎组和L精氨酸(L A rg)治疗组。假手术组于术后6 h,胰腺炎组和L A rg组分别于术后3、6、12 h取静脉血和肝组织,测定血清丙氨酸转氨酶(ALT)、天冬氨酸转氨酶(A ST)、淀粉酶和肝组织NO,逆转录聚合酶链反应(RT PCR)方法检测不同时间点肝组织肿瘤坏死因子α(TNFα)、TLR 2/4mRNA表达变化。结果与假手术组比较,胰腺炎组大鼠3 h肝组织TLR 2/4 mRNA表达开始增高〔(1.970±0.362)×10-3,(175.000±0.111)×1-0 3比(1.150±0.725)×10-6,(11.450±1.724)×10-4,〕12 h表达达峰值〔(2.940±0.316)×1-0 3,(2 673.000±88.380)×1-0 3,P均<0.01〕;血清淀粉酶ALT、A ST 3 h后即升高,肝损伤加重,肝组织TNFαmRNA表达升高,NO浓度逐渐降低...

目的探讨一氧化氮(NO)对于急性出血坏死性胰腺炎(AHNP)时肝脏中T o ll样受体2/4(TLR 2/4)mRNA表达的影响。方法采用牛磺胆酸钠(TAC)逆行胰胆管注射制备AHNP肺损伤大鼠模型。动物分为假手术组、胰腺炎组和L精氨酸(L A rg)治疗组。假手术组于术后6 h,胰腺炎组和L A rg组分别于术后3、6、12 h取静脉血和肝组织,测定血清丙氨酸转氨酶(ALT)、天冬氨酸转氨酶(A ST)、淀粉酶和肝组织NO,逆转录聚合酶链反应(RT PCR)方法检测不同时间点肝组织肿瘤坏死因子α(TNFα)、TLR 2/4mRNA表达变化。结果与假手术组比较,胰腺炎组大鼠3 h肝组织TLR 2/4 mRNA表达开始增高〔(1.970±0.362)×10-3,(175.000±0.111)×1-0 3比(1.150±0.725)×10-6,(11.450±1.724)×10-4,〕12 h表达达峰值〔(2.940±0.316)×1-0 3,(2 673.000±88.380)×1-0 3,P均<0.01〕;血清淀粉酶ALT、A ST 3 h后即升高,肝损伤加重,肝组织TNFαmRNA表达升高,NO浓度逐渐降低(P<0.05或P<0.01);给予L A rg治疗后,NO浓度升高(P<0.05),TLR 2/4 mRNA表达降低〔3 h:(0.351±0.153)×1-0 3,(135.000±22.310)×1-0 3;6 h:(2.100±0.535)×10-3,(187.000±26.850)×1-0 3;12 h:(2.620±0.208)×1-0 3,(1 959.000±270.000)×10-3;P<0.05或P<0.01〕,血清淀粉酶、ALT、A ST均降低,肝损伤减轻,肝组织TNFαmRNA降低(P<0.05或P<0.01)。结论AHNP时,肝组织内TLR 2/4 mRNA表达上调,肝组织损伤加重;NO可以明显抑制AHNP肝组织TLR 2/4 mRNA的表达。

 
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