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慢性点燃
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  chronic epileptic
     Effect of YuXianLing(YXL) Granules on the Ca~(2+) Concentration of Hippocampi Synaptosomes in the Pentylenetetrazol Model of the Chronic Epileptic Rats
     愈痫灵颗粒剂对戊四氮慢性点燃大鼠海马突触体内钙离子浓度的影响
短句来源
     Effect of YuXianLing(YXL) Drug Granules on the Amino-acid Neurotransmitter and Their Receptors' Gene Expression in the Hippocampus of the Chronic Epileptic Rats
     愈痫灵对慢性点燃大鼠海马区氨基酸类神经递质及其受体基因表达的影响
短句来源
     The chronic epileptic model was induced in rats by intraperitoneal injection of Coriaria lectone. Lectin histochemistry for the detection of microglial cells and their morphological changes were studied. Ricinus communis agglutinin 120 (RCA 1) was used as histochemical marker on sections of hippocampus and cerebral cortex.
     在腹腔注射马桑内酯所致的大鼠慢性点燃癫痫模型上 ,应用小胶质细胞的特异性标记物蓖麻凝集素 - 12 0(Ricinus comm unis agglutinin- 12 0 ,RCA- 1)对大鼠海马和大脑皮质小胶质细胞及其形态学进行凝集素组化研究。
短句来源
  chronic kindling
     To Investigate a Chronic Kindling Epilepsy Animal Model with Presynaptic Membrane Protein (Munc-18) Antibody
     神经突触前膜蛋白(Munc-18)抗体慢性点燃癫癎动物模型的探索
短句来源
     Experiment Ⅲ:6 infant rats were intraperitoneally injected with 45mg/kg of NMDA from PN 18 to PN 30 respectively. The establishment of chronic kindling model was observed.
     实验三:6只乳鼠分别于生后18~30天腹腔注射NMDA(45mg/kg),观察慢性点燃模型建立情况。
短句来源
     Conclusion: To stimulate wistar rats at amygdalas with constant pulsating current may induces chronic kindling epilepsy and to establish relatively stable animal model.
     结论:通过微电极刺激Wistar大鼠杏仁核可以建立稳定的大鼠癫痫慢性点燃模型。
短句来源
     Conclusions Munc18 antibody might induce epilepsy by chronic kindling way on rat,suggesting that the mechanism might be linked to neurons’s apoptosis and lose in hippocampus area.
     结论Munc18抗体能慢性点燃致大鼠,其机制可能与其诱导整个海马区神经细胞的凋亡和丢失有关。
短句来源
     Objective To study the effect of saikosaponins, the active ingredients of Bupleurum chinense DC, on glial fibrillary acidic protein (GFAP) expression in hippocampal astrocytes of chronic kindling rats induced by pentetrazole (PTZ).
     目的研究柴胡总皂甙对戊四氮(PTZ)慢性点燃癫痫模型大鼠海马区星形胶质细胞的胶质纤维酸性蛋白(GFAP) 表达的影响。
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  chronically kindling
     The Study of Presynaptic Membrane Protein (Munc18) Antibody Chronically Kindling Rat and Its Mechanism
     神经突触前膜胞内蛋白(Munc18)抗体慢性点燃致痫大鼠及其致痫机制的研究
短句来源
     Presynaptic membrane protein (Munc-18) antibody chronically kindling epileptiform rat and its mechanism
     神经突触前膜胞内蛋白(Munc-18)抗体慢性点燃致痫大鼠及其机制
短句来源
  “慢性点燃”译为未确定词的双语例句
     Methods SD rats were devided randomly into four roups: control group, epilepsy Group I (PTX, 1.5?mg·kg -1 ·d -1 ),epilepsy Group II (PTX, 2?mg·kg -1 ·d -1 ) and the phenobarbital group. The kindling model of rats was induced by daily injection of PTX intraperitoneally.
     方法 :采用PTX腹腔注射制作SD鼠慢性点燃模型 ,并将其分为对照组、癫痫I组 (PTX ,1 5mg·kg- 1 ·d- 1 )、癫痫II组 (PTX ,2mg- 1 ·d- 1 )和苯巴比妥钠干预组 ;
短句来源
     Objective: To study the effects of Annao tablet(main component is β-asarone) on S100B and NPY of cortex in chronic epilepsy rats.
     目的:研究安脑片(主成分β-细辛醚)对慢性点燃癫痫大鼠脑皮质S100B蛋白和神经肽Y(NPY)的影响。
短句来源
     Method: The decoction was made by traditional decoction,and was tested in PTZ,MES acute seizue model in mice and PTZ kindling model in rats.
     方法 :采用传统水煎法得到石菖蒲水煎剂 ,分别试验戊四唑 (PTZ)、最大电休克 (MES)急性惊厥模型及PTZ慢性点燃模型。
短句来源
     Conclusion Saikosaponins can influence the Glu level in hippocampal CA1 of PTZ-induced slow-kindling rats,thus inhibiting PTZ-induced seizure.
     结论柴胡总皂甙可以影响PTZ点燃大鼠海马CA1区的Glu表达水平,从而抑制PTZ慢性点燃大鼠的痫性发作。
短句来源
     Resaults: 1. YXL can effectively prolong the potential time and shorten last-time of epilepsy-attacks, decreasing the frequency and amplitudes of waves of epileptic discharge in rats' brain and there was significant diffirence compared with model group(P<0.05,P<0.01).
     结果:1.与模型组比较,愈痫灵颗粒剂可有效延长慢性点燃大鼠的癫痫发作潜伏期,减少发作持续时间,降低痫性放电波幅,减少痫性放电频率(P<0.05,P<0.01);
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  chronic epileptic
This treatment will not affect the incidence of late seizures or PTE, which are both caused by a chronic epileptic focus.
      
Increased expression of GAD mRNA during the chronic epileptic syndrome due to intrahippocampal tetanus toxin
      
Surgical pathology of chronic epileptic seizure disorders: experience with 63 specimens from extratemporal corticectomies, lobec
      
In the present study a unilateral intrahippocampal injection of tetanus toxin into the ventral hippocampus was used to produce a chronic epileptic syndrome characterised by brief seizures that recurred intermittently for 6-8 weeks.
      
A report is presented on a test in cats for the examination of animals with chronic epileptic processes (Kopeloff-method).
      
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The changes of membrane potential and discharging modes of corticocerebral and hippocampal neurons were investigated in chronically kindled epileptic and acute convulsive rats induced by abdominal injection of coriaria lactone.EEG in the cortex showed obvious episodic discharges in epileptic rats.Several kinds of discharging modes of corticocerebral and hippocampal neurons were observed with intracellular recorders,including membrane depolarization alone,membrane depolarization with discontinuous and/or sustained...

The changes of membrane potential and discharging modes of corticocerebral and hippocampal neurons were investigated in chronically kindled epileptic and acute convulsive rats induced by abdominal injection of coriaria lactone.EEG in the cortex showed obvious episodic discharges in epileptic rats.Several kinds of discharging modes of corticocerebral and hippocampal neurons were observed with intracellular recorders,including membrane depolarization alone,membrane depolarization with discontinuous and/or sustained discharges and alternation of membrane depolarization and hyperpolarization.The results showed that coriaria lactone could induce changes in electric activity of corticocerebral and hippocampal neurons with different modes and mainly cause membrane depolarization.

在腹腔注射马桑内酯所致的急性癫痫和慢性点燃癫痫大鼠模型上,观察了大脑皮层和海马神经元膜电位的变化及放电模式。结果显示:皮层脑电图出现明显的阵发性痫样放电,胞内微电极记录皮层及海马神经元放电的模式为:①单纯去极化;②去极化并伴有阵发性或持续性放电;③去极化与超极化交替出现等。表明马桑内酯可以引起大脑皮层及海马神经元生物电活动的改变,其改变形式不一,但主要引起去极化。

In order to explore the suppressive effect of glucocorticoid (GC) and its mechanism on chronically kindled epileptic rats, the behavior and EEG of the kindled rats induced by pentylenetetrazole were observed. The coexistence of glucorticoid receptor (GR) and GABA in the neurons of cerebral cortex was investigated by immunocytochemic double staining. The results showed that dexamethasone pretreatment (4mg/kg i.v.) at the day 3 or day 15 after kindling followed by injcetion of pentylenetetrazole (30mg/kg i.p)...

In order to explore the suppressive effect of glucocorticoid (GC) and its mechanism on chronically kindled epileptic rats, the behavior and EEG of the kindled rats induced by pentylenetetrazole were observed. The coexistence of glucorticoid receptor (GR) and GABA in the neurons of cerebral cortex was investigated by immunocytochemic double staining. The results showed that dexamethasone pretreatment (4mg/kg i.v.) at the day 3 or day 15 after kindling followed by injcetion of pentylenetetrazole (30mg/kg i.p) could reduce the degree of epileptic activity or suppress completely the epilepsy activity. Also, the coexistence of GR and GABA in a part of neurons of cerebral cortex was proved. The results suggest that GC could produce anti epileptic effect on the chonical epileptic rat by regulating synthesis of GABA in the same neurons of cerebral cortex.\;

为了探讨糖皮质激素的抗癫痫效应和作用机制, 本研究观察了糖皮质激素对戊四氮诱导的慢性点燃型癫痫大鼠的行为和脑电图的影响, 并应用免疫细胞化学双重染色技术探查了大脑皮质神经元内糖皮质激素受体(GR) 与γ- 氨基丁酸(GABA) 的共存情况。结果显示, 在慢性点燃型癫痫大鼠, 在点燃后的第3 天或第15 天, 先经静脉给予地塞米松(4m g/kg), 再经腹腔注射戊四氮(30m g/kg) 可明显减弱或完全抑制癫痫发作。免疫细胞化学双重染色证明, GR和GABA共存于大脑皮质部分神经元。以上结果提示, 糖皮质激素具有抗慢性癫痫的效应, 其作用机制可能与GR调节同一神经元内GABA的合成有关。

Objective To study the relationship between epileptic seizure and the concentration of γ aminobytyric acid (GABA), and the activity of glutamate decarboxylase (GAD). Methods High performance liquid chromatography (HPLC) was used to determine the GABA content and the activity of GAD in the temporal tissue of Sprague Dawley(SD) rats; SD rats were devided randomly into four groups: control group (Group A), epilepsy group in ictal period (Group B), epilepsy group in interictal period (Group C) and...

Objective To study the relationship between epileptic seizure and the concentration of γ aminobytyric acid (GABA), and the activity of glutamate decarboxylase (GAD). Methods High performance liquid chromatography (HPLC) was used to determine the GABA content and the activity of GAD in the temporal tissue of Sprague Dawley(SD) rats; SD rats were devided randomly into four groups: control group (Group A), epilepsy group in ictal period (Group B), epilepsy group in interictal period (Group C) and the phenobarbital group (Group D). The kindling model of rats was induced by daily injection of picrotoxin (PTX) intraperitoneally. Results The average value of temporal GABA content (3.82±0.81 μmol/mg) and GAD activity (11.85±2.56 μmol/mg) in Group B was much higher than that in Group A ( P <0.05). In comparison with Group B, the temporal GABA content (3.27±1.42 μmol/mg) and GAD activity (8.91±2.44 μmol/mg) in Group C were decreased, but higher than that in Group A. The results of GABA content and GAD activity between Group A and Group D revealed no significant difference. Conclusions The changes of GABA and GAD were closely related to the epileptic seizure. The results suggest that the augmented GABAergic neurotransmission is the selfprotective and anticonvulsive mechanism in this model. Phenobarbital sodium can protect it from seizure provided that the SD rats were given daily injection of PTX intraperitoneally.

目的 探索癫痫发生与γ 氨基丁酸 (GABA)浓度、谷氨酸脱羧酶 (GAD)活性的关系。方法 采用印防己毒 (PTX)腹腔注射制做SD鼠慢性点燃模型 ,并将其分为对照组、癫痫发作组、发作间期组和苯巴比妥钠干预组。用高效液相色谱仪 (HPLC)测定颞区脑组织GABA浓度和GAD活性。结果 注药第 2 0d ,71%的鼠达到完全点燃状态 ;癫痫发作组鼠颞区脑组织GABA浓度为 (3.82±0 .81) μmol g ,GAD活性为 (11.85± 2 .5 6 ) μmol g ,较对照组〔(2 .15± 0 .37) μmol g ,(7.6 9± 2 .12 ) μmol g〕明显增高 ;癫痫发作间期组鼠颞区脑组织GABA浓度为 (3.2 7± 1.42 ) μmol g ,GAD活性为 (8.91±2 .44 ) μmol g,较发作期〔(3.82± 0 .81) μmol g ,(11.85± 2 .5 6 ) μmol g〕有所下降 ,但高于对照组。苯巴比妥钠干预组鼠颞区脑组织GABA浓度〔(2 .36± 0 .6 1) μmol g〕、GAD活性〔(6 .96± 2 .5 8) μmol ...

目的 探索癫痫发生与γ 氨基丁酸 (GABA)浓度、谷氨酸脱羧酶 (GAD)活性的关系。方法 采用印防己毒 (PTX)腹腔注射制做SD鼠慢性点燃模型 ,并将其分为对照组、癫痫发作组、发作间期组和苯巴比妥钠干预组。用高效液相色谱仪 (HPLC)测定颞区脑组织GABA浓度和GAD活性。结果 注药第 2 0d ,71%的鼠达到完全点燃状态 ;癫痫发作组鼠颞区脑组织GABA浓度为 (3.82±0 .81) μmol g ,GAD活性为 (11.85± 2 .5 6 ) μmol g ,较对照组〔(2 .15± 0 .37) μmol g ,(7.6 9± 2 .12 ) μmol g〕明显增高 ;癫痫发作间期组鼠颞区脑组织GABA浓度为 (3.2 7± 1.42 ) μmol g ,GAD活性为 (8.91±2 .44 ) μmol g,较发作期〔(3.82± 0 .81) μmol g ,(11.85± 2 .5 6 ) μmol g〕有所下降 ,但高于对照组。苯巴比妥钠干预组鼠颞区脑组织GABA浓度〔(2 .36± 0 .6 1) μmol g〕、GAD活性〔(6 .96± 2 .5 8) μmol g〕无明显变化。结论 在该模型中 ,GABA浓度、GAD活性与癫痫发作密切相关 ,提示GABA能神经元的抑制功能增强是机体重要的内源性抗癫痫机制。用苯巴比妥钠和PTX同时腹腔注射 ,可以预防癫痫的发生。

 
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